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1 eye movements, but not in a control region (occipital cortex).
2 the healthy comparison group in the superior occipital cortex.
3 increased during 20S(-), most notably in the occipital cortex.
4 h as other regions in the insular and medial occipital cortex.
5 al lobe and with other areas of parietal and occipital cortex.
6 s of the inhibitory neurotransmitter GABA in occipital cortex.
7 and middle frontal gyri, the insula, and the occipital cortex.
8 c lamina of frontal, parietal, temporal, and occipital cortex.
9 n cooperation with perceptual systems of the occipital cortex.
10 same effect in an adjacent area, the lateral occipital cortex.
11 was correlated with rCBF in the amygdala and occipital cortex.
12 tive N1 enhancement was localized to lateral occipital cortex.
13 sses that generate the hallucinations in the occipital cortex.
14 rrupting connections between the eye and the occipital cortex.
15 which the figure region is routed to lateral occipital cortex.
16 nd intact implicit encoding in earlier-stage occipital cortex.
17 parahippocampal cortex, fusiform gyrus, and occipital cortex.
18 ivity to color, objects and faces in ventral occipital cortex.
19 ed performance nor diminished BOLD signal in occipital cortex.
20 in relative brain GABA concentration in the occipital cortex.
21 r brain areas, including the pre-frontal and occipital cortex.
22 raine attack have been shown to occur in the occipital cortex.
23 brainstem structures preceded activation of occipital cortex.
24 for myo-inositol or other metabolites in the occipital cortex.
25 erent times using an input function from the occipital cortex.
26 r medial prefrontal cortex, hippocampus, and occipital cortex.
27 nged from -33% in the caudate to -20% in the occipital cortex.
28 the anterior and posterior superior-parieto-occipital cortex.
29 regions and with expression of NR(1) in the occipital cortex.
30 well as relative metabolic increases in the occipital cortex.
31 ed to be very common throughout parietal and occipital cortex.
32 ered in widespread regions of hyperexcitable occipital cortex.
33 nor global attention was lateralized in the occipital cortex.
34 periaqueductal gray matter, cerebellum, and occipital cortex.
35 al controls during visual stimulation of the occipital cortex.
36 smaller in the gray and white matter of the occipital cortex.
37 bust multivariate decoding being reported in occipital cortex.
38 subcortically in bilateral basal ganglia and occipital cortex.
39 n no longer convey visual information to the occipital cortex.
40 pramarginal gyri as well as the left lateral occipital cortex.
41 MRS data were acquired from occipital cortex.
42 be, the right anterior insula, and bilateral occipital cortex.
43 ty, especially in the cerebellum and parieto-occipital cortex.
44 res correlated with metabolic changes in the occipital cortex.
45 ps, LO1 and LO2, in object-selective lateral occipital cortex.
46 rper tuned profile in more posterior ventral occipital cortex.
47 pagation from the V1 to the V2 region of the occipital cortex.
48 nd lower in sulcal, perirolandic, and medial occipital cortex.
49 stimulus size was reflected by activation in occipital cortex.
50 on compared with healthy control subjects in occipital cortex.
52 evaluations showed significant decreases in occipital cortex (9.7% and 10%) and significant relative
56 t OFC, right dorsolateral prefrontal cortex, occipital cortex, ACC, ventral striatum/nucleus accumben
57 rimary visual cortex, increased extrastriate occipital cortex activation selectively during maintaine
59 from right and left temporo-parietal cortex, occipital cortex and a central voxel incorporating basal
60 tain their connectivity with the reorganized occipital cortex and as a result influence processing of
61 dose DHA groups had greater decreases in the occipital cortex and cerebellar cortex, respectively.
62 ices in the frontal, parietal, temporal, and occipital cortex and in the subcortical region were obta
64 a different functional role than that in the occipital cortex and may be part of the neuronal mechani
66 ns of the dorsolateral prefrontal cortex and occipital cortex and quantitative true color image analy
67 set, face-selective sources in right lateral occipital cortex and right fusiform gyrus and sources in
69 al information flow between the left lateral occipital cortex and right intraparietal sulcus, as indi
70 roscopy data were acquired from the parietal-occipital cortex and supplementary motor area in all sub
72 rmalities involve a circuit encompassing the occipital cortex and the cortical/subcortical systems ph
74 tree approach revealed the centrality of the occipital cortex and the peculiar aggregation of cerebel
75 omponent, which include the superior parieto-occipital cortex and the rostral superior parietal lobul
77 Glucose concentrations in gray matter-rich occipital cortex and white matter-rich periventricular t
81 s between the anterior temporal lobe and the occipital cortex, and between bilateral occipital poles.
82 ses in limbic/paralimbic areas, temporal and occipital cortex, and cerebellum were also significantly
84 lts suggest that FEF and IPS modulate visual occipital cortex, and FEF modulates IPS, in relation to
85 temporal gyrus, middle temporal gyrus, right occipital cortex, and inferior frontal cortex was found
88 nial magnetic stimulation (TMS) to the human occipital cortex are immune to saccadic suppression, whe
90 ared to ATs, in the object sensitive lateral occipital cortex as well as in the face-sensitive occipi
91 c TMS pulses were delivered over lateralized occipital cortex at 100, 200, or 400 ms into the retenti
92 a (rOFA), a face-selective region in lateral occipital cortex, at different latencies up to 210 ms af
93 d reduced cerebral metabolism in the primary occipital cortex (BA 17) that was revealed only by 3D-SS
94 nsorimotor cortex, superior temporal cortex, occipital cortex, basal ganglia, limbic system and hypot
96 tic stimulation (rTMS) was applied to medial occipital cortex before presentation of the same task.
97 as 1 (LO1) and 2 (LO2), in the human lateral occipital cortex between the dorsal part of visual area
98 ving words, these subjects activated lateral occipital cortex bilaterally in addition to the language
99 es in apoD immunoreactivity were detected in occipital cortex (Brodmann's area 18) in either group, o
100 h significant posterior flow deficits in the occipital cortex (Brodmann's areas 18 and 19), usually s
103 onal partitioning of visual processes in the occipital cortex by Riddoch), but there were also other
105 recorded from the scalp overlying the human occipital cortex can be entrained to the second or third
106 ce a muscle twitch or block movement; TMS of occipital cortex can produce visual phosphenes or scotom
107 his animal model, unilateral ablation of the occipital cortex causes unequivocal apoptosis of cortico
108 refrontal cortex, amygdala, temporal cortex, occipital cortex, cerebellum and thalamus (P<0.05 correc
110 rom candidate pseudoreference regions (i.e., occipital cortex, cerebellum, and whole brain) to obtain
111 e that underpins size knowledge: the lateral occipital cortex codes perceptually based aspects (stati
112 in the frontoparietal cortex and 60% in the occipital cortex compared to age-matched normal controls
113 uptake in the posterior temporoparietal and occipital cortex compared to clinically normal controls,
114 ophy additionally showing elevated uptake in occipital cortex compared with early-onset Alzheimer's d
115 which in humans, is more highly expressed in occipital cortex compared with the remainder of cortex t
116 guously and slowly (3.5 +/- 1.1 mm/min) over occipital cortex, congruent with the retinotopy of the v
118 ncrease) were observed in the gray matter of occipital cortex contralateral to the affected visual he
119 reases of alpha-band activity were seen over occipital cortex contralateral to the direction of the t
120 cortical activity in portions of the ventral occipital cortex, corresponding to known object areas wi
122 roups, and the subgenual anterior cingulate, occipital cortex (cuneus), and posterior cerebellum were
124 ulate; bilateral parahippocampal gyrus; left occipital cortex) demonstrated indistinguishable activit
125 ol, the normal alpha rhythm (8-13 Hz) in the occipital cortex disappears and a frontal alpha rhythm e
126 affecting distance judgments, while rTMS to occipital cortex disrupted distance but not roughness ju
127 PCr) and inorganic phosphate (Pi) within the occipital cortex during (activation) and after (recovery
129 ion of compensatory increases in rCBF of the occipital cortex during incremental learning and the lef
130 ecuneus, middle temporal gyrus, and superior occipital cortex during the anticipation of potential re
131 functional MRI to map activity in the human occipital cortex, during local and global attention, wit
132 dorsal occipital cortex or the right ventral occipital cortex, during the brief presentation of a tra
134 ranscranial magnetic stimulation of the left occipital cortex evoked contraction of right hand muscle
135 hese independently recorded variables, i.e., occipital cortex excitability and reactivity and EEG pha
137 tion of the changes in thickness across DF's occipital cortex, finding the most substantial loss in t
139 hey showed relative hypoactivity in the left occipital cortex for the low spatial frequency faces.
140 s are distributed widely across the inferior occipital cortex, fusiform areas, and the cingulate gyru
141 ients also appeared to have persistently low occipital cortex GABA after chronic benzodiazepine treat
142 panic disorder had a 22% reduction in total occipital cortex GABA concentration (GABA plus homocarno
143 of this study was to determine whether these occipital cortex GABA concentrations are altered after a
144 report, using this technique, suggested that occipital cortex GABA concentrations are reduced in pati
145 Depressed subjects had significantly lower occipital cortex GABA concentrations compared with healt
150 s of proton magnetic resonance spectroscopy, occipital cortex GABA concentrations were measured in 11
152 GABA neuronal response (blunted reduction of occipital cortex GABA level) to acute benzodiazepine adm
153 etic resonance spectroscopic measurements of occipital cortex GABA levels across the menstrual cycle
154 cts, who exhibited a significant decrease in occipital cortex GABA levels after this intervention.
155 ere were no significant correlations between occipital cortex GABA levels and measures of illness or
156 ated a highly significant (52%) reduction in occipital cortex GABA levels compared with the group of
158 a parallel-group, repeated-measures design, occipital cortex GABA responses to acute oral, open-labe
160 results indicate that superior IPS, but not occipital cortex, has a central role in VSTM storage.
162 t a silent zone at the posterior pole of the occipital cortex, implying a lack of complete cortical r
163 ntral striatum, inferior temporal gyrus, and occipital cortex in both depression and schizophrenia in
164 e EEG recordings from frontal, parietal, and occipital cortex in freely moving rats (n = 11) during a
165 y alpha-band activity (8-15 Hz) over parieto-occipital cortex in humans plays an important role in su
166 d potential responses were recorded over the occipital cortex in patients with schizophrenia and in a
167 , posterior cingulate cortex, precuneus, and occipital cortex in patients with TLE as compared with h
168 r cingulate cortices and deactivation in the occipital cortex in response to the drug-related stimulu
171 ontal and parietal control regions to visual occipital cortex in visuospatial attention, the goal mot
172 uperior parietal lobule and the left lateral occipital cortex) included the default mode network seed
175 parietal cortices without any change in the occipital cortex, indicating that Fgf2 is necessary to r
178 to those with persistent ADHD in the medial occipital cortex, insula, parahippocampus, and prefronta
179 daptation for small shape changes in lateral occipital cortex irrespective of category membership, co
181 emianopic patients and suggest that when the occipital cortex is damaged or inhibited, and the visual
182 corroborative clinical evidence suggest that occipital cortex is engaged in tactile tasks requiring f
184 at the spatial pattern of propagation in the occipital cortex is non-concentric with a variable exten
185 pping activations in the lateral and ventral occipital cortex [known as the lateral occipital complex
186 siform gyrus, and insula, and extending into occipital cortex (left hemisphere) and orbitofrontal cor
187 to localize object-specific ROIs in lateral occipital cortex (LO) and scene-specific ROIs in the par
188 probe whether dorsal regions of the lateral occipital cortex (LO) are activated in tactile recogniti
189 ent fMRI studies have identified the lateral occipital cortex (LO) as a potential neural origin of th
190 with scrambled line drawings in the lateral occipital cortex (LO) of the ventral stream, an area tha
191 (2) a decrease in activation of the lateral occipital cortex (LO), and (3) a decrease in the dorsal
192 region, such as the shape-selective lateral occipital cortex (LO), must still base its activation on
193 pattern in both the retinotopic and lateral occipital cortex (LOC) in humans contains category infor
194 l FFA and bilateral object-selective lateral occipital cortex (LOC) predicted the participants' abili
195 Neurostimulation was targeted at the lateral occipital cortex (LOC), a key region for object percepti
200 on of a mid-level visual region [the lateral occipital cortex (LOC)] in a population much younger tha
203 rch suggests an enhanced excitability in the occipital cortex may underlie this reaction during IPS,
206 pital face area, fusiform face area, lateral occipital cortex, mid fusiform, parahippocampal place ar
207 scranial magnetic stimulation (TMS) over the occipital cortex modulated the impact of memory on searc
208 ions between the left temporal lobe and left occipital cortex not showing evidence of development unt
209 a mean tissue-to-plasma efflux constant for occipital cortex obtained in 10 subjects (ki=0.037 min(-
211 relative to total creatine (GABA/Cr) in the occipital cortex (OC), anterior cingulate cortex (ACC),
213 e report motor cortical function in the left occipital cortex of a subject who suffered a left middle
217 m the dorsolateral prefrontal cortex and the occipital cortex of elderly patients with schizophrenia
220 n the dorsolateral prefrontal cortex and the occipital cortex of patients with schizophrenia than in
225 the right supraorbital area], but not of the occipital cortex or sham stimulation, increased the prop
226 or parietal cortex, the right or left dorsal occipital cortex or the right ventral occipital cortex,
227 me trials by (1) pretrial phase synchrony of occipital cortex oscillations in the 8-9 Hz (low alpha)
229 anterior cingulate cortex (P =.003) and the occipital cortex (P =.01) in the depressed subjects.
230 cerebellum, with intermediate values in the occipital cortex, parietal cortex, and caudate putamen.
232 ge of pERK1/2 levels in the temporal cortex, occipital cortex, parietal cortex, midbrain, and medulla
233 hat in congenitally blind subjects the right occipital cortex participates in a functional network fo
234 owed significant metabolic reductions in the occipital cortex, particularly in the primary visual cor
235 Furthermore, these findings suggest the occipital cortex plays a key role in supporting mental r
236 erior cingulate cortex (ACC) and the parieto-occipital cortex (POC) in adolescents (n=30) and emergin
238 metic problem complexity in ventral temporal-occipital cortex, posterior parietal cortex, and medial
239 al cerebellum and in a region in the lateral occipital cortex presumably corresponding to the area KO
250 cortex was unresponsive to Braille words and occipital cortex responded to spoken words but not diffe
253 seline BOLD response in the bilateral middle occipital cortex, selectively during the stimulus-proces
254 ntrast, superior parietal lobule and parieto-occipital cortex showed greater activation to the apart
255 recruited by patients only: the left dorsal occipital cortex showed systematic activation in all con
256 Electroencephalogram recorded over the left occipital cortex showed: 1) coherence with electromyogra
257 trastriate occipital cortex, whereas primary occipital cortex shows consistent activity across all sc
258 ttsburgh Compound B retention selectively in occipital cortex, sparing regions typically labeled in A
260 sponse decrements in right frontal and right occipital cortex, strongly supporting the compensatory r
261 ntrol (medulla and pons), and vision (dorsal occipital cortex, superior colliculi and lateral genicul
263 tion of the ipsilateral field within lateral occipital cortex that is normally associated with visual
265 , after suppressing the activity in the left occipital cortex, the congruency-dependent response faci
266 cts activated association areas in the right occipital cortex, the foci of which were similar to area
267 ical areas, including portions of the medial occipital cortex, the lateral parietal cortex, and the s
269 information being encoded (fusiform/lateral occipital cortex), they each exerted opposite effects on
270 simplified reference tissue method with the occipital cortex time activity curve as an input functio
272 second method was also graphic; however, the occipital cortex time-activity curve was used as the inp
273 ium was taken to represent bound tracer, the occipital cortex time-activity curve was used to represe
274 hod, the difference between the striatal and occipital cortex time-activity curves at secular equilib
275 oops that are dominated by flow from parieto-occipital cortex to integrative frontal areas in the hig
276 scranial magnetic stimulation (TMS) over the occipital cortex to interfere with attentional reorienti
277 s in excitability affect the function of the occipital cortex to other, less provocative visual stimu
279 ed negative BOLD response (NBR) in the human occipital cortex, triggered by stimulating part of the v
280 Here we show that disrupting function of the occipital cortex using focal transcranial magnetic stimu
284 brain regions, including the ventrotemporal occipital cortex (VTOC), the posterior parietal cortex,
288 unctional activation (measured with fMRI) in occipital cortex was more extensive when participants vi
289 By contrast, pattern similarity in lateral occipital cortex was related to memory only when context
290 positron emission tomographic scanning, the occipital cortex was strongly activated in the congenita
291 tal, left superior parietal, and left middle occipital cortex) was evaluated using fMRI datasets acqu
292 ht posterior insula, anterior cingulate, and occipital cortex were examined in subjects at rest.
293 Granger causality from FEF and IPS to visual occipital cortex were significantly greater than both bo
294 in the frontoparietal cortex and 27% in the occipital cortex when compared with young (3-6 months) a
295 scenes approximately 1 s before extrastriate occipital cortex, whereas primary occipital cortex shows
297 istration in overlapping areas in the middle occipital cortex while performing the same task conditio
298 ctive cathodal-inhibitory tDCS over the left occipital cortex, while, in control Experiment 2, partic
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