ライフサイエンスコーパス: oesophagus

1 and overtreatment in patients with Barrett's oesophagus.

2 stinal metaplasia and dysplasia in Barrett's oesophagus.

3 othelial growth factors (VEGFs) in Barrett's oesophagus.

4 ces and transports the particles towards the oesophagus.

5 of particle transport from the rakers to the oesophagus.

6 might prevent the precursor lesion Barrett's oesophagus.

7 ated and reliable in patients with Barrett's oesophagus.

8 a pectoris but generally originates from the oesophagus.

9 Acid was infused into the lower oesophagus.

10 eral cortical projections to the pharynx and oesophagus.

11 which receive vagal afferent input from the oesophagus.

12 afferent feedback from the face, pharynx and oesophagus.

13 that are specific to patients with Barrett's oesophagus.

14 n for multi-layered epithelium and Barrett's oesophagus.

15 tructural and functional regeneration of the oesophagus.

16 n whose expression is highly enriched in the oesophagus.

17 risk factor for the development of Barrett's oesophagus.

18 ndoscopic therapies for dysplastic Barrett's oesophagus.

19 ar smooth-muscle fibres within the abdominal oesophagus.

20 transport it back to the throat and into the oesophagus.

21 ed by approximately 70% compared with normal oesophagus.

22 in the perception of pain originating in the oesophagus.

23 risk of neoplastic progression in Barrett's oesophagus.

24 orded pressures in the hypopharynx and upper oesophagus.

25 d precursor lesions in people with Barrett's oesophagus--a metaplastic disorder that confers a high r

26 c, histologically confirmed carcinoma of the oesophagus (adenocarcinoma, squamous-cell, or undifferen

27 Surveillance of Barrett's oesophagus allows us to study the evolutionary dynamics

28 ample comprised 6167 patients with Barrett's oesophagus and 4112 individuals with oesophageal adenoca

29 up-regulated in the progression of Barrett's oesophagus and beta-catenin mediated transcription of c-

30 nfined to people with adenocarcinomas of the oesophagus and gastro-oesophageal junction (Siewert type

31 ort consisted of 468 patients with Barrett's oesophagus and intestinal metaplasia.

32 s necessary for the development of Barrett's oesophagus and its progression to cancer, and new stride

33 from dysfunction at the mouth, pharynx, and oesophagus and may predispose individuals to aspiration

34 e T-helper 1 response in normal and inflamed oesophagus and normal intestine.

35 the number of known risk loci for Barrett's oesophagus and oesophageal adenocarcinoma and revealed n

36 genome-wide association studies of Barrett's oesophagus and oesophageal adenocarcinoma available in P

37 tium investigating the genetics of Barrett's oesophagus and oesophageal adenocarcinoma, we aimed to i

38 ey molecules in the development of Barrett's oesophagus and oesophageal adenocarcinoma, which might e

39 sk variants for the development of Barrett's oesophagus and oesophageal adenocarcinoma.

40 ress the devices during delivery through the oesophagus and other narrow orifices in the digestive sy

41 types of surgery used to treat cancer of the oesophagus and summarise the available data about their

42 cinomas (SqCCs) of the lungs, head and neck, oesophagus, and cervix account for up to 30% of cancer d

43 nt of non-melanoma skin cancer and Barrett's oesophagus, and improved photosensitising drugs are in d

44 enetic variations from MFD-1, tumour, normal oesophagus, and leucocytes were analysed with SNP6.

45 cancers of the oral cavity, pharynx, larynx, oesophagus, and liver, and causes a small increase in th

46 of several complex tissues such as trachea, oesophagus, and skeletal muscle in animal models and hum

47 osed lower oesophagus, the non-exposed upper oesophagus, and the cutaneous area of pain referral, bef

48 orts to screen patients at risk of Barrett's oesophagus, and whether such efforts avert cancer death.

49 al nociceptive-like fibres in the guinea-pig oesophagus are derived from two embryonically distinct s

50 structures such as the respiratory tract and oesophagus are diverse, comprising several subtypes of f

51 The metaplastic cells of Barrett's oesophagus are predisposed to develop these cancer hallm

52 ium (believed to be a precursor of Barrett's oesophagus) are both characterized by the expansion of t

53 case-control pairs of the head and neck and oesophagus, as well as 443 additional controls.

54 Barrett oesophagus (BE) is a premalignant condition which predis

55 hanges that have been described in Barrett's oesophagus can be categorised according to the predomina

56 High-grade dysplasia in columnar-lined oesophagus can be eradicated by endoscopic photodynamic

57 The precancerous lesion known as Barrett's oesophagus can evolve to oesophageal adenocarcinoma in d

58 ctive data on head and neck cancer (HNC) and oesophagus cancer are limited.

59 samples and evaluated in relation to HNC and oesophagus cancer risk and post-diagnosis all-cause mort

60 Achalasia is a rare motility disorder of the oesophagus characterised by loss of enteric neurons lead

61 Obesity increases the risk of cancers in the oesophagus, colorectum, breast, endometrium, and kidney.

62 uctions in HRQoL scores related to Barrett's oesophagus compared with controls from the general popul

63 The vagal nociceptive-like fibres in the oesophagus comprise two distinct subtypes dictated by th

64 fficacy of a dedicated service for Barrett's oesophagus, cost-effectiveness and appropriateness of cu

65 However, only a few patients with Barrett's oesophagus develop adenocarcinoma, which complicates cli

66 6 x 10(-8)) and was independent of Barrett's oesophagus development (p=0.45).

67 essed in all tissues examined, including the oesophagus, eye, liver, intestine, posterior and anterio

68 infusion, their pain threshold in the upper oesophagus fell further and for longer (mean fall in are

69 idy in a cohort of 350 people with Barrett's oesophagus followed for 20,770 person-months.

70 electrical stimuli applied to the pharynx or oesophagus had no effect on the response to cortical sti

71 pic treatment of early neoplastic lesions in oesophagus has evolved as a valid and less invasive alte

72 ffuse oesophageal spasm and hypercontracting oesophagus, have no well defined pathology and could rep

73 in microbial communities occur in the lower oesophagus in Barrett's carcinogenesis, which can be det

74 in microbial communities occur in the lower oesophagus in Barrett's carcinogenesis, which can be det

75 ue to assess biomechanical properties of the oesophagus in human subjects.

76 o orthotopically replace the entire cervical oesophagus in immunocompetent rats.

77 and passive biomechanical properties of the oesophagus in normal healthy humans.

78 lusive: no model completely mimics Barrett's oesophagus in terms of the presence of intestinal goblet

79 s and extracellular matrix to regenerate the oesophagus in vivo in a human being to re-establish swal

80 n mice causes hyperplasia selectively in the oesophagus, in association with increased cell prolifera

81 the major cell and tissue components of the oesophagus including functional epithelium, muscle fibre

82 In the case of Barrett's oesophagus, intestinal metaplasia occurs at the gastro-o

83 Barrett's oesophagus is a chronic precancerous condition that pred

84 Barrett's oesophagus is a metaplastic change of the lining of the

85 Barrett's oesophagus is characterised by replacement of reflux-dam

86 Hypocontracting oesophagus is generally caused by weak musculature commo

87 tro-oesophageal reflux disease and Barrett's oesophagus is increasing.

88 The mouse embryonic oesophagus is initially lined with a simple columnar epi

89 Barrett's oesophagus is the main precursor to oesophageal adenocar

90 Barrett's oesophagus is the premalignant precursor of oesophageal

91 thy volunteers, acid infusion into the lower oesophagus lowered the pain threshold in the upper oesop

92 agus lowered the pain threshold in the upper oesophagus (mean decrease 18.2% [95% CI 10.4 to 26.0]; p

93 en oesophageal squamous mucosa and Barrett's oesophagus mucosa.

94 n-dysplastic [n=24] and dysplastic Barrett's oesophagus [n=23], and oesophageal adenocarcinoma [n=19]

95 temperature can be reliably measured in the oesophagus, nasopharynx, mouth, and bladder.

96 the dysplastic epithelium in columnar-lined oesophagus occurred after light activation.

97 stologically confirmed adenocarcinoma of the oesophagus, oesophagogastric junction, or stomach that h

98 ylidene-2'-deoxyguanosine were higher in the oesophagus of Aldh2-knockout mice than in wild-type mice

99 hypothalamus, pituitary, pancreas, lungs and oesophagus of mouse embryos using in situ hybridization.

100 Moderate balloon distension of the oesophagus of the rat (14-18 mmHg) provoked a significan

101 l drinking increased ALDH2 production in the oesophagus of wild-type mice.

102 The true effect of Barrett's oesophagus on life expectancy and the efficacy of long-t

103 w risk loci associated with either Barrett's oesophagus or oesophageal adenocarcinoma, within or near

104 ence of complex tissues such as the trachea, oesophagus, or skeletal muscle have few therapeutic opti

105 endometrium, kidney, liver, lung, lymphoid, oesophagus, ovary, pancreas, skin, soft tissue and thyro

106 Barrett's oesophagus predisposes to adenocarcinoma.

107 rade dysplasia in columnar-lined (Barrett's) oesophagus presents a difficult therapeutic dilemma.

108 from 86 patients representing the Barrett's oesophagus progression sequence (normal squamous control

109 The cancer hallmarks of Barrett's oesophagus provide a framework to categorise the genetic

110 eration and functional roles of ALDH2 in the oesophagus remain elusive.

111 ive stress-strain relationships of the human oesophagus resemble those of other soft biological tissu

112 The scores of patients with Barrett's oesophagus seem to be similar to those of patients with

113 st-to-hip ratio, and length of the Barrett's oesophagus segment.

114 ing nuclear GFP into the epithelium of E15.5 oesophagus, some cells expressed both K8 and GFP.

115 emical techniques with retrogradely labelled oesophagus-specific neurones and performing extracellula

116 , including most of those originating in the oesophagus, stomach, biliary tract and pancreas, but not

117 ant diseases, including cancers of the lung, oesophagus, stomach, breast, and kidney.

118 ted with an increased risk of cancers of the oesophagus, stomach, larynx, lung, and urinary bladder.

119 is a metaplastic change of the lining of the oesophagus, such that the normal squamous epithelium is

120 were monitored within the acid-exposed lower oesophagus, the non-exposed upper oesophagus, and the cu

121 alterations of RA biosynthesis in Barrett's oesophagus, the precursor lesion to oesophageal adenocar

122 prediction of the transition from Barrett's oesophagus to oesophageal adenocarcinoma.

123 distributed in the digestive tract from the oesophagus to the colon.

124 ed for the growth and differentiation of the oesophagus, trachea and lung, and suggest that mutations

125 ohort study (BEST2), patients with Barrett's oesophagus underwent the Cytosponge test before their su

126 nsport of tiny concentrated particles to the oesophagus using a hydrodynamic tongue.

127 stents were removed after 3.5 years and the oesophagus was assessed by endoscopy, biopsy, endoscopic

128 d early steps in the initiation of Barrett's oesophagus, we assessed the expression, location and fun

129 ordings from the isolated vagally innervated oesophagus, we show that in the guinea-pig, the vagus ne

130 ra-bag pressure and ultrasound images of the oesophagus were recorded simultaneously.

131 latency reflex responses in the pharynx and oesophagus, were used to condition the cortical response

132 in stratified epithelia, most notably in the oesophagus where the Nanog promoter is hypomethylated.

133 However, most patients with Barrett's oesophagus will not progress and endoscopic surveillance

134 the guinea-pig, the vagus nerves supply the oesophagus with a large population of nociceptive-like a

135 isorder characterized by infiltration of the oesophagus with eosinophils.

136 that the vagus nerves supply the guinea-pig oesophagus with nociceptors in addition to tension mecha

137 ntenance of the structural morphology of the oesophagus with off-the-shelf non-biological scaffold an

138 al we saw full-thickness regeneration of the oesophagus with stratified squamous epithelium, a normal