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1 ells were killed as a result of necrosis and oncosis.
2 form of stress-activated cell death known as oncosis.
3 ent apoptosis, while higher CPE doses induce oncosis.
4 and that morphologically resembles cellular oncosis.
5 lable to the cells induced cell death due to oncosis.
6 ent Shigella infection of HMDM and is termed oncosis.
7 apoptosis, others induce cell starvation, or oncosis.
8 death, apoptosis, autophagy, pyroptosis, and oncosis.
9 optosis but is similar to that described for oncosis, a form of cell death accompanied by the membran
10 c relevance, these results suggest that both oncosis and apoptosis may occur in the intestines during
12 This strain caused rapid host cell death by oncosis, and few bacterial CFU were recovered after incu
13 ath differentially mediated by apoptosis and oncosis effectors, with IVIg protecting target cells by
14 n this study for demonstrating apoptosis and oncosis have pathophysiologic relevance, these results s
20 d morphologically by cell shrinkage, whereas oncosis is a form of cell death induced by energy deplet
22 r these conditions neurons rapidly underwent oncosis (necrosis) with a complete loss in viability occ
24 e expression, membrane damage, apoptosis and oncosis of living sensing organisms can provide insight
25 sis, the sensitivity of spectral analysis to oncosis or ischemic cell death had not previously been s
28 antibodies, designated anti-Porimin (for pro-oncosis receptor inducing membrane injury), was found to
29 crosis is depicted by a gain in cell volume (oncosis), swelling of organelles, plasma membrane leakag
31 osis was associated with PARP-1 cleavage and oncosis was associated with a rapid decline in intracell
32 by reperfusion leads to necrotic cell death (oncosis), which often occurs within minutes of reperfusi
33 edominant form of cell death in all lines is oncosis, which is driven by the loss of plasma membrane
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