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1 hesitis, axial joint disease, and persistent oligoarthritis.
2 established linkage of the locus to juvenile oligoarthritis.
3 TNFalpha in the etiopathogenesis of juvenile oligoarthritis.
4 B1 was also observed in female patients with oligoarthritis.
5 ers clinical course but usually manifests as oligoarthritis.
6 ps of persistent oligoarthritis and extended oligoarthritis.
7 irmed as independent susceptibility loci for oligoarthritis.
8 , and DRB1 in UK Caucasian JIA patients with oligoarthritis.
9 locus is linked and associated with juvenile oligoarthritis.
10 ypes containing this haplotype with extended oligoarthritis.
11 ly acquired ReA and 31 with undifferentiated oligoarthritis.
12 tients recruited to the study, 266 (53%) had oligoarthritis.
13 rse clinical course but usually manifests as oligoarthritis.
14 ways was evident in patients with persistent oligoarthritis.
15 es from 15 patients (8 with undifferentiated oligoarthritis, 3 with ReA, 1 with rheumatoid arthritis,
16 ad Reiter's syndrome, 7 had undifferentiated oligoarthritis, 4 had undifferentiated monarthritis, 6 h
17 -related arthritis [ERA], 42 with persistent oligoarthritis, 45 with rheumatoid factor [RF]-negative
18                                 Furthermore, oligoarthritis again occurred on day 10 (median; IQR 9-1
19 0 days), developed acute and severe mono- or oligoarthritis almost exclusively in the limb closest to
20 Rheumatology-defined subgroups of persistent oligoarthritis and extended oligoarthritis.
21    Linkage to HLA-A and HLA-DRB1 was seen in oligoarthritis and in the International League of Associ
22 ogical examinations revealed severe poly- or oligoarthritis and moderate to severe cortical hyperplas
23                         In 358 children with oligoarthritis and rheumatoid factor-negative polyarthri
24 sociation of multiple TNF SNPs with juvenile oligoarthritis and to construct and analyze SNP-tagged T
25 o establish linkage of HLA-A and HLA-DRB1 in oligoarthritis and to show that the 2 loci contribute in
26 ation of dactylitis of a toe, heel pain, and oligoarthritis appears to be strongly suggestive of psor
27 teroids are an effective treatment for early oligoarthritis, but there is still a high level of long-
28                                              Oligoarthritis has a significant impact on function and
29 e associations between HLA loci and juvenile oligoarthritis have previously been documented, evidence
30 vative therapy in patients with recent-onset oligoarthritis in a randomized controlled trial.
31 m that are responsible for susceptibility to oligoarthritis in UK Caucasian patients with JIA.
32 umented, evidence for linkage of HLA loci in oligoarthritis in UK Caucasians with juvenile idiopathic
33 pear to be a major cause of undifferentiated oligoarthritis or ReA.
34 8 patients with either ReA, undifferentiated oligoarthritis, or other forms of arthritis, and from 24
35 ted States, intermittent or chronic mono- or oligoarthritis, particularly affecting the knee, is the
36         Children with persistent or extended oligoarthritis, polyarthritis (either rheumatoid factor
37 compared with those from the ERA, persistent oligoarthritis, RF-negative polyarthritis, and systemic
38                  In patients with persistent oligoarthritis, RF-negative polyarthritis, and systemic
39                          For acute mono- and oligoarthritis, rheumatologists performed arthrocentesis
40 the intronic +851 TNF SNP and the persistent oligoarthritis subgroup (OR 3.86, 95% CI 1.6-9.2).
41 A, as compared with patients with persistent oligoarthritis, the milder subtype (P = 0.046).
42 umbers were higher in patients with extended oligoarthritis, the more severe subtype of JIA, as compa
43                                              Oligoarthritis was defined as <5 tender and/or swollen j
44                                  After viral oligoarthritis was observed in 11 of the first 51 vaccin
45 , rheumatoid arthritis, and undifferentiated oligoarthritis, was assayed by polymerase chain reaction
46 ive polyarthritis, systemic JIA, or extended oligoarthritis were eligible for the study.
47 ling subgroups of JIA, systemic and extended oligoarthritis, were underrepresented in that study.
48 of unselected patients with undifferentiated oligoarthritis, which further supports the hypothesis th

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