ライフサイエンスコーパス: oncotic pressure

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1 effects of (in)Abeta depended on changes in oncotic pressure.

2 icantly faster in groups 2 to 4, and colloid oncotic pressure after resuscitation was greater in grou

3 This suggests that plasma oncotic pressure alone may be a more important determina

4 We tested the hypothesis that plasma oncotic pressure alone, not the plasma-to-lymph oncotic

5 tic pressure (piGC) from knowledge of plasma oncotic pressure and the filtration fraction revealed th

6 ratio, plasma protein concentration, plasma oncotic pressure, and myocardial contractility.

7 Total solids, colloid oncotic pressure, arterial oxygen content, Hb, lactate,

8 e to hemodilution caused by its high colloid oncotic pressure, but may facilitate diffusive oxygen tr

9 vestigate this further, Lp and the effective oncotic pressure difference (f3DeltaPi) acting across th

10 Thus, the effective oncotic pressure difference (sigma black triangle down p

11 hese results support our hypothesis that the oncotic pressure difference across the tumor microvascul

12 otic pressure alone, not the plasma-to-lymph oncotic pressure difference, modulates pulmonary transva

13 ar fluid filtration than the plasma-to-lymph oncotic pressure difference.

14 c pressures were estimated as the sum of the oncotic pressure due to HA alone plus the oncotic pressu

15 ficients to HA and A, there was an effective oncotic pressure (E pi) of between 3.46 and 6.0 cm H2O o

16 Oncotic pressure, effective at inducing VDAC closure, al

17 ncentration and the decreased transcapillary oncotic pressure gradient.

18 rol animals, even though the plasma-to-lymph oncotic pressure gradients were equal.

19 Starling's equation indicates that reduced oncotic pressure gradients will favor edema formation, a

20 4.5 and 17.7 +/- 2.2 mm Hg; plasma-to-lymph oncotic pressure gradients, respectively, were 4.4 +/- 0

21 Whereas oncotic pressure in blood plasma of various species is k

22 um total protein is the chief determinant of oncotic pressure in humans.

23 ecies is known, no data are available on the oncotic pressure in the interstitial space of tumors.

24 ecause of the leaky nature of tumor vessels, oncotic pressure in tumor interstitium should be close t

25 The high oncotic pressure in tumors is consistent with the elevat

26 aised left atrial pressure elevation, plasma oncotic pressures in dextran and control sheep, respecti

27 We found interstitial oncotic pressures in four human tumor xenografts to be h

28 ic wick method for the direct measurement of oncotic pressures in the interstitial fluid of tumors gr

29 Neither the plasma oncotic pressure nor the mean arterial pressure differed

30 he oncotic pressure due to HA alone plus the oncotic pressure of albumin (A) in an HA matrix.

31 ross the endothelial glycocalyx and that the oncotic pressure of interstitial fluid does not directly

32 cromolecules was assessed from the effective oncotic pressure (omega delta pi) exerted by the perfusa

33 as determined, blood was sampled, and plasma oncotic pressure (pi(A)) was measured.

34 Computation of glomerular intracapillary oncotic pressure (piGC) from knowledge of plasma oncotic

35 min (HSA) serves not only as a physiological oncotic pressure regulator and a ligand carrier but also

36 ntial contribution of medullary interstitial oncotic pressure to the net balance of forces influencin

37 ents in serum albumin, immunoglobulin G, and oncotic pressure values were 31, 32, and 26%, respective

38 glomerular filtration during HPP, perfusate oncotic pressure was reduced by lowering the concentrati

39 Total interstitial oncotic pressures were estimated as the sum of the oncot

40 ack triangle down pi was near 70% of luminal oncotic pressure when the tissue concentration equalled

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