1 tected against testis infection, injury, and oligospermia.

2 nished testosterone and inhibin B levels and oligospermia.

3 spermia (OR 1.22, 95% CI 1.11-1.34), and for oligospermia (1.14, 1.04-1.25), but age at diagnosis and

4 rkably, several AR mutations associated with oligospermia and androgen insensitivity syndrome map to

5 nk2a2 has been disrupted are infertile, with oligospermia and globozoospermia ('round-headed' spermat

6  with infertility secondary to her partner's oligospermia and her chronic anovulation presented 13 da

7 permia, 8480 mg/m(2) (4264) in patients with oligospermia, and 6626 mg/m(2) (3576) in patients with n

8 overlap of CED associated with normospermia, oligospermia, and azoospermia.

9 ) progenitors had reduced testicular weight, oligospermia, and decreased sperm mobility.

10 rcentage of males with azoospermia or severe oligospermia, and its homology with a Drosophila male in

11  10% of males with idiopathic azoospermia or oligospermia have microdeletions in this region.

12 ome infertile men with azoospermia or severe oligospermia have small deletions in regions of the Y ch

13 a was noted in 53 (25%) of 214 participants, oligospermia in 59 (28%), and normospermia (sperm concen

14 that include testicular and tubular atrophy, oligospermia, Leydig cell hyperproliferation and increas

15 hould be suspected in an infertile male with oligospermia or azoospermia with low ejaculate volume, n

16            Odds ratios (ORs) and 95% CIs for oligospermia (sperm concentration >0 and <15 million per

17  the infertile men had azoospermia or severe oligospermia (sperm concentration, <5 million per millil

18 ration, <5 million per milliliter), four had oligospermia (sperm concentration, 5 million to <20 mill