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1 ies with either synchronous and asynchronous oogenesis.
2 a on the size of the mtDNA bottleneck during oogenesis.
3 norhabditis elegans germline development and oogenesis.
4 Drosophila SAGA are differently required in oogenesis.
5 eraction in photoreceptor axon targeting and oogenesis.
6 xes Iml1/GATOR1 and GATOR2 during Drosophila oogenesis.
7 a loss-of-function mouse model for STAG3 in oogenesis.
8 al mRNAs and proteins that accumulate during oogenesis.
9 and the latter has yet to be established in oogenesis.
10 cells called border cells during Drosophila oogenesis.
11 ion between Spir and Capu is required during oogenesis.
12 dynein-mediated processes during Drosophila oogenesis.
13 ssed by, the Matrimony (Mtrm) protein during oogenesis.
14 ential for establishing an actin mesh during oogenesis.
15 licle cell differentiation during Drosophila oogenesis.
16 of key components of Notch signaling during oogenesis.
17 pmental gene amplification during Drosophila oogenesis.
18 tant targets of EGFR signaling in Drosophila oogenesis.
19 licle cells into the amplification stages of oogenesis.
20 ally occurs during either spermatogenesis or oogenesis.
21 ed genes enhance apoptosis during C. elegans oogenesis.
22 r development during Drosophila melanogaster oogenesis.
23 ls to adopt the sperm fate at the expense of oogenesis.
24 oposed to repress expression of genes during oogenesis.
25 the diversification of BMP signaling during oogenesis.
26 tyltransferase with an essential function in oogenesis.
27 he promotion of membrane organization during oogenesis.
28 anterior localization of bicoid mRNA in late oogenesis.
29 enorhabditis briggsae, GLD-1 acts to promote oogenesis.
30 changes are not responsible for the block in oogenesis.
31 seh1 has a crucial germline function during oogenesis.
32 membrane biogenesis and organization during oogenesis.
33 er called the chromocenter during Drosophila oogenesis.
34 dy axes are established in the oocyte during oogenesis.
35 tructural nucleoporin Seh1 during Drosophila oogenesis.
36 CP190, these proteins are not essential for oogenesis.
37 nges in ovarian physiology that occur during oogenesis.
38 s reveal critical functions for this gene in oogenesis.
39 ecause they also function at other stages of oogenesis.
40 ffective at silencing gene expression during oogenesis.
41 assembly of an actin mesh during Drosophila oogenesis.
42 ngation from a sphere to an ellipsoid during oogenesis.
43 owed the analysis of baz function throughout oogenesis.
44 ed mechanism that controls key events during oogenesis.
45 erm-associated genes during normal postnatal oogenesis.
46 D-2-binding partner, GLD-3, to ensure normal oogenesis.
47 the cytoplasm of cells undergoing IC during oogenesis.
48 the posteriorly localized germ plasm during oogenesis.
49 e maternal provision of Nv-Hb protein during oogenesis.
50 m for activation of cell death in Drosophila oogenesis.
51 FMRP controls germline proliferation during oogenesis.
52 aintain their localized distributions during oogenesis.
53 ermatogenesis, whereas GLD-2/RNP-8 specifies oogenesis.
54 ent and follicle formation during Drosophila oogenesis.
55 n and microtubule dynamics during Drosophila oogenesis.
56 and Peg3, which all become methylated during oogenesis.
57 rmation can be identified in D. melanogaster oogenesis.
58 blishing the DNA methylation imprints during oogenesis.
59 eal places that these actin regulators shape oogenesis.
60 ly from chromosome segregation errors during oogenesis.
61 ith spermatogenesis and transitions later to oogenesis.
62 esis of the Drosophila follicle cells during oogenesis.
63 sterior cortex of the oocyte from stage 7 of oogenesis.
64 hase and genomic stability during Drosophila oogenesis.
65 KDM1B had no effect on mouse development and oogenesis.
66 GFR and BMP signaling pathways in Drosophila oogenesis.
67 ual sites of Stl metalloprotease function in oogenesis.
68 iotic initiation in both spermatogenesis and oogenesis.
69 lication and mitochondrial biogenesis during oogenesis.
70 to regulate ovarian germline stem cells and oogenesis.
71 mutational mechanisms in spermatogenesis and oogenesis.
72 clear actin must be tightly regulated during oogenesis.
73 erning to the posterior of the oocyte during oogenesis.
74 of germ-line-derived nurse cells during late oogenesis.
75 targets and influencing transcription during oogenesis.
76 recocious transition from spermatogenesis to oogenesis.
77 are maintained at a constant diameter before oogenesis.
78 jire as a cofactor of Notch signaling during oogenesis.
79 distribution of 5862 mRNAs during Drosophila oogenesis.
80 tegrating egg chambers, indicating defective oogenesis.
81 the tight suppression of transposons during oogenesis.
82 iptome assembly at different stages of mouse oogenesis.
83 poral restriction of actin remodeling during oogenesis.
84 erm cells for their survival past stage 5 of oogenesis.
85 cell nuclei and germinal vesicle during mid-oogenesis.
86 ry assimilated from the maternal diet during oogenesis.
87 on by Egg is required for multiple stages of oogenesis.
88 First, KHC is temporally regulated during oogenesis.
89 that H3K9 methylation by Egg is required for oogenesis.
90 ferentiation, or other processes involved in oogenesis.
91 d microtubule organization during Drosophila oogenesis.
92 ty in D. sechellia due to maternal arrest of oogenesis.
93 amounts of excess histones generated during oogenesis [9], and the maternal supplies of core histone
99 iched with Me(199)Hg, spanning the period of oogenesis, allowing us to differentiate between mercury
100 trapment-mediated localization of nos during oogenesis, also function in da neurons for formation and
101 omotes translation of Cyclin A, beginning in oogenesis, an earlier onset than previously recognized.
102 hey are ineffective in gene knockdown during oogenesis, an important model system for the study of ma
103 t to an interplay between unique features of oogenesis and a host of endogenous and exogenous factors
104 hylation of the DMRs of Peg3 and Xist during oogenesis and also in the maintenance of unmethylation s
105 tion allows females to assess the demands of oogenesis and alter their behavior and metabolic state t
106 re depend on centrosome positioning early in oogenesis and are independent of anterior-posterior axis
107 the animal-vegetal axis is determined during oogenesis and at ovulation, the egg is radially symmetri
109 -184 leads to multiple severe defects during oogenesis and early embryogenesis, culminating in the co
113 ffect genes, these factors accumulate during oogenesis and enable the activation of the embryonic gen
114 is important for dorso-ventral patterning in oogenesis and for anterior-posterior pattern formation d
115 in is critical for normal endocytosis during oogenesis and for embryogenesis in the sea star and that
116 erized the metabolic mechanisms that support oogenesis and found that mitochondria in mature Drosophi
117 wth factor-like signaling in regulating both oogenesis and immune system activity, and propose a sign
120 s to extensive glycogen accumulation late in oogenesis and is required for the developmental competen
121 teroplasmic flies was decreased, both during oogenesis and over multiple generations, at the restrict
122 effects of H3.3R26 and H3.3K27 in modulating oogenesis and partitioning cells to the inner cell mass
123 tand transcriptional regulation during human oogenesis and preimplantation development, we defined st
124 ese tightly regulated processes begin during oogenesis and proceed through gastrulation to establish
125 f oskar localization that occurs during late oogenesis and results in amplification of the germ plasm
126 , and females are sterile, showing disrupted oogenesis and severe defects in follicle cell differenti
127 ophila female germ line epigenome throughout oogenesis and show that the oocyte has a unique, dynamic
128 e dot chromosome during early embryogenesis, oogenesis and spermatogenesis resemble that of the curre
129 1 function interferes with the completion of oogenesis and spermatogenesis through sexually dimorphic
130 e mutants are sterile, displaying defects in oogenesis and spermatogenesis, and analysis of DNA synth
133 ss expression of maternal transcripts during oogenesis and suggest that interplay between OMA-1 and o
134 ified between ventral gene expression during oogenesis and the activation of the protease cascade in
135 in germ cells throughout spermatogenesis and oogenesis and thus, focused further efforts on this fami
136 nimals, indole induces genes associated with oogenesis and, accordingly, extends fecundity and reprod
139 ce indicates that production of new oocytes (oogenesis) and their enclosure by somatic cells (follicu
140 maternal mRNAs, Zar2 was present throughout oogenesis, and endogenous Zar2 co-immunoprecipitated end
141 pecific DNA hypermethylation acquired during oogenesis, and its expression silencing was reversible o
142 versification of BMP signaling in Drosophila oogenesis, and they provide insight into a mechanism und
143 The piRNA pathway is essential in early oogenesis, and we find that nuclear localization of Zfrp
147 cells differentiated early during Drosophila oogenesis--are arrested at G2 phase and can serve as a m
151 the disordered character of transport at mid-oogenesis, as revealed by streaming, is an important com
152 s of germ cell-specific markers and in vitro oogenesis, as well as the use of intraovarian transplant
153 g wdr-5.1/wdr-5.2 function fail to switch to oogenesis at 25 degrees C, resulting in a masculinizatio
154 66 proteins likely act uniquely during late oogenesis, because they are up-regulated at maturation a
155 uration are factors needed not only for late oogenesis but also completion of meiosis and early embry
156 fic SEX-LETHAL (SXL) protein is required for oogenesis, but how Sxl interfaces with the genetic circu
157 that distinguish various stages of wild-type oogenesis, but that developing egg chambers fail to migr
158 blished along the animal-vegetal axis during oogenesis, but the underlying molecular mechanisms are p
161 ion of the mechanical stress associated with oogenesis by conferring stability and elasticity to germ
162 regulate actin remodeling during Drosophila oogenesis by controlling Ena localization/activity, such
163 nterior-posterior axis is established during oogenesis by the localization of bicoid and oskar mRNAs
167 silencing of MISO abolishes the increase in oogenesis caused by mating in blood-fed females, causes
170 nctions of three individual H3.3 residues in oogenesis, cleavage-stage embryogenesis and early develo
175 ages in spermatogenesis and then switches to oogenesis during late stages of larval development.
177 emale reproductive structure and the site of oogenesis, fertilization, and maturation of the embryo a
178 A replication during Drosophila melanogaster oogenesis, finding that mtDNA replication commenced befo
180 the EcR-B1 isoform is required during early oogenesis for follicle cell survival and that disruption
185 short hairpin RNAs (shRNAs) effective during oogenesis has provided an alternative to producing germl
187 to-nuclear incompatibility during Drosophila oogenesis has severe consequences for egg production and
188 to the vegetal cortex during Xenopus laevis oogenesis have been reported to function in germ layer p
191 Additionally, preoogenesis Hg and during-oogenesis Hg were transferred proportionally to eggs, su
192 anism of centriole elimination during female oogenesis, highlighting a protective role for Polo kinas
194 arian follicle maturation through studies of oogenesis in both vertebrate and invertebrate systems, l
196 for cell-autonomous roles in development and oogenesis in Drosophila, but the function of its evoluti
203 d to adversely affect two distinct stages of oogenesis in the developing ovary: the events of prophas
204 for early development is established during oogenesis in the form of the maternal transcriptome.
206 to drive meiotic prophase I progression and oogenesis; in the absence of food, the resultant inactiv
207 pensable for the early MT-dependent steps of oogenesis, including cell division, and that dTBCB is no
208 ndent developmental events during Drosophila oogenesis, indicating ligand-independent Notch activity
211 follicular epithelium (FE) around stage 6 of oogenesis is essential for entry into the endocycle and
212 ulation of gene expression during Drosophila oogenesis is essential for patterning the anterior-poste
213 g of the follicular epithelium in Drosophila oogenesis is required for the formation of three-dimensi
218 t traverses the Drosophila oocyte during mid-oogenesis, is essential for proper establishment of the
219 A is sequestered at the anterior pole during oogenesis, is not translated until fertilization, and pr
220 teroid signaling regulates multiple steps in oogenesis, it is not known whether it regulates Drosophi
221 y stages are determined by the female during oogenesis, it is unknown whether reproducibility is perp
228 Understanding the unique mechanisms of human oogenesis necessitates the development of an in vitro sy
231 these mutations, and compare the effects on oogenesis of both strong loss-of-function and weak hypom
232 RNA system exist: somatic cells that support oogenesis only employ Piwi, whereas germ cells utilize a
234 e Drosophila oocyte anterior from stage 9 of oogenesis onwards to provide a local source for Bicoid p
238 demonstrates that vha-12 is not required for oogenesis or spermatogenesis in the adult germ line, but
243 dicating that lack of PARP-1 function during oogenesis predisposes the female gamete to genome instab
244 y exploiting the asymmetric cell division of oogenesis--present a potent selective pressure favoring
245 enzyme is a broad-spectrum regulator of the oogenesis program that acts within an RNA regulatory net
251 uently, failure of oskar localization during oogenesis results in embryos lacking germ cells and abdo
255 Analysis of bcd mRNA during late stages of oogenesis suggested a model for steady-state bcd localiz
258 Our findings link two universal features of oogenesis, the Bb and the chromosomal bouquet, to oocyte
259 ation from stage 9 to stage 10 in Drosophila oogenesis, the egg chamber increases in length by approx
264 egulates expression of maternal mRNAs during oogenesis, the oocyte to embryo transition, and early em
267 follicle cells switch into endocycles during oogenesis they repress the apoptotic response to DNA dam
268 eld haplo-X oocytes, during XX hermaphrodite oogenesis they segregate to the first polar body to yiel
269 ndogenous gene during Caenorhabditis elegans oogenesis; this process is referred to as germ-line cosu
270 cumulates at the oocyte posterior during mid-oogenesis through a well-studied process involving kines
271 turbation of the oocyte's epigenome in early oogenesis, through depletion of the dKDM5 histone demeth
275 ates and inhibits Dicer during meiosis I for oogenesis to proceed normally in Caenorhabditis elegans
276 trate that bucky ball functions during early oogenesis to regulate polarity of the oocyte, future egg
278 owing the canonical pattern during XX female oogenesis to yield haplo-X oocytes, during XX hermaphrod
280 ese hubs were analyzed further in Drosophila oogenesis, using targeted germline RNAi, and adhesion wa
281 ation are enriched in the germ plasm at late oogenesis via a diffusion and entrapment mechanism, the
282 female 20E-interacting protein that promotes oogenesis via mechanisms also favoring Plasmodium surviv
283 g, are dictated by the sex of the germ line (oogenesis vs. spermatogenesis) in Caenorhabditis elegans
285 ional analysis of the tail during Drosophila oogenesis we have gained an understanding of how KHC ach
289 le cell polarity both during early stages of oogenesis, when follicle cells undergo the mitotic cell
290 echanism for such coordination in Drosophila oogenesis, when the expression of the transcription fact
291 restrict disulfide bond formation until late oogenesis, when the oocyte no longer experiences large v
292 lly wild-type fly, it is insufficient during oogenesis, where Brk must utilize CtBP and 3R to pattern
293 ical structures of embryos originates during oogenesis, which is when the expression of maternally pr
294 usually produced during spermatogenesis and oogenesis, which take place in the testis and the ovary,
295 tone acetyltransferase (HAT) activity blocks oogenesis, while loss of the H2B deubiquitinase (DUB) ac
296 6 mutation was selectively eliminated during oogenesis within four generations, whereas the milder CO
299 late sufficient nutrient reserves to support oogenesis without the requirement for a blood meal.
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