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1 ac1 signaling results in abnormal cerebellar ontogenesis.
2 changes in size and shape of the body during ontogenesis.
3 e parallel but independent programs of human ontogenesis.
4 a functional role of Notch signaling in LCH ontogenesis.
5 ost fundamental processes of higher organism ontogenesis.
6 ithin plant mesophyll cells during haustoria ontogenesis.
7 determination during early embryogenesis and ontogenesis.
8 ized that PPARdelta might regulate epidermal ontogenesis.
9 dicating only a transient delay in epidermal ontogenesis.
10 del systems for the earliest stages of human ontogenesis.
11 ere progressively increased during epidermal ontogenesis.
12 ed by incorporating knowledge about atypical ontogenesis.
13 CSTase and SSase activities during in vitro ontogenesis.
18 oupled CXCR4 regulates cell migration during ontogenesis and disease states including cancer and infl
19 a key survival pathway during normal B-cell ontogenesis and in a subset of diffuse large B-cell lymp
20 tor expression is critical for physiological ontogenesis and its expression is restricted postnatally
21 12 plays an essential role during epidermal ontogenesis and normal hair follicle cycling and that it
22 n vivo under physiological conditions (e.g., ontogenesis and pregnancy) and in pathology (allergic an
24 e hand, on the anamnestic retention of their ontogenesis and, on the other, on the emergence of novel
25 mouse somatic growth and maintenance during ontogenesis, and IGF-I pathway is likely to be a target
27 ors accelerate not only permeability barrier ontogenesis, but also the expression of structural prote
28 eceptor family regulate permeability barrier ontogenesis by stimulating lipid metabolism and the form
29 Moreover, hormones that accelerate barrier ontogenesis (e.g. glucocorticoids, thyroid hormone, and
31 was to develop an in vitro model of barrier ontogenesis in order to identify those factors critical
32 cholesterol sulfate cycle enzymes during SC ontogenesis is a component of the fetal epidermal differ
39 tion of crucial neurotrophic pathways during ontogenesis, leading to aberrant parasympathetic innerva
40 able as a valuable resource for studying the ontogenesis, morphogenesis and function of the mammalian
43 evelopment, we examined the distribution and ontogenesis of expression of prolactin receptors (PRLRs)
44 ly understandings provide a platform for the ontogenesis of further, deeper achievements in the human
47 ry component may therefore contribute to the ontogenesis of hypertension in the pre-hypertensive SH r
51 t a direct or indirect role for PROP1 in the ontogenesis of pituitary gonadotropes, as well as somato
54 CSTase and SSase activities during in vitro ontogenesis precisely mirrored those obtained in utero.
57 effects of air exposure on epidermal barrier ontogenesis, we compared the activities of several key e
58 her these enzymes are induced during barrier ontogenesis, we examined their activity in epidermis of
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