ライフサイエンスコーパス: oomycete

1 cialized intercellular structures (fungi and oomycetes).

2 in the protection against the fungus and the oomycete.

3 oactivity against plant pathogenic fungi and oomycetes.

4 independently in plant pathogenic fungi and oomycetes.

5 , protects plants against diseases caused by oomycetes.

6 ced by plant pathogenic bacteria, fungi, and oomycetes.

7 infection structures of pathogenic fungi and oomycetes.

8 production and bioactivity against fungi and oomycetes.

9 ted for RxLR-effectors from plant pathogenic oomycetes.

10 ttern of cross-kingdom HGT between fungi and oomycetes.

11 to pathogen Phytophthora infestans and other oomycetes.

12 s is not the case for several subfamilies in oomycetes.

13 t the fungal kingdom, and in the fungus-like oomycetes.

14 ved in the perception of bacteria, fungi and oomycetes.

15 e encoded by the genomes of plant pathogenic oomycetes.

16 e population structure within these obligate oomycetes.

17 or delivery are uncharacterized in fungi and oomycetes.

18 e are shared with plant-associated fungi and oomycetes.

19 ies and catalogues the effector secretome of oomycetes.

20 in avirulence proteins from three different oomycetes.

21 t appear to be widespread and diverse in the oomycetes.

22 from the same or closely related species of oomycetes.

23 , SWEETs had not been identified in fungi or oomycetes.

24 or future P450 annotations in newly explored oomycetes.

25 and 31 P450 subfamilies were newly found in oomycetes.

26 ee hundred and fifty-six P450s were found in oomycetes.

27 homology data revealed P450 family blooms in oomycetes.

28 ution of phytopathogenic traits in fungi and oomycetes.

29 gen-associated molecular patterns (PAMPs) in oomycetes.

30 hogen of potato and a model organism for the oomycetes, a distinct lineage of fungus-like eukaryotes

31 diseases of plants and animals are caused by oomycetes, a group of eukaryotic pathogens important to

32 Oomycetes accomplish parasitic colonization of plants by

33 ve secondary metabolites, including the anti-oomycete and antifungal haterumalide, oocydin A and the

34 for the biosynthesis of the antifungal, anti-oomycete and antitumor haterumalide, oocydin A (ooc).

35 Oomycete and fungal effectors with RXLR and RXLR-like mo

36 Effector proteins secreted by oomycete and fungal pathogens have been inferred to ente

37 is to unrelated (hemi)biotrophic filamentous oomycete and fungal pathogens.

38 Many oomycete and fungal plant pathogens are obligate biotrop

39 Bacterial, oomycete and fungal plant pathogens establish disease by

40 nal variants of the RXLR motif, and that the oomycete and fungal RXLR motifs enable binding to the ph

41 Oomycete and fungal symbionts have significant impacts o

42 ant species in resistance against bacterial, oomycete and viral pathogens.

43 o mutualism) with their hosts include fungi, oomycetes and actinomycete bacteria.

44 her than the so-called Crinkler effectors of oomycetes and fungi, these effectors are encoded by othe

45 onal secreted proteins from plant pathogenic oomycetes and its similarity to a host-targeting signal

46 last 150 yr between plant pathogens (fungi, oomycetes and plasmodiophorids) and vascular plants.

47 d nematodes), common host-targeting signals (oomycetes and protozoans) and specialized intercellular

48 d economical impact of the animal pathogenic oomycetes and review the recent advances in this emergin

49 he understanding of the relationship between oomycetes and their host plants.

50 The resource is focused on fungi, protists (oomycetes) and bacterial plant pathogens that have genom

51 stance by challenging EP plants with fungal, oomycete, and bacterial pathogens and an insect pest.

52 ease resistance of plants against bacterial, oomycete, and fungal pathogens and has a unique mode of

53 d by other plant resistance genes and virus, oomycete, and nematode effectors and for host susceptibi

54 olite not previously shown to be produced by oomycetes, and two proteins with homology to vertebrate

55 bium meliloti as well as with the pathogenic oomycete Aphanomyces euteiches.

56 Oomycetes are a phylogenetically distinct group of organ

57 sequences similar to those seen in fungi and oomycetes are also found in the animal kingdom, but rath

58 Oomycetes are fungal-like eukaryotic microbes in the kin

59 in bacteria, equivalent systems in fungi and oomycetes are poorly understood.

60 Oomycetes are responsible for multi-billion dollar damag

61 able to identify known sequences present in oomycetes as well as identify novel sequences.

62 e, we report the identification of SWEETs in oomycetes as well as SWEETs and a potential SemiSWEET in

63 toplasm, consistent with the hypothesis that oomycetes, as is the case with bacteria and fungi, activ

64 keleton confers resistance against fungi and oomycetes, AtADF4 is not involved in resistance against

65 ggests a molecular "arms race" as plants and oomycetes attempt to achieve and evade detection, respec

66 ily of 700 proteins with similarity to known oomycete avirulence genes.

67 Similar to other oomycete avirulence proteins, AVR3aKI carries a signal p

68 Recent characterization of four oomycete Avr genes revealed that they encode effector pr

69 defense responses that are effective against oomycete, bacterial and viral pathogens, pointing to a c

70 are consistent with the hypothesis that some oomycetes became successful plant parasites by multiple

71 Research revealed that oomycetes belonging to different orders contain distinct

72 nes resistance to downy mildew caused by the oomycete Bremia lactucae carrying the cognate avirulence

73 icals to control plant and animal pathogenic oomycetes cannot be used anymore; due to resistance in t

74 The three examined plant pathogenic oomycetes carry complex and diverse sets of RXLR effecto

75 Oomycetes cause devastating plant diseases of global imp

76 Many species of oomycetes cause economic and environmental damage owing

77 Phytopathogenic oomycetes cause some of the most devastating diseases af

78 Oomycete cell wall carbohydrates were recognized by fish

79 T has played a role in shaping how fungi and oomycetes colonize plant hosts.

80 ranches of life (ascomycete, eubacteria, and oomycete) converge onto the Arabidopsis TCP14 transcript

81 gests that prostaglandins may be involved in oomycete development.

82 s, we outline some of the reasons fungal and oomycete diseases cause such significant losses to tropi

83 ing effective long-term control measures for oomycete diseases.

84 ast 10 Dm genes conferring resistance to the oomycete downy mildew fungus Bremia lactucae map to the

85 We study the oomycete downy mildew pathogen of Arabidopsis, Hyalopero

86 amily includes all experimentally identified oomycete effector and avirulence genes, and its rapid pa

87 Recently, four oomycete effector genes have been isolated, and several

88 Recently, the first oomycete effector genes with cultivar-specific avirulenc

89 ted programmed cell death upon bacterial and oomycete effector recognition as well as decreased resis

90 Here the latest findings on oomycete effector secretion, delivery and function are d

91 For several oomycete effectors (i.e., the RxLR-effectors) it has bee

92 Oomycete effectors identified to date contain a targetin

93 lusion that RXLR and dEER serve to transduce oomycete effectors into host cells indicates that the >3

94 ctions of the very large number of predicted oomycete effectors that contain them.

95 at protein belonging to an ancient family of oomycete effectors that rapidly evolves to escape host d

96 displays similarity to the WY domain core in oomycete effectors.

97 otifs, RXLR and EER, present in translocated oomycete effectors.

98 idly moving toward genome-wide catalogues of oomycete effectors.

99 tionally interchangeable with RxLR motifs of oomycete effectors.

100 ponse induced by non-host flagellins and the oomycete elicitor INF1.

101 kthroughs in live-cell imaging of fungal and oomycete encounter sites, including live-cell imaging of

102 ng in several distinct lineages of fungal or oomycete-feeding nematodes.

103 The oomycetes form a phylogenetically distinct group of euka

104 The oomycetes form one of several lineages within the eukary

105 ubset of these genes is conserved in related oomycetes from the Phytophthora genus.

106 mato, potato and barley to viral, bacterial, oomycete, fungal and nematode infections.

107 yotic proteins, which include effectors from oomycetes, fungi and other parasites.

108 ve major pathogen groups (viruses, bacteria, oomycetes, fungi, and nematodes), has contributed to our

109 ety of eukaryotic plant pathogens, including oomycetes, fungi, and nematodes.

110 din biosynthesis have been identified in the oomycete genome.

111 Oomycete genomes display a strongly bipartite organizati

112 The high level of synteny between these oomycete genomes extends to the ABC superfamily, where 1

113 fector genes have been isolated, and several oomycete genomes have been sequenced.

114 tative effectors have now been identified in oomycete genomes, the sequences of which show evidence o

115 e of Phytophthora infestans, a member of the oomycete group of fungus-like microbes and the cause of

116 Although research on plant pathogenic oomycetes has flourished in recent years, the animal pat

117 ished in recent years, the animal pathogenic oomycetes have received less attention.

118 uring the compatible interaction between the oomycete Hyaloperonospora arabidopsidis (Hpa) and its ho

119 Here, we demonstrate that the biotrophic oomycete Hyaloperonospora arabidopsidis (Hpa) exhibits a

120 Here, we report the genome sequence of the oomycete Hyaloperonospora arabidopsidis (Hpa), an obliga

121 the tandem WY-domain effector ATR1 from the oomycete Hyaloperonospora arabidopsidis through direct a

122 eudomonas syringae pv tomato, the biotrophic oomycete Hyaloperonospora arabidopsidis, and the necrotr

123 domonas syringae pv. tomato DC3000 or to the oomycete Hyaloperonospora arabidopsidis.

124 basal defense against RKN as well as to the oomycete Hyaloperonospora arabidopsidis.

125 in response to infections by the pathogenic oomycete Hyaloperonospora parasitica.

126 conditions and treatment with the pathogenic oomycete, Hyaloperonospora parasitica, wild type had a h

127 he presence or absence of the mycelia of the oomycetes in both shaking and static conditions.

128 only form of extracellular SOD in fungi and oomycetes, in stark contrast to the extracellular Cu/Zn-

129 The eukaryotic microbes called oomycetes include many important saprophytes and pathoge

130 The genus Aphanomyces (Saprolegniales, Oomycetes) includes species with a variety of ecologies

131 When plant-pathogenic oomycetes infect their hosts, they employ a large arsena

132 tants were more susceptible and resistant to oomycete infection, respectively, showing that the inten

133 mportantly, for restriction of bacterial and oomycete infections.

134 Current research is helping us learn how oomycetes interact with host and environment, understand

135 We suggest that bacteria/oomycete interactions should be considered not only in t

136 tion of RxLR effectors from plant pathogenic oomycetes into the cytoplasm of their host is currently

137 nt that the effector secretome of pathogenic oomycetes is more complex than expected, with perhaps se

138 largest group of translocated proteins from oomycetes is the RxLR effectors, defined by their conser

139 TIR-NBS-LRR R genes specifying resistance to oomycetes, is dependent on a functional EDS1 allele for

140 te having limited secondary metabolism, many oomycetes make chemicals for communicating within their

141 unparalleled opportunities to determine how oomycetes manipulate hosts to establish infection.

142 Understanding oomycete metabolism is fundamental to understanding thes

143 ss effective, there is a need for studies on oomycete metabolism to help identify promising and more

144 biotic gene transfer events have diversified oomycete metabolism, resulting in biochemical pathways t

145 le progress has been made in identifying the oomycete molecules that trigger them.

146 act as ecological amplifiers for fungal and oomycete mycelial networks in soils, extending their pot

147 ngdom as well as in phylogenetically distant oomycetes or "pseudofungi" species.

148 The eukaryotic oomycetes, or water molds, contain several species that

149 Oomycete P450 patterns suggested host influence in shapi

150 that is distinct from that triggered by the oomycete PAMP INF1.

151 ogens Pseudomonas syringae pv tomato and the oomycete parasite Peronospora parasitica, bos1 exhibits

152 syringae pv. tomato DC3000(avrRpm1) and the oomycete parasite Peronospora parasitica.

153 o their response to a bacterial pathogen and oomycete parasite.

154 otein RAC1 that determines resistance to the oomycete pathogen Albugo candida.

155 se can inhibit germination of spores of this oomycete pathogen and inhibit tobacco leaf infection by

156 that the effector protein HaRxL106 from the oomycete pathogen Hyaloperonospora arabidopsidis co-opts

157 nced disease resistance against the virulent oomycete pathogen Hyaloperonospora arabidopsidis Noco2,

158 e toward a virulent strain of the biotrophic oomycete pathogen Hyaloperonospora arabidopsidis Noco2.

159 d plants was also more resistant against the oomycete pathogen Hyaloperonospora arabidopsidis.

160 terial pathogen Pseudomonas syringae and the oomycete pathogen Hyaloperonospora arabidopsidis.

161 terial pathogen Pseudomonas syringae and the oomycete pathogen Hyaloperonospora arabidopsidis.

162 Hyaloperonospora parasitica is a native oomycete pathogen of Arabidopsis and is related to other

163 R1(NdWsB) in Hyaloperonospora parasitica, an oomycete pathogen of Arabidopsis.

164 ctor protein Avr1b of Phytophthora sojae, an oomycete pathogen of soybean (Glycine max), we show that

165 lar hybrid strains of Phytophthora sojae, an oomycete pathogen of soybean, high frequency mitotic gen

166 ce and are compromised for resistance to the oomycete pathogen Peronospora parasitica in mutants with

167 sistance to a highly virulent isolate of the oomycete pathogen Peronospora parasitica.

168 mination and blue mold disease caused by the oomycete pathogen Peronospora tabacina.

169 spore germination and leaf infection by the oomycete pathogen Peronospora tabacina.

170 um) nuclear proteome during infection by the oomycete pathogen Phytophthora capsici.

171 Late blight, caused by the oomycete pathogen Phytophthora infestans, is the most de

172 The oomycete pathogen Phytophthora infestans, the agent of t

173 causes increased susceptibility only to the oomycete pathogen Phytophthora infestans.

174 Resistance of soybean against the oomycete pathogen Phytophthora sojae is conferred by a s

175 ble interactions of soybean tissues with the oomycete pathogen Phytophthora sojae or the bacterial pa

176 esponse to inoculation with the downy mildew oomycete pathogen Sclerospora graminicola.

177 ellular proteins in Phytophthora and Pythium oomycete pathogen species.

178 RPP8 confers resistance to an oomycete pathogen, Peronospora parasitica.

179 The oomycete pathogen, therefore, is under selection pressur

180 but also show defects in colonization by an oomycete pathogen, with the absence of appressoria forma

181 E2 contributes to innate immunity against an oomycete pathogen.

182 e microdomains in plant susceptibility to an oomycete pathogen.

183 nd presentation of information on fungal and Oomycete pathogenicity genes and their host interactions

184 icipates in nonhost resistance to fungal and oomycete pathogens and is required for full penetration

185 Oomycete pathogens cause serious damage to a wide spectr

186 Fungal and oomycete pathogens cause some of the most devastating di

187 Oomycete pathogens contain large complements of predicte

188 Many fungal and oomycete pathogens differentiate a feeding structure nam

189 This is a novel strategy that oomycete pathogens exploit to modulate host defense.

190 cally and agronomically important fungal and Oomycete pathogens for intervention with synthetic chemi

191 factors that mediate plant susceptibility to oomycete pathogens is relatively unexplored.

192 Known effectors in oomycete pathogens possess an RXLR-EER motif in their am

193 different queries with a focus on fungal and oomycete pathogens were performed, leading to 510 up-reg

194 nhanced resistance to virulent bacterial and oomycete pathogens, contains a gain-of-function mutation

195 ulence and effector genes from 54 fungal and Oomycete pathogens, of which 176 are from animal pathoge

196 ch determine resistance to viral, fungal and oomycete pathogens, respectively.

197 s are released into plants upon infection by oomycete pathogens, suggesting they may elicit plant def

198 In the oomycete pathogens, two conserved N-terminal motifs, RXL

199 nd effector genes from bacterial, fungal and Oomycete pathogens, which infect human, animal, plant, i

200 nificantly reduced infection capacity of the oomycete pathogens.

201 enhanced resistance to virulent bacterial or oomycete pathogens.

202 disease resistance to virulent bacterial and oomycete pathogens.

203 of evolution of QoI resistance in fungal and oomycete pathogens.

204 th, and enhances resistance to bacterial and oomycete pathogens.

205 nhanced resistance to virulent bacterial and oomycete pathogens.

206 nhanced resistance to virulent bacterial and oomycete pathogens.

207 istance of Arabidopsis against bacterial and oomycete pathogens.

208 ative analysis of P450s in 13 newly explored oomycete pathogens.

209 tance is effective against both the virulent oomycete Peronospora and the bacterial pathogen Pseudomo

210 ee RPP genes directed against the pathogenic oomycete Peronospora parasitica.

211 to five different isolates of the biotrophic oomycete, Peronospora parasitica (causal agent of downy

212 syringae and Xanthomonas campestris, and an oomycete, Peronospora parasitica.

213 on between Phytophthora pathogens, which are oomycetes, phylogenetically distinct from fungi, has bee

214 hogen of Arabidopsis and is related to other oomycete phytopathogens that include several species of

215 ecretion, because effector proteins from the oomycete Phytophthora infestans and virulence determinan

216 The oomycete Phytophthora infestans causes late blight, a ra

217 The oomycete Phytophthora infestans causes late blight, the

218 Filamentous pathogens such as the oomycete Phytophthora infestans infect plants by develop

219 on using a different pathogen of tomato, the oomycete Phytophthora infestans that is distantly relate

220 g a secreted protein from the hemibiotrophic oomycete Phytophthora infestans that is specifically exp

221 The oomycete Phytophthora infestans, causal agent of the tom

222 or proteins (GIPs), that are secreted by the oomycete Phytophthora sojae, a pathogen of soybean, and

223 e to virulent strains of P. syringae and the oomycete Phytophthora sojae.

224 ring sexual development in the heterothallic oomycete, Phytophthora infestans, were identified by sup

225 Fungal and oomycete plant parasites are among the most devastating

226 s to a common virulence strategy between the oomycete plant pathogen P. infestans and several mammali

227 Transformation of the diploid oomycete plant pathogen Phytophthora infestans with anti

228 Late blight, caused by the oomycete plant pathogen Phytophthora infestans, is a dev

229 and Nuk12 candidate effector proteins of the oomycete plant pathogen Phytophthora infestans.

230 developed and applied to 2147 ESTs from the oomycete plant pathogen Phytophthora infestans.

231 aliana) ecotype Columbia-0 is nonhost to the oomycete plant pathogen Phytophthora sojae and the funga

232 Here, we show that two effectors from the oomycete plant pathogen Phytophthora sojae suppress RNA

233 We recently found that the oomycete plant pathogen Phytophthora sojae uses nuclear

234 Phytophthora palmivora is a devastating oomycete plant pathogen.

235 cesses that lead to speciation of fungal and oomycete plant pathogens and provide an outline of how s

236 The sequenced genomes of oomycete plant pathogens contain large superfamilies of

237 Oomycete plant pathogens deliver effector proteins insid

238 effector proteins produced by bacterial and oomycete plant pathogens have been elucidated in recent

239 f cytochrome b mutations found in fungal and oomycete plant pathogens resistant to Q(o) inhibitors (Q

240 They are closely related to other oomycete plant pathogens such as Phytophthora species an

241 Fungal and oomycete plant pathogens translocate effector proteins i

242 hyper-susceptible to fungal, bacterial, and oomycete plant pathogens, demonstrating that Pip1 is an

243 We used the draft genome sequences of three oomycete plant pathogens, Phytophthora sojae, Phytophtho

244 cations of population genomics to fungal and oomycete plant pathogens.

245 Since the natural oomycete population is too low to reach a quorum necessa

246 One type of plant pathogens, oomycetes, produces effector proteins with N-terminal RX

247 ent in these AVR proteins and other secreted oomycete proteins, is similar to a host-cell-targeting s

248 cation in the DNA recognition helices of the oomycete proteins.

249 rasites, including viruses, bacteria, fungi, oomycetes, protozoa, insects and nematodes.

250 ated zoospores that are produced by the soil oomycete Pythium aphanidermatum as a biological vector,

251 fection of rice (Oryza sativa) with the root oomycete Pythium graminicola.

252 We show that the hyphae of the mycelial soil oomycete Pythium ultimum function as active translocatio

253 rt of the PAH fluorene (FLU) by the mycelial oomycete Pythium ultimum that was grown along the air-wa

254 to extreme susceptibility to a root-rotting oomycete (Pythium spp), demonstrating that these genes a

255 Two oomycetes, Pythium oligandrum and Pythium aphanidermatum

256 While oomycete R2R3 proteins contain c-Myb-like helices, R1R2R

257 P. ultimum sequences with similarity to oomycete RXLR and Crinkler effectors, Kazal-like and cys

258 determine the myriad virulence functions of oomycete RXLR effector proteins.

259 hts into structure/function relationships of oomycete RXLR effectors and how these proteins engage wi

260 structures of the effector domains from two oomycete RXLR proteins, Phytophthora capsici AVR3a11 and

261 e present in at least half of the identified oomycete RXLR-dEER effector candidates, and we show that

262 A related pathogenic oomycete's HT signal export is dependent on PI(3)P bindi

263 uckeri, Flavobacterium columnare, and/or the oomycete Saprolegnia ferax.

264 UV-B exposure and infection by a pathogenic oomycete, Saprolegnia ferax.

265 Oomycetes secrete both extracellular and intracellular e

266 Fungi and oomycetes secrete many specialized effector proteins for

267 bition of Pythium ultimum, a phytopathogenic oomycete sensitive to pyoluteorin.

268 sweet2 mutants were more susceptible to the oomycete, showing impaired growth after infection.

269 rium catenoides, a free-living sister of the oomycetes, shows that these transfers largely converge w

270 tionarily conserved effectors from different oomycete species can suppress immunity in plant species

271 Oomycetes such as these Phytophthora species share the k

272 Plant pathogenic oomycetes, such as the Irish potato famine pathogen Phyt

273 Plant pathogenic oomycetes, such as the potato (Solanum tuberosum) and to

274 to an enhanced susceptibility to a virulent oomycete, suggesting a role for BAP1 in basal defense re

275 a-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSCEPTIBILITY1 (IOS1) as a critical PTI player

276 a-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSCEPTIBILITY1 (IOS1) as a critical PTI player

277 abidopsis thaliana) receptor kinase IMPAIRED OOMYCETE SUSCEPTIBILITY1 (IOS1) as required for successf

278 This is supported by the observation that an oomycete that does not form zoospores, Hyaloperonospora

279 Saprolegnia parasitica is a freshwater oomycete that is capable of infecting several species of

280 guish isolates within several species of the oomycetes that cause downy mildew diseases.

281 Fungi and oomycetes that colonize living plant tissue form extensi

282 ers largely converge within the radiation of oomycetes that colonize plant tissues.

283 ng function has been recruited by pathogenic oomycetes to facilitate their own invasion.

284 ed by eukaryotic microbes, such as fungi and oomycetes, to host plants and contribute to the establis

285 During infection, oomycetes translocate effector proteins into host cells,

286 Understanding the mechanisms underlying oomycete virulence and the genomic processes by which th

287 Several common mechanisms underlying oomycete virulence, including protein toxins and cell-en

288 lcNAc) epitopes were not identified when the oomycete was grown in vitro or while infecting the roots

289 lar and molecular events in response to this oomycete, which has a broad host range.

290 ning and biochemical studies have shown that oomycetes, which belong to the kingdom Stramenopila, sec

291 Unlike oomycetes, which employ RXLR effectors to suppress host

292 quences and other sequenced plant pathogenic oomycetes with 91% of the hybrid assembly derived sequen

293 ering effectors, have emerged from comparing oomycetes with different genome characteristics, parasit

294 plant defenses against pathogenic fungi and oomycetes with limited, indirect evidence.

295 ularly destructive group of plant pathogenic oomycete, with the goal of understanding the mechanisms

296 lacking chromalveolates such as ciliates and oomycetes would be explained by plastid loss in these li