1 accines and case reports of human autoimmune oophoritis.

2 lts in the failure to induce both autoimmune oophoritis and autoantibody production.

3 evealed that Aod1 controls susceptibility to oophoritis and comprises two linked QTL with opposing al

4 ction of noninfectious ovarian inflammation (oophoritis) and serum antibody to ovarian and placental

5 es, antiovarian autoantibody responsiveness, oophoritis, and atrophy, to five quantitative trait loci

6 e development of antiovarian autoantibodies, oophoritis, and atrophy.

7                                 AOD began as oophoritis at 3 wk.

8                  Mice with amAb often had no oophoritis; but more importantly, bilateral ovariectomy

9 e detectable ZP3 T cell response (day 5) and oophoritis (day 7) failed to prevent the amAb response,

10    A ZP3 peptide contains both an autoimmune oophoritis-inducing T cell epitope and a B cell epitope

11 hibin-alpha-targeted experimental autoimmune oophoritis is initiated by CD4(+) Th1 T cells that stimu

12 s diseases including hearing loss, orchitis, oophoritis, mastitis, and pancreatitis.

13 ng phenomenon based on the murine autoimmune oophoritis model.

14 k, ovaries engrafted at 5 wk had more severe oophoritis than ovaries engrafted at 6 or 12 wk; moreove

15 rferon gamma in vitro, were able to transfer oophoritis to normal recipients.

16 al follicles, and the resultant interstitial oophoritis was associated with unimpaired ovarian functi

17             The mice OX at 2 wk did not have oophoritis whereas approximately 80% of mice OX at 3 or

18  T cells and induces experimental autoimmune oophoritis with a unique biphasic phenotype characterize