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1 ved bilateral quisqualic acid lesions of the nucleus basalis magnocellularis.
2 o detected in the striatum (4.2%) and in the nucleus basalis magnocellularis (19.2%) 3-12 h following
3 no marked loss of cholinergic neurons in the nucleus basalis magnocellularis after intracerebroventri
4     Results demonstrated that lesions of the nucleus basalis magnocellularis and frontal cortex selec
5 as suggested that cholinergic neurons in the nucleus basalis magnocellularis and substantia innominat
6 ticopetal cholinergic lesions applied to the nucleus basalis magnocellularis and substantia innominat
7 nt changes in trkA mRNA were detected in the nucleus basalis magnocellularis, and no significant chan
8 rizontal limb of the diagonal band of Broca, nucleus basalis magnocellularis, and striatum of gonadec
9 h the medial septum, diagonal band of Broca, nucleus basalis magnocellularis, and striatum were proce
10                                    Since the Nucleus Basalis Magnocellularis (Meynert in humans and p
11 es of evidence have supported a role for the nucleus basalis magnocellularis (NB) in attentional mech
12     Rats with quisqualic acid lesions of the nucleus basalis magnocellularis (nBM) and control rats w
13     Rats with 192 IgG-saporin lesions of the nucleus basalis magnocellularis (NBM) and sham-operated
14      Previous research has demonstrated that nucleus basalis magnocellularis (nbm) corticopetal choli
15 termine if a selective cholinergic lesion of nucleus basalis magnocellularis (Nbm) could affect the n
16 s tested the hypothesis that the cholinergic nucleus basalis magnocellularis (NBM) is involved in sol
17 ic lesions of the medial septum area (MS) or nucleus basalis magnocellularis (NBM) on amplitude and p
18 ed in aging rats: cholinergic lesions of the nucleus basalis magnocellularis (NBM) produce larger dec
19 LS, animals were lesioned bilaterally in the nucleus basalis magnocellularis (nBM) using either quisq
20  of the diagonal band of Broca (HDB) and the nucleus basalis magnocellularis (NBM) were counted.
21                          Fluctuations in the nucleus basalis magnocellularis (NBM) were highly variab
22 ith bilateral 192 IgG-saporin lesions to the nucleus basalis magnocellularis (nBM) were tested on olf
23 c immunotoxic- or sham-lesion surgery of the nucleus basalis magnocellularis (NBM), the basal forebra
24 F receptor immunoreactive neurons within the nucleus basalis magnocellularis (NBM), the horizontal li
25                      The BLA projects to the nucleus basalis magnocellularis (NBM), which sends broad
26 ormation and its release is regulated by the nucleus basalis magnocellularis (NBM).
27 ith a 2-week-old right-sided ablation of the nucleus basalis magnocellularis (NBM).
28 ilateral injection of ibotenic acid into the nucleus basalis magnocellularis, or unilateral transecti
29              Injection of lidocaine into the nucleus basalis magnocellularis produced a profile of be
30 ell bodies in the medial septal area and the nucleus basalis magnocellularis, radiofrequency lesions
31 A) and the cholinergic substantia innominata/nucleus basalis magnocellularis (SI/nBM) is important fo
32 A from the cholinergic substantia innominata/nucleus basalis magnocellularis (SI/nBM) on performance
33 s in the sublenticular substantia innominata/nucleus basalis magnocellularis (SI/nBM), as well as cer
34 nfusions of colchicine or vehicle in the rat nucleus basalis magnocellularis, the time course of chan
35                               Lesions of the nucleus basalis magnocellularis were without effect on t

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