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1 r run-on assay and of chromatin structure by nuclease protection assay.
2 ease in TPO mRNA levels as measured by an S1 nuclease protection assay.
3 s in intestinal lymphocytes were assessed by nuclease protection assays.
4 evels using reverse transcription-PCR and S1 nuclease protection assays.
5 tivities were consistent with the results of nuclease protection assays.
6 length of DNA that is protected by RAD52 in nuclease protection assays.
7 ' rapid amplification of cDNA end-PCR and S1 nuclease protection assays.
8 (NTRs) were assayed for RNA production by S1 nuclease protection assays.
9 mRNA with the use of solution hybridization-nuclease protection assays.
11 ifically methylated U1939 as determined by a nuclease protection assay and by methylation assays usin
16 CE (rapid amplification of cDNA ends) and S1 nuclease protection assays and was at the site of an act
17 d metabolism of btuB RNA were analyzed by S1 nuclease protection assays, and mutations that alter the
18 ption/polymerase chain reaction analysis and nuclease protection assay, BAT was demonstrated to expre
21 n start site was identified in rat kidney by nuclease protection assay defining a 5' untranslated reg
22 d amplification of cDNA ends coupled with S1 nuclease protection assays demonstrate that the M3 prote
25 nscripts by primer extension and modified S1 nuclease protection assays demonstrated that transcripti
26 A expression studies, based on a multiplexed-nuclease protection assay, demonstrated that cell cycle-
27 them to high-throughput genomic quantitative nuclease protection assay for quantifying simultaneously
29 osing the basic mechanistic principle of the nuclease protection assay into this biosensor framework,
30 g-1 cDNA, and it was further demonstrated by nuclease protection assay, Northern blotting, and immuno
31 amined by a variety of methods, including S1 nuclease protection assays, Northern blotting, Western b
33 transcription-PCR analyses, coupled with S1 nuclease protection assays, provided evidence that gene
41 ession studies, again based on a multiplexed-nuclease protection assay, showed that TGF-beta-related
42 ormed using a novel multiplexed quantitative nuclease protection assay that involves customized DNA m
43 apid amplification of cDNA ends as well as a nuclease protection assay to map the transcriptional sta
48 y 5'-rapid amplification of cDNA ends and S1 nuclease protection assay, we determined that the transc
49 of cDNA ends, primer extension analysis, and nuclease protection assay, we identified transcription s
50 polymerase chain reaction, and quantitative nuclease protection assays, we assessed the ability of s
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