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1 ts and increases the median survival time of nude mice.
2 ancer cell proliferation and tumor growth in nude mice.
3 dard in vivo ectopic osteoinduction assay in nude mice.
4 on and a matrigel plug angiogenesis assay in nude mice.
5 blot assays, or injected subcutaneously into nude mice.
6 istant human ovarian cancer cells in athymic nude mice.
7 bited their tumorigenicity when engrafted in nude mice.
8 Calif) and injected in both flanks of eight nude mice.
9 a A375 human melanoma tumor model in athymic nude mice.
10 ion of breast cancer cell line xenografts in nude mice.
11 tocin-induced 8- to 10-week-old male athymic nude mice.
12 doses of a (90)Y-labeled GRPr antagonist in nude mice.
13 d into the distal posterior rectum of BALB/c-nude mice.
14 cell line and in vivo in PC-3 tumor-bearing nude mice.
15 nospheres in soft agar and nodules/tumors in nude mice.
16 gnificantly suppressed by NOS2 inhibition in nude mice.
17 in culture and promotes xenograft growth in nude mice.
18 duced tumorigenesis in an allograft model of nude mice.
19 ther subcutaneously or intraperitoneally, in nude mice.
20 ells in vitro and induces tumor formation in nude mice.
21 tion with the capacity to form metastases in nude mice.
22 orsphere assays and were less tumorigenic in nude mice.
23 ess differentiated tumors when injected into nude mice.
24 sitive neovasculature when transplanted into nude mice.
25 man pancreatic adenocarcinomas propagated in nude mice.
26 and orthotopic xenografts of CL1-5 cells in nude mice.
27 rongly reduced the growth of ccRCC tumors in nude mice.
28 to enhanced lethality of tumor xenografts in nude mice.
29 tides targeting HE4 arrested tumor growth in nude mice.
30 d enhanced metastatic potential in allograft-nude mice.
31 eration in vitro and HCC xenograft growth in nude mice.
32 lations markedly inhibited tumorigenicity in nude mice.
33 oliferation in vitro and after grafting onto nude mice.
34 the AKT pathway in the CAM model, as well as nude mice.
35 cells that were subcutaneously implanted in nude mice.
36 nd diminishes tumorigenesis of xenografts in nude mice.
37 n promotes tumor growth of AFP- HCC cells in nude mice.
38 nospheres in soft agar and nodules/tumors in nude mice.
39 nvasion, migration, and xenograft tumors, in nude mice.
40 proliferation, migration and tumor growth in nude mice.
41 1 in vitro but also enhanced tumor growth in nude mice.
42 nograft growth and invasion of CRC tumors in nude mice.
43 gar, and increased xenograft tumor growth in nude mice.
44 s and generated transplantable xenografts in nude mice.
45 ream of EN, and abolishes tumor formation in nude mice.
46 ration and tumor formation and metastasis in nude mice.
47 reased growth of tumor xenografts in athymic nude mice.
48 inhibition of HCC xenograft tumor growth in nude mice.
49 the human osteosarcoma MNNG/HOS xenograft in nude mice.
50 rived xenografts expressing SLC13A5-shRNA in nude mice.
51 rowth inhibition of LoVo xenografts grown in nude mice.
52 t growth in 3T3 cells and tumor formation in nude mice.
53 of mammospheres and reduced tumor volume in nude mice.
54 ed the growth of human leukemia xenograft in nude mice.
55 th of xenograft tumors from HCT-116 cells in nude mice.
56 -amplified neuroblastoma xenograft growth in nude mice.
57 elial MCF-10A cells with malignant growth in nude mice.
58 culture, and tumor growth and metastasis in nude mice.
59 osis, and marked regression of xenografts in nude mice.
60 skin on the muzzles and tails of athymic NCr nude mice.
61 colorectal xenograft tumors was examined in nude mice.
62 ing human thymus tissue into athymic C57BL/6 nude mice.
63 nhibits tumor growth in HCT116 xenografts in nude mice.
64 ion and growth of subcutaneous xenografts in nude mice.
65 its the growth of breast tumor xenografts in nude mice.
66 cancer cells in vitro and tumor formation in nude mice.
67 tively target to mucin4-expressing tumors in nude mice.
68 on in soft agar, and in vivo tumor growth in nude mice.
69 suppressed the growth of tumor xenografts in nude mice.
70 colorectal cancer xenograft model in athymic nude mice.
71 slows the growth of HCC xenograft tumors in nude mice.
72 not different between wild type and athymic nude mice.
73 o and inhibits PCa xenograft tumor growth in nude mice.
74 strated by PET imaging in HT29 tumor-bearing nude mice.
75 th upon transplantation into immunodeficient nude mice.
76 arly after microinjection of OVCAR3 cells in nude mice.
77 ed versus untreated HCC cells was studied in nude mice.
78 nd their metastatic potential was studied in nude mice.
79 een with P1.204 or seen with P815 growing in nude mice.
80 giogenesis when injected subcutaneously into nude mice.
81 ts in a delay of tumor growth in xenografted nude mice.
82 KB-3-1 and COLO-205 tumor xenograft-bearing nude mice.
83 xenografts in the mammary fat pads of female nude mice.
84 to be nearly abolished in immunocompromised nude mice.
85 llomavirus has shown broad tissue tropism in nude mice.
86 CSC-like cells formed subcutaneous tumors in nude mice.
87 nd promotes the formation of fibrosarcoma in nude mice.
88 on and peritoneal tumor formation in athymic nude mice.
89 cells (A2780) implanted orthotopically into nude mice.
90 s ATF4 expression and its tumor formation on nude mice.
91 HV (TIVE-KSHV) into hyperglycemic and normal nude mice.
92 lation, Ki-67 expression and tumor growth in nude mice.
93 ly into NTR1-positive HT29 xenograft-bearing nude mice.
94 anted into calvarial defects created in CD-1 nude mice.
95 inhibition of HCC xenograft tumor growth in nude mice.
96 ctionally less effective at wound closure in nude mice.
97 l migration and abolished lung metastasis in nude mice.
98 bits proteasome function and tumor growth in nude mice.
99 cycle analysis and in vivo tumorigenesis in nude mice.
100 -expressing human lung cancer cell line into nude mice.
101 he multidrug resistant MCF-7/ADR xenografted nude mice.
102 plates, colonies in soft agar, and tumors in nude mice.
103 ses tumor growth and metastatic potential in nude mice.
104 rived orthotopic xenograft tumors in athymic nude mice.
105 ggressive orthotopic tumor models in athymic nude mice: a human PC-3 M-luc-C6 prostate tumor and a hu
108 cells, and in both eight-patient bloods and nude mice administered with the labeled CTCs in comparis
109 K1/2 inhibitor given to TNF-alpha-pretreated nude mice after human SSRBC infusion and onset of vasooc
110 experiments were performed on tumor-bearing nude mice after subcutaneous injection of RIN-m5F cells.
111 rapid tumor formation and growth in athymic nude mice after subcutaneous inoculation of the transfec
113 ent growth, slower xenograft tumor growth in nude mice and have decreased phosphorylation of AKT.
114 tifying the biodistribution of antibodies in nude mice and provides an alternative to PET analysis in
116 ChA-1 cells were injected into the flanks of nude mice and treated with miR-24 inhibitor or inhibitor
117 ibits growth of A375M melanoma xenografts in nude mice and whether rimcazole treatment changes (18)F-
119 family mutation efficiently form sarcomas in nude mice, and a Ras-ZEB1-Akt pathway then causes transi
120 ces hypoxia in FaDu and HCT116 xenografts in nude mice, and causes a significant tumour growth delay
121 lorectal cancer metastases were generated in nude mice, and epifluorescence imaging of ICG, as well a
122 and H1975) were subcutaneously implanted in nude mice, and growth was followed by caliper measuremen
124 matrix proteins, subcutaneous tumor size in nude mice, and invasive behavior, including bone marrow
125 nd cervical cancer cell xenograft in vivo in nude mice, and suppress cervical cancer cell migration a
126 nt growth inhibition of MOLM13 xenografts in nude mice, and the activity correlates with inhibition o
127 bodies and transferred into infected SCID or nude mice, and the animals received the same antibody ev
128 2 were injected into the pancreas of athymic nude mice, and their local and distant spread was monito
129 molecules were then determined in naive CD-1 nude mice, and tumor targeting was assessed in CD-1 nude
131 ously and orthotopically implanted tumors in nude mice, and was accompanied by c-SRC downregulation.
141 ging experiments were performed with athymic nude mice bearing A33 antigen-expressing, SW1222 colorec
142 dent SSM3 mouse mammary tumors, male athymic nude mice bearing androgen-dependent CWR22 prostate canc
143 trigel suspension model was established with nude mice bearing cells equally infected with each repor
145 ncer xenografts, and male and female athymic nude mice bearing estrogen-independent MDA-MB-231 human
146 jected intravenously into the BALB/c athymic nude mice bearing folate receptor (FR)-overexpressing KB
147 nd biodistribution studies were performed in nude mice bearing HCC4006 and A549 xenograft tumors.
149 icacy of PEG-GIRLRG peptide was evaluated in nude mice bearing heterotopic cervical (HT3), esophageal
150 ce, and tumor targeting was assessed in CD-1 nude mice bearing high-HER2-expressing NCI-N87 tumors an
154 and PLX4720 induced tumor regression in both nude mice bearing melanoma xenografts and in a genetical
155 In vivo antitumor efficacy was tested in nude mice bearing PSMA+ PC3 PIP or PSMA- PC3 flu flank x
156 tion and SPECT/CT imaging experiments, using nude mice bearing PSMA-positive PC-310 and PSMA-negative
161 of tumor cure while being well tolerated by nude mice bearing subcutaneous GPA33-positive SW1222 xen
162 One-hour dynamic PET scans were performed on nude mice bearing subcutaneous human head and neck tumor
163 cs of (89)Zr-DFO-AC-10 was studied in BALB/c nude mice bearing subcutaneous human Karpas 299 tumors (
164 nd biodistribution studies were performed on nude mice bearing U87MG and MDA-MB-231 xenografted tumor
166 /kg MEK1/2 inhibitor to TNF-alpha-pretreated nude mice before human SSRBC infusion inhibited SSRBC ad
167 antly inhibited the growth of U937 tumors in nude mice by both intraperitoneal and oral administratio
171 scles were subcutaneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles Ri
172 117085 significantly reduces tumor growth in nude mice compared with control untreated mice or either
174 nhibited orthotopic prostate tumor growth in nude mice, compared with monotherapy, by reversing the e
175 significantly reduced cell proliferation in nude mice, compared with untreated controls (p = 0.02).
176 taneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles River Laboratories
183 cells were subcutaneously transplanted into nude mice, DPSC/CTL cells induced mineralized tissue for
184 drastically reduced tumor growth in athymic nude mice, due to down regulation of fibroblast growth f
186 utside the primary tumor microenvironment in nude mice, exhibited signatures of immune evasion, incre
187 ics and induced complete tumor regression in nude mice explanted with lung cancer patient xenografts.
188 vivo an orthotopic liver injection model in nude mice further demonstrated that knockdown of RhoE en
189 ntal pulp on poly-l-lactic acid scaffolds in nude mice gave rise to perfect heterotopic ossicles in v
190 ication involving the TKD into the brains of nude mice generated high-grade astrocytomas with short l
195 ral administration of compound 28 to athymic nude mice implanted with human tumor xenografts afforded
196 ild cardiac hypertrophy was also observed in nude mice implanted with IDH2(R140Q)-expressing xenograf
199 uding HUVEC-mediated trophoblast invasion in nude mice, in vitro three-dimensional capillary tube for
200 tants strongly decreased tumor metastases in nude mice, indicating the requirement of PTTG for STAT3-
201 mplantation of heat-exposed HEPG2 cells into nude mice induced significantly larger, more aggressive
202 ties, SPECT and CT scans of HT29-xenografted nude mice injected with (177)Lu-3BP-227 were acquired, a
203 tically attenuates tumor growth in xenograft nude mice injected with human K562 leukemia cells and ce
204 Subcutaneous injection of TRIM24 iHMECs in nude mice led to growth of intermediate to high-grade tu
205 id gland scaffolds into the renal capsule of nude mice led to the differentiation of transplanted hDF
206 rs and xenografts established in NOD-SCID or nude mice, low MCPIP1 levels correlated strongly with in
216 e absence of a reliable islet potency assay, nude mice (NM) transplantation is the criterion standard
218 ce fulfilled criteria for the model but only nude mice offered sufficient availability for large ther
220 ve transfer of congenic T cells into athymic nude mice prior to infection did not alter lesion size.
221 implantation of LOX-treated neocartilage in nude mice promoted further maturation of the neotissue,
222 uman prostate xenograft model established in nude mice, RAD001 alone or in combination with docetaxel
224 ansplantation of hERG1-expressing cells into nude mice resulted in an increased incidence of tumors.
225 5alpha-depleted pancreatic cancer cells into nude mice resulted in markedly reduced tumorigenicity (P
226 KD SCC1 cells into the floor of the mouth in nude mice resulted in the formation of significantly sma
227 ne was sufficient to induce tumorigenesis in nude mice resulting in short lifespan irrespective of wh
229 D25(+)IL7Ralpha(+)) after grafting recipient nude mice revealed that MPP3 cells were the most effecti
230 ografts of MKN45/5FU cells in the stomach of nude mice revealed that these cells had a high potential
231 Tri-Modality Reporter Mouse derived MSCs in nude mice showed linear correlation with the cell number
232 cancer cells injected intra-peritoneally in nude mice showed significant reduction (p<0.002) in tumo
233 SCs transplanted to the ischemic hindlimb of nude mice showed significantly higher BLI and PET signal
235 lonization of melanoma cells in the lungs of nude mice, suggesting an increase in metastatic potentia
236 pressed DNAJB6a formed tumors more slowly in nude mice than control cells or cells that expressed a m
240 expressing versican siRNA were injected into nude mice, the resulting tumors displayed significantly
241 n PMP tissue was i.p. grafted and grown into nude mice, then constituted into reliable and reproducib
242 and AGS, which do and do not form tumors in nude mice, to identify their genomic differences relevan
245 an ovarian SKOV3 tumors cells into 14 female nude mice, treatment with vehicle or pazopanib (2.5 mg p
246 e than WT cybrids, however, when injected in nude mice, tRNAmut cybrids produced larger tumours and s
248 sing head and neck cancer xenograft model in nude mice, tumor growth was inhibited by CXCR7-targeting
250 ia conditions, were then assessed in healthy nude mice using the left kidney and spleen as reference
253 Growth of PLC5 tumor xenografts in BALB/c nude mice was inhibited by daily oral treatment with nil
254 of WT HBx-expressing cells to form tumors in nude mice was significantly higher than that of mutant H
257 ceous glands in Fatp4 null skin grafted onto nude mice were found to be dystrophic and enwrapped by t
261 and preliminary in vivo xenograft studies in nude mice were performed to evaluate target binding.
262 otently inhibited DMS-53 xenograft growth in nude mice when given by oral gavage, with no marked alte
263 on in PCa cells and induces larger tumors in nude mice, whereas its silencing decreased proliferation
264 sulted in marked regression of xenografts in nude mice, whereas the delivery of an miR-125a inhibitor
265 , n = 6) were xenografted into castrate male nude mice which were then treated for 35 days with vehic
266 g in aggressive tumor formation in xenograft nude mice, which could be suppressed by combined treatme
268 ive in suppressing xenograft tumor growth in nude mice, which underlines the translational potential
269 ous cell carcinoma upon inoculation into the nude mice, while parental HaCaT cells remain non-tumorig
270 -fragment dose for imaging was determined in nude mice with a subcutaneous head and neck carcinoma mo
272 avenously administered every 3 d for 16 d to nude mice with AR42J tumor xenografts that were approxim
273 OIS and enabled tumor transplants to grow in nude mice with characteristic cell morphology of anaplas
274 uman breast carcinoma MX-1 xenograft-bearing nude mice with compounds 18b and 28c achieved more than
275 In vivo treatment of A375 xenograft-bearing nude mice with cryptolepine (10 mg/Kg body weight, i.p.)
276 t overexpress MFSD2A were transferred to CD1 nude mice with dextran sodium sulfate-induced colitis, w
279 uorescence imaging to detect ccRCC tumors in nude mice with RCC xenografts by using mAb girentuximab
281 ing product, (18)F-FVIIai, was injected into nude mice with subcutaneous human pancreatic xenograft t
282 11B6 uptake was performed on NMRI and BALB/c nude mice with subcutaneous LNCaP xenografts up to 14 d
283 ouse) was injected intravenously into BALB/c nude mice with subcutaneous PSMA-expressing LNCaP tumors
284 111)In-labeled ADCs were performed on BALB/c nude mice with subcutaneous PSMA-positive LS174T-PSMA xe
286 Methods: Two groups of 6 adult female BALB/c nude mice with subcutaneously implanted tumors underwent
288 ary tumors, in an NFATc1-dependent manner in nude mice with T-cell deficiency, revealing an addiction
289 targeting potential and antitumor effects in nude mice with tumors that were sensitive or resistant t
294 mouse models, including intact or castrated nude mice xenografted with androgen-sensitive LNCaP cell
295 -3 MBq) were injected via the tail vein into nude mice xenografted with BxPC3 (integrin alphavbeta6-p
296 androgen-insensitive C81 cells and castrated nude mice xenografted with castration-resistant C4-2 and
297 ble for small-animal PET studies in multiple nude mice xenografted with the A431 carcinoma cell line.
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