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2 1/2 inhibitors in reversing vasoocclusion in nude and humanized SCD mouse models of acute vasoocclusi
8 d time-dependent tumor uptake was studied in nude BALB/c mice bearing a subcutaneous HER3 overexpress
9 p to 6 d after intravenous administration to nude BALB/c mice bearing high EpCAM-expressing HT-29 col
10 umor growth was significantly decreased when nude BALB/c mice were injected with Msi1-knockdown BCCs.
13 in Xenopus egg extracts, we show that Nudel/NudE facilitates the binding of Lis1 to dynein, which en
14 was performed in tumour xenografts in 15 NCr nude immunodeficient mice, which were treated with eithe
15 r the cytoplasmic dynein regulators LIS1 and NudE/L (Nde1/Ndel1), but not for the dynactin p150(Glued
17 scles were subcutaneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles Ri
22 cells, and in both eight-patient bloods and nude mice administered with the labeled CTCs in comparis
23 K1/2 inhibitor given to TNF-alpha-pretreated nude mice after human SSRBC infusion and onset of vasooc
24 experiments were performed on tumor-bearing nude mice after subcutaneous injection of RIN-m5F cells.
26 tifying the biodistribution of antibodies in nude mice and provides an alternative to PET analysis in
28 ChA-1 cells were injected into the flanks of nude mice and treated with miR-24 inhibitor or inhibitor
39 dent SSM3 mouse mammary tumors, male athymic nude mice bearing androgen-dependent CWR22 prostate canc
40 trigel suspension model was established with nude mice bearing cells equally infected with each repor
42 ncer xenografts, and male and female athymic nude mice bearing estrogen-independent MDA-MB-231 human
43 jected intravenously into the BALB/c athymic nude mice bearing folate receptor (FR)-overexpressing KB
44 nd biodistribution studies were performed in nude mice bearing HCC4006 and A549 xenograft tumors.
46 icacy of PEG-GIRLRG peptide was evaluated in nude mice bearing heterotopic cervical (HT3), esophageal
47 ce, and tumor targeting was assessed in CD-1 nude mice bearing high-HER2-expressing NCI-N87 tumors an
51 In vivo antitumor efficacy was tested in nude mice bearing PSMA+ PC3 PIP or PSMA- PC3 flu flank x
52 tion and SPECT/CT imaging experiments, using nude mice bearing PSMA-positive PC-310 and PSMA-negative
57 of tumor cure while being well tolerated by nude mice bearing subcutaneous GPA33-positive SW1222 xen
58 One-hour dynamic PET scans were performed on nude mice bearing subcutaneous human head and neck tumor
59 cs of (89)Zr-DFO-AC-10 was studied in BALB/c nude mice bearing subcutaneous human Karpas 299 tumors (
60 nd biodistribution studies were performed on nude mice bearing U87MG and MDA-MB-231 xenografted tumor
62 /kg MEK1/2 inhibitor to TNF-alpha-pretreated nude mice before human SSRBC infusion inhibited SSRBC ad
65 117085 significantly reduces tumor growth in nude mice compared with control untreated mice or either
71 vivo an orthotopic liver injection model in nude mice further demonstrated that knockdown of RhoE en
72 ntal pulp on poly-l-lactic acid scaffolds in nude mice gave rise to perfect heterotopic ossicles in v
73 ication involving the TKD into the brains of nude mice generated high-grade astrocytomas with short l
76 ild cardiac hypertrophy was also observed in nude mice implanted with IDH2(R140Q)-expressing xenograf
79 ties, SPECT and CT scans of HT29-xenografted nude mice injected with (177)Lu-3BP-227 were acquired, a
80 tically attenuates tumor growth in xenograft nude mice injected with human K562 leukemia cells and ce
81 Subcutaneous injection of TRIM24 iHMECs in nude mice led to growth of intermediate to high-grade tu
82 id gland scaffolds into the renal capsule of nude mice led to the differentiation of transplanted hDF
89 ce fulfilled criteria for the model but only nude mice offered sufficient availability for large ther
91 ve transfer of congenic T cells into athymic nude mice prior to infection did not alter lesion size.
92 implantation of LOX-treated neocartilage in nude mice promoted further maturation of the neotissue,
94 ansplantation of hERG1-expressing cells into nude mice resulted in an increased incidence of tumors.
95 5alpha-depleted pancreatic cancer cells into nude mice resulted in markedly reduced tumorigenicity (P
96 KD SCC1 cells into the floor of the mouth in nude mice resulted in the formation of significantly sma
97 ne was sufficient to induce tumorigenesis in nude mice resulting in short lifespan irrespective of wh
98 D25(+)IL7Ralpha(+)) after grafting recipient nude mice revealed that MPP3 cells were the most effecti
99 ografts of MKN45/5FU cells in the stomach of nude mice revealed that these cells had a high potential
100 SCs transplanted to the ischemic hindlimb of nude mice showed significantly higher BLI and PET signal
102 pressed DNAJB6a formed tumors more slowly in nude mice than control cells or cells that expressed a m
109 ia conditions, were then assessed in healthy nude mice using the left kidney and spleen as reference
111 Growth of PLC5 tumor xenografts in BALB/c nude mice was inhibited by daily oral treatment with nil
112 of WT HBx-expressing cells to form tumors in nude mice was significantly higher than that of mutant H
118 and preliminary in vivo xenograft studies in nude mice were performed to evaluate target binding.
119 -fragment dose for imaging was determined in nude mice with a subcutaneous head and neck carcinoma mo
121 avenously administered every 3 d for 16 d to nude mice with AR42J tumor xenografts that were approxim
122 OIS and enabled tumor transplants to grow in nude mice with characteristic cell morphology of anaplas
123 In vivo treatment of A375 xenograft-bearing nude mice with cryptolepine (10 mg/Kg body weight, i.p.)
124 t overexpress MFSD2A were transferred to CD1 nude mice with dextran sodium sulfate-induced colitis, w
127 uorescence imaging to detect ccRCC tumors in nude mice with RCC xenografts by using mAb girentuximab
129 ing product, (18)F-FVIIai, was injected into nude mice with subcutaneous human pancreatic xenograft t
130 11B6 uptake was performed on NMRI and BALB/c nude mice with subcutaneous LNCaP xenografts up to 14 d
131 ouse) was injected intravenously into BALB/c nude mice with subcutaneous PSMA-expressing LNCaP tumors
132 111)In-labeled ADCs were performed on BALB/c nude mice with subcutaneous PSMA-positive LS174T-PSMA xe
134 Methods: Two groups of 6 adult female BALB/c nude mice with subcutaneously implanted tumors underwent
135 ary tumors, in an NFATc1-dependent manner in nude mice with T-cell deficiency, revealing an addiction
136 targeting potential and antitumor effects in nude mice with tumors that were sensitive or resistant t
140 ble for small-animal PET studies in multiple nude mice xenografted with the A431 carcinoma cell line.
144 family mutation efficiently form sarcomas in nude mice, and a Ras-ZEB1-Akt pathway then causes transi
145 ces hypoxia in FaDu and HCT116 xenografts in nude mice, and causes a significant tumour growth delay
146 lorectal cancer metastases were generated in nude mice, and epifluorescence imaging of ICG, as well a
147 and H1975) were subcutaneously implanted in nude mice, and growth was followed by caliper measuremen
149 matrix proteins, subcutaneous tumor size in nude mice, and invasive behavior, including bone marrow
150 nd cervical cancer cell xenograft in vivo in nude mice, and suppress cervical cancer cell migration a
151 nt growth inhibition of MOLM13 xenografts in nude mice, and the activity correlates with inhibition o
152 bodies and transferred into infected SCID or nude mice, and the animals received the same antibody ev
153 2 were injected into the pancreas of athymic nude mice, and their local and distant spread was monito
154 molecules were then determined in naive CD-1 nude mice, and tumor targeting was assessed in CD-1 nude
155 ously and orthotopically implanted tumors in nude mice, and was accompanied by c-SRC downregulation.
157 nhibited orthotopic prostate tumor growth in nude mice, compared with monotherapy, by reversing the e
158 taneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles River Laboratories
160 cells were subcutaneously transplanted into nude mice, DPSC/CTL cells induced mineralized tissue for
162 utside the primary tumor microenvironment in nude mice, exhibited signatures of immune evasion, incre
164 uding HUVEC-mediated trophoblast invasion in nude mice, in vitro three-dimensional capillary tube for
165 tants strongly decreased tumor metastases in nude mice, indicating the requirement of PTTG for STAT3-
166 rs and xenografts established in NOD-SCID or nude mice, low MCPIP1 levels correlated strongly with in
168 uman prostate xenograft model established in nude mice, RAD001 alone or in combination with docetaxel
169 lonization of melanoma cells in the lungs of nude mice, suggesting an increase in metastatic potentia
171 expressing versican siRNA were injected into nude mice, the resulting tumors displayed significantly
172 n PMP tissue was i.p. grafted and grown into nude mice, then constituted into reliable and reproducib
173 and AGS, which do and do not form tumors in nude mice, to identify their genomic differences relevan
174 an ovarian SKOV3 tumors cells into 14 female nude mice, treatment with vehicle or pazopanib (2.5 mg p
175 e than WT cybrids, however, when injected in nude mice, tRNAmut cybrids produced larger tumours and s
176 sing head and neck cancer xenograft model in nude mice, tumor growth was inhibited by CXCR7-targeting
177 on in PCa cells and induces larger tumors in nude mice, whereas its silencing decreased proliferation
178 sulted in marked regression of xenografts in nude mice, whereas the delivery of an miR-125a inhibitor
179 g in aggressive tumor formation in xenograft nude mice, which could be suppressed by combined treatme
181 ive in suppressing xenograft tumor growth in nude mice, which underlines the translational potential
182 ous cell carcinoma upon inoculation into the nude mice, while parental HaCaT cells remain non-tumorig
267 ggressive orthotopic tumor models in athymic nude mice: a human PC-3 M-luc-C6 prostate tumor and a hu
269 nsional culture and in an in vivo orthotopic nude mouse model of HNSCC through a novel transcription-
283 resent study, we generated a novel strain of nude NOD/SCID/IL2rg(-/-) (NSIN) mice by knocking out Fox
284 was studied in normal and immune-deficient (nude, nu/nu) BALB/c mice infected with vaccinia virus (V
287 premalignancy and (b) an in vivo orthotopic nude rat lung cancer model to evaluate response to epige
295 ved grafting into the spinal cord of athymic nude rats with no signs of overgrowth and with a very si
296 bcutaneous cancer esophageal xenografts, and nude rats with orthotopic esophageal cancers in four stu
297 lop into NK cells, but DKO BM transfers into nude recipients and NK cells in E4BP4/Rag-1/IL-7 triple-
298 cell immune system since it did not occur in nude, severe combined immunodeficiency, or T-cell deplet
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