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1 , and previous hysterectomy (with or without oophorectomy).
2 in the same population who had not undergone oophorectomy.
3 he efficacy of prophylactic hysterectomy and oophorectomy.
4 are these with women who had not received an oophorectomy.
5 rospectively for breast cancer incidence and oophorectomy.
6 not undergone hysterectomy, with or without oophorectomy.
7 dy participants were censored at the time of oophorectomy.
8 from 3.34 to 4.65 years, depending on age at oophorectomy.
9 iestrogen therapy, i.e., progesterone and/or oophorectomy.
10 , irrespective of whether HRT was used after oophorectomy.
11 undergoing both prophylactic mastectomy and oophorectomy.
12 e of hormone replacement therapy (HRT) after oophorectomy.
13 8 and 8.6 years after bilateral prophylactic oophorectomy.
14 for ovarian cancer or risk-reducing salpingo-oophorectomy.
15 horectomy and 55 with a concurrent bilateral oophorectomy.
16 as adjusted for age, smoking, and unilateral oophorectomy.
17 bilateral mastectomies and 68%, prophylactic oophorectomy.
18 bdominal hysterectomy and bilateral salpingo-oophorectomy.
19 develop in a woman years after prophylactic oophorectomy.
20 ephrectomy; 134985, hysterectomy; and 27445, oophorectomy.
21 th total hysterectomy and bilateral salpingo-oophorectomy.
22 g surgery in the form of unilateral salpingo-oophorectomy.
23 procedures, ie, bilateral mastectomy and/or oophorectomy.
24 ecular bone sustained by rats within 6 wk of oophorectomy.
25 t of premenopausal women underwent bilateral oophorectomy.
26 ed for women who did and who did not undergo oophorectomy.
27 e that developed in women after menopause or oophorectomy.
28 106 women had a hysterectomy with bilateral oophorectomy.
29 ucocele 2 years after risk-reducing salpingo-oophorectomy.
30 1 mutation, survival was much improved after oophorectomy.
31 undergone a hysterectomy with or without an oophorectomy.
32 prior hormone-sensitive cancers or bilateral oophorectomy.
33 at menopause, or history of hysterectomy or oophorectomy.
34 047 (10.4%) hysterectomies, and 1782 (6.5%) oophorectomies.
35 to have mastectomies and 33% (4/12) to have oophorectomies.
36 (95% CI, 1.34-1.50) after oophorectomy vs no oophorectomy, 0.88 (95% CI, 0.85-0.90) after hysterectom
40 Among premenopausal women who had unilateral oophorectomy, 21 percent were on HRT at 3 months, increa
41 6 years with both tamoxifen and prophylactic oophorectomy, 3.5 years with prophylactic mastectomy, an
42 en seen at our clinic underwent prophylactic oophorectomy, 33 of whom had a calculated risk of carryi
43 with tamoxifen, 2.6 years with prophylactic oophorectomy, 4.6 years with both tamoxifen and prophyla
44 ad undergone prophylactic bilateral salpingo-oophorectomy (47 women) were matched with mutation-posit
45 with tamoxifen, 4.4 years with prophylactic oophorectomy, 6.3 years with tamoxifen and oophorectomy,
46 with BRCA1/2 mutations undergo prophylactic oophorectomy after completion of childbearing, decide ab
48 s, with and without quality adjustment, were oophorectomy alone and oophorectomy and mastectomy, resp
49 an incremental cost-effectiveness ratio over oophorectomy alone of 2352 dollars per life-year for BRC
52 omy: 42 without oophorectomy or a unilateral oophorectomy and 55 with a concurrent bilateral oophorec
53 ately 4 years with hysterectomy and salpingo-oophorectomy and adherence to colorectal cancer screenin
54 ptibility genes BRCA1 or BRCA2, prophylactic oophorectomy and bilateral mastectomy have emerged as pr
55 h prior or concurrent bilateral prophylactic oophorectomy and by approximately 90% in women with inta
56 and age of diagnosis, country, and bilateral oophorectomy and censoring at contralateral mastectomy,
57 on at baseline between years since bilateral oophorectomy and common carotid artery intima-media thic
58 on-based sample of women who had received an oophorectomy and compare these with women who had not re
59 plained by greater frequency of hysterectomy/oophorectomy and earlier age at surgery after endometrio
60 women, 31 to 56 years old, who had undergone oophorectomy and hysterectomy received conjugated equine
61 history of premenopausal surgery, bilateral oophorectomy and hysterectomy without oophorectomy were
64 men who had undergone bilateral prophylactic oophorectomy and in 292 matched controls who had not und
67 ures (including a laparotomy and at least an oophorectomy and omental biopsy) in each group of the st
70 he 98 women who chose risk-reducing salpingo-oophorectomy and peritoneal cancer was diagnosed in 1 wo
71 tisite randomized clinical trial of adjuvant oophorectomy and tamoxifen for 5 years or observation an
72 in a randomized controlled trial of adjuvant oophorectomy and tamoxifen or observation who had estrog
73 may favorably influence response to adjuvant oophorectomy and tamoxifen treatment in patients with es
76 9 women who underwent bilateral prophylactic oophorectomy and who were studied to determine the risk
77 c oophorectomy, 6.3 years with tamoxifen and oophorectomy, and 2.6 years with mastectomy, or with bot
78 unilateral oophorectomy, 1097 with bilateral oophorectomy, and 2390 referent women were eligible for
79 (95% probability interval, 4 to 25 days) for oophorectomy, and 6 days (95% probability interval, 3 to
80 bdominal hysterectomy and bilateral salpingo-oophorectomy, and administration of six cycles of intrav
81 cologic procedures including tubal ligation, oophorectomy, and partial hysterectomy have been demonst
82 es (cholecystectomy, colectomy, hysterectomy/oophorectomy, and prostatectomy) between 1987 and 2004.
83 bout effects of HRT, effects of prophylactic oophorectomy, and risks of cancer associated with BRCA1/
84 and/or amenorrhea lasting >6 months, and/or oophorectomy, and/or increased follicle-stimulating horm
85 ," "preventive," "bilateral," "mastectomy," "oophorectomy," and "ovariectomy," a MEDLINE search of th
87 hat women who undergo prophylactic bilateral oophorectomy are at increased risk of death for all caus
88 ancer risk reductions conferred by bilateral oophorectomy are not strongly confounded by failure to a
91 ctomy, partial nephrectomy, hysterectomy, or oophorectomy at 1370 hospitals in the United States from
92 ldbearing, decide about short-term HRT after oophorectomy based largely on quality-of-life issues rat
93 cted on women who had undergone prophylactic oophorectomies because of the elevated risk of ovarian c
94 omen who had received prophylactic bilateral oophorectomy before the age of 45 years than in referent
96 f-reported receipt of bilateral prophylactic oophorectomy (BPO) and utilization of CA-125 and transva
98 onsider the option of bilateral prophylactic oophorectomy (BPO), in the hope that removal of healthy
100 ons between hysterectomy, bilateral salpingo-oophorectomy (BSO), and incidence of diabetes in postmen
101 omy, by 2.8 to 3.4 years; and mastectomy and oophorectomy, by 3.3 to 6.0 years over surveillance.
102 n women with a BRCA1 or BRCA2 mutation after oophorectomy, by age of oophorectomy; to estimate the im
103 age or older who had not undergone bilateral oophorectomy chose to undergo either surveillance for ov
105 breast cancer risk associated with bilateral oophorectomy could be affected by common conditions that
106 was ignored, the strong protective effect of oophorectomy, coupled with the high prevalence of the pr
107 omen without surgery; risk-reducing salpingo-oophorectomy decreased breast cancer incidence by 37% to
109 at premenopausal hysterectomy with bilateral oophorectomy decreases the risk of breast cancer in blac
110 7 years of life expectancy from prophylactic oophorectomy, depending on their cumulative risk of canc
113 the 33 mutation-positive women who underwent oophorectomy during follow-up developed breast cancer, c
114 Prophylactic mastectomy and prophylactic oophorectomy, effective in retrospective clinical experi
115 rst birth, oral contraceptive use, bilateral oophorectomy, estrogen plus progestin use, and height.
116 50-87, we analysed those who had received an oophorectomy for a non-cancer indication before the onse
117 ce surgical menopause following hysterectomy/oophorectomy for noncancerous conditions; it is also com
118 rning the efficacy of bilateral prophylactic oophorectomy for reducing the risk of gynecologic cancer
119 dentified through risk-reducing prophylactic oophorectomy from three women with germline BRCA1 mutati
120 ause, and cancer treatments such as surgical oophorectomy, gonadotropin-releasing hormone agonists, c
121 years since hysterectomy in the no bilateral oophorectomy group (beta = 0.005 (standard error, 0.023)
122 ynecologic cancer was longer in the salpingo-oophorectomy group, with a hazard ratio for subsequent b
124 o-oophorectomy, women who underwent salpingo-oophorectomy had a lower risk of ovarian cancer, includi
125 eries (prophylactic bilateral mastectomy and oophorectomy) had an incremental cost-effectiveness rati
126 nd ovarian cancer, but it is unclear whether oophorectomy has an impact on survival in women with BRC
128 ered after randomisation to have a bilateral oophorectomy, have ovarian cancer, or had exited the reg
130 nal hypertension for hemorrhagic stroke, and oophorectomy, HDP, preterm delivery, and stillbirth for
131 ween women who underwent hysterectomy and/or oophorectomy (higher odds for less educated women) and t
132 last births, age at menopause, hysterectomy, oophorectomy, hormone therapy use, and body mass index (
134 s of age (HR 2.36, P = 0.01), and unilateral oophorectomy (HR 9.76, P < 0.0001) were independent dete
136 hylactic hysterectomy; and 52%, prophylactic oophorectomy if they tested positive for a mutation.
137 according to her cancer risks, prophylactic oophorectomy improved survival by 0.4 to 2.6 years; mast
138 Among women who had undergone bilateral oophorectomy, IMT was significantly related to years sin
140 s recorded in women who underwent unilateral oophorectomy in either overall or stratified analyses.
141 the efficacy of prophylactic mastectomy and oophorectomy in preventing breast and ovarian cancer to
146 ylactic hysterectomy with bilateral salpingo-oophorectomy is an effective strategy for preventing end
158 fen, oral contraceptives, bilateral salpingo-oophorectomy, mastectomy, both surgeries, or surveillanc
160 age, a more conservative unilateral salpingo-oophorectomy may be performed, assuming that careful sta
161 ysterectomy status with or without bilateral oophorectomy might increase risk for CVD, but most studi
162 al gains in life expectancy and prophylactic oophorectomy more limited gains for young women with BRC
163 h lower odds of breast cancer (for bilateral oophorectomy, multivariable-adjusted odds ratios = 0.60,
164 terval: 0.47, 0.77; for hysterectomy without oophorectomy, multivariable-adjusted odds ratios = 0.68,
165 primary hysterectomy and bilateral salpingo-oophorectomy, often using minimally invasive approaches
167 tomy; to estimate the impact of prophylactic oophorectomy on all-cause mortality; and to estimate 5-y
169 viduals who underwent risk-reducing salpingo-oophorectomy, one early-stage ovarian neoplasm and one e
170 en women reported a hysterectomy: 42 without oophorectomy or a unilateral oophorectomy and 55 with a
171 in the smaller group of women with bilateral oophorectomy or hysterectomy with one ovary retained.
172 sociated with breast cancer risk, but either oophorectomy or hysterectomy, or both, and the timing of
173 Exclusion criteria were previous bilateral oophorectomy or ovarian malignancy, increased risk of fa
174 o patient in the genetic group had undergone oophorectomy or was taking prophylactic agents such as t
175 sing hormone analogue triptorelin, bilateral oophorectomy, or bilateral ovarian irradiation were used
177 gnant tumor are not uncommon in prophylactic oophorectomies performed in women at very high risk for
179 my (PM) at various ages, and/or prophylactic oophorectomy (PO) at ages 40 or 50 years in 25-year-old
181 hypothesized that population differences in oophorectomy prevalence might significantly influence br
182 ncy or cancer prevention through castration (oophorectomy), preventing chemically induced mouse carci
187 bdominal hysterectomy and bilateral salpingo-oophorectomy revealed similar estimates (HR, 0.59; 95% C
191 Therefore, although risk-reducing salpingo-oophorectomy (RRSO) is standard treatment among women wi
194 s risk reduction from risk-reducing salpingo-oophorectomy (RRSO), by CJM and self-identified Jewish s
195 duction, particularly risk-reducing salpingo-oophorectomy (RRSO), has become an important component o
198 ancer was diagnosed at surgery, prophylactic oophorectomy significantly reduced the risk of coelomic
199 lated tumor predisposition, and explains why oophorectomy significantly reduces breast cancer risk an
200 s of these mutations, risk-reducing salpingo-oophorectomy significantly reduces morbidity and mortali
201 rian cancer, prophylactic bilateral salpingo-oophorectomy significantly reduces the incidence of this
202 een determined; however, estrogen reduction (oophorectomy) significantly reduces recurrence in premen
206 Women with a hysterectomy (regardless of oophorectomy status) had an adverse risk profile at base
208 we observed that hysterectomy, regardless of oophorectomy status, was associated with increased risk
210 nalysis of ovarian tissues from prophylactic oophorectomies, suggest that depletion of ovarian follic
211 gainst prophylactic surgery (eg, mastectomy, oophorectomy); these surgeries are an option for mutatio
212 BRCA2 mutation after oophorectomy, by age of oophorectomy; to estimate the impact of prophylactic oop
213 n who did not undergo risk-reducing salpingo-oophorectomy, undergoing salpingo-oophorectomy was assoc
216 y stroke were 1.42 (95% CI, 1.34-1.50) after oophorectomy vs no oophorectomy, 0.88 (95% CI, 0.85-0.90
217 peritoneal cancer associated with bilateral oophorectomy was 0.20 (95% CI, 0.13 to 0.30; P < .001).
218 mortality to age 70 years associated with an oophorectomy was 0.23 (95% CI, 0.13 to 0.39; P < .001).
219 uted to breast cancer in women who underwent oophorectomy was 0.38 (95% CI, 0.19-0.77; P = .007) for
220 isk of breast cancer; risk-reducing salpingo-oophorectomy was associated with a lower risk of ovarian
225 use, the finding that years since bilateral oophorectomy was associated with increasing atherosclero
226 g salpingo-oophorectomy, undergoing salpingo-oophorectomy was associated with lower all-cause mortali
229 idence of IBTR in carriers who had undergone oophorectomy was not significantly different from that i
230 k demonstrated that the protective effect of oophorectomy was strongest among women who were premenop
231 ter severity of lung disease, menopause, and oophorectomy were associated with greater decline in BMD
232 ateral oophorectomy and hysterectomy without oophorectomy were associated with lower odds of breast c
233 ence and all-cause mortality associated with oophorectomy were evaluated using time-dependent surviva
234 ho did not have previous cancer or bilateral oophorectomy were followed-up for an average of 5.3 year
235 re postmenopausal or who underwent bilateral oophorectomy were less likely to have hot flashes if the
236 ctor of IBTR when carriers who had undergone oophorectomy were removed from analysis (HR, 1.99; P = .
237 women who underwent unilateral or bilateral oophorectomy while residing in Olmsted County, MN, USA,
242 red prophylactic mastectomy and prophylactic oophorectomy with no prophylactic surgery among women wh
243 comparison of women who underwent bilateral oophorectomy with referent women provided evidence for a
244 ompared the effect of risk-reducing salpingo-oophorectomy with that of surveillance for ovarian cance
245 after hysterectomy with or without bilateral oophorectomy with the changes observed up to and after n
246 -Meier curves to compare women who underwent oophorectomy with those who had ovarian preservation.
247 n who did not undergo risk-reducing salpingo-oophorectomy, women who underwent salpingo-oophorectomy
248 %, respectively, that bilateral prophylactic oophorectomy would reduce ovarian cancer risk by 45%, an
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