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1 ae in blood (lymphatic filariasis) and skin (onchocerciasis).
2 at promise as a simple tool for diagnosis of onchocerciasis.
3 omes many difficulties in identifying active onchocerciasis.
4 Here, we demonstrate NETs formation in human onchocerciasis.
5 rategy in some foci to control and eliminate onchocerciasis.
6 nt regimens against lymphatic filariasis and onchocerciasis.
7 es with a new program that aims to eliminate onchocerciasis.
8 been the drug of choice for the treatment of onchocerciasis.
9 Onchocerca volvulus, the causative agent of onchocerciasis.
10 mosum, which is an important vector of human onchocerciasis.
11 ilariasis, and skin disease and blindness in onchocerciasis.
12 88-2001), only 19.6% of cases were caused by onchocerciasis.
13 sms contribute to the pathogenesis of ocular onchocerciasis.
14 iod; 1283 (5.2%) of these deaths were due to onchocerciasis.
15 red during this period; 29.7% were caused by onchocerciasis.
16 tory reactions after ivermectin treatment of onchocerciasis.
17 to play a role in the development of ocular onchocerciasis.
18 le for antibody in the development of ocular onchocerciasis.
19 Africans who could have been coinfected with onchocerciasis.
20 lein-1 ELISA detected antibodies in 34 of 41 onchocerciasis (83%), 38 of 88 leishmaniasis (43%), 18 o
23 ion, 14 patients treated with ivermectin for onchocerciasis acquired >10 years ago during temporary r
25 available and emerging diagnostic tests for onchocerciasis and considers how they might be best empl
26 review the development of models concerning onchocerciasis and discuss the various approaches that h
27 ivermectin administration campaigns against onchocerciasis and lymphatic filariasis being conducted
31 matory responses seen following treatment of onchocerciasis and suggest new targets for modulating th
32 n Ov16 with serum samples from patients with onchocerciasis and with various types of control serum s
33 ontribute to the eye defects associated with onchocerciasis and, because there is no counterpart in m
34 a from parasitic (leishmaniasis, Chagas, and onchocerciasis), and infectious diseases (leprosy and So
35 d, and needed now, in the fight to eliminate onchocerciasis are new tools, such as preventive and the
40 utility of molecular xenomonitoring (MX) for onchocerciasis as elimination efforts expand into Africa
42 r filaricidal agents and/or vaccines against onchocerciasis based on immunological and rational hypot
43 s mortality has been noted among people with onchocerciasis, but it is not clear whether this effect
46 the absence of a gold standard, whereas the onchocerciasis case focuses on the identification of vil
47 ssociated adverse reactions in patients with onchocerciasis, changes in plasma tryptase levels and sk
49 doxycycline on a "test and treat" basis for onchocerciasis control and confirm doxycycline as a pote
55 and host mortality with data obtained by the Onchocerciasis Control Programme in West Africa from 231
56 amined, by use of data collected, during the Onchocerciasis Control Programme in western Africa (OCP)
58 implications for a noninvasive host-specific onchocerciasis diagnostic but provides a basis for the m
60 orporated into transmission models to inform onchocerciasis elimination efforts in Africa and residua
62 posttreatment sera from patients treated for onchocerciasis enhanced eosinophil survival; both GM-CSF
63 development of an effective vaccine against onchocerciasis has been the focus of a research program
64 vaccine candidates and drug targets against onchocerciasis has so far been confronted with several l
65 um damnosum s.l., the vector of West African onchocerciasis, has been the target of a major insect co
66 eveloped that include doxycycline to control onchocerciasis in areas where infections persist despite
67 ectin treatment for lymphatic filariasis and onchocerciasis in areas where L. loa infection is endemi
69 s was determined in a murine model of ocular onchocerciasis in which Ags from the parasitic worm Onch
70 ailable for five NTDs (lymphatic filariasis, onchocerciasis, intestinal helminthiasis, schistosomiasi
72 velopment of chemotherapeutic agents against onchocerciasis, lymphatic filariasis, and heartworm.
73 patients with loiasis, lymphatic filariasis, onchocerciasis, mansonellosis, or other helminthiases an
74 r regulatory submission for the treatment of onchocerciasis, might serve as an alternative to the wid
76 ty treatment strategy for the elimination of onchocerciasis or lymphatic filariasis has been delayed
81 ive in clearing microfilariae in the skin of onchocerciasis patients with persistent microfilaridermi
84 the rate of blindness from causes other than onchocerciasis remained approximately constant during fo
85 will support future basic and translational onchocerciasis research, with particular relevance for o
86 dentify potential vaccine candidates against onchocerciasis resulted in the cloning of recombinant pr
87 nematodes that cause lymphatic filariasis or onchocerciasis, resulting in blocked worm development an
88 ematodes, including lymphatic filariasis and onchocerciasis (river blindness) has transformed our app
89 gue) and CCR1 using a murine model of ocular onchocerciasis (river blindness) in which neutrophils an
91 bundant in the filarial nematodes that cause onchocerciasis (river blindness), including the larvae (
92 us as a natural model or 'analogue' of human onchocerciasis (river blindness), which is caused by Onc
94 ion of drugs to target lymphatic filariasis, onchocerciasis, schistosomiasis, soil-transmitted helmin
95 , schistosomiasis, lymphatic filariasis, and onchocerciasis, suggests that many of them could be cont
96 Since chemotherapy is widely used to treat onchocerciasis, the utility of PCR in assessing response
97 ed methods are needed for field diagnosis of onchocerciasis, to support efforts aimed at elimination
100 es-for example, for eye health (trachoma and onchocerciasis), ulcer care (leprosy), or renal support
101 summarizes the progress made to advance the onchocerciasis vaccine from the research laboratory into
103 without a history of control measures where onchocerciasis was endemic, microfilariae (MF) prevalenc
105 plains locale-specific infection patterns in onchocerciasis (whereas acquired protective immunity has
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