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1 ved bilateral quisqualic acid lesions of the nucleus basalis magnocellularis.
2 o detected in the striatum (4.2%) and in the nucleus basalis magnocellularis (19.2%) 3-12 h following
3 no marked loss of cholinergic neurons in the nucleus basalis magnocellularis after intracerebroventri
5 as suggested that cholinergic neurons in the nucleus basalis magnocellularis and substantia innominat
6 ticopetal cholinergic lesions applied to the nucleus basalis magnocellularis and substantia innominat
7 nt changes in trkA mRNA were detected in the nucleus basalis magnocellularis, and no significant chan
8 rizontal limb of the diagonal band of Broca, nucleus basalis magnocellularis, and striatum of gonadec
9 h the medial septum, diagonal band of Broca, nucleus basalis magnocellularis, and striatum were proce
11 es of evidence have supported a role for the nucleus basalis magnocellularis (NB) in attentional mech
12 Rats with quisqualic acid lesions of the nucleus basalis magnocellularis (nBM) and control rats w
15 termine if a selective cholinergic lesion of nucleus basalis magnocellularis (Nbm) could affect the n
16 s tested the hypothesis that the cholinergic nucleus basalis magnocellularis (NBM) is involved in sol
17 ic lesions of the medial septum area (MS) or nucleus basalis magnocellularis (NBM) on amplitude and p
18 ed in aging rats: cholinergic lesions of the nucleus basalis magnocellularis (NBM) produce larger dec
19 LS, animals were lesioned bilaterally in the nucleus basalis magnocellularis (nBM) using either quisq
22 ith bilateral 192 IgG-saporin lesions to the nucleus basalis magnocellularis (nBM) were tested on olf
23 c immunotoxic- or sham-lesion surgery of the nucleus basalis magnocellularis (NBM), the basal forebra
24 F receptor immunoreactive neurons within the nucleus basalis magnocellularis (NBM), the horizontal li
28 ilateral injection of ibotenic acid into the nucleus basalis magnocellularis, or unilateral transecti
30 ell bodies in the medial septal area and the nucleus basalis magnocellularis, radiofrequency lesions
31 A) and the cholinergic substantia innominata/nucleus basalis magnocellularis (SI/nBM) is important fo
32 A from the cholinergic substantia innominata/nucleus basalis magnocellularis (SI/nBM) on performance
33 s in the sublenticular substantia innominata/nucleus basalis magnocellularis (SI/nBM), as well as cer
34 nfusions of colchicine or vehicle in the rat nucleus basalis magnocellularis, the time course of chan
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