ライフサイエンスコーパス: odontogenic

1 Despite the high incidence of odontogenic abscesses in pet rabbits, published data on

2 1 and Tbx2 in turn antagonistically regulate odontogenic activity that leads to EK formation and to m

3 amelysin (MMP-20), kallikrein-4 (KLK-4), and odontogenic ameloblast-associated protein (ODAM) using q

4 d the ability to be differentiated to osteo-/odontogenic and adipogenic pathways, resembling mesenchy

5 of CD146+ pericytes are likely controlled by odontogenic and Bmp2-dependent VegfA production in odont

6 racts with Notch signaling and promotes both odontogenic and osteogenic differentiation of SCAPs.

7 ithelium are required for the development of odontogenic and skeletogenic mesenchyme cells.

8 Odontogenic-associated genes and proteins were detected

9 alence of, and sensitivity for, detection of odontogenic bacteremia.

10 n inducible in Aves and suggest that loss of odontogenic Bmp4 expression may be responsible for the e

11 tion of cranial neural crest is specified as odontogenic by Dlx-1 and Dlx-2 genes.

12 This case report describes an unusual odontogenic carcinoma, which was detected during routine

13 olecular evidence of cftr mRNA expression by odontogenic cells strongly suggest that CFTR plays an im

14 me cells of the mandibular arch give rise to odontogenic cells, while more caudal cells form the dist

15 APC staining was not seen in any other odontogenic cells.

16 with potent capacities to differentiate into odontogenic cells.

17 uctive mesenchyme is capable of inducing the odontogenic commitment of both dental and non-dental epi

18 eem to have evolved through the extension of odontogenic competence from the external dermis to inter

19 most cases CT study in patients suspected of odontogenic craniofacial infection revealed presence of

20 The odontogenic keratocyst is a common odontogenic cyst which accounts for 10% of all oral cyst

21 val cyst of the adult are both developmental odontogenic cysts located predominantly in the mandibula

22 ific and closely resemble other inflammatory odontogenic cysts.

23 It represents 3% to 12% of all odontogenic cysts.

24 hibition of Ca(2+) influx and suppression of odontogenic differentiation and mineralization as demons

25 ortantly, Phen-treated DPSCs showed enhanced odontogenic differentiation and mineralization in vivo.

26 evaluated the role of ORAI1 in mediating the odontogenic differentiation and mineralization of dental

27 howed that ORAI1 plays critical roles in the odontogenic differentiation and mineralization of DPSCs

28 The gene expression for odontogenic differentiation markers such as osteocalcin,

29 During the odontogenic differentiation of DPSCs, the expression of

30 t BMP signaling activity is required for the odontogenic differentiation of MSCs.

31 ciated with the odontogenic region, promotes odontogenic differentiation of MSCs.

32 red and evaluated the pro-osteogenic and pro-odontogenic effects of 4 small bioactive compounds- phen

33 It is a benign aggressive tumor of odontogenic epithelial origin with a high rate of recurr

34 ocally invasive benign neoplasm derived from odontogenic epithelium and presents with diverse phenoty

35 In contrast, high levels of Shh in odontogenic epithelium arrests tooth development at the

36 ental hard tissue interspersed in a field of odontogenic epithelium in a glandular configuration, con

37 including the skin, vagina, forestomach, and odontogenic epithelium.

38 al maturation of both colonic epithelium and odontogenic epithelium.

39 al cystic neoplasm thought to arise from the odontogenic epithelium.

40 factors that are expressed in early Astyanax odontogenic epithelium.

41 ed by reduced cell proliferation and loss of odontogenic fate marker Pitx2 expression in the dental e

42 o to investigate the mechanism that sustains odontogenic fate of dental mesenchyme during tooth devel

43 ium, with Wnt signaling also controlling the odontogenic fate.

44 Expansion of odontogenic field in Osr2-deficient mice required Msx1,

45 nstream of Pax9 and patterns the mesenchymal odontogenic field through protein-protein interactions w

46 to their molars because of expansion of the odontogenic field.

47 ivation of Osr2, a negative regulator of the odontogenic function of the Bmp4-Msx1 signaling pathway,

48 oral epithelial protein signal that directs odontogenic gene expression in mesenchyme cells of the d

49 ow-derived cells all responded by expressing odontogenic genes.

50 the genes envisaged as participating in an 'odontogenic homeobox gene code' by specifying molar deve

51 The molecular events of odontogenic induction are beginning to be elucidated, bu

52 lecule RhoA, and its overexpression prevents odontogenic induction.

53 THIS STUDY WAS TO ASSESS PATHWAYS, BY WHICH ODONTOGENIC INFECTIONS SPREAD INTO RESPECTIVE DEEP HEAD

54 es appear to be risk factors for pain due to odontogenic infections, comparatively little is known ab

55 teria for Gorlin syndrome, although none had odontogenic jaw keratocysts.

56 onal biopsy revealing contents typical of an odontogenic keratocyst (OKC).

57 d clinical knowledge base for the peripheral odontogenic keratocyst and assist clinicians in the iden

58 An odontogenic keratocyst can develop at virtually any site

59 The odontogenic keratocyst is a common odontogenic cyst whic

60 ribes the rare peripheral presentation of an odontogenic keratocyst localized to the maxillary anteri

61 ggressive clinical course for the peripheral odontogenic keratocyst.

62 This paper reviews odontogenic keratocysts and presents a case treated by c

63 Odontogenic keratocysts are believed to arise from remna

64 Odontogenic keratocysts are one component of the basal c

65 Odontogenic keratocysts in humans are aggressive, noninf

66 al cell nevus syndrome and all patients with odontogenic keratocysts should be evaluated for this syn

67 cell carcinomas, palmar and/or plantar pits, odontogenic keratocysts, skeletal and developmental anom

68 by multiple cutaneous basal cell carcinomas, odontogenic keratocysts, skeletal anomalies and facial d

69 ssues have been described to have osteogenic/odontogenic-like differentiation capacity, but little at

70 ol2a1, runx2a and chondromodulin, as well as odontogenic marker dlx2b.

71 s directly involved in the activation of the odontogenic mesenchyme has not been characterized.

72 of a lateral signaling center over competent odontogenic mesenchyme led to loss of teeth in avians wh

73 for activin betaA is expressed in the early odontogenic mesenchyme of all murine teeth but mutant mi

74 tooth formation in the presumptive oral and odontogenic mesenchyme of the maxillary and mandibular p

75 that antagonizes Msx1-mediated activation of odontogenic mesenchyme, was significantly upregulated in

76 , 37 years; age range, 13-55 years), who had odontogenic orbital cellulitis underwent clinical evalua

77 Infections of odontogenic origin are the most common cause of inflamma

78 t (GCA) is an uncommon developmental cyst of odontogenic origin most frequently seen near mandibular

79 ence of a subperiosteal abscess suggested an odontogenic origin of orbital infection.

80 The odontogenic origin of the orbital infection was not clin

81 ival cysts in the adult are unusual cysts of odontogenic origin with less than 100 reported, includin

82 periodontal cyst (LPC) is an unusual cyst of odontogenic origin, most frequently encountered in the m

83 They are uncommon cysts of odontogenic origin, noted primarily in the fifth and six

84 ) cervicofacial infection were of mandibular odontogenic origin.

85 hown to be bioactive, capable of stimulating odontogenic/osteogenic differentiation as observed by ge

86 hat the ephrinB1-EphB2 interaction regulates odontogenic/osteogenic differentiation from dental pulp

87 lications for the mechanisms contributing to odontogenic pain and management strategies.

88 view here the latest ideas on the control of odontogenic patterning and morphogenesis.

89 The proposal that odontogenic potential extends into the trunk neural cres

90 aeal ectoderm and endoderm demonstrated that odontogenic potential extends not only further caudally

91 portion of the intermediate region, but that odontogenic potential extends to its most caudal limit.

92 synergizes with Msx1 to activate mesenchymal odontogenic potential for tooth morphogenesis and sequen

93 are known to be essential for the switch in odontogenic potential from the epithelium to the mesench

94 f tooth development depends on activation of odontogenic potential in the mesenchyme by signals from

95 n tooth development depends on activation of odontogenic potential in the presumptive dental mesenchy

96 The odontogenic potential of 12 individual single-colony-der

97 in vitro culture system was used to test the odontogenic potential of more caudal regions of neural c

98 ated to cyclic RGD peptides can increase the odontogenic potential of these cells.

99 the maxillary molar epithelium has lost its odontogenic potential.

100 of the dental lamina that contains quiescent odontogenic progenitors that can be activated during phy

101 ator dental laminae, which contain quiescent odontogenic progenitors.

102 indicate that activation of the mesenchymal odontogenic program during early tooth development requi

103 n factor, Klf4, which is associated with the odontogenic region, promotes odontogenic differentiation

104 2, Barx1, and Pax9 defines a potential chick odontogenic region.

105 egrate Dlx1, Dlx2 and Fgf3 and Fgf8 into the odontogenic regulatory hierarchy along with Msx1, Msx2 a

106 e and embryonic oral epithelium stimulate an odontogenic response in the stem cells.

107 s were absent from the epithelium of the non-odontogenic second branchial arch.

108 hat Bmp4 is a key Msx1-dependent mesenchymal odontogenic signal for driving tooth morphogenesis throu

109 morphogenetic protein 4 (Bmp4), an essential odontogenic signal, in the developing tooth mesenchyme.

110 activin and Bmp4 act as crucial mesenchymal odontogenic signals during early tooth development.

111 ption factors are required for activation of odontogenic signals, including Bmp4 and Fgf3, in the ear

112 nt genes and molecular pathways using normal odontogenic tissue (human "dentome") for comparison.

113 compare it with gene expression of different odontogenic tissues ("dentome").

114 ow that this population alters its fate from odontogenic to become chondrogenic.

115 paction-induced changes in cell shape induce odontogenic transcription factors (Pax9, Msx1) and a che

116 e occurrence and treatment of an adenomatoid odontogenic tumor (AOT) presenting as a periodontal intr

117 nship between ameloblastoma and keratocystic odontogenic tumor (KCOT) by means of a genome-wide expre

118 Keratocystic Odontogenic Tumor (KCOT) is a locally aggressive develop

119 The keratocystic odontogenic tumor (KCOT) is a relatively rare, benign ne

120 The squamous odontogenic tumor (SOT) is a rare, benign, locally infil

121 A primary human keratocystic odontogenic tumor cell population, KCOT-1, has been esta

122 The mutant mice developed an odontogenic tumor of dental epithelium origin.

123 s of a young woman with a giant keratocystic odontogenic tumor of the mandible.

124 AOT is a rare odontogenic tumor usually associated with unerupted teet

125 f ameloblastomas, the most common epithelial odontogenic tumor, and in this tumor's likely precursor

126 uding the WNT pathway, might be the cause of odontogenic tumorigenesis and maintenance.

127 Adenomatoid odontogenic tumors (AOT) make up 3% of odontogenic tumor

128 Keratocystic odontogenic tumors (KCOT) may occur sporadically or asso

129 Keratocystic odontogenic tumors (KCOTs) of the jaw affect more than 6

130 structures similar to calcifying epithelial odontogenic tumors in humans.

131 r 80% of ameloblastomas, locally destructive odontogenic tumors of the jaw, by genomic analysis of ar

132 Odontogenic tumors originate from the remains of migrati

133 atoid odontogenic tumors (AOT) make up 3% of odontogenic tumors.