ライフサイエンスコーパス: oncogenic mechanism

1 antiviral vaccines, and by identifying their oncogenic mechanism.

2 eloma cell lines, regardless of transforming oncogenic mechanism.

3 an thyroid cells, consistent with a dominant oncogenic mechanism.

4 s of heterozygosity remained the predominant oncogenic mechanism.

5 ion on certain Ser/Thr-Pro motifs is a major oncogenic mechanism.

6 hogenesis and highlight a new miRNA-mediated oncogenic mechanism.

7 recruit CBP/p300 as an important step of its oncogenic mechanism.

8 endent pathways which also contribute to the oncogenic mechanism.

9 mutant cancers as well as insight into their oncogenic mechanisms.

10 ficantly contributed to our understanding of oncogenic mechanisms.

11 ther these subtypes use similar or divergent oncogenic mechanisms.

12 vival than CM, is needed to delineate non-UV oncogenic mechanisms.

13 luster into subgroups suggestive of distinct oncogenic mechanisms.

14 nitiation is a key determinant of subsequent oncogenic mechanisms.

15 is study also indicates that one model of an oncogenic mechanism, activation of a proto-oncogene by t

16 However, the direct oncogenic mechanisms and downstream effector pathways im

17 Identifying methylated genes elucidates oncogenic mechanisms and provides valuable biomarkers; t

18 of tyrosine kinases represents an important oncogenic mechanism, and yet the majority of such events

19 g in differentiation stage (cell of origin), oncogenic mechanisms, and clinical outcome.

20 Potential TBC1D3 oncogenic mechanisms are discussed in light of these res

21 topic can lead to a better understanding of oncogenic mechanisms, as well as the discovery of novel

22 ectopic expression of WNT target genes - an oncogenic mechanism associated with mutations in Adenoma

23 irst step in the biochemical analysis of the oncogenic mechanism associated with translocation (6;9),

24 developments call for a re-evaluation of the oncogenic mechanisms behind PI3K pathway alterations.

25 of DLX5 can facilitate our understanding of oncogenic mechanisms driven by overexpression of DLX5.

26 ity at tumor-suppressor loci is an important oncogenic mechanism first discovered in retinoblastomas.

27 rant Notch signaling represents an important oncogenic mechanism for T cell acute lymphoblastic leuke

28 , which will facilitate investigation of the oncogenic mechanisms for this DNA virus.

29 Our studies thus uncover a novel oncogenic mechanism in Ewing sarcoma, involving post-tra

30 n of de novo DNA methylation may be a common oncogenic mechanism in leukemogenesis.

31 by polysomy 8 might represent an alternative oncogenic mechanism in lipoblastoma.

32 nal activation, which underlies the BRD4-NUT oncogenic mechanism in NMC.

33 The oncogenic mechanism in these fusion genes is akin to CDH

34 , since PIK3CA amplification is an important oncogenic mechanism in thyroid cancer, particularly FTC.

35 in bone, and our findings implicate a novel oncogenic mechanism in which deregulated USP6 transcript

36 Our findings reveal a novel oncogenic mechanism in which the chromosome 19 transloca

37 ear to be alternative and mutually exclusive oncogenic mechanisms in GISTs.

38 has greatly accelerated our understanding of oncogenic mechanisms in lymphomagenesis.

39 We now report a recurrent oncogenic mechanism, in IMTs, in which tropomyosin (TPM)

40 ibition to other tumor types driven by other oncogenic mechanisms is established.

41 Thus, Ack1 activated by surface signals or oncogenic mechanisms may directly enhance AR transcripti

42 Gaining further insights into PIK3CA oncogenic mechanisms may produce new biomarkers and help

43 ant implications on the understanding of the oncogenic mechanism of CRD-BP as well as in the future d

44 between wound healing and cancer and for the oncogenic mechanism of cyclin D.

45 lation of p100 processing may be part of the oncogenic mechanism of HTLV-I.

46 To decipher the presumed oncogenic mechanism of miR-125b, we used transplantation

47 inoma may have implications for studying the oncogenic mechanism of other BRD4-associated tumors.

48 muscle cells in medullomyoblastomas and the oncogenic mechanism of OTX2 as a repressor of diverse di

49 Although the oncogenic mechanism of TBC1D3 has yet to be elucidated,

50 cyclin A2 by RARalpha-PLZF may represent an oncogenic mechanism of this chimeric protein and contrib

51 rely on arguably incomplete knowledge of the oncogenic mechanisms of cyclin D1.

52 ned from these studies may shed light on the oncogenic mechanisms of other herpesviruses.

53 The oncogenic mechanisms of overnutrition, a confirmed indep

54 as their human counterparts with respect to oncogenic mechanisms of plasma cell transformation.

55 rogeneous properties and apparently distinct oncogenic mechanisms of these tumors.

56 there are important differences between the oncogenic mechanisms of these two viruses.

57 However, the oncogenic mechanisms of YAP activity are unclear.

58 carcinogenesis through induction of multiple oncogenic mechanisms, perhaps contributing to the poor p

59 To gain greater insight into oncogenic mechanisms regulated by TFAP2C, we examined ma

60 However, the MCPyV life cycle and its oncogenic mechanism remain poorly understood.

61 human hepatocellular carcinoma (HCC) but its oncogenic mechanisms remain unknown.

62 matological cancers; however, the underlying oncogenic mechanism remains elusive.

63 mutations in histone H3, yet the underlying oncogenic mechanism remains poorly characterized.

64 f Spry2 and c-Met signaling may be a pivotal oncogenic mechanism responsible for unrestrained activat

65 We describe a constitutive Akt1 oncogenic mechanism resulting from interaction of overex

66 These findings indicate that other oncogenic mechanism(s) may be involved in the developmen

67 that integration may represent an additional oncogenic mechanism through direct effects on cancer-rel

68 ring development, providing insight into the oncogenic mechanisms through which NF1 loss contributes

69 eir downstream signaling pathways is a major oncogenic mechanism underlying hematopoietic malignancie

70 Our studies shed light on the oncogenic mechanism underlying t(15;19) and provide furt

71 To investigate the underlying oncogenic mechanisms, we have cross bred transgenic stra

72 Several new and unexpected oncogenic mechanisms were suggested by the pattern of so

73 ese data imply that CoAA may share a similar oncogenic mechanism with oncogene EWS and that CoAA dere