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1 emented with nuts, or a control diet (advice on a low-fat diet).
2 overall survival (35 versus 63 days for mice on a folate-restricted diet).
3 els increased dramatically following 28 days on a LP diet.
4 a polysaccharide-rich standard diet but not on a PD diet.
5 own periodicity that was longer than animals on a chow diet.
6 ion of weight gain in mice on an HFD but not on a chow diet.
7 not reduced by the inflammation in the mice on a chow diet.
8 protein tolerance and lethal hyperammonemia on a chow diet.
9 es of glucose synthesis compared to athletes on a mixed diet.
10 ture, likely enables giant pandas to survive on a bamboo diet.
11 ts on HFD than in controls and diabetics fed on a normal diet.
12 umor-derived angiogenesis in mice maintained on a normal diet.
13 iver-specific PPARgamma null mice maintained on a normal diet.
14 low cytometry than did those of apoE-/- mice on a normal diet.
15 as comparable to that of wild-type (WT) mice on a normal diet.
16 D diet were also observed in Lrh-1(-/-) mice on a normal diet.
17 ciated with normal aging compared with those on a normal diet.
18 ckground, become obese on a high fat but not on a low fat diet.
19 significant larger decreases for TT carriers on a low-fat diet.
20 iption omega-3-acid ethyl esters in patients on a low-fat diet.
21 ffer cells is evident in eNos(-/-) mice even on a low-fat diet.
22 Mice lacking MGAT2 have a normal phenotype on a low-fat diet.
23 imals fed a high-salt diet compared to those on a regular diet.
24 assium ATPase (H,K-ATPase) compared to those on a regular diet.
25 dy weight, food intake or energy expenditure on a regular diet.
26 luconeogenic genes and hyperglycemia in mice on a regular diet.
27 PON3Tg/LDLR null mice than in LDLR null mice on a western diet.
28 ted in CD11d(-/-)/ApoE(-/-) mice after 16 wk on a Western diet.
29 severe hepatic steatosis following placement on a high fat diet.
30 reduced autophagy had worn off after 7 weeks on a high fat diet.
31 d body weight compared to the wild-type mice on a high fat diet.
32 s Bim and Puma had been ablated were studied on a high-fat diet.
33 produces significantly enhanced weight gain on a high-fat diet.
34 sensitivity and are resistant to weight gain on a high-fat diet.
35 d insulin resistance when animals are placed on a high-fat diet.
36 ultimately corresponding to less weight gain on a high-fat diet.
37 ate phenotype seen in heterozygous null mice on a high-fat diet.
38 ocytes, fatty livers, and insulin resistance on a high-fat diet.
39 TS-c potently decreasing weight gain in mice on a high-fat diet.
40 their susceptibility to obesity when placed on a high-fat diet.
41 on and control of body weight in mice placed on a high-fat diet.
42 bited absence of Pparg circadian rhythmicity on a high-fat diet.
43 atherosclerosis development in apoE-/- mice on a high-fat diet.
44 in our samples that increased in proportion on a high-fat diet.
45 therosclerosis development in ApoE(-/-) mice on a high-fat diet.
46 k for fatty liver disease in mice maintained on a high-fat diet.
47 weight gain in recipient wild-type mice fed on a high-fat diet.
48 tion imbalance, and reduced animal longevity on a high-fat diet.
49 ration was blunted in mice lacking PER1/2 or on a high-fat diet.
50 s in the aortic sinus following 10 and 16 wk on a high-fat diet.
51 tics of rats on a high-fat diet toward those on a standard diet.
52 taining 25% palm oil and compared with those on a standard diet.
53 eta expression in the macula densa increases on a high-salt diet.
54 mined after C75 injection in rats maintained on a ketogenic diet.
55 terns, hyperphagia, or excessive weight gain on a palatable diet.
56 activity that restricts insect reproduction on a synthetic diet.
57 , gain weight and accumulate excess body fat on a chronic HF diet.
58 ice to development of cholesterol gallstones on a lithogenic diet.
59 given to restore these bacteria to patients on a low FODMAP diet.
60 18-70 years who had coeliac disease and were on a gluten-free diet.
61 lities or their presenting clinical features on a gluten-free diet.
62 the vaccine in patients with coeliac disease on a gluten-free diet.
63 ogy at diagnosis and after at least one year on a gluten-free diet.
64 nes in the liver and serum, compared to mice on a normal chow diet.
65 ed with batf3(-/-) or wt bone marrow and put on a western type diet.
66 -density lipoprotein receptor-deficient mice on a Western-type diet.
67 iated enhanced inhibition of ENaC in animals on a high-potassium diet.
68 ccumulate excess cholesterol when maintained on a high-cholesterol diet.
69 reduction in plaque burden in Ldlr(-/-) mice on a high-cholesterol diet.
70 and effects on NAFLD, with special emphasis on a low-carbohydrate diet.
71 y ganglionic blockade or by weaning the mice on a carbohydrate-free diet.
72 gree to which subjects developed fatty liver on a choline-deficient diet.
73 or samples from subjects with celiac disease on a gluten-containing diet.
74 m and at 50% in liver TR1 of mice maintained on a selenium deficient diet.
75 effectors produced only females when raised on a limited tetracycline diet.
76 bserved an increase in survival time in mice on a glutamine restriction diet.
92 = 10) and healthy (n = 9) controls after 3 d on a eucaloric diet and again after 3 d on a diet of sim
94 mice also fail to develop hepatic steatosis on a high fat diet and manifest half-normal serum choles
95 Hig2 expression increased in livers of mice on a high-fat diet and during fasting, two states associ
96 ted Na(+) retention, elevated blood pressure on a high-Na(+) diet and increased surface expression of
98 /-), and CGD-sensitive C57L mice were placed on a lithogenic diet and then analyzed for CGD at the bi
101 0b) have less adipose tissue when maintained on a normal chow diet and are resistant to diet-induced
102 Also, we found that OP rats were hyperphagic on a regular chow diet and gained more weight compared t
103 - mice deficient in macrophage p38alpha MAPK on a Western diet and found that they had a marked incre
104 ensitivity], 10 subjects with celiac disease on a gluten-containing diet, and 52 presumed healthy ind
105 d substantially improved glucose homeostasis on a high-fat diet, and hyperinsulinemic-euglycemic clam
106 hat compared with wild-type and apoE-/- mice on a normal diet, apoE-/- mice on a Western high-fat die
107 and littermate LDLR(-/-) mice were initiated on a high-fat diet at 6 months of age, followed 1 week l
108 e-deficient (Apoe(-/-)) strains, was started on a Western diet at 6 weeks of age and maintained on th
109 ldosterone was measured in diabetic patients on a high-sodium diet, before and after angiotensin II s
114 undetectable in biopsy samples from patients on a gluten-free diet but expanded rapidly and specifica
115 sed coeliac patients that initially improved on a gluten-free diet but then relapsed despite a strict
116 ngth of jejunal villi and have normal growth on a normal diet but were less susceptible (P<0.01) to H
117 nockin mice had normal levels of hepatic fat on a chow diet, but when challenged with a high-sucrose
118 ats on a high-potassium diet than from those on a control diet, but this was not a result of altered
119 e8 is increased in human obesity and in mice on a high-fat diet, but its role in obesity is unknown.
120 tin degradation occurred also in uremic mice on a normal-phosphate diet, but they did not develop AMC
121 e show that SMRT(+/-) mice are normal weight on a regular diet, but develop increased adiposity on a
122 oth wild-type and RELMbeta KO mice were lean on a standard chow diet, but, upon switching to a high-f
123 carotid lesions were induced in FVB mice fed on a high-fat diet by streptozotocin injection followed
124 NMR profiling demonstrates that maintenance on a ketogenic diet causes a 25% reduction of myocardial
131 We showed that iNKT cell-deficient mice on a low-fat diet, considered a normal diet for mice, di
132 sponse to cocaine in rats that gained weight on a 'junk-food' diet, consistent with greater responsiv
134 which develop significant insulin resistance on a high-fat diet, despite no difference in adiposity.
135 Ksp-Cre mice rapidly developed hyperkalemia on a high-K+ diet, despite a 10-fold increase in serum a
139 demonstrated that Fyn knockout (FynKO) mice on a standard chow diet display increased glucose cleara
142 ation became obese (with increased fat mass) on a chow diet due to increased food intake and reduced
143 mt1(+/+) and Shmt1(-/-) dams were maintained on a standard AIN93G diet (Dyets), an AIN93G diet lackin
145 pancreatic insulin content, MIP-CreERT mice on a chow diet exhibited normal ambient glycemia, glucos
149 during short-term treatment, wild-type mice on a calorie-restricted diet experienced significant dec
150 mpaired glucose tolerance if their dams were on a high-fat diet, experienced prenatal stress, or both
152 s) were divided into groups that were placed on a normal chow diet, fasted for 24 hours, or fasted fo
155 salt homeostasis in male Wistar rats placed on a cholate-rich diet for 5 days and in cultured primar
156 ononuclear cells, healthy humans were placed on a controlled diet for 1 week, then given fish oil and
159 ockout mice and wild-type animals were place on a high-carbohydrate diet for 16 weeks, and mitochondr
162 r-deficient (Ldlr(-/-)) mice were maintained on a high-fat diet for 3 weeks followed by 6 weeks of or
163 Furthermore, FAT-D2KO animals that were kept on a high-fat diet for 8 weeks gained more body weight a
171 methionine- and choline-deficient diet, mice on a high-fat diet given streptozotocin, and mice with d
172 we previously reported that during refeeding on a low-fat diet, glucose tolerance is normal but insul
174 ts [BMI (kg/m(2)): 30.6 +/- 1.2] were placed on a hypercaloric diet (>1000 kcal simple carbohydrates/
176 ype mice, interleukin (IL) 10-deficient mice on a normal diet had decreased levels of colonic H2S syn
179 tant and consistent influence, larvae raised on a good-quality diet having substantially higher PO ac
180 Analysis of the effects after placing mice on a high fat diet (HFD) regimen demonstrated that runni
181 mpared with wild-type littermates (controls) on a high fat diet (HFD), Tg mice exhibited increased ad
182 -) mice gain more weight than wild-type mice on a high-fat diet (HFD) and that key liver and white ad
183 a in rats of both sexes that were maintained on a high-fat diet (HFD) for 6 weeks prior to DRG inflam
184 a group fed on the same diet or a group fed on a high-fat diet (HFD) rich in saturated fats for 3 we
187 ance, and insulin resistance when maintained on a high-fat diet (HFD), and were unable to maintain mu
191 is, KO mice gain significantly more fat mass on a high-fat diet (HFD), yet are surprisingly resistant
196 ent of TICs and tumorigenesis in mice placed on a Western diet high in cholesterol and saturated fat
197 ic hypertension relative to wild types (WTs) on a high-salt diet (HSD); this was attenuated by a PGI(
198 sulin sensitivity in these mice was improved on a standard chow diet in the absence of any weight dif
199 4 d postweaning in mice that were maintained on a normal diet (including increases in Proteobacteria
200 t's general condition improved substantially on a lactose-free diet, including hypercalcaemia, hyperc
202 tory receptor neurons, and long-term feeding on a camphor diet led to reversible downregulation of TR
203 d reduced adiposity, protection from obesity on a high-fat diet, low plasma lipid levels, and a neuro
208 d a large number of brain neurons affordable on a cooked diet may thus have been a major positive dri
209 lack of support for protein leverage effects on a low-protein diet may stem from the fact that protei
214 gh-fat diet (with or without fructose), mice on a Western-type diet, mice on a methionine- and cholin
215 nted by co-housing with offspring of mothers on a regular diet (MRD) and transferable to germ-free mi
218 he increased weight gain of SMRT(mRID1) mice on a high-fat diet occurs predominantly in fat with adip
219 ral administration of NPY in the Acb in rats on a free-choice diet of saturated fat, 30% sucrose solu
221 nse of females feeding for two and five days on a control diet or a diet containing either a low or a
224 type littermate control mice were maintained on a standard chow diet or subjected to 4 weeks of high-
225 EF, CON, and SUP rats were either maintained on a control diet, or the choline-supplemented diet.
226 lia of diabetic individuals, mice maintained on a high-fat diet, or cultured corneal epithelial cells
227 osis, col(V) sensitization of ApoE(-/-) mice on a regular chow diet overcame IL-10-mediated inhibitio
235 double knockouts) or placing Abcb4(-/-) mice on a high-fat diet protected against liver injury, with
237 e A(2b)AR ligand BAY 60-6853 in control mice on a high-fat diet reduced the lipid profile and atheros
238 dequate calcium intake is not achieved while on a nondairy diet, requiring investigation into optimal
239 When these animals were subsequently placed on a normal diet, resonance ratios reverted to those see
241 onstrate that mice lacking Irf5, when placed on a high-fat diet, show no difference in the growth of
243 in vitro and ex vivo results, Efnb1 KO mice on a high salt diet showed a statistically significant h
245 y been found to be altered in Npc1(-/-) mice on a high fat diet, showed related, but small, changes i
251 ShK-186 did not alter weight gain in mice on a chow diet, suggesting that the obesity-inducing die
252 POMC neurons became defective in obese mice on a high-fat diet, suggesting that loss of glucose sens
253 teatosis, insulin resistance, or dysglycemia on a sucrose-enriched diet, suggesting that elevated ins
256 he CaV1.1 pore gain more body weight and fat on a chow diet than control mice, without changes in foo
257 was lower in renal tissue obtained from rats on a high-potassium diet than from those on a control di
258 ediate maple syrup urine disease mice placed on a high protein diet that mimics the catabolic stress
259 al proximal and distal tubule transport, but on a low NaCl diet the increased RAAS activity prevents
262 ent of adipose tissue (AT) mass accumulation on a high-fat diet, the FynKO mice remained fully glucos
265 itive rats became hypertensive after 4 weeks on a high-NaCl diet, their plasma marinobufagenin levels
266 dult obese mice, 2 months after being placed on a high-fat diet, there was a striking increase of lep
269 a 4% diet, these mice have no phenotype, but on a 60% fat diet, they resist diet-induced obesity beca
270 IL-1alpha/beta-deficient bone marrow and fed on a high-cholesterol diet, they had markedly decreased
271 aner and shorter than the wild-type controls on a regular chow diet; they were also resistant to high
275 shifts the metabolic characteristics of rats on a high-fat diet toward those on a standard diet.
277 -density lipoprotein receptor-deficient mice on a Western-type diet using RNA sequencing and in silic
280 VitD3-replete diet (VitD(HI) mice) and mice on a VitD3-deficient diet (VitD(LO) mice), the inflammat
281 rculosis burdens did not differ between mice on a VitD3-replete diet (VitD(HI) mice) and mice on a Vi
282 say detected individuals with celiac disease on a gluten-containing diet vs controls with an area und
284 eatosis and insulin resistance in CBA/J mice on a sucrose-enriched diet was paralleled by increased h
285 that experienced prenatal stress and/or were on a high-fat diet weighed more beginning on postnatal d
287 y lipoprotein receptor-null (Ldlr(-/-)) mice on a high-fat diet were orally administered with vehicle
288 MMP14 haploinsufficient mice, animals placed on a high-fat diet were unable to remodel fat pad collag
290 nonobese, adult male C57BL/6 mice maintained on a normal chow diet were subjected to a microbiome dep
292 ive pediatric patients (18 years or younger) on a gluten-containing diet who tested positive for TGA-
293 nium into proteins and had the same lifespan on a chemically defined diet with or without selenium su
294 lues identified subjects with celiac disease on a gluten-containing diet with 100% sensitivity (95% C
295 ose levels in imprinting control region mice on a high fat diet with diet-induced obesity following s
296 ed cholesterol efflux, and injection of mice on a western-type diet with locked nucleic acid-antisens
297 (controls) and 9 mouse models of NAFLD: mice on a high-fat diet (with or without fructose), mice on a
299 atherosclerotic lesions in ApoE-p50-DKO mice on a normal chow diet without preexisting atherosclerosi
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