ライフサイエンスコーパス: offensive

1 adolescence increased nearly all measures of offensive aggression (i.e., upright offensive attacks, l

2 utoradiography, the hamsters were tested for offensive aggression after microinjections of AVP in com

3 ated adolescent fluoxetine administration on offensive aggression and the development of the serotoni

4 at received AAS for 3, 7, 14, or 28 days for offensive aggression and then examined the hamsters for

5 ult male hamsters, individual differences in offensive aggression are correlated with differences in

6 ssive adults from P28 to P42, and tested for offensive aggression as young adults (P45).

7 and are suspected to inhibit AVP-facilitated offensive aggression by an activation of 5-HT1A receptor

8 Offensive aggression did not follow a clear stereotyped

9 AAS-treated hamsters were tested for offensive aggression following the administration of the

10 When tested for offensive aggression in a resident/intruder paradigm, re

11 Offensive aggression in golden hamsters is inhibited by

12 inine vasopressin (AVP) in the regulation of offensive aggression in golden hamsters.

13 tal and neuroplastic changes correlated with offensive aggression in hamsters.

14 ) exposure throughout adolescence stimulates offensive aggression in hamsters.

15 (AAS) exposure during adolescence stimulates offensive aggression in male Syrian hamsters.

16 anterior hypothalamus (AH) in the control of offensive aggression in Syrian golden hamsters.

17 ids (AAS) throughout adolescence facilitates offensive aggression in Syrian hamsters.

18 The action of microinjected AVP to increase offensive aggression is blocked by the pretreatment of h

19 In golden hamsters, offensive aggression is facilitated by vasopressin and i

20 We measured the offensive aggression of wild caught territorial monogamo

21 AAS) exposure during adolescence facilitates offensive aggression that is correlated with the enhance

22 AAS) exposure during adolescence facilitates offensive aggression that is modulated, in part, by sero

23 econd experiment, mated males exhibited more offensive aggression to a male intruder but more defensi

24 f cocaine throughout adolescence, scored for offensive aggression using the resident-intruder paradig

25 ters administered fluoxetine were tested for offensive aggression using the resident/intruder test, s

26 of AAS exposure, no concomitant increases in offensive aggression were observed compared to oil-treat

27 rast, by Day 14 of AAS treatment, AH AVP and offensive aggression were significantly higher than oil-

28 dose-dependent inhibition of AVP-facilitated offensive aggression, CGS was ineffective.

29 infant attachment, and modulation of memory, offensive aggression, defensive fear reactions, and rewa

30 P) acting through a V1A receptor facilitates offensive aggression, i.e., the initiation of attacks an

31 ndrogenic steroid (AAS) exposure facilitates offensive aggression, in part by altering the developmen

32 g adolescence predisposes Syrian hamsters to offensive aggression, with demonstrable neurophysiologic

33 active features and projections with overall offensive aggression.

34 rank, may be mediating the inter-individual offensive aggression.

35 e involved in adolescent cocaine-facilitated offensive aggression.

36 olescence (P27-P56) display highly escalated offensive aggression.

37 are more anxious and males display increased offensive aggressiveness.

38 This contact initiates complex offensive and defensive strategies by both parties.

39 Bacterial type VI secretion is an offensive and defensive weapon that utilizes a molecular

40 From a tree's perspective, wood is an offensive and defensive weapon used against neighbors in

41 In the case where a product has an offensive and strong aroma, many consumers might not app

42 s were dramatically reduced and male-typical offensive attacks were rarely displayed by ERKO males.

43 male-typical aggressive behavior, including offensive attacks, but they also showed higher levels of

44 sures of offensive aggression (i.e., upright offensive attacks, lateral attacks, flank/rump bites, pu

45 Offensive attacks, on the other hand, depend specificall

46 lunge and bite aggression, but rarely showed offensive attacks.

47 that under normal circumstances, potentially offensive autoreactive cells are silenced by mechanisms

48 erotonergic system shapes the development of offensive behaviors in male golden hamsters.

49 venting the enhancement and proliferation of offensive biological warfare programs.

50 biology has great potential to be misused in offensive biological weapons programs.

51 nvention where all countries agreed to cease offensive biological weapons research.

52 EON) group, indicating that AEON is the most offensive boar taint compound for sensitive assessors.

53 nce/absence of tadpoles and salamanders with offensive (broadened gape width) or non-offensive phenot

54 sense subjects clearly classify intruders as offensive, but fail to attack.

55 d intensity of fighting based on unambiguous offensive fighting behaviors.

56 of defense that coevolve with corresponding offensive lines of the parasite.

57 ensive capsule and genes for secretion of an offensive lysin or toxin suggest a heretofore unknown pa

58 Almost all offensive odorous compounds, particularly benzyl isothio

59 t lag impaired major parameters of home-team offensive performance, for example, slugging percentage,

60 tage, but did not similarly affect away-team offensive performance.

61 ncreased predation on tadpoles likely causes offensive phenotype individuals to have strong impacts o

62 eir size nor larval period differed from non-offensive phenotype individuals.

63 Moreover, in the presence of tadpoles, offensive phenotype salamanders metamorphosed 25% faster

64 Offensive phenotype salamanders reduced tadpole survival

65 Thus, offensive phenotype salamanders reduced the number of ta

66 amorphosed 25% faster and 5% larger than non-offensive phenotype salamanders, but in their absence, n

67 salamander treatments and 6% larger than non-offensive phenotype treatments.

68 Offensive phenotypes also caused tadpoles to metamorphos

69 with offensive (broadened gape width) or non-offensive phenotypes in pond enclosures.

70 potential fitness advantages, the impacts of offensive phenotypes on frog and salamander life histori

71 treatments, and by over 30% compared to non-offensive phenotypes.

72 pulation and community level consequences of offensive phenotypes.

73 niles, high doses of fluoxetine only reduced offensive responses (i.e., frequency and repetition of a

74 As serotonin inhibits offensive responses in adult hamsters, it is hypothesize

75 Most noted offensive sludge odors that interfere with daily activit

76 e also characterized two potential alternate offensive strategies by associated variation in network

77 e the parasite deploys complex defensive and offensive strategies to combat immune attack.

78 vealed that pathogens utilize many universal offensive strategies to overcome host defenses, irrespec

79 ne whether we can assess differences in team offensive strategy by their network properties.

80 iously perceived by different individuals as offensive ("sweaty, urinous"), pleasant ("sweet, floral"

81 Offensive tail flipping, attacks, and approaches by both

82 pe tail flips and a fall in the frequency of offensive tail flips of the new subordinate.

83 sula is known to be involved in responses to offensive tastes.