ライフサイエンスコーパス: oligodendrocyte progenitor

1 ssion from Sox2(+) 'early' to Sox2(-) 'late' oligodendrocyte progenitor.

2 sm involving crosstalk between microglia and oligodendrocyte progenitors.

3 ick and mouse models and in vitro culture of oligodendrocyte progenitors.

4 uted population of glial cells that serve as oligodendrocyte progenitors.

5 f4, plays an important role in maturation of oligodendrocyte progenitors.

6 ntiated mitosis in primary cultures of mouse oligodendrocyte progenitors.

7 , and CC1-, indicated a close resemblance to oligodendrocyte progenitors.

8 presence of a susceptible population of late oligodendrocyte progenitors.

9 els, but we could detect the alpha(1A)-AR in oligodendrocyte progenitors.

10 sed markers for astrocytes and for neural or oligodendrocyte progenitors.

11 cerebral white matter and targeted death of oligodendrocyte progenitors.

12 es the expression of myelin basic protein in oligodendrocyte progenitors.

13 ritical role in remyelination via effects on oligodendrocyte progenitors.

14 observed in the survival or proliferation of oligodendrocyte progenitors.

15 cluding reactive microglia, macrophages, and oligodendrocyte progenitors.

16 owth factor receptor alpha (Pdgfra)-positive oligodendrocyte progenitors.

17 s that the ectopic astrocytes originate from oligodendrocyte progenitors.

18 dendrocyte development was delayed such that oligodendrocyte progenitors accumulated inappropriately

19 results showing defective differentiation of oligodendrocyte progenitors after silencing specific HDA

20 results showing defective differentiation of oligodendrocyte progenitors after silencing specific HDA

21 evel, lack of yy1 arrests differentiation of oligodendrocyte progenitors after they exit from the cel

22 Furthermore, the oligodendrocyte progenitors also become progressively de

23 Depletion of oligodendrocyte progenitors and demyelination are major

24 , revealed that it is similarly expressed in oligodendrocyte progenitors and not oligodendrocytes.

25 sis using gene expression profiling of A2B5+ oligodendrocyte progenitors and O4+ oligodendrocytes.

26 w that Nkx2.2 is also expressed in mammalian oligodendrocyte progenitors and that the differentiation

27 xpressed exclusively in oligodendrocytes and oligodendrocyte progenitors, and Olig1 can promote forma

28 ndently inhibited the maturation of purified oligodendrocyte progenitors, and pharmacological inhibit

29 pMN domain of the ventral VZ where Pdgfra(+) oligodendrocyte progenitors--and motoneurones--originate

30 Immunohistochemical studies showed that late oligodendrocyte progenitors appear at gestational age 22

31 n of neural progenitors and specification of oligodendrocyte progenitors are completed with the forma

32 ignificance statement: It is recognized that oligodendrocyte progenitors are mechanosensitive cells.

33 hibition, we demonstrate that specified NG2+ oligodendrocyte progenitors are plastic cells, whose dec

34 iation is attributed in part to apoptosis of oligodendrocyte progenitors as they exit the cell cycle.

35 ed in apoptosis and reduced proliferation of oligodendrocyte progenitors, as well as arrested maturat

36 ury, including both reactive macrophages and oligodendrocyte progenitors; astrocytes were not identif

37 cytotoxic effects on actively proliferating oligodendrocyte progenitors but much less on immature ol

38 NFIA is expressed in oligodendrocyte progenitors, but not differentiated olig

39 Moreover, EGFR(+) oligodendrocyte progenitors, but not neuroblasts, expres

40 In the adult mammalian brain, oligodendrocyte progenitors can differentiate into matur

41 Generating patient-specific oligodendrocyte progenitors capable of repairing myelina

42 Recently, we showed oligodendrocyte progenitor cell (OPC) accumulation and r

43 In oligodendrocyte progenitor cell (OPC) cultures, IL-11 re

44 d Ca(2+) channels (L-VOCCs) are required for oligodendrocyte progenitor cell (OPC) development, we ge

45 n by acting at several critical steps during oligodendrocyte progenitor cell (OPC) development.

46 The effect of olesoxime on oligodendrocyte progenitor cell (OPC) differentiation an

47 d increases both adult mouse and adult human oligodendrocyte progenitor cell (OPC) differentiation, i

48 he ventral telencephalon were generated, but oligodendrocyte progenitor cell (OPC) generation was sev

49 se (MAPK)-dependent pathway is implicated in oligodendrocyte progenitor cell (OPC) lineage progressio

50 re infants results in inflammation, arrested oligodendrocyte progenitor cell (OPC) maturation, and re

51 ibute to remyelination failure by perturbing oligodendrocyte progenitor cell (OPC) maturation.

52 This work describes the role of oligodendrocyte progenitor cell (OPC) microRNAs (miRNAs)

53 two nf1 orthologs in zebrafish and show that oligodendrocyte progenitor cell (OPC) numbers are increa

54 Oligodendrocyte death and oligodendrocyte progenitor cell (OPC) proliferation duri

55 We have previously demonstrated that oligodendrocyte progenitor cell (OPC) proliferation is c

56 Human oligodendrocyte progenitor cell (OPC) specification and

57 Both oligodendrocyte progenitor cell commitment and oligodend

58 strates a unique role for Olig1 in promoting oligodendrocyte progenitor cell commitment, differentiat

59 mportant roles for Sox17 in controlling both oligodendrocyte progenitor cell cycle exit and different

60 elevant, FDA-approved compounds that promote oligodendrocyte progenitor cell differentiation and indu

61 endrocyte lineage cells completely inhibited oligodendrocyte progenitor cell differentiation and myel

62 Treg directly promoted oligodendrocyte progenitor cell differentiation and myel

63 tory supernatants also resulted in decreased oligodendrocyte progenitor cell differentiation without

64 myelin debris, which contains inhibitors of oligodendrocyte progenitor cell differentiation.

65 d neither a direct nor an indirect impact on oligodendrocyte progenitor cell differentiation.

66 enhancer (PIKE) expression by shRNA prevents oligodendrocyte progenitor cell differentiation.

67 ess and promotes precocious neurogenesis and oligodendrocyte progenitor cell elaboration.

68 process formation were also inhibited in the oligodendrocyte progenitor cell line CG-4 after suppress

69 of transplanted cells co-labeled for NG2, an oligodendrocyte progenitor cell marker, but not for neur

70 urons and astrocytes remains the same, early oligodendrocyte progenitor cell markers are decreased in

71 As shown recently, failure in oligodendrocyte progenitor cell maturation contributes t

72 Oligodendrocyte progenitor cell number decreased with ag

73 infiltrates and demyelination, and increased oligodendrocyte progenitor cell proliferation and BDNF+

74 Oligodendrocyte progenitor cell proliferation was observ

75 /or indirectly (via astrocytes) impact human oligodendrocyte progenitor cell survival and differentia

76 enriched population of cells expressing the oligodendrocyte progenitor cell-specific marker NG2.

77 se embryonic and lung fibroblasts to induced oligodendrocyte progenitor cells (iOPCs) using sets of e

78 al and mesenchymal glioblastoma, relative to oligodendrocyte progenitor cells (Oli-Neu).

79 e found that AXIN2 was expressed in immature oligodendrocyte progenitor cells (OLPs) in white matter

80 erived growth factor receptor alpha-positive oligodendrocyte progenitor cells (OPC) located within th

81 ntiate human embryonic stem cells (hESCs) to oligodendrocyte progenitor cells (OPCs) according to dev

82 se genes was measured in primary neurons and oligodendrocyte progenitor cells (OPCs) after inflammato

83 Microarray analysis in oligodendrocyte progenitor cells (OPCs) after Sox17 atte

84 AMPA stimulation of cultured oligodendrocyte progenitor cells (OPCs) also caused an i

85 in vitro primary rat embryonic cell model of oligodendrocyte progenitor cells (OPCs) and a mouse N20.

86 ation at the same site, eventually depleting oligodendrocyte progenitor cells (OPCs) and exhausting t

87 hogenetic protein (BMP) signaling pathway in oligodendrocyte progenitor cells (OPCs) and suppresses r

88 expression in the developing CNS identifies oligodendrocyte progenitor cells (OPCs) and whose activa

89 , both the proliferation and total number of oligodendrocyte progenitor cells (OPCs) appeared normal

90 ination and why remyelination is absent when oligodendrocyte progenitor cells (OPCs) are present.

91 In the postnatal brain, oligodendrocyte progenitor cells (OPCs) arise from the s

92 termination of proliferation determines when oligodendrocyte progenitor cells (OPCs) can initiate dif

93 Oligodendrocyte progenitor cells (OPCs) can repair demye

94 Remyelination by oligodendrocyte progenitor cells (OPCs) can restore thes

95 ular mechanisms that drive the maturation of oligodendrocyte progenitor cells (OPCs) during the remye

96 l myelinating glial cells, centrally derived oligodendrocyte progenitor cells (OPCs) ectopically exit

97 Here, we report that oligodendrocyte progenitor cells (OPCs) express function

98 fish, we observed that prior to myelination, oligodendrocyte progenitor cells (OPCs) extend processes

99 factor-1 (IGF-1) provides neuroprotection to oligodendrocyte progenitor cells (OPCs) following cerebr

100 y also be a radial component of migration of oligodendrocyte progenitor cells (OPCs) from a ventral s

101 Migration of oligodendrocyte progenitor cells (OPCs) from proliferati

102 mice also exhibited an increased density of oligodendrocyte progenitor cells (OPCs) in CNS white mat

103 Oligodendrocyte progenitor cells (OPCs) in demyelinated

104 e show that the expression of Sox2 occurs in oligodendrocyte progenitor cells (OPCs) in rodent models

105 fficulties in generating pure populations of oligodendrocyte progenitor cells (OPCs) in sufficient qu

106 ion, and may contribute to the production of oligodendrocyte progenitor cells (OPCs) in the dorsal co

107 Oligodendrocyte progenitor cells (OPCs) in the postnatal

108 of human embryonic stem cell (hESC)-derived oligodendrocyte progenitor cells (OPCs) into adult rat s

109 Differentiation of oligodendrocyte progenitor cells (OPCs) into mature olig

110 n obligatory step for the differentiation of oligodendrocyte progenitor cells (OPCs) into myelinating

111 in disorders can be treated by transplanting oligodendrocyte progenitor cells (OPCs) into the affecte

112 Differentiation and maturation of oligodendrocyte progenitor cells (OPCs) involve the asse

113 cal changes, while in developing neurons and oligodendrocyte progenitor cells (OPCs) it induces cellu

114 lutamatergic synapses onto adult-born NG2(+) oligodendrocyte progenitor cells (OPCs) migrating from t

115 t oligodendrocytes, whether by transplanting oligodendrocyte progenitor cells (OPCs) or by mobilizing

116 n which Tsc1 is deleted by Cre expression in oligodendrocyte progenitor cells (OPCs) or in premyelina

117 Neonatal engraftment by oligodendrocyte progenitor cells (OPCs) permits the myel

118 Oligodendrocyte progenitor cells (OPCs) persist in human

119 We show how Sox2 is expressed in oligodendrocyte progenitor cells (OPCs) preparing to und

120 In experimental models, oligodendrocyte progenitor cells (OPCs) rather than prev

121 Oligodendrocyte progenitor cells (OPCs) recruited to dem

122 Oligodendrocyte progenitor cells (OPCs) that are positiv

123 isoprenoid and cholesterol synthesis, causes oligodendrocyte progenitor cells (OPCs) to migrate past

124 er, direct injection of neural stem cells or oligodendrocyte progenitor cells (OPCs) to the lesion si

125 in which we targeted Notch1 inactivation to oligodendrocyte progenitor cells (OPCs) using Olig1Cre a

126 ation by blocking the differentiation of rat oligodendrocyte progenitor cells (OPCs) via modulation o

127 Many chronically demyelinated lesions have oligodendrocyte progenitor cells (OPCs) within their bor

128 mong three macroglial progenitor populations-oligodendrocyte progenitor cells (OPCs), astrocytes and

129 In oligodendrocyte progenitor cells (OPCs), Lrp1 is require

130 In cultured oligodendrocyte progenitor cells (OPCs), Sox17 levels we

131 tly characterized by scarce undifferentiated oligodendrocyte progenitor cells (OPCs), suggesting the

132 gene expression profiling on purified murine oligodendrocyte progenitor cells (OPCs), the remyelinati

133 rve conduction, and the ectopic migration of oligodendrocyte progenitor cells (OPCs), the resident my

134 rotein fibronectin perturb the maturation of oligodendrocyte progenitor cells (OPCs), thereby impedin

135 Since one role of NG2 glia is that of oligodendrocyte progenitor cells (OPCs), we investigated

136 cells produces first motor neurons and then oligodendrocyte progenitor cells (OPCs), which migrate,

137 and generate de novo synapses with recruited oligodendrocyte progenitor cells (OPCs), which, early af

138 iated neuroinflammation, and an expansion of oligodendrocyte progenitor cells (OPCs).

139 the generation of new oligodendrocytes from oligodendrocyte progenitor cells (OPCs).

140 liferation, migration and differentiation of oligodendrocyte progenitor cells (OPCs).

141 modulator of intracellular Ca(2+) levels in oligodendrocyte progenitor cells (OPCs).

142 lay a fundamental role in the development of oligodendrocyte progenitor cells (OPCs).

143 ig1-Cre-expressing cells reduces the pool of oligodendrocyte progenitor cells (OPCs).

144 glial-restricted progenitors cells, known as oligodendrocyte progenitor cells (OPCs).

145 cytes arise from migratory and proliferative oligodendrocyte progenitor cells (OPCs).

146 ult human forebrain contain large numbers of oligodendrocyte progenitor cells (OPCs).

147 h activity could promote formation of excess oligodendrocyte progenitor cells (OPCs).

148 emyelination by impairing differentiation of oligodendrocyte progenitor cells (OPCs).

149 endent on recruitment and differentiation of oligodendrocyte progenitor cells (OPCs).

150 myelin and myelin repair by differentiating oligodendrocyte progenitor cells (OPCs).

151 most PLP-EGFP-expressing cells gave rise to oligodendrocyte progenitor cells (OPCs).

152 Proliferative oligodendrocyte progenitor cells (OPs) express large, de

153 Genetic deletion of TACE in oligodendrocyte progenitor cells (OPs) induces premature

154 Parenchymal oligodendrocyte progenitor cells (pOPCs) are considered

155 of betaT4-positive cells with A2B5-positive oligodendrocyte progenitor cells after transplantation (

156 peroxia showed a reduced capacity to protect oligodendrocyte progenitor cells against the toxic effec

157 e of the TUJ-1-positive cells, A2B5-positive oligodendrocyte progenitor cells and A2B5-negative cells

158 by an expression pattern resembling that of oligodendrocyte progenitor cells and carries a distincti

159 Both parenchymal oligodendrocyte progenitor cells and endogenous adult ne

160 In the developing spinal cord, early oligodendrocyte progenitor cells are induced from the ve

161 Oligodendrocyte progenitor cells are stem cells in the c

162 ation that human embryonic stem cell-derived oligodendrocyte progenitor cells are susceptible to JC v

163 nical translation: first, transplantation of oligodendrocyte progenitor cells as a means of treating

164 vides direct evidence that targeting EGFR in oligodendrocyte progenitor cells at a specific time afte

165 However, myosin-1d was undetectable in oligodendrocyte progenitor cells at early and late time

166 pression of syntaxin 4 but not syntaxin 3 in oligodendrocyte progenitor cells but not immature oligod

167 PIKE-L expression is up-regulated when oligodendrocyte progenitor cells commit to differentiati

168 and constitutive ablation of NR1 in neonatal oligodendrocyte progenitor cells did not interrupt their

169 ous system (CNS) most often is the result of oligodendrocyte progenitor cells differentiating into my

170 TACE deficiency in oligodendrocyte progenitor cells following demyelination

171 brinogen inhibits nerve repair by preventing oligodendrocyte progenitor cells from differentiating in

172 cal studies revealed that the recruitment of oligodendrocyte progenitor cells in response to demyelin

173 e fifth major cell population that serves as oligodendrocyte progenitor cells in the postnatal CNS.

174 eration of human oligodendrocytes from human oligodendrocyte progenitor cells in vitro.

175 ly tune axonal diameter, promote re-entry of oligodendrocyte progenitor cells into the cell cycle, or

176 on rather than uncontrolled proliferation of oligodendrocyte progenitor cells may have important impl

177 immature oligodendrocyte-lineage cells, with oligodendrocyte progenitor cells more vulnerable to inju

178 In the absence of ERK1/ERK2 signaling NG2(+) oligodendrocyte progenitor cells proliferated and differ

179 On the other hand, it is possible that oligodendrocyte progenitor cells remain undifferentiated

180 Differentiation of oligodendrocyte progenitor cells requires activation of

181 noted because Nkx2.2 promotes maturation of oligodendrocyte progenitor cells specified by expression

182 w that miconazole and clobetasol function in oligodendrocyte progenitor cells through mitogen-activat

183 ibility of human embryonic stem cell-derived oligodendrocyte progenitor cells to infection with JC vi

184 y changes during the transition from A2B5(+) oligodendrocyte progenitor cells to premyelinating GalC(

185 man adult brain-derived oligodendrocytes and oligodendrocyte progenitor cells under conditions of met

186 Whereas LINGO-1 expressed by oligodendrocyte progenitor cells was previously identifi

187 two populations of NG2 proteoglycan-positive oligodendrocyte progenitor cells were identified that ex

188 Oligodendrocyte progenitor cells were not decreased in d

189 se developing axon-free nerves, 25 to 33% of oligodendrocyte progenitor cells were proliferating.

190 rability of O4+ preoligodendrocytes, whereas oligodendrocyte progenitor cells were resistant to insul

191 o explore the mechanism of redistribution of oligodendrocyte progenitor cells with compensatory myeli

192 ng inflammatory injury, oligodendrocytes and oligodendrocyte progenitor cells within lesion sites are

193 r for cell proliferation), NG2 (a marker for oligodendrocyte progenitor cells) and brain-derived neur

194 arative capabilities, and transplantation of oligodendrocyte progenitor cells, have generated substan

195 ed to the expansion of genetically wild-type oligodendrocyte progenitor cells, oligodendrocyte differ

196 Nonproliferating oligodendrocyte progenitor cells, oligodendrocytes, and

197 uced myelin were examined for remyelination, oligodendrocyte progenitor cells, reactive astrocytes, a

198 sis, remyelination can fail despite abundant oligodendrocyte progenitor cells, suggesting impairment

199 d with a truncated proliferative response of oligodendrocyte progenitor cells, suggesting that deplet

200 ds for enhancing myelination from endogenous oligodendrocyte progenitor cells, we screened a library

201 ze the effects of BCNU on clonal cultures of oligodendrocyte progenitor cells-one of the best-charact

202 require technologies to generate functional oligodendrocyte progenitor cells.

203 lates cell cycle exit and differentiation in oligodendrocyte progenitor cells.

204 otential for recovery mediated by endogenous oligodendrocyte progenitor cells.

205 O mice revealed a specific deficit of NG2(+) oligodendrocyte progenitor cells.

206 ves differentiation of oligodendrocytes from oligodendrocyte progenitor cells.

207 mote production of new oligodendrocytes from oligodendrocyte progenitor cells.

208 tire CNS after they have differentiated from oligodendrocyte progenitor cells.

209 s have dramatically increased numbers of CNS oligodendrocyte progenitor cells.

210 mmunocytochemically for oligodendrocytes and oligodendrocyte progenitor cells.

211 white matter is synthesized by proliferating oligodendrocyte progenitor cells.

212 ouse for voltage-operated Ca(2+) channels in oligodendrocyte progenitor cells.

213 mouse pluripotent epiblast stem-cell-derived oligodendrocyte progenitor cells.

214 ndent on the survival and differentiation of oligodendrocyte progenitor cells.

215 compression on the function and survival of oligodendrocyte progenitor cells/oligodendrocytes and ax

216 els to selectively delete TACE expression in oligodendrocyte progenitors cells (OPs), we found that T

217 alpha)-expressing stromal cells derived from oligodendrocytes progenitor cells (OPC) were discovered

218 senchymal stem cells, mouse lymphocytes, rat oligodendrocyte progenitor CG-4 cells, and human cervica

219 te and determined whether transplanted adult oligodendrocyte progenitors could remyelinate the chroni

220 found that, in the absence of Schwann cells, oligodendrocyte progenitors cross ventral root transitio

221 FGF2 -/- and +/+ mice had similar oligodendrocyte progenitor densities in normal adult CNS

222 efects in PLP1 mRNA expression and splicing, oligodendrocyte progenitor development, and oligodendroc

223 We observed that oligodendrocyte progenitors did not accumulate, prolifer

224 treatment, and was associated with enhanced oligodendrocyte progenitor differentiation and epigeneti

225 effect of three mechanical stimuli on mouse oligodendrocyte progenitor differentiation and identify

226 est that NFIA participates in the control of oligodendrocyte progenitor differentiation and may contr

227 indicate that NFIA is sufficient to suppress oligodendrocyte progenitor differentiation during adult

228 scription factor nuclear factor IA (NFIA) in oligodendrocyte progenitor differentiation during develo

229 tral nervous system lesions are required for oligodendrocyte progenitor differentiation into remyelin

230 Yin Yang 1 (YY1) as a critical regulator of oligodendrocyte progenitor differentiation.

231 l enlargement of the Olig2/Nkx2.2-expressing oligodendrocyte progenitor domain, whereas conditional H

232 so enhanced with precocious neurogenesis and oligodendrocyte progenitor elaboration.

233 Taken together, these data suggest that for oligodendrocyte progenitors, ErbB2 signaling plays a rol

234 hey remain in the cortex as undifferentiated oligodendrocyte progenitors for up to 3 weeks before mye

235 ptional network regulating the transition of oligodendrocyte progenitors from cell cycle exit to diff

236 ides a favorable environment for SVZ-derived oligodendrocyte progenitor generation.

237 regions of proneural tumors were enriched in oligodendrocyte progenitor genes, whereas the NE regions

238 We also determined that A2B5(+) oligodendrocyte progenitors grown in conditioned media d

239 which to accelerate differentiation of human oligodendrocyte progenitors (hOPCs) directly, we used CD

240 complex is insufficient for the induction of oligodendrocyte progenitors in developing spine; however

241 of neurons, and may regulate the function of oligodendrocyte progenitors, interneurons, GABA, and NMD

242 The last step of differentiation of oligodendrocyte progenitors into myelin-forming cells is

243 CNS laminin is to promote the development of oligodendrocyte progenitors into myelin-forming oligoden

244 is inhibited by impaired differentiation of oligodendrocyte progenitors into myelin-producing oligod

245 Although the oligodendrocyte progenitor is established as the major s

246 transition from multipotential precursors to oligodendrocyte progenitors is associated with the progr

247 genes is abolished, whereas the formation of oligodendrocyte progenitors is not affected.

248 Maturation-dependent vulnerability of late oligodendrocyte progenitors is thought to account for th

249 nd Distalless-related homeobox (DLX), or the oligodendrocyte progenitor marker Nkx2.2.

250 droitin sulfate proteoglycan-NG2(+) and late oligodendrocyte progenitor marker(+)), and terminal-diff

251 ssociation with myelin, without reexpressing oligodendrocyte progenitor markers or reentering the cel

252 ly rarely found to be immunoreactive against oligodendrocyte progenitor markers such as NG2 or PDGFRa

253 Myelin regeneration requires endogenous oligodendrocyte progenitor migration and activation of t

254 be a critical molecule in the regulation of oligodendrocyte progenitor migration and myelination.

255 ere analyzed to establish the time course of oligodendrocyte progenitor migration, proliferation, and

256 ransfer glycolytic substrates to neurons and oligodendrocyte progenitors (NG2(+) cells) exhibit enhan

257 re population has long been considered to be oligodendrocyte progenitors, numerous NG2(+) cells are p

258 nick end labeling and a marked depletion of oligodendrocyte progenitors of 71 +/- 8%.

259 ng regions overlapping with ventral sites of oligodendrocyte progenitor (OLP) generation.

260 Klf6 is rapidly induced in oligodendrocyte progenitors (OLP) by gp130 factors, and

261 In mouse neural tube, newly specified oligodendrocyte progenitors (OLPs) are first exposed to

262 Specification of oligodendrocyte progenitors (OLPs) begins early in devel

263 gated the relationship of Olig2+ and Nkx2.2+ oligodendrocyte progenitors (OLPs) by comparing the expr

264 the transcription factor OLIG2 and generate oligodendrocyte progenitors (OLPs) in culture.

265 The developmental origin of oligodendrocyte progenitors (OLPs) in the forebrain has

266 (TGFbeta) signaling is crucial for allowing oligodendrocyte progenitor (OP) cell cycle withdrawal, a

267 Proliferation of oligodendrocyte progenitor (OP) cells is a crucial proce

268 s provides a mechanism for such precision in oligodendrocyte progenitor (OP) proliferation.

269 Oligodendrocyte progenitors (OPCs) are electrically resp

270 ite matter progenitors respond to injury, as oligodendrocyte progenitors (OPCs) proliferate.

271 mice with sustained activation of ERK1/2 in oligodendrocyte progenitors (OPCs), oligodendrocytes, an

272 Oligodendrocyte progenitors (OPCs), which normally matur

273 nction between subcortical white matter NG2+ oligodendrocyte progenitors (OPs) and O4+ preoligodendro

274 ) expression and survival of differentiating oligodendrocyte progenitors (OPs) in proinflammatory cyt

275 ion of both postmitotic oligodendrocytes and oligodendrocyte progenitors (OPs) is the major hallmark

276 The abundance and widespread distribution of oligodendrocyte progenitors (OPs) within the adult CNS a

277 changes in the proliferation or survival of oligodendrocyte progenitor or mature cells.

278 NG2(+) glial cells (also called oligodendrocyte progenitors or polydendrocytes) also pro

279 rsely, overexpression of MRF within cultured oligodendrocyte progenitors or the chick spinal cord pro

280 emyelination is not limited by an absence of oligodendrocyte progenitors or their failure to generate

281 ncluding radial glia (GFAP+, vimentin+), and oligodendrocyte progenitors (PDGFR-alpha+) were prolifer

282 local proliferation and mobilization of the oligodendrocyte progenitor pool.

283 inating deficits despite an expansion of the oligodendrocyte progenitor pool.

284 ponses to acute death of premyelinating late oligodendrocyte progenitors (preOLs).

285 ndrocyte regeneration, possibly by enhancing oligodendrocyte progenitor proliferation and/or differen

286 In response to preOL death, early oligodendrocyte progenitors rapidly proliferate and diff

287 Oligodendrocyte progenitors respond to biophysical or me

288 In the developing chicken CNS, oligodendrocyte progenitors selectively express Nkx2.2 h

289 stration of rmTIMP-1 to A2B5(+) immunopanned oligodendrocyte progenitors significantly increased the

290 Unlike oligodendrocyte progenitors, SPNs displayed pronounced r

291 Therefore, axon-oligodendrocyte progenitor synapse formation is a transi

292 and LINC complex, mediate nuclear changes in oligodendrocyte progenitors that favor a default pathway

293 We show here that the ability of oligodendrocyte progenitors to acquire the identity of m

294 ates were found to promote the transition of oligodendrocyte progenitors to newly formed oligodendroc

295 , the presence of susceptible populations of oligodendrocyte progenitors underlies regional predilect

296 -N spinal cord, premature differentiation of oligodendrocyte progenitors was observed.

297 Importantly, Nf1-/- spinal cord-derived oligodendrocyte progenitors, which are amplified 12-fold

298 erivation and prospective isolation of human oligodendrocyte progenitors, which, upon transplantation

299 Transfection of oligodendrocyte progenitors with an MSP-green fluorescen

300 ta was also detected in NG2 cells (potential oligodendrocyte progenitors) within the white matter and