ライフサイエンスコーパス: occipital lobe

1 (relative sparing of the hippocampus and/or occipital lobe).

2 ons measured (temporal, frontal, and parieto-occipital lobes).

3 ith fibres of the lateral pathway within the occipital lobe.

4 llow damage to primary visual cortex, in the occipital lobe.

5 r colour in more dorsolateral regions of the occipital lobe.

6 intracranial electrodes placed on the human occipital lobe.

7 ure and can be traced posteriorly around the occipital lobe.

8 opographically organized visual areas of the occipital lobe.

9 rface coil to measure a 14-cm3 volume in the occipital lobe.

10 gamma peak frequency and amplitude from the occipital lobe.

11 in extrastriate visual cortex of the ventral occipital lobe.

12 emporal lobe, posterior parietal cortex, and occipital lobe.

13 m away from the most posterior aspect of the occipital lobe.

14 its for simple targets are restricted to the occipital lobe.

15 bserved in the parietal lobe followed by the occipital lobe.

16 of foveal and peripheral signals within the occipital lobe.

17 rsistent white matter changes in the parieto-occipital lobes.

18 w density in the white matter of the parieto-occipital lobes.

19 elieved to require integrity of the parietal-occipital lobes.

20 ith age but it decreased in the parietal and occipital lobes.

21 ory periphery in the parietal, temporal, and occipital lobes.

22 olandic operculum, and superior and inferior occipital lobes.

23 d right and left superior and right inferior occipital lobes.

24 d, localized to portions of the temporal and occipital lobes.

25 emotor regions, and also in the parietal and occipital lobes.

26 prefrontal, the right parietal and the left occipital lobes.

27 ntion would be disproportionately greater in occipital lobes.

28 nd parietal lobes but not in the frontal and occipital lobes.

29 ickening was demonstrated in the frontal and occipital lobes.

30 specially the frontal, lateral temporal, and occipital lobes.

31 us and regions of the temporal, parietal and occipital lobes.

32 across the subcortex, frontal, parietal and occipital lobes.

33 ical white matter of the frontal and parieto-occipital lobes.

34 urons were stained in frontal, parietal, and occipital lobes.

35 ost commonly affected region was the parieto-occipital lobes (100%), however, other atypical regions

36 nimals selectively activated the left medial occipital lobe--a region involved in the earliest stages

37 iated with reduced gray matter volume in the occipital lobe and left cerebellar lobule VIIb, which is

38 spheric communication between regions of the occipital lobe and of the superior parietal lobules.

39 pathways, including the lingual gyrus in the occipital lobe and the inferior parietal lobe, which had

40 ent spatial frequencies were mapped over the occipital lobe and then over the entire brain.

41 matter from frontal, parietal, temporal, and occipital lobes and cerebellum as well as basal ganglia.

42 in anterior temporal lobes, parietal lobes, occipital lobes and cerebellum.

43 nts showed activation in the fusiform gyrus, occipital lobe, and inferior frontal cortex relative to

44 nterior-posterior stretch in the frontal and occipital lobes, and superior-inferior stretch in right

45 dle frontal gyri bilaterally and in the left occipital lobe as a control region.

46 bplate, and intermediate zones of the monkey occipital lobe as early as E70.

47 gyrus, lateral and medial temporal lobe, and occipital lobe as well as caudate and putamen nuclei, af

48 adoxical ability of some human subjects with occipital lobe brain damage to discriminate unseen stimu

49 ller gray matter volumes in the temporal and occipital lobes, but no difference between groups was fo

50 g light results in similar activation in the occipital lobes by fMRI.

51 bolic reductions were most pronounced in the occipital lobe, caudate nucleus, and temporal lobe.

52 least two characteristics localizing to the occipital lobe (clinical symptoms, interictal focus, ict

53 reased in the precuneus and the parietal and occipital lobes compared with subjects with subjective c

54 The occipital lobe contains retinotopic representations of t

55 The lateral temporal, frontal, parietal, and occipital lobe cortices appeared normal for age 66 years

56 revealed that activity in large parts of the occipital lobe covaried with each.

57 disorder; and neuroimaging demonstration of occipital lobe damage.

58 gyri than sulci and in frontal compared with occipital lobes, decreasing consistently throughout the

59 seizure control, outcome after resection of occipital lobe developmental abnormalities is less unifo

60 anscranial magnetic stimulation of the human occipital lobe disrupts the normal perception of objects

61 od flow increases were observed in the right occipital lobe during the memory task relative to the co

62 Medically refractory occipital lobe epilepsies are increasingly treated with

63 s had unilateral frontal lobe epilepsy, five occipital lobe epilepsy (OLE), six parietal lobe epileps

64 main pathological substrates of uncontrolled occipital lobe epilepsy are gliomas and developmental ab

65 n was present, intact cortex in the lesioned occipital lobe exhibited conventional retinotopic organi

66 fiber tracking (FT) were used to measure the occipital lobe fiber tracts connecting the two hemispher

67 ammonis region 1, but not in the thalamus or occipital lobes from the same brains.

68 lear magnetic resonance spectroscopy, serial occipital lobe GABA and homocarnosine concentrations wer

69 icipants with WS showed reduced thalamic and occipital lobe gray matter volumes and reduced gray matt

70 bodies (DLB) have both been associated with occipital lobe hypometabolism on (18)F-FDG PET, whereas

71 calized to the temporal lobe in 9 and to the occipital lobe in 1; seizure onset was poorly localized.

72 phere could be traced posteriorly around the occipital lobe in 45 (90%).

73 eactivity was significantly decreased in the occipital lobe in both presymptomatic (mean BOLD change

74 shown in the cortex of the left parietal and occipital lobe in the first patient and symmetrically in

75 Demyelination involved parietal-occipital lobes in 90%, leading to visual and auditory p

76 ost prominent in the frontal, cingulate, and occipital lobes in all high-risk individuals compared wi

77 l lobes had a lower uptake than parietal and occipital lobes in general.

78 P levels were found in frontal, parietal and occipital lobes in MCI and late AD compared to controls

79 ease in F(4)-NPs was present in parietal and occipital lobes in MCI compared to controls and a signif

80 ional thinning of the parietal, temporal and occipital lobes in the VLBW group, whereas regional thic

81 he cuneus, lingual and fusiform gyri, middle occipital lobe, inferior parietal lobule, and also cingu

82 timulation is performed there, activation of occipital lobes is seen by fMRI.

83 (total and frontal, parietal, temporal, and occipital lobes), lateral ventricular cerebrospinal flui

84 ippocampus and right middle temporal gyrus), occipital lobe (left lingual gyrus), anterior cingulate,

85 st RT group than in the slow RT group in the occipital lobes, left sensorimotor cortices, and supplem

86 ), right posterior cingulate gyrus, and left occipital lobe (lingual gyrus).

87 erns of hypometabolism involving the lateral occipital lobe, lingual gyrus, cuneus, precuneus, poster

88 erns of hypometabolism involving the lateral occipital lobe, lingual gyrus, cuneus, precuneus, poster

89 linical variables were also unrelated to the occipital lobe location of abnormality.

90 Outcome and occipital lobe location were analyzed with respect to su

91 ionship to etiology, pathological substrate, occipital lobe location, surgical approach, and electroc

92 from those where the line sloped upwards in occipital lobe (lvPPA-high), representing unusually high

93 bjects in which the line sloped downwards in occipital lobe (lvPPA-low), representing low uptake, wer

94 y decreased cortical grey matter rCBF in the occipital lobe (mean difference -11.1 mL/100 g per min,

95 lective involvement of parietal, frontal and occipital lobes might explain some of the clinical and n

96 In the occipital lobe, NAA and ADP are 11.9 +/- 1.9mM and 32 +/

97 hates, and ADP levels in the hippocampus and occipital lobe of 15 healthy volunteers.

98 icant grey matter atrophy bilaterally in the occipital lobe of PDD patients compared with Parkinson's

99 ective areas in the fusiform gyrus (FFA) and occipital lobe (OFA).

100 g of the retinal nerve fibre layer following occipital lobe/optic radiation damage due to stroke.

101 showed greater hypometabolism in the medial occipital lobe, orbitofrontal cortex, anterior temporal

102 y differ with respect to location within the occipital lobe (overall medial [50%] or lateral [38%]);

103 nt activation decreases in the left anterior occipital lobe (P=.016), right posterior temporal lobe (

104 n bilateral, frontal, parietal, temporal and occipital lobes (P < 0.001), while FTD patients had a th

105 the posterior P2 component over the parieto-occipital lobe peaked significantly earlier for the uniq

106 Occipital lobe perfusion defects have been identified on

107 a R = +0.62, p < 3 x 10(-4), whereas, in the occipital lobe, R = +0.67, p < 5 x 10(-5).

108 f actual to expected hemorrhages = 1.37) and occipital lobes (ratio = 1.45, p < 0.0001).

109 the thalamus and decreased rCBF in the left occipital lobe, right cerebellum, and temporal cortex bi

110 ns of interest in anterior temporal lobe and occipital lobe sections corresponding closely to those u

111 Initial signs or symptoms suggested occipital lobe seizure onset in 13 of 16 patients.

112 tients who underwent surgery for intractable occipital lobe seizures were retrospectively evaluated.

113 sterior parietal cortex or the right ventral occipital lobe selectively induced a significant shift t

114 c variances in the central sulcus region and occipital lobe suggest potential difficulties in control

115 logy of the rolandic cortex) and the ICOE-G (occipital lobe symptomatology), while the autonomic clin

116 6.47) gyri, and one right ILF end point, the occipital lobe (T = 5.36).

117 The caudal occipital lobe, temporal pole, genu and prefrontal white

118 ose hypometabolism (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebell

119 ose hypometabolism (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebell

120 hat have shown that following lesions of the occipital lobe, the retinal nerve fibre layer thickness

121 campus and parahippocampal gyrus, and in the occipital lobe, the right frontal lobe and the left pari

122 The activation volumes in both occipital lobes, the left sensorimotor cortex, and the s

123 ately 8% in the frontal lobes and 12% in the occipital lobes-these regions also showed significant vo

124 dema of the white matter of the parietal and occipital lobes; this syndrome is potentially reversible

125 Whereas resection of occipital lobe tumors associated with chronic epilepsy p

126 aseline and during visual stimulation in the occipital lobe using (31)P magnetic resonance spectrosco

127 the foot as a control, and activation in the occipital lobes was not observed.

128 g transcranial magnetic stimulation over the occipital lobe, we showed a clear impairment in the accu

129 The occipital lobes were obtained after death from six adult

130 rences in the brains were small, even in the occipital lobe where only a small region of the primary

131 cts were most prominent for the parietal and occipital lobe, which showed divergent effects as early

132 rmal optic nerves and only subtle changes to occipital lobe white and gray matter.

133 us, right inferior temporal gyrus, and right occipital lobe white matter.