ライフサイエンスコーパス: nutritional intervention

1 s diseases can be reversed or ameliorated by nutritional intervention.

2 20-25% was observed which was refractory to nutritional intervention.

3 d at enrollment and, for cases, after a 5-mo nutritional intervention.

4 bating infection in aged individuals through nutritional intervention.

5 ether pigs and humans respond similarly to a nutritional intervention.

6 IBS-36 questionnaire assessed the impact of nutritional intervention.

7 anabolic signaling that may require targeted nutritional intervention.

8 This process may limit the efficacy of nutritional intervention.

9 to alleviate complex disorders via tailored nutritional intervention.

10 reatment of severe malnutrition in emergency nutritional interventions.

11 tor learning and memory can be reversed with nutritional interventions.

12 eered filtering systems to phytogenetics and nutritional interventions.

13 ta immaturity that is not rescued by current nutritional interventions.

14 ill drive future development of personalized nutritional interventions.

15 tially ameliorated following two widely used nutritional interventions.

16 and detoxifier) can be reversed by targeted nutritional interventions.

17 infections, smoking cessation, and possibly nutritional interventions.

18 isease states and thus could be a target for nutritional interventions.

19 5 and 2015 was performed to identify RCTs of nutritional interventions administered to critically ill

20 evaluation is required to determine whether nutritional interventions and exercise training may pres

21 Nutritional interventions and the IL-6 response to exerc

22 The applicability of many nutritional interventions and their effects on outcomes

23 dogenous lipase activities may be altered by nutritional interventions, and further, that accelerated

24 is possible to reverse these effects through nutritional interventions applied later in development.

25 ematic reviews evaluating dietary intake and nutritional interventions are becoming common but are re

26 The best way to determine if nutritional interventions are effective is to compare th

27 clinical and animal studies have identified nutritional intervention as a viable method to curtail t

28 The role of nutritional interventions as adjunct therapies to ART me

29 rcent coverage of the same environmental and nutritional interventions, as envisioned by the MDGs, wo

30 nominated friends increased adoption of the nutritional intervention by 12.2% compared with random t

31 stic rationale and experimental evidence for nutritional interventions commonly used in PMDs, includi

32 Targeted nutritional interventions could therefore save millions

33 t their illness and enhance adjustment, or a nutritional intervention, designed to promote a more hea

34 Nutritional interventions directed at these clinical con

35 OFC gains in SGA term infants improve after nutritional intervention during the first 9 mo of life a

36 ck of high-quality evidence that proves that nutritional interventions during critical illness reduce

37 Nutritional interventions during pregnancy have been suc

38 Thus, whether nutritional interventions early in an intensive care uni

39 utritional treatment may be efficacious as a nutritional intervention for preschoolers aged 2 to 6 ye

40 is framework are presented in the context of nutritional interventions for chronic diseases.

41 ewborn provision of vitamin K, are important nutritional interventions for infants and children.

42 lth outcomes, and investigating the value of nutritional interventions for mitochondrial disease cond

43 irm the considerable potential for a role of nutritional interventions for osteoarthritis, but they e

44 tophagy/lysosomal system through appropriate nutritional intervention has significant beneficial effe

45 gression, this suggests limited evidence for nutritional interventions having an impact on delaying H

46 Hg (HR, 6.9; 95% CI, 1.5-32.1), and use of a nutritional intervention (HR, 2.3; 95% CI, 1.3-4.1) were

47 uch growth is complex and may be affected by nutritional interventions imposed many years previously.

48 Behavioral and nutritional intervention improved energy intake and HAZ

49 The road to nutritional intervention in cancer prevention has led to

50 perhaps, in view of the adjunctive nature of nutritional intervention in critical illness, be focused

51 Fetal body composition may be modifiable via nutritional intervention in the mother and thus may play

52 and economic efficiencies when investigating nutritional interventions in chronic diseases.

53 Given such complex pathways, nutritional interventions in CKD, when applied in concer

54 tical challenges reduce the effectiveness of nutritional interventions in complex emergencies, and im

55 lateral sclerosis, and support the study of nutritional interventions in larger randomised controlle

56 heterogeneity remain amongst many studies of nutritional interventions in surgical patients.

57 timates of delta occur frequently in RCTs of nutritional interventions in the critically ill that are

58 suggest that future research should focus on nutritional interventions in the pre- and postdischarge

59 The results may be used to provide tailored nutritional interventions in these patients.

60 Combined with other studies of choline and nutritional interventions in this population, this study

61 Nutritional interventions included general ration distri

62 However, in the United States, several nutritional interventions, including high-protein diets,

63 Findings of controlled trials indicate that nutritional interventions, including vitamin A palmitate

64 Furthermore, nutritional intervention increased renal, hepatic, and i

65 Nutritional interventions involve provision of regular m

66 This nutritional intervention is likely to affect local gut i

67 gy expenditure and energy intake change, but nutritional intervention is not necessarily beneficial.

68 ve studies and longitudinal studies in which nutritional intervention is provided before cognitive de

69 will serve to re-define how the efficacy of nutritional interventions is assessed.

70 ce supporting similar metabolic responses to nutritional interventions is lacking.

71 tochondrial disorders (PMDs), and the use of nutritional interventions is routine in their supportive

72 sensitive period of the life cycle in which nutritional intervention may reap the greatest benefits.

73 Nutritional intervention may therefore selectively influ

74 Early nutritional interventions may modulate health risks in p

75 ese methods to estimate the causal effect of nutritional interventions on clinical outcomes among cri

76 graphy for evaluating the effect of prenatal nutritional interventions on components of fetal growth.

77 fically answer the question on the impact of nutritional interventions on HIV disease progression and

78 Pediatrics statement on the effects of early nutritional interventions on the development of atopic d

79 Nutritional intervention reduced hemolysis-related renal

80 Nutritional intervention requires repeated measurement o

81 ing leads, and describes future prospects in nutritional intervention research.

82 , the search plan for a systematic review of nutritional interventions should be comprehensive.

83 art defect risk would help to refine current nutritional intervention strategies to reduce risk and m

84 ects by personalizing drug prescriptions and nutritional intervention strategies.

85 Most nutritional intervention studies in CHD patients traditi

86 Nutritional intervention studies performed in animals an

87 eviewed the results of randomized controlled nutritional intervention studies that have assessed the

88 The first generation of phase III nutritional intervention studies to prevent cancer has b

89 ition studies, possible extensions of N-of-1 nutritional intervention studies, and areas of future re

90 In this nutritional intervention study 46 healthy and non-obese

91 articipants of the prospective German Infant Nutritional Intervention study after 10 years of follow-

92 amples and 29 quality control samples from a nutritional intervention study.

93 Pancreatic insufficiency was managed with nutritional interventions that included high-calorie die

94 plasma ANGPTL4 concentrations after various nutritional interventions that increase NEFA concentrati

95 We detail various nutritional interventions that may affect the IL-6 respo

96 ted to result in dietary recommendations and nutritional interventions that optimize individual healt

97 s for randomized controlled trials (RCTs) of nutritional interventions that used mortality as the pri

98 The relationships between the type of nutritional intervention, the gut microbiota, and therap

99 >90 d survival), creating an opportunity for nutritional intervention to stop or reverse cachexia.

100 Nutritional interventions to complement breastfeeding af

101 Studies addressing pretransplant nutritional interventions to reduce AML relapse rates ar

102 These results emphasize the need for early nutritional interventions to reduce daily sugar intake i

103 nal pathology, providing a basis for testing nutritional interventions to reduce malaria-associated m

104 dings warrant further studies of therapeutic nutritional interventions to restore arginine metabolism

105 f the abnormal metabolic profile, a targeted nutritional intervention trial with folinic acid, betain

106 vestigations of the complexities inherent to nutritional intervention trials.

107 ction (PCR), were assessed over 5 years of a nutritional intervention, which promoted adherence to th

108 The long-term effect of nutritional intervention with hydrolysate infant formula

109 status after treatment suggest that targeted nutritional intervention with methylcobalamin and folini

110 A set of real samples obtained after human nutritional intervention with placebo or polyphenol-rich

111 Emphasis on combination of nutritional interventions with exercise and newer bone-s

112 a water, sanitation, handwashing (WSH), and nutritional intervention would slow TL attrition during