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1 hat results from the development of in utero obstructive uropathy.
2 rosis and chronic renal failure secondary to obstructive uropathy.
3 nt females died at 4-6 months of age from an obstructive uropathy.
4 understanding the pathobiology of congenital obstructive uropathy.
5 which includes ischemic hypoxic insults, and obstructive uropathy.
6 the understanding of the pathophysiology of obstructive uropathy.
7 tones, or renal insufficiency as a result of obstructive uropathy.
8 d increasing use of medications for treating obstructive uropathy.
9 weight/obesity associated significantly with obstructive uropathy.
10 masses, urinary tract infection, trauma, and obstructive uropathy.
11 ovides valuable insight into a wide range of obstructive uropathies.
12 rovides unique insights into a wide range of obstructive uropathies and has been demonstrated to be u
13 ecessive disease characterized by congenital obstructive uropathy and abnormal facial expression.
17 ity was ischemic bowel (75%); the lowest was obstructive uropathy-associated urinary tract infection
19 tenatal intervention has been done for fetal obstructive uropathy for over a decade, yet little is kn
20 ing kidney in the fetus as it is affected by obstructive uropathy has had no significant advances in
21 In the current study, a model of chronic obstructive uropathy in the mouse is established and the
24 se models of postischemic renal fibrosis and obstructive uropathy, treatment with N-terminal Slit2 be
25 y Network criteria), exclusion criteria were obstructive uropathy, urothelial carcinoma, and metastat
26 those fetuses with the most severe forms of obstructive uropathy, usually associated with a fatal ne
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