ライフサイエンスコーパス: nucleus pulposus cell

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1 cules, and that this regulation is unique to nucleus pulposus cells.

2 fect of HIFs on GlcAT-1 promoter function in nucleus pulposus cells.

3 umber of human studies focusing primarily on nucleus pulposus cells.

4 ommon target gene for HIF-1 and HIF-2 in the nucleus pulposus cells.

5 T-1 promoter activity and expression only in nucleus pulposus cells.

6 CCN3 and suppressed its promoter activity in nucleus pulposus cells.

7 e-dependent decrease in the proliferation of nucleus pulposus cells.

8 on and promoter activity was observed in rat nucleus pulposus cells after TGFbeta treatment.

9 crease in promoter activity was seen both in nucleus pulposus cells and in N1511 chondrocytes.

10 lyses were used to measure ANK expression in nucleus pulposus cells from rats and humans.

11 disc disease may restore NOTCH signaling and nucleus pulposus cell function.

12 ssion and the suppression of CCN2 by CCN3 in nucleus pulposus cells further the paradigm that these C

13 Results of this study indicate that in nucleus pulposus cells, HIF-2 and HIF-1 modulate their o

14 Here, we show that in nucleus pulposus cells, hypoxia robustly induces PHD3 ex

15 transcriptional coactivator of HIF-1alpha in nucleus pulposus cells independent of the PKM2-JMJD5 axi

16 Adenovirus-mediated gene transfer to nucleus pulposus cells may be the initial stage of a new

17 Adaptive response to hypoxia in nucleus pulposus cells of the intervertebral disc is reg

18 ation through controlling ADAMTS activity in nucleus pulposus cells of the intervertebral disc.

19 ss of all the PHDs is maintained in isolated nucleus pulposus cells regardless of the disease state.

20 Since nucleus pulposus cells reside under conditions of hypoxi

21 erexpression of HIF-1alpha and HIF-2alpha in nucleus pulposus cells resulted in a significant suppres

22 stochemically in disc tissue, and numbers of nucleus pulposus cells staining positive for ADAMTS 4, 5

23 suppressed GlcAT-1 promoter activity only in nucleus pulposus cells, suggesting a cell type-specific

24 ng CCN3 expression in Smad3-null mice and in nucleus pulposus cells transduced with lentiviral short

25 Above 330 mosmol/kg, cultured nucleus pulposus cells up-regulated target genes TauT, B

26 xygen-dependent changes in ANK expression in nucleus pulposus cells were minimal.

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