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1 clude Ras, Dbl family, and G-protein-coupled oncogene proteins.
2 st in their more celebrated cousins, the Ras oncogene proteins.
3 ents for the transforming actions of diverse oncogene proteins.
4 observed by overexpressing c-Myc (Myc proto-oncogene protein), a downstream target of Pim kinases.
7 l molecule and a disordered peptide from the oncogene protein c-Myc, we describe a "specific-diffuse"
10 transforming proteins and include Dbl family oncogene proteins, G protein-coupled receptors and G pro
12 ression of the HGF receptor, the c-met proto-oncogene protein, is uniformly found in the human bronch
13 ng (activation of v-akt murine thymoma viral oncogene/protein kinase B [AKT], inhibition of glycogen
14 on by forming a novel complex with two other oncogenes, protein kinase C, iota and epithelial cell tr
15 Due to their ability to function as dominant oncogenes, protein kinases have become favored targets i
16 the tumor suppressor protein p53, the viral oncogene protein pp60src, or a ubiquitin activating enzy
17 rs of transformation by a diverse variety of oncogene proteins that include Ras, Dbl family, and G-pr
20 il activator (ENA-78) and the growth-related oncogene proteins, was markedly suppressed in PMN from s
21 aps due to an association of the HER-2 proto-oncogene protein with resistance to hormone and/or chemo
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