ライフサイエンスコーパス: occult

1 tients, referred for the evaluation of OGIB (occult, 25 patients [43%]; overt, 33 patients [57%]), un

2 >/= 0.2 logMAR in 4 (24 %) classic, 9 (47 %) occult, 6 (33 %) pigment epithelial detachment, 6 (55 %)

3 aluation of nonaccidental trauma to identify occult abdominal organ injuries.

4 right ventricle (RV) was utilized to detect occult abnormalities in regional and global contractilit

5 There were no occult adrenocortical carcinomas.

6 associated with clinically detected ovarian, occult, and peritoneal cancers diagnosed in the cohort.

7 dministered to these 60 patients to identify occult antigen exposure known to cause hypersensitivity

8 ional cancer detected at MR imaging that was occult at mammography.

9 diagnoses were screen-detected and six were occult at RRSO).

10 If these events are due to occult atheroma, the risk-factor profile and coronary pr

11 f these cases were attributed to exposure of occult avian antigens from commonly used feather bedding

12 , sometimes associated with radiographically occult avulsion fractures.

13 Between 2001 and 2010, 759 true occult bacteremia cases were identified, including 65 pa

14 ents were selected from 694 ED patients with occult bacteremia other than S. aureus.

15 ependent predictors of mortality among adult occult bacteremia patients.

16 the incidence of bacterial meningitis and of occult bacteremia since the advent of Haemophilus influe

17 Among patients with occult bacteremia, S. aureus infections had significantl

18 Occult bacterial infections represent a worldwide health

19 the clinical setting to identify previously occult biomarkers of drug sensitivity that can aid in th

20 nting with overt bleeding than in those with occult bleeding (21/54 [39%] vs 16/82 [20%]; difference,

21 size of 136 patients (54 overt bleeding, 82 occult bleeding) was enrolled.

22 stinal end point was a composite of overt or occult bleeding, symptomatic gastroduodenal ulcers or er

23 e collected and assessed for consistency and occult bleeding.

24 pg/ml (odds ratio [OR]: 7.3), positive fecal occult blood (OR: 13.2), hemoglobin < or =90 g/l (OR: 6.

25 tric ulcers, and a higher incidence of fecal occult blood loss.

26 e = 3,520; IRR = 0.87 (0.80-0.96) and Faecal Occult Blood Number eligible = 6,566; 0.86 (0.78-0.94).

27 etter screening compliance compared to fecal occult blood or endoscopic screening.

28 ing test for colorectal neoplasia; the fecal occult blood test (FOBT) detects neoplasias with low lev

29 The use of the fecal occult blood test (FOBT) for colorectal cancer (CRC) scr

30 1.38; 95% CI: 1.31, 1.45) but not with fecal occult blood test (HR, 1.00; 95% CI: 0.91, 1.10) than th

31 165.19), or having undergone a recent fecal occult blood test (OR, 13.69; 95% CI: 3.66, 51.29).

32 ical smear test, 2) a mammogram, 3) a faecal occult blood test and 4) a prostate specific antigen tes

33 rs (for example, simplifying access to fecal occult blood test cards), or made system-level changes (

34 anol, RNAlater Stabilization Solution, fecal occult blood test cards, and fecal immunochemical test t

35 agnostic indications, such as positive fecal occult blood test result (OR, 0.33; 95% CI, 0.19-0.57),

36 assing the first screening round of a faecal occult blood test screening programme in a single geogra

37 lonoscopy, flexible sigmoidoscopy, and fecal occult blood test were 27.9, 0.6, and 29.5 per 1000 pers

38 colorectal cancer (CRC) by the guaiac fecal occult blood test, interval cancers develop in 48% to 55

39 ography colonography, the guaiac-based fecal occult blood test, the fecal immunochemical test, the mu

40 lonoscopy or sigmoidoscopy (year 1) or fecal occult blood testing (FOBT) in year 1 and FOBT, colonosc

41 opy plus either sensitive unrehydrated fecal occult blood testing (FOBT) or fecal immunochemical test

42 f once-only flexible sigmoidoscopy and fecal occult blood testing (FOBT).

43 years, annual highly sensitive guaiac fecal occult blood testing (HSFOBT), annual fecal immunochemic

44 f biennial screening with guaiac-based fecal occult blood testing (n = 419,966) showed reduced CRC-sp

45 Screening Programme (asymptomatic but faecal occult blood testing [FOBt] positive).

46 d clinical trials to reduce mortality: fecal occult blood testing and flexible sigmoidoscopy.

47 Screening with sensitive guaiac-based fecal occult blood testing, fecal immunochemical testing (FIT)

48 ears to be lower than that with guaiac fecal occult blood testing.

49 Rates of patient completion of fecal occult blood tests (FOBTs) are often low.

50 Consecutive rounds of fecal occult blood tests (FOBTs) are used to screen for colore

51 , with less invasive tests (sigmoidoscopy or occult blood tests) for lower-risk persons and colonosco

52 Fecal tests (occult blood, methylation) engender excellent patient co

53 The effect of screening with fecal occult-blood testing on colorectal-cancer mortality pers

54 In randomized trials, fecal occult-blood testing reduces mortality from colorectal c

55 r to annual or biennial screening with fecal occult-blood testing.

56 survival over a median of 6.3 years, whereas occult bone marrow metastasis, although rare, was associ

57 One patient with occult brain metastasis had grade 4 intracranial hemorrh

58 an detect subcentimeter and mammographically occult breast cancer, with a sensitivity and specificity

59 BSGI examinations detected mammographically occult breast cancer.

60 can aid detection of small mammographically occult breast cancers (cancer detection rate, 0.8-10 can

61 st cancer and the number of mammographically occult breast cancers detected per 1,000 women screened.

62 ulated as the percentage of mammographically occult BSGI-detected breast cancer and the number of mam

63 chnic venous thrombosis (SVT) is a marker of occult cancer and a prognostic factor for cancer surviva

64 Its significance as a first symptom of occult cancer and as a prognostic factor for cancer surv

65 Pericarditis may be a marker of occult cancer and augurs increased mortality after a can

66 domization, 33 (3.9%) had a new diagnosis of occult cancer between randomization and the 1-year follo

67 Risk factors predictive of occult cancer detection in patients with a first unprovo

68 moking status may be important predictors of occult cancer detection in patients with first unprovoke

69 ssess the effect of specific risk factors on occult cancer detection within 1 year of a diagnosis of

70 CI, 1.24-6.33; P= .014) were associated with occult cancer detection.

71 versity in practices regarding screening for occult cancer in a person who has an unprovoked venous t

72 To estimate the prevalence of occult cancer in patients with unprovoked VTE, including

73 Occult cancer is detected in 1 in 20 patients within a y

74 ess the efficacy of a screening strategy for occult cancer that included comprehensive computed tomog

75 Clinically occult cancer was detected among 2.6% of high-risk women

76 The prevalence of occult cancer was low among patients with a first unprov

77 organ unnoticed by the host, referred to as 'occult cancer'.

78 t because it may lead to the diagnosis of an occult cancer, and a substantial number of patients, mai

79 .6 years, and found that SVT was a marker of occult cancer.

80 creatic ductal adenocarcinoma, a notoriously occult cancer.

81 ical practice and can sometimes be a sign of occult cancer.

82 ts were randomly assigned to undergo limited occult-cancer screening (basic blood testing, chest radi

83 t, cervical, and prostate cancer) or limited occult-cancer screening in combination with CT.

84 In the primary outcome analysis, 4 occult cancers (29%) were missed by the limited screenin

85 years between initial treatment and relapse, occult cancers are maintained in an apparent state of do

86 65 days after prior screen, with modeling of occult cancers detected at RRSO.

87 gical age, and anatomic origin of clinically occult cancers detected at surgery.

88 screen was 81.3% (95% CI, 54.3% to 96.0%) if occult cancers were classified as false negatives and 87

89 Of the six occult cancers, five (83.3%) were stage I to II (CI, 35.

90 luence the progression of early subclinical (occult) cancers.

91 dy sought to evaluate the natural history of occult cardiac dysfunction in Duchenne muscular dystroph

92 he consequence (rather than the cause) of an occult cardiomyopathy, which persists despite a signific

93 We hypothesized that CMR would identify occult cardiotoxicity characterized by structural and fu

94 In an asymptomatic population, there is occult cardiovascular disease which can be detected by c

95 ult disease are identified, the low rates of occult CBC do not support the use of CPM in average-risk

96 Occult CBC was identified in 24 (6%) patients, and 114 (

97 is safe and efficacious for the diagnosis of occult CBD stones in patients with intermediate risk for

98 estimation of tumor thickness and predicting occult cervical nodal metastasis.

99 tween campus facilities, and highlights that occult changes in microbiota should be considered when i

100 Stage 3 resembles occult choroidal neovascularization, occurs primarily in

101 66), 28.6% (76/266), and 9.8% (26/266) among occult, classic, retinal angiomatous proliferation, and

102 e the ribonuclease H (RH) domain contains an occult cleavage site located near its center, cleavage m

103 se simultaneous Apc loss frequently leads to occult clonal expansion without morphological transforma

104 dal neovascularization (CNV) than those with occult CNV (P < 0.001).

105 n of PCV and differentiation between PCV and occult CNV in this selected clinic population.

106 chments (PEDs) attributable to either PCV or occult CNV were retrospectively reviewed by a grader mas

107 rization (CNV) leakage area, smaller area of occult CNV, and presence of subretinal fluid (SRF).

108 ization (CNV) (aHR, 3.1; CI, 2.4-3.9) versus occult CNV, blocked fluorescence (aHR, 1.4; CI, 1.1-1.8)

109 thalmologists to distinguish between PCV and occult CNV, decreasing the need for ICGA and the risks r

110 o 10.90) compared with minimally classic and occult CNV, whereas the hazard ratio of fibrosis develop

111 Senescent HTRA1 mice developed occult CNV, which likely resulted from the degradation o

112 n ICGA-confirmed diagnosis of PCV and 14 had occult CNV.

113 as noted when classic CNVM was compared with occult CNVM (chi(2) = 10.4, df = 2, P < 0.005).

114 prokaryotic organism that is a frequent and occult contaminant of cell cultures.

115 repurification, indicating interference from occult contaminants.

116 to identify factors predictive of HRL and/or occult contralateral breast cancer (CBC).

117 ive magnetic resonance imaging (MRI) detects occult contralateral breast cancers (CBCs) in women with

118 Nevertheless, an occult contribution of PV-expressing interneurons to the

119 nondiabetic renal transplant recipients for occult defects in insulin sensitivity or secretion.

120 markers in predicting the presence of early occult disease and/or the screening and monitoring of in

121 Until reliable predictors for occult disease are identified, the low rates of occult C

122 tic modality enabling us to correctly detect occult disease in 74% of patients and to differentiate l

123 Molecular detection of occult disease in regional lymph nodes is associated wit

124 llege of Surgeons Oncology Group to evaluate occult disease in SLNs and bone marrow of early-stage br

125 ollected 2 to 5 years before diagnosis, when occult disease is probably present.

126 chool children, to identify risk factors for occult disease, and to assess the value of laboratory te

127 y recurrent micrometastases and for treating occult disease.

128 identify the individuals most likely to have occult disease.

129 ught to be at risk for potentially harboring occult disease.

130 e, however, may identify patients with early occult disseminated disease, who are at risk for relapse

131 The presence of diabetes mellitus (DM) or occult DM within the cohort confounded previous studies.

132 be indistinguishable; the need to eliminate occult environmental factors known to cause pulmonary fi

133 elanoma on the head and neck, where clinical occult extension is common, were studied at an academic

134 y defined clinical margins and unpredictable occult extension.

135 esents a genome-wide copy number analysis of occult fallopian tube carcinomas identified through risk

136 which were associated with radiographically occult fibular avulsion fractures.

137 antly associated with whiplash injuries were occult fracture (P<.01), bone marrow contusion of the ve

138 ligament and disk injuries and contusion or occult fracture) for traumatic injuries.

139 d 25.0% for patients with overt GI bleeding, occult GI bleeding, abdominal pain, chronic diarrhea, an

140 estinal (GI) bleeding, chronic diarrhea, and occult GI bleeding, accounting for 57.9%, 12.4%, and 9.7

141 trasonography and CT cannot reliably exclude occult groin abnormalities.

142 Occult HBV characterization clarifies new facets of HBV

143 Occult HBV infection (OBI) is defined by the persistence

144 ed pregnant women with isolated anti-HBc and occult HBV infection have very low HBV DNA levels and ar

145 was to compare the prevalence of previous or occult HBV infection in a cohort of hepatitis B surface

146 In the United States, neither previous nor occult HBV infection is an important factor in HCC devel

147 None of the women with isolated anti-HBc and occult HBV infection transmitted HBV to their infants.

148 Occult HBV infection was identified in 24% (95% CI, 18%-

149 HBV DNA (as a marker of occult HBV infection) was detected in the livers of 10.7

150 men with isolated anti-HBc were assessed for occult HBV infection, defined as HBV DNA levels >15 IU/m

151 virus (HBV)infection but might also signify occult HBV infection.

152 actors associated with isolated anti-HBc and occult HBV infection.

153 In order to determine the prevalence of occult HBV reactivation in a large cohort of patients du

154 suppressive therapies, in order to detect an occult HBV reactivation.

155 g by detecting window period infections and 'occult' HBV infections (OBIs), characterized by undetect

156 Of these 14 patients, 9 were assessed for occult HCV infection by reverse transcription quantitati

157 These findings indicate the presence of occult HCV infection in some patients with abnormal leve

158 CV infection after liver transplantation had occult HCV infections.

159 ly transmissible, and may be associated with occult health-threatening conditions.

160 Occult hepatitis B virus (HBV) infection (OBI) is define

161 hether universal infant immunization affects occult hepatitis B virus (HBV) infection (OBI), serum sa

162 y to hepatitis B core antigen (anti-HBc) and occult hepatitis B virus (HBV) infection are not well kn

163 Genotype D occult hepatitis B virus (HBV) infections (OBIs) have a

164 agnosis of hernia, MRI correctly detected an occult hernia in 10 of 11 cases (91%).

165 Early diagnosis and treatment of occult hernias are essential in relieving symptoms and i

166 Occult hernias are symptomatic but not palpable on physi

167 upport a model in which early development of occult hippocampal hyperexcitability may contribute to t

168 nt study, we hypothesized that HIV-1-induced occult HIV-associated nephropathy (HIVAN) would become a

169 giotensin system, promote the progression of occult HIVAN to apparent HIVAN.

170 xycycline for 3 weeks (to develop clinically occult HIVAN) followed by doxycycline-free water during

171 s from an endemic area with either active or occult infection can also transmit coccidioidomycosis.

172 ET/CT may be clinically useful for detecting occult infection foci in end-stage renal disease patient

173 Occult infection with hepatitis C virus (HCV) is defined

174 lope gene are associated with immune escape, occult infection, and resistance to therapy.

175 ed that 55% of these patients (n = 5) had an occult infection, with the detection of negative strand

176 It is unclear if women with occult infections are at risk of transmitting HBV to the

177 Such occult infections are common during pregnancy but their

178 new, emerging roles in detecting clinically occult inflammation (in asymptomatic patients) and infla

179 east in part be explained by the presence of occult inflammatory stimuli due to the absence of T cell

180 e values of US, CT, and MRI for detection of occult inguinal hernia.

181 There were no serious occult injuries that required immediate therapy.

182 A new regimen for screening of occult injuries to make allowance for this is proposed.

183 serous carcinoma appears to develop from an occult intraepithelial carcinoma in the fimbria of the f

184 The study evaluated the possibilities of occult invasion detected by immunohistochemistry, sectio

185 Few reports demonstrate occult invasion with immunohistochemistry staining, whic

186 nd Measures: Assessment of the occurrence of occult invasion, diagnosis of invasion by immunohistoche

187 Objective: To investigate the occurrence of occult invasive disease within in situ melanoma by using

188 intraepithelial neoplasia after exclusion of occult invasive disease.

189 Results: Occult invasive melanoma was detected in 11 of 33 consec

190 ned on 8 March 2015 ut as the limb of Europa occulted Io.

191 n psoriatic arthritis or unmasked previously occult joint disease.

192 than conventional cytology for detection of occult leptomeningeal lymphoma; however, some FCM-negati

193 surrounding retina but failed to identify 1 occult lesion that was detected with infrared imaging an

194 c lesions were associated with worse VA than occult lesions (66 vs. 69 letters; P=0.0003).

195 Occult lesions became inactive more slowly than classic

196 had >90% subretinal growth pattern, whereas occult lesions had a more variable growth pattern.

197 cipants with classic, minimally classic, and occult lesions were randomized in a 2:1 ratio to EMBT or

198 cipants with classic, minimally classic, and occult lesions were randomized to receive (a) EMBT and 2

199 classic lesions may be more responsive than occult lesions, although generally both subgroups are in

200 stically significant (favoring controls) for occult lesions, but not for predominantly classic and mi

201 c and minimally classic lesions, but not for occult lesions.

202 s, optimize the day of surgery, and identify occult lesions.

203 e visual acuity, most probably in cases with occult lesions.

204 sed assays in the diagnosis of cytologically occult lung neoplasms.

205 tive and observational analyses suggest that occult lymph-node metastases are an important prognostic

206 Occult macular dystrophy was diagnosed based on genetic

207 extramacular commotio retinae may represent occult macular injury or previously undiagnosed visual i

208 rence tomography are invaluable in revealing occult macular pathology that may not be apparent to cli

209 aware that they can be the first sign of an occult malignancy and that early recognition is vital fo

210 in patients randomized in the Screening for Occult Malignancy in Patients with Idiopathic Venous Thr

211 x cancer was the only factor associated with occult malignancy in the CPM (OR 2.88, P = 0.04).

212 ty invasive index cancer was associated with occult malignancy in the CPM; however, lack of standardi

213 The identification of reliable predictors of occult malignancy or HRL in the contralateral breast may

214 reliminary study, a small number of cases of occult malignancy were subsequently diagnosed among preg

215 fection is a strong indicator for underlying occult malignancy.

216 anchnic vein thrombosis (SVT) as a marker of occult malignant disease.

217 use multiparameter flow cytometry to detect occult marrow disease (OMD) in patients with solitary pl

218 laboratory features suggest the presence of 'occult' mastocytosis or another haematologic neoplasm, a

219 n on noninvasive prenatal testing (NIPT) and occult maternal malignancies may explain results that ar

220 e patient underwent surgical excision of the occult melanoma without evidence of other sites of metas

221 eg, lack of residual membrane or presence of occult membrane), thus affecting additional surgical man

222 rders, such as Alzheimer's disease, in which occult mesial temporal lobe seizures are suspected to pl

223 Several small studies suggest that occult metastases (OMs) in pleura, bone marrow (BM), or

224 nel lymph nodes were centrally evaluated for occult metastases deeper in the blocks.

225 essed by colorectal tumors that could reveal occult metastases in lymph nodes and better estimate rec

226 Occult metastases were an independent prognostic variabl

227 nificant difference between patients in whom occult metastases were detected and those in whom no occ

228 s of overall survival among patients in whom occult metastases were detected and those without detect

229 Occult metastases were detected in 15.9% (95% confidence

230 etastases were detected and those in whom no occult metastases were detected with respect to overall

231 urative surgery by identifying patients with occult metastases.

232 In this respect, early detection of occult metastasis invisible to current imaging methods w

233 fication of oral cancer according to risk of occult metastasis, guiding treatment strategies.

234 his finding resulted in identification of an occult metastatic melanoma involving the axillary lymph

235 Occult microinvasion was demonstrated in up to one-third

236 are variable, potentially due to undetected occult micrometastases (OM).

237 tal question arises as to whether clinically occult micrometastases survive in a state of balanced pr

238 arin-resistant microthrombi in patients with occult, mucinous adenocarcinomas.

239 CT images reveal otherwise occult muscle depletion.

240 y (n=27), myocarditis or sarcoidosis (n=22), occult myocardial infarction (n=13), and hypertrophic ca

241 ic lung resection performed in patients with occult N2 disease was 10.8% (18 of 166) (8.1% in the EBU

242 he rate of nontherapeutic lung resection for occult N2 disease, with comparison between the EBUS grou

243 rate of nontherapeutic operations because of occult N2 nodal disease.

244 an reported in the literature because of its occult nature.

245 determining the outgrowth versus dormancy of occult neoplasia and suggest a potential long-term dange

246 were associated with detection of clinically occult neoplasms at RRSO.

247 The primary occult NET was localized by (18)F-FDOPA PET/CT in 12 pat

248 ata raise the possibility that persistent or occult neurologic and lymphoid disease may occur followi

249 t consistent on a cytomorphologic basis with occult nevi.

250 is study evaluates frequency and patterns of occult nipple involvement in a large contemporary cohort

251 ificantly aids in the detection of otherwise occult nodal and bone metastases.

252 tion was used to estimate the probability of occult nodal disease as a function of total number of LN

253 des the first empirically based estimates of occult nodal disease risk in patients after surgery for

254 To rule out occult nodal disease with 90% confidence, six, nine, and

255 dal metastasis and tumors more than 1 cm had occult nodal metastasis.

256 Detection of OC and EC and even clinically occult OC was achieved, making it a potential tool of si

257 This also included one patient with occult OC.

258 Thirty-eight tumors were occult on mammograms.

259 tecting primary neuroendocrine tumors (NETs) occult on morphologic and functional imaging, in relatio

260 aging tool for the detection of primary NETs occult on SRS, especially tumors with a well-differentia

261 ssic lesions, and 0.30 mm/year for eyes with occult only lesions (P < 0.01).

262 nflammation could represent individuals with occult opportunistic infections in need of additional sc

263 ng DNA methylation profiles to determine the occult original cancer in cases of cancer of unknown pri

264 immune antibodies in 25 cases of acute zonal occult outer retinopathy (AZOOR) identified using the cl

265 Acute zonal occult outer retinopathy (AZOOR) remains a challenging d

266 Therefore, acute zonal occult outer retinopathy (AZOOR) was diagnosed.

267 ical entity that best represents acute zonal occult outer retinopathy (AZOOR).

268 or a distinct entity within the acute zonal occult outer retinopathy complex.

269 Acute zonal occult outer retinopathy should be considered in patient

270 classic (p = 0.105), 0.000 (-1.15, 0.20) in occult (p = 0.005), -0.200 (-1.20, 0.60) in cases with s

271 implicated in cryptogenic stroke, including occult paroxysmal atrial fibrillation, patent foramen ov

272 progress, or regress, especially during the occult phase.

273 ciations found in a patient with acute zonal occult photoreceptor loss.

274 bitor-induced activation of wild-type Raf in occult precursor skin lesions.

275 PET/CT imaging uncovered previously occult primary nonocular cancers (11/18, 61%), revealed

276 , although generally rare, may also indicate occult prostate cancer that may need to be further scrut

277 g right heart catheterization would identify occult pulmonary venous hypertension (OPVH).

278 We aimed to investigate occult, putative causes in the environments of patients

279 To minimize bias by occult recurrence, we excluded patients who recurred or

280 tients, infrared imaging and SD-OCT detected occult retinal astrocytic hamartomas that were not obser

281 or who have complex cytogenetics should have occult RT excluded before initiating venetoclax therapy.

282 entional measures of function, suggestive of occult RV myocardial disease.

283 ere used to assess the independent effect of occult S. aureus bacteremia on patient mortality.

284 Evaluation revealed an occult small-cell lung cancer.

285 t case is an infiltrative breast cancer with occult sonography findings in a patient with a history o

286 emiology and outcomes of adult patients with occult Staphylococcus aureus bacteremia who were inadver

287 igment epithelial detachment associated with occult subfoveal choroidal neovascularization with intra

288 or lymphocytes is associated with clinically occult transition to donor-derived immunity.

289 Ocular Neovascularization; Minimally Classic/Occult Trial of the Anti-VEGF Antibody Ranibizumab in th

290 roenvironment (niche) may affect the fate of occult tumor cells, including their biological and genet

291 of several techniques designed to locate an occult tumor, including cross-sectional anatomic imaging

292 use paraneoplastic disorders often herald an occult tumor.

293 y eradicating tumors or policing a state of (occult) tumor dormancy.

294 With the high potential for occult tumors in common conditions such as fibroids, val

295 ult in side effects, including the growth of occult tumors.

296 latives older than age 50 were found to have occult tumors; the tumors were cleared surgically from 8

297 nique opportunity to study the morphology of occult type 1 neovascular membranes in AMD and allows pr

298 ntaining the TL PICC was performed to detect occult venous thrombosis.

299 independent readers assessed the presence of occult vertebral body and facet fractures, vertebral bod

300 Occult wild-type transthyretin cardiac amyloid had a pre