ライフサイエンスコーパス: oogenesis

1 ies with either synchronous and asynchronous oogenesis.

2 a on the size of the mtDNA bottleneck during oogenesis.

3 norhabditis elegans germline development and oogenesis.

4 Drosophila SAGA are differently required in oogenesis.

5 eraction in photoreceptor axon targeting and oogenesis.

6 xes Iml1/GATOR1 and GATOR2 during Drosophila oogenesis.

7 a loss-of-function mouse model for STAG3 in oogenesis.

8 al mRNAs and proteins that accumulate during oogenesis.

9 and the latter has yet to be established in oogenesis.

10 cells called border cells during Drosophila oogenesis.

11 ion between Spir and Capu is required during oogenesis.

12 dynein-mediated processes during Drosophila oogenesis.

13 ssed by, the Matrimony (Mtrm) protein during oogenesis.

14 ential for establishing an actin mesh during oogenesis.

15 licle cell differentiation during Drosophila oogenesis.

16 of key components of Notch signaling during oogenesis.

17 pmental gene amplification during Drosophila oogenesis.

18 tant targets of EGFR signaling in Drosophila oogenesis.

19 licle cells into the amplification stages of oogenesis.

20 ally occurs during either spermatogenesis or oogenesis.

21 ed genes enhance apoptosis during C. elegans oogenesis.

22 r development during Drosophila melanogaster oogenesis.

23 ls to adopt the sperm fate at the expense of oogenesis.

24 oposed to repress expression of genes during oogenesis.

25 the diversification of BMP signaling during oogenesis.

26 tyltransferase with an essential function in oogenesis.

27 he promotion of membrane organization during oogenesis.

28 anterior localization of bicoid mRNA in late oogenesis.

29 enorhabditis briggsae, GLD-1 acts to promote oogenesis.

30 changes are not responsible for the block in oogenesis.

31 seh1 has a crucial germline function during oogenesis.

32 membrane biogenesis and organization during oogenesis.

33 er called the chromocenter during Drosophila oogenesis.

34 dy axes are established in the oocyte during oogenesis.

35 tructural nucleoporin Seh1 during Drosophila oogenesis.

36 CP190, these proteins are not essential for oogenesis.

37 nges in ovarian physiology that occur during oogenesis.

38 s reveal critical functions for this gene in oogenesis.

39 ecause they also function at other stages of oogenesis.

40 ffective at silencing gene expression during oogenesis.

41 assembly of an actin mesh during Drosophila oogenesis.

42 ngation from a sphere to an ellipsoid during oogenesis.

43 owed the analysis of baz function throughout oogenesis.

44 ed mechanism that controls key events during oogenesis.

45 erm-associated genes during normal postnatal oogenesis.

46 D-2-binding partner, GLD-3, to ensure normal oogenesis.

47 the cytoplasm of cells undergoing IC during oogenesis.

48 the posteriorly localized germ plasm during oogenesis.

49 e maternal provision of Nv-Hb protein during oogenesis.

50 m for activation of cell death in Drosophila oogenesis.

51 FMRP controls germline proliferation during oogenesis.

52 aintain their localized distributions during oogenesis.

53 ermatogenesis, whereas GLD-2/RNP-8 specifies oogenesis.

54 ent and follicle formation during Drosophila oogenesis.

55 n and microtubule dynamics during Drosophila oogenesis.

56 and Peg3, which all become methylated during oogenesis.

57 rmation can be identified in D. melanogaster oogenesis.

58 blishing the DNA methylation imprints during oogenesis.

59 eal places that these actin regulators shape oogenesis.

60 ly from chromosome segregation errors during oogenesis.

61 ith spermatogenesis and transitions later to oogenesis.

62 esis of the Drosophila follicle cells during oogenesis.

63 sterior cortex of the oocyte from stage 7 of oogenesis.

64 hase and genomic stability during Drosophila oogenesis.

65 KDM1B had no effect on mouse development and oogenesis.

66 GFR and BMP signaling pathways in Drosophila oogenesis.

67 ual sites of Stl metalloprotease function in oogenesis.

68 iotic initiation in both spermatogenesis and oogenesis.

69 lication and mitochondrial biogenesis during oogenesis.

70 to regulate ovarian germline stem cells and oogenesis.

71 mutational mechanisms in spermatogenesis and oogenesis.

72 clear actin must be tightly regulated during oogenesis.

73 erning to the posterior of the oocyte during oogenesis.

74 of germ-line-derived nurse cells during late oogenesis.

75 targets and influencing transcription during oogenesis.

76 recocious transition from spermatogenesis to oogenesis.

77 are maintained at a constant diameter before oogenesis.

78 jire as a cofactor of Notch signaling during oogenesis.

79 distribution of 5862 mRNAs during Drosophila oogenesis.

80 tegrating egg chambers, indicating defective oogenesis.

81 the tight suppression of transposons during oogenesis.

82 iptome assembly at different stages of mouse oogenesis.

83 poral restriction of actin remodeling during oogenesis.

84 erm cells for their survival past stage 5 of oogenesis.

85 cell nuclei and germinal vesicle during mid-oogenesis.

86 ry assimilated from the maternal diet during oogenesis.

87 on by Egg is required for multiple stages of oogenesis.

88 First, KHC is temporally regulated during oogenesis.

89 that H3K9 methylation by Egg is required for oogenesis.

90 ferentiation, or other processes involved in oogenesis.

91 d microtubule organization during Drosophila oogenesis.

92 ty in D. sechellia due to maternal arrest of oogenesis.

93 amounts of excess histones generated during oogenesis [9], and the maternal supplies of core histone

94 During oogenesis a maternal factor is localized to the vegetal

95 During Drosophila melanogaster oogenesis, a germline stem cell divides forming a cyst o

96 During Drosophila oogenesis, a gradient of EGFR activation patterns the fo

97 During Drosophila oogenesis, a prominent metabolic checkpoint occurs at th

98 During Drosophila oogenesis, activation of Notch signaling in the follicul

99 iched with Me(199)Hg, spanning the period of oogenesis, allowing us to differentiate between mercury

100 trapment-mediated localization of nos during oogenesis, also function in da neurons for formation and

101 omotes translation of Cyclin A, beginning in oogenesis, an earlier onset than previously recognized.

102 hey are ineffective in gene knockdown during oogenesis, an important model system for the study of ma

103 t to an interplay between unique features of oogenesis and a host of endogenous and exogenous factors

104 hylation of the DMRs of Peg3 and Xist during oogenesis and also in the maintenance of unmethylation s

105 tion allows females to assess the demands of oogenesis and alter their behavior and metabolic state t

106 re depend on centrosome positioning early in oogenesis and are independent of anterior-posterior axis

107 the animal-vegetal axis is determined during oogenesis and at ovulation, the egg is radially symmetri

108 n the FMR1 gene that appears to occur during oogenesis and during early embryogenesis.

109 -184 leads to multiple severe defects during oogenesis and early embryogenesis, culminating in the co

110 rather than asymmetrically, localized during oogenesis and early embryogenesis.

111 xpression in the posterior germ plasm during oogenesis and early embryogenesis.

112 s of transcription in heterochromatin during oogenesis and early embryonic development.

113 ffect genes, these factors accumulate during oogenesis and enable the activation of the embryonic gen

114 is important for dorso-ventral patterning in oogenesis and for anterior-posterior pattern formation d

115 in is critical for normal endocytosis during oogenesis and for embryogenesis in the sea star and that

116 erized the metabolic mechanisms that support oogenesis and found that mitochondria in mature Drosophi

117 wth factor-like signaling in regulating both oogenesis and immune system activity, and propose a sign

118 Centrioles are lost during oogenesis and inherited from the sperm at fertilization.

119 nd nucleoplasmin methylation appears late in oogenesis and is most abundant in the laid egg.

120 s to extensive glycogen accumulation late in oogenesis and is required for the developmental competen

121 teroplasmic flies was decreased, both during oogenesis and over multiple generations, at the restrict

122 effects of H3.3R26 and H3.3K27 in modulating oogenesis and partitioning cells to the inner cell mass

123 tand transcriptional regulation during human oogenesis and preimplantation development, we defined st

124 ese tightly regulated processes begin during oogenesis and proceed through gastrulation to establish

125 f oskar localization that occurs during late oogenesis and results in amplification of the germ plasm

126 , and females are sterile, showing disrupted oogenesis and severe defects in follicle cell differenti

127 ophila female germ line epigenome throughout oogenesis and show that the oocyte has a unique, dynamic

128 e dot chromosome during early embryogenesis, oogenesis and spermatogenesis resemble that of the curre

129 1 function interferes with the completion of oogenesis and spermatogenesis through sexually dimorphic

130 e mutants are sterile, displaying defects in oogenesis and spermatogenesis, and analysis of DNA synth

131 d migration--to the sex-specific pathways of oogenesis and spermatogenesis.

132 tabolism with the precursor l-DOPA, resuming oogenesis and stimulating egg production.

133 ss expression of maternal transcripts during oogenesis and suggest that interplay between OMA-1 and o

134 ified between ventral gene expression during oogenesis and the activation of the protease cascade in

135 in germ cells throughout spermatogenesis and oogenesis and thus, focused further efforts on this fami

136 nimals, indole induces genes associated with oogenesis and, accordingly, extends fecundity and reprod

137 t may play important regulatory roles during oogenesis and/or early embryogenesis.

138 ght to determine whether ASUN plays roles in oogenesis and/or embryogenesis.

139 ce indicates that production of new oocytes (oogenesis) and their enclosure by somatic cells (follicu

140 maternal mRNAs, Zar2 was present throughout oogenesis, and endogenous Zar2 co-immunoprecipitated end

141 pecific DNA hypermethylation acquired during oogenesis, and its expression silencing was reversible o

142 versification of BMP signaling in Drosophila oogenesis, and they provide insight into a mechanism und

143 The piRNA pathway is essential in early oogenesis, and we find that nuclear localization of Zfrp

144 Oogenesis appeared to be more sensitive compared to sper

145 RNAs localized to the vegetal cortex during oogenesis are known to be essential.

146 female tissues and the maternal diet during oogenesis are significant sources of Hg.

147 cells differentiated early during Drosophila oogenesis--are arrested at G2 phase and can serve as a m

148 Using Drosophila oogenesis as a model system, we show that mutations in s

149 Here we use Drosophila oogenesis as an in vivo model system to determine the ex

150 migration of border cells during Drosophila oogenesis as our model system.

151 the disordered character of transport at mid-oogenesis, as revealed by streaming, is an important com

152 s of germ cell-specific markers and in vitro oogenesis, as well as the use of intraovarian transplant

153 g wdr-5.1/wdr-5.2 function fail to switch to oogenesis at 25 degrees C, resulting in a masculinizatio

154 66 proteins likely act uniquely during late oogenesis, because they are up-regulated at maturation a

155 uration are factors needed not only for late oogenesis but also completion of meiosis and early embry

156 fic SEX-LETHAL (SXL) protein is required for oogenesis, but how Sxl interfaces with the genetic circu

157 that distinguish various stages of wild-type oogenesis, but that developing egg chambers fail to migr

158 blished along the animal-vegetal axis during oogenesis, but the underlying molecular mechanisms are p

159 osophila is protected from DNA damage during oogenesis by a mechanism involving short RNAs.

160 ed appears to be negatively regulated during oogenesis by Abelson kinase.

161 ion of the mechanical stress associated with oogenesis by conferring stability and elasticity to germ

162 regulate actin remodeling during Drosophila oogenesis by controlling Ena localization/activity, such

163 nterior-posterior axis is established during oogenesis by the localization of bicoid and oskar mRNAs

164 Starting in mid-oogenesis, Cad74A is expressed in the follicle cells tha

165 Gro insufficiency during oogenesis can be explained by its downregulation in Brk-

166 Stage 10 of Drosophila oogenesis can be subdivided into stages 10A and 10B base

167 silencing of MISO abolishes the increase in oogenesis caused by mating in blood-fed females, causes

168 the absence of dTBCB during later stages of oogenesis causes major defects in cell polarity.

169 In contrast, during oogenesis, CGH-1 forms patr-1-independent mRNA storage b

170 nctions of three individual H3.3 residues in oogenesis, cleavage-stage embryogenesis and early develo

171 ation and in physiological demand control of oogenesis, coordinating these processes.

172 During Caenorhabditis elegans oogenesis, cytoplasmic RNPs can transition among diffuse

173 During Drosophila oogenesis diverse cell behaviors are seen in the soma an

174 In mouse oogenesis, DNA methylation establishment occurs on a lar

175 ages in spermatogenesis and then switches to oogenesis during late stages of larval development.

176 During oogenesis, EGG-4 and EGG-5 assemble at the oocyte cortex

177 emale reproductive structure and the site of oogenesis, fertilization, and maturation of the embryo a

178 A replication during Drosophila melanogaster oogenesis, finding that mtDNA replication commenced befo

179 During Drosophila oogenesis, follicle cells sequentially undergo three dis

180 the EcR-B1 isoform is required during early oogenesis for follicle cell survival and that disruption

181 During oogenesis, germline expression of I-2 results in the acc

182 In oogenesis, graded JAK activity stimulated by Upd specifi

183 of egg-chamber elongation during Drosophila oogenesis has always been mysterious.

184 Although Drosophila oogenesis has been intensively studied, the genetic and

185 short hairpin RNAs (shRNAs) effective during oogenesis has provided an alternative to producing germl

186 In particular, research on Drosophila oogenesis has provided critical insights into piRNA biog

187 to-nuclear incompatibility during Drosophila oogenesis has severe consequences for egg production and

188 to the vegetal cortex during Xenopus laevis oogenesis have been reported to function in germ layer p

189 ents for the SMC5/6 complex during mammalian oogenesis have not previously been examined.

190 The spermatogenesis/oogenesis helix-loop-helix (SOHLH) proteins SOHLH1 and S

191 Additionally, preoogenesis Hg and during-oogenesis Hg were transferred proportionally to eggs, su

192 anism of centriole elimination during female oogenesis, highlighting a protective role for Polo kinas

193 During oogenesis, hundreds of maternal RNAs are selectively loc

194 arian follicle maturation through studies of oogenesis in both vertebrate and invertebrate systems, l

195 Met and SRC regulate both vitellogenesis and oogenesis in C. lectularius.

196 for cell-autonomous roles in development and oogenesis in Drosophila, but the function of its evoluti

197 maintenance and egg chamber polarity during oogenesis in Drosophila.

198 es of Kinesin-dependent streaming during mid-oogenesis in Drosophila.

199 d, how filamin mechanosensing contributes to oogenesis in Drosophila.

200 P107-associated protein SEH1 is required for oogenesis in Drosophila.

201 ed the dysregulation of proteins involved in oogenesis in female livers.

202 During oogenesis in feminized animals (fem-3), a single pair of

203 d to adversely affect two distinct stages of oogenesis in the developing ovary: the events of prophas

204 for early development is established during oogenesis in the form of the maternal transcriptome.

205 We previously established Drosophila oogenesis, in particular nurse cell dumping, as a new mo

206 to drive meiotic prophase I progression and oogenesis; in the absence of food, the resultant inactiv

207 pensable for the early MT-dependent steps of oogenesis, including cell division, and that dTBCB is no

208 ndent developmental events during Drosophila oogenesis, indicating ligand-independent Notch activity

209 eaks (ttk(twk)) reduces TTK69 levels late in oogenesis, inhibiting this expansion.

210 Drosophila melanogaster oogenesis is an ideal system for studying IC.

211 follicular epithelium (FE) around stage 6 of oogenesis is essential for entry into the endocycle and

212 ulation of gene expression during Drosophila oogenesis is essential for patterning the anterior-poste

213 g of the follicular epithelium in Drosophila oogenesis is required for the formation of three-dimensi

214 o downregulation of the RAS-ERK pathway, and oogenesis is stalled.

215 er, the function of Ago1 in other aspects of oogenesis is still elusive.

216 Oogenesis is the process by which ovarian germ cells und

217 orm germline cysts, but the role of cysts in oogenesis is unknown.

218 t traverses the Drosophila oocyte during mid-oogenesis, is essential for proper establishment of the

219 A is sequestered at the anterior pole during oogenesis, is not translated until fertilization, and pr

220 teroid signaling regulates multiple steps in oogenesis, it is not known whether it regulates Drosophi

221 y stages are determined by the female during oogenesis, it is unknown whether reproducibility is perp

222 TTK69 is required again later in oogenesis: it controls the volume of the dorsal-appendag

223 letal organization and egg morphology during oogenesis, leading to female sterility.

224 developmental progression through meiosis in oogenesis, little is known about Cyclin A.

225 During oogenesis, mammalian eggs accumulate proteins required f

226 on and a female Mating-Induced Stimulator of Oogenesis (MISO) protein.

227 We show that, during oogenesis, mitochondria accumulate at the oocyte posteri

228 Understanding the unique mechanisms of human oogenesis necessitates the development of an in vitro sy

229 Critical events of oogenesis occur during three distinct developmental stag

230 Neo-oogenesis occurred early after transplantation, as evide

231 these mutations, and compare the effects on oogenesis of both strong loss-of-function and weak hypom

232 RNA system exist: somatic cells that support oogenesis only employ Piwi, whereas germ cells utilize a

233 females direct nutritional resources towards oogenesis only when inseminated.

234 e Drosophila oocyte anterior from stage 9 of oogenesis onwards to provide a local source for Bicoid p

235 During oogenesis, oocytes are arrested in prophase and resume m

236 ne-derived nurse cells during Drosophila mid-oogenesis or follicle development.

237 whether reproducibility is perpetuated from oogenesis or reacquired by the zygote.

238 demonstrates that vha-12 is not required for oogenesis or spermatogenesis in the adult germ line, but

239 female, or male) and type of gametogenesis (oogenesis or spermatogenesis).

240 tal life as they commit to, and prepare for, oogenesis or spermatogenesis.

241 fetal ovary or testis and hence to embark on oogenesis or spermatogenesis.

242 ng cbl gene dosage by half rescues the dFmr1 oogenesis phenotypes.

243 dicating that lack of PARP-1 function during oogenesis predisposes the female gamete to genome instab

244 y exploiting the asymmetric cell division of oogenesis--present a potent selective pressure favoring

245 enzyme is a broad-spectrum regulator of the oogenesis program that acts within an RNA regulatory net

246 Drosophila oogenesis provides a developmental system with which to

247 During Drosophila oogenesis, RCs connect the maturing oocyte to nurse cell

248 faces with the genetic circuitry controlling oogenesis remains unknown.

249 d lead necessarily toward spermatogenesis or oogenesis, respectively.

250 However, YY1 KD during oogenesis resulted in the loss of DNA methylation on Peg

251 uently, failure of oskar localization during oogenesis results in embryos lacking germ cells and abdo

252 At a specific time in oogenesis, somatic follicle cells transition from genomi

253 ive repertoire of CSR-1 small RNAs targeting oogenesis-specific mRNAs.

254 During Drosophila melanogaster oogenesis, spindle assembly occurs without centrosomes a

255 Analysis of bcd mRNA during late stages of oogenesis suggested a model for steady-state bcd localiz

256 pression in all cells during early stages of oogenesis that diminishes in later stages.

257 Therefore, in oogenesis, the activities of Upd and Upd3 both appear to

258 Our findings link two universal features of oogenesis, the Bb and the chromosomal bouquet, to oocyte

259 ation from stage 9 to stage 10 in Drosophila oogenesis, the egg chamber increases in length by approx

260 During stages 8 to 10 of oogenesis, the FC epithelium transitions between simple

261 During Drosophila oogenesis, the follicular epithelial cells undergo two m

262 Later in oogenesis, the GATOR2 components Mio and Seh1 are requir

263 Later in oogenesis, the gradient of EGFR activation is split into

264 egulates expression of maternal mRNAs during oogenesis, the oocyte to embryo transition, and early em

265 During oogenesis, the roles of patterning and morphogenesis by

266 We demonstrate that during Drosophila oogenesis, the two-domain expression pattern of Broad, a

267 follicle cells switch into endocycles during oogenesis they repress the apoptotic response to DNA dam

268 eld haplo-X oocytes, during XX hermaphrodite oogenesis they segregate to the first polar body to yiel

269 ndogenous gene during Caenorhabditis elegans oogenesis; this process is referred to as germ-line cosu

270 cumulates at the oocyte posterior during mid-oogenesis through a well-studied process involving kines

271 turbation of the oocyte's epigenome in early oogenesis, through depletion of the dKDM5 histone demeth

272 Mechanisms that couple later steps of oogenesis to environmental conditions remain largely und

273 We used oogenesis to explore developmental roles of two importan

274 dysferlin RNA and protein are expressed from oogenesis to gastrulation.

275 ates and inhibits Dicer during meiosis I for oogenesis to proceed normally in Caenorhabditis elegans

276 trate that bucky ball functions during early oogenesis to regulate polarity of the oocyte, future egg

277 Continued YY1 KD from oogenesis to the blastocyst stage caused further loss in

278 owing the canonical pattern during XX female oogenesis to yield haplo-X oocytes, during XX hermaphrod

279 During oogenesis, TspanC8 genes were up-regulated in border cel

280 ese hubs were analyzed further in Drosophila oogenesis, using targeted germline RNAi, and adhesion wa

281 ation are enriched in the germ plasm at late oogenesis via a diffusion and entrapment mechanism, the

282 female 20E-interacting protein that promotes oogenesis via mechanisms also favoring Plasmodium surviv

283 g, are dictated by the sex of the germ line (oogenesis vs. spermatogenesis) in Caenorhabditis elegans

284 We show that during C. elegans oogenesis WAPL-1 antagonizes binding of cohesin containi

285 ional analysis of the tail during Drosophila oogenesis we have gained an understanding of how KHC ach

286 During C. elegans oogenesis, we find that two groups of PUF RNA binding pr

287 Whereas Setd1a plays no role in oogenesis, we report that Setd1b deficiency causes femal

288 Because effects on both stages of oogenesis were elicited using doses that yield circulati

289 le cell polarity both during early stages of oogenesis, when follicle cells undergo the mitotic cell

290 echanism for such coordination in Drosophila oogenesis, when the expression of the transcription fact

291 restrict disulfide bond formation until late oogenesis, when the oocyte no longer experiences large v

292 lly wild-type fly, it is insufficient during oogenesis, where Brk must utilize CtBP and 3R to pattern

293 ical structures of embryos originates during oogenesis, which is when the expression of maternally pr

294 usually produced during spermatogenesis and oogenesis, which take place in the testis and the ovary,

295 tone acetyltransferase (HAT) activity blocks oogenesis, while loss of the H2B deubiquitinase (DUB) ac

296 6 mutation was selectively eliminated during oogenesis within four generations, whereas the milder CO

297 mo, and thus are sufficient to sustain adult oogenesis without a source of renewal.

298 that the core Brahma complex can function in oogenesis without any of these accessory subunits.

299 late sufficient nutrient reserves to support oogenesis without the requirement for a blood meal.

300 a number of biological processes, including oogenesis, wound healing, and cytokinesis.