1 arded migration of the epithelium across the open wound.

2 is the migration of keratinocytes across the open wound.

3 ssure to rapidly stanch blood loss and close open wounds.

4 ibuted similarly between the treatment arms (Open: wound 0.5%, liver 5.8%, lung 4.6%, other 8.4%; Lap

5                                              Open wound and epithelialization rates and angiogenesis

6 ncanonical members of the JNK pathway caused open wounds, as did several genes involved in actin cyto

7  Both groups had comparable readmissions for open wounds, but patients treated with CEA required more

8 itating the healing process by protecting an open wound from bacterial insult.

9                                     Using an open wound infection model, we show that deletion of mGs

10 ertension, dyspnea, diabetes, renal failure, open wounds/infection, or advanced American Society of A

11       Whole mount confocal microscopy showed open wounds on the ocular surface of mice at 1 and 2 wee

12 ng of rectal swab samples, swab samples from open wounds or drainages, and urine samples from patient

13                         Infections occur via open wounds or following ingestion of contaminated seafo

14 close the wound gap, resulting in persistent open wounds or partially covered sinus tracts.

15 litative morphological classification of the open wound phenotypes and evaluation of JNK activation s

16 om the nares, oropharynx or trachea, and any open wound routinely on admission to the intensive care

17  healing assays including time to closure of open wounds, tensile strength of healed incisional wound

18 e surgical and medical care of contaminated, open wounds with immunization and immunoglobulin therapy