ライフサイエンスコーパス: ocular infection

1 ence in rabbits (based on survival following ocular infection).

2 of infected mice between days 1 and 5 after ocular infection.

3 fic effector T (T(eff)) cells induced during ocular infection.

4 oss lymphoid and extralymphoid tissues after ocular infection.

5 mice than in BALB/c and CBA/J mice following ocular infection.

6 le of CXCL10 during the acute phase of HSV-1 ocular infection.

7 d disease occurs in humans following primary ocular infection.

8 imulation in susceptibility to P. aeruginosa ocular infection.

9 nd enteric washes and were protected against ocular infection.

10 ymphotropic dissemination of HSV-1 following ocular infection.

11 for the treatment of symptomatic adenoviral ocular infection.

12 ndicating the critical role of HVEM in HSV-1 ocular infection.

13 ding virulence and immune responses to viral ocular infection.

14 n models: mouse footpad infection and rabbit ocular infection.

15 s into immunopathological responses to viral ocular infection.

16 tics in order to gain effective control over ocular infection.

17 SE by acyclovir treatment provided 4 d after ocular infection.

18 of either receptor attenuates disease after ocular infection.

19 against herpes simplex virus type 1 (HSV-1) ocular infection.

20 ls of testican-1 after P. aeruginosa-induced ocular infection.

21 ratitis (AK) is a rare but sight-threatening ocular infection.

22 ne model of naturally-acquired P. aeruginosa ocular infection.

23 monas aeruginosa are the leading isolates in ocular infections.

24 therapy for the treatment of adenoviral (Ad) ocular infections.

25 teria are the major contributor of bacterial ocular infections.

26 ence showing excellent potency in the war on ocular infections.

27 was aimed to review the bacterial profile of ocular infections.

28 f this antiviral drug in treating adenoviral ocular infections.

29 ibility pattern among patients with external ocular infections.

30 s hospital infections and community-acquired ocular infections.

31 ibacterials used for empirical management of ocular infections.

32 marily on innate immunity to protect against ocular infections.

33 n be a vector for transmission of adenovirus ocular infections.

34 live attenuated vaccines against genital and ocular infections.

35 is known about the role of magA in secondary ocular infections.

36 lens could be used as a treatment for fungal ocular infections.

37 differentiate Fusarium spp. responsible for ocular infections.

38 al wild-type adenoviral serotypes that cause ocular infections.

39 unoglobulin for treatment and prophylaxis of ocular infections.

40 he role of NK cells in the modulation of CMV ocular infection, 9.0 x 10(2) plaque-forming units of th

41 Following ocular infection, all three viruses produced similar acu

42 simplex virus type 1 (HSV-1) during primary ocular infection and after reactivation of latent infect

43 of CD4 knockout (KO) mice after both primary ocular infection and challenge with RH tachyzoites.

44 ns that cause sexually transmitted diseases, ocular infections and atypical pneumonias.

45 Pneumococcus is also a major cause of human ocular infections and is commonly isolated in cases of b

46 Bacteria are major cause of ocular infections and possible loss of vision.

47 ed in the eyes of control mice after primary ocular infection, and near-normal histology with few tac

48 s conducted among 160 patients with external ocular infections at Borumeda hospital, Northeast Ethiop

49 Most of the isolates of Fusarium ocular infections belong to the F. solani or F. oxysporu

50 s latent HSV infection in the mouse model of ocular infection but has no impact on the maintenance of

51 of explant reactivation at the lower dose of ocular infection but not at the higher dose.

52 ratory tract suspected to be responsible for ocular infections but no well-described case of D. pigru

53 Ocular infection by Chlamydia trachomatis is the leading

54 are highly antibiotic resistant, and primary ocular infection by ESBL E coli has rarely been reported

55 ns of DCs to the protection afforded against ocular infection by immunization against HSV-1 and their

56 PPK1 is essential for a successful ocular infection by P. aeruginosa.

57 Ocular infection by this organism is the leading cause o

58 ion of neutrophils during primary chlamydial ocular infection by using the guinea pig model of Chlamy

59 -associated MRSA is an important pathogen of ocular infections; CA-MRSA and HA-MRSA ocular infections

60 arises during Pseudomonas aeruginosa-induced ocular infection can trigger tissue damage resulting in

61 Fungal ocular infections cause significant ocular morbidity, pa

62 Trachoma, an ocular infection caused by Chlamydia trachomatis, is the

63 y and duration of Chlamydia trachomatis (Ct) ocular infections decrease with age, suggesting developm

64 Successful outcome for patients with ocular infection depends on close collaboration between

65 en of ocular infections; CA-MRSA and HA-MRSA ocular infections differ demographically and clinically,

66 The rare descriptions, in the literature, of ocular infections due to Pasteurella multocida include:

67 ch has targeted immune mechanisms in primary ocular infections, events that could impact chronic resp

68 rachomatis is a leading cause of genital and ocular infections for which no vaccine exists.

69 Following primary ocular infection, herpes simplex virus type 1 (HSV-1) es

70 Following ocular infection, herpes simplex virus type 1 (HSV-1) es

71 After ocular infection, HSV-specific CD8+ T cells migrate to a

72 Ocular infections, if left untreated, can damage the str

73 with the peripheral nervous system following ocular infection in mice.

74 is a dearth of information on prevalence of ocular infection in our population.

75 nesis of herpes simplex virus type 1 (HSV-1) ocular infection in the mouse.

76 Following ocular infection in vivo, clusters formed exclusively in

77 tunistic pathogen Serratia marcescens causes ocular infections in healthy individuals.

78 4, has only been reported to cause wound and ocular infections in humans.

79 a topical antiviral treatment for adenoviral ocular infections in the target population.

80 he role of neutrophils in primary chlamydial ocular infection, indicates a previously unappreciated r

81 External ocular infection is a public health problem in Ethiopia.

82 that the anatomic host site in pneumococcal ocular infections is important.

83 l as well as molecular investigation on HAdV ocular infections is rather absent in Greece, which has

84 ), a subgroup D virus associated with severe ocular infections, is unable to use CAR efficiently to i

85 icroflora isolates, 0.80 (CI, 0.54-0.94) for ocular infection isolates, and 1.0 (CI, 0.45-1.0) for st

86 Among ocular infection isolates, invasive and cytotoxic strain

87 ied a potential target for modulation during ocular infection, macrophage migration inhibitory factor

88 In immunized mice during primary HSV-1 ocular infection, macrophages play an important role in

89 anti-HSV-1 mechanisms of murine IFN-beta in ocular infection, mice were transduced with an adenovira

90 hese phenotypes are also mirrored in a mouse ocular infection model.

91 st to provide evidence that in P. aeruginosa ocular infection, mouse strains favoring development of

92 In the 7-day period after ocular infection of BALB/c mice, the replication of HSV-

93 At 11 days postinfection, ocular infection of C57BL/6 mice with all of the strains

94 ollowing herpes simplex virus type 1 (HSV-1) ocular infection of C57BL/6 mice, activated CD8(+) T cel

95 We have reported previously that ocular infection of different strains of mice with recom

96 Following footpad or ocular infection of mice lacking type I IFN receptors, H

97 dministration begun at different times after ocular infection of mice with HSV could influence the se

98 n) failed to infect ganglionic neurons after ocular infection of mice.

99 cessfully infecting ganglionic neurons after ocular infection of mice.

100 lactic Delta41Delta29 vaccination on primary ocular infection of NIH inbred mice with HSV-1 McKrae, a

101 ut most importantly in virulence as shown by ocular infection of nonhuman primates.

102 Following ocular infection of rabbits, dLAT1.5 reactivated at a lo

103 an wild-type or marker-rescued viruses after ocular infection of rabbits.

104 l vaccine, gD2 alum, protected against acute ocular infection only.

105 cular necrosis in response to either primary ocular infection or challenge.

106 To mitigate the burden of bacterial ocular infections, physicians should regard on risk redu

107 However, its regulatory role in ocular infection remains unclear.

108 ing IL-18 to the cornea of mice before HSV-1 ocular infection resulted in reduced angiogenesis and di

109 IL-10(-/-) animals depleted of nTregs before ocular infection showed more severe SK lesions as compar

110 r System, Central Nervous System Infections, Ocular Infections, Soft Tissue Infections of the Head an

111 In Pseudomonas aeruginosa ocular infection, T-helper cell 1-responsive mouse strai

112 C. trachomatis infection causes trachoma, an ocular infection that leads to blindness, and sexually t

113 tible C57BL/6J (B6) mouse to resistant after ocular infection through modulation of the inflammatory

114 Sixty days after ocular infection, trigeminal ganglia (TG) were removed f

115 ated population prevalence of C. trachomatis ocular infection was approximately 17.5%.

116 When ocular infection was caused by an invasive strain in viv

117 moral immunity in herpes simplex virus (HSV) ocular infections was studied in immunoglobulin mu chain

118 potheses, guinea pigs with primary C. caviae ocular infections were depleted of neutrophils by using

119 All patients with suspected external ocular infections were examined under slit lamp microsco

120 Although isolates from ocular infections were found in all four groups, they ha

121 We identified 274 patients with MRSA ocular infections, which comprised 181 CA-MRSA and 93 HA

122 oxin (Ac-CT) or with saline, before or after ocular infection with A. castellanii.

123 implex virus type 1 (HSV-1) during the acute ocular infection with and without AED treatment focusing

124 scertain the disease pattern of trachoma and ocular infection with C trachomatis in a trachoma hypere

125 ll households were examined for trachoma and ocular infection with C. trachomatis at baseline, and 6

126 s where trachoma is mesoendemic suggest that ocular infection with Chlamydia trachomatis can be elimi

127 Ocular infection with Chlamydia trachomatis can lead to

128 It is caused by recurrent ocular infection with Chlamydia trachomatis in childhood

129 tibiotic treatment could reduce trachoma and ocular infection with Chlamydia trachomatis in hyperende

130 reventable blindness, is produced by chronic ocular infection with Chlamydia trachomatis, an obligate

131 Trachoma, caused by repeated ocular infection with Chlamydia trachomatis, is an impor

132 The host inflammatory response to ocular infection with herpes simplex virus (HSV) can be

133 erpetic stromal keratitis (HSK) that follows ocular infection with herpes simplex virus (HSV) is sugg

134 Ocular infection with herpes simplex virus (HSV) results

135 Ocular infection with herpes simplex virus (HSV) sets of

136 inding immunoinflammatory reaction caused by ocular infection with herpes simplex virus (HSV).

137 ammatory lesion stromal keratitis induced by ocular infection with herpes simplex virus (HSV).

138 tory stromal keratitis (SK) lesion caused by ocular infection with herpes simplex virus (HSV).

139 f immunization strategies to protect against ocular infection with herpes simplex virus 1 (HSV-1) mus

140 Ocular infection with herpes simplex virus 1 (HSV-1) res

141 Ocular infection with herpes simplex virus 1 (HSV-1) set

142 Ocular infection with herpes simplex virus 1 can result

143 Stromal keratitis resulting from ocular infection with herpes simplex virus is a common c

144 Ocular infection with herpes simplex virus leads to an i

145 plicated in the modulation of the outcome of ocular infection with herpes simplex virus type 1 (HSV-1

146 Treatment was begun at different times after ocular infection with HSV and the outcome was assessed c

147 Ocular infection with HSV causes a chronic T cell-mediat

148 Ocular infection with HSV causes corneal neovascularizat

149 Ocular infection with HSV may result in the blinding imm

150 Ocular infection with HSV results in a blinding immunoin

151 sion was up-regulated (10- to 20-fold) after ocular infection with HSV, an event that involved the pr

152 e between VEGF-A and sVR-1 that occurs after ocular infection with HSV, which causes prominent neovas

153 Mixed ocular infection with HSV-1 strains CJ394 and OD4 yield

154 octamer (8mer) as an eye drop 1 hour before ocular infection with HSV-1 was investigated.

155 ny-stimulating factor 1 (CSF-1) DNA prior to ocular infection with HSV-1.

156 5-80%) experiencing development of HSE after ocular infection with HSV-1.

157 e are highly susceptible to HSV-1 infection, ocular infection with HSV-IL-4 resulted in 100% survival

158 ical changes and their implication following ocular infection with HSV.

159 A in the cornea and draining lymph node upon ocular infection with HSV.

160 s potential importance, the role of IL-12 in ocular infection with P. aeruginosa remains unexplored a

161 EXPOSURE Ocular infection with recombinant adeno-associated viral

162 After ocular infection with the HSV-1 strain McKrae, virus rep

163 study was conducted to determine whether the ocular infection with this recombinant virus induces opt

164 prevalent among staphylococcal isolates from ocular infections, with many strains demonstrating multi

165 Bacteria are the major contributor of ocular infections worldwide.

166 This impedes therapies for ocular infections, wound healing, and dry-eye disease th