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1 low cytometry to evaluate the changes in the numbers of leukocytes.
2 antly higher levels of IFN-gamma, had higher numbers of leukocytes accumulated in the lungs, and clea
3 crophage activity was less striking, but the number of leukocytes adherent to the sinusoidal lining h
4 ce resulted in a significant increase in the number of leukocytes adhering to endothelial cells and e
5 nd 22, mice were killed and analyzed for the number of leukocytes and colony-forming units (CFU) in b
6 us administration of pancreatic ascites, the number of leukocytes and the protein concentration withi
7 WT mice developed similar levels of AHR and numbers of leukocytes and cytokine levels in BAL fluid a
8 ociated bacterial infections, and had higher numbers of leukocytes and higher plasma levels of C-reac
10 ocardium, CD4 KO mice displayed higher total numbers of leukocytes and proinflammatory monocytes (18.
11 ces a significant and persistent increase in numbers of leukocytes at the site of the delayed-type hy
12 al loads in blood and tissues, and increased numbers of leukocytes, B cells, and total serum IgM.
13 Whereas there was no difference in absolute number of leukocytes (CD45high), CD4 T cells, or CD8 T c
14 rst 24 h after inoculation yet decreased the numbers of leukocytes containing intracellular (mCherry-
15 fter transfusion of blood containing defined numbers of leukocytes expressing selected mismatched ant
16 , or CD3 cell counts, HIV-1 RNA viremia, the number of leukocytes in CVL fluid, or HIV-1 therapy.
18 he patient's underlying disease and age, the number of leukocytes in the graft, the degree of HLA dis
19 Flt3L administration, on average, the total number of leukocytes in the peripheral blood increased f
21 ured units were directly proportional to the number of leukocytes in the range of 0.028-4.2 U L(-1) (
22 in these regions and had strikingly reduced numbers of leukocytes in the bronchoalveolar lavage flui
24 n, IFN-gammaR-/- mice have recruited greater numbers of leukocytes into their lungs, with neutrophils
25 mice were decreased by 72% and 93%, and the number of leukocytes invading the subdermal ear tissue w
26 o 3 h after injury revealed that the reduced number of leukocytes is not due to a reduction in random
28 infection of the bovine mammary gland, large numbers of leukocytes migrate into the udder, resulting
35 M-1 nor anti-LFA-1 significantly reduced the number of leukocytes rolling on venule endothelial surfa
36 ng intravital microscopy, we showed that the number of leukocytes rolling per minute in unstimulated
37 the monoclonal antibody CD11b/18 (P < .05); numbers of leukocytes sequestered in the ischemic hemisp
40 s apply to the immune system, in which small numbers of leukocytes, such as pulmonary dendritic cells
41 ood flow does not result in a representative number of leukocyte TEM as compared to pulsatile flow, w
42 nt with sulfasalazine markedly decreased the number of leukocytes that accumulated in the inflamed (c
43 e of PGIA, we found a steady increase in the number of leukocytes that exhibited firm adherence to th
46 on donor endothelial cells would reduce the number of leukocytes transmigrating into the allograft,
48 ed expression of Hoxa3 reduces the excessive number of leukocytes within the db wound; however, the e
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