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1 1a1, FnEDA, and TIMP1 mRNA expression in the obstructed kidney.
2 ced significant upregulation of FnEDA in the obstructed kidney.
3 ntiapoptotic and antifibrotic effects in the obstructed kidney.
4 filtration of T cells and macrophages in the obstructed kidney.
5 ndogenous beta-galactosidase activity in the obstructed kidney.
6 is greater in the adult than in the neonatal obstructed kidney.
7 and progressive interstitial fibrosis of the obstructed kidney.
8 the fibrotic response observed in innervated obstructed kidneys.
9 r-1(+) inflammatory DCs were also present in obstructed kidneys.
10 ha and vascular endothelial growth factor in obstructed kidneys.
11 bone marrow-derived fibroblast precursors in obstructed kidneys.
12 downregulation of both laminin and TINag in obstructed kidneys.
13 ta expression or Smad 2/3 phosphorylation in obstructed kidneys.
14 tion and nuclear translocation of smad2/3 in obstructed kidneys.
15 differentiated from obstructed and partially obstructed kidneys.
16 obstructed CSF-1-deficient compared with WT obstructed kidneys.
17 erved renal mass and gross morphology of the obstructed kidneys.
18 pletely abolished alpha SMA induction in the obstructed kidneys.
19 prevented renal interstitial fibrosis in the obstructed kidneys.
20 with the expert panel in 92% (12/13) of the obstructed kidneys, 93% (38/41) of the unobstructed kidn
22 b accumulated at lower concentrations in the obstructed kidney and exhibited a more diffuse whole-bod
23 and alpha-smooth muscle actin expression in obstructed kidneys and enhanced tubulointerstitial injur
24 3 expression increased in the fibroblasts of obstructed kidneys and was associated with increased Ca(
25 al cells upregulated the chemokine CXCL16 in obstructed kidneys, and circulating fibroblast precursor
26 of nonobstructed kidneys, in 92% (33/36) of obstructed kidneys, and in 45% (13/29) of equivocal kidn
28 nsin-(1-7) to mice with UUO caused injury in obstructed kidneys compared with controls and increased
33 increased apoptosis and macrophage influx in obstructed kidneys from Mas(-/-) mice, compared with unt
36 from beta6(-/-) mice showed less injury than obstructed kidneys from wild-type (WT) mice, associated
37 hospho-FAK-positive CD11b macrophages in the obstructed kidneys from WT mice was clearly attenuated i
43 hat downregulation of SnoN expression in the obstructed kidney is mediated by an enhanced ubiquitin-d
47 hi and T cells and less apoptotic TEC in the obstructed kidneys of Csf1(op)/Csf1(op) mice compared wi
50 ) collagen - was substantially higher in the obstructed kidneys of mice with Agtr1(-/-) marrow than i
51 teinase-9 (MMP-9) induction was found in the obstructed kidneys of tPA(-/-) mice, which led to a dram
53 in the neonatal rat delays maturation of the obstructed kidney, possibly in part through suppressed e
54 dies: the unobstructed kidney; the partially obstructed kidney, proximally or distally obstructed, wi
55 cantly higher levels of each molecule in the obstructed kidney than in the nonobstructed kidney, the
56 expression and synthesis of periostin in the obstructed kidney that associated with the progression o
57 ation and extracellular matrix remodeling in obstructed kidneys, thus ameliorating tubulointerstitial
61 wth factors, UUO stimulates apoptosis in the obstructed kidney, which is quantitatively greater in th
62 analysis suggested resident DC maturation in obstructed kidneys with increased CD11b and less F4/80 e
63 tion and impairment of function; the totally obstructed kidney, with arrested renal function; and the
64 ce developed more severe renal damage in the obstructed kidney, with marked tubular atrophy and inter
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