ライフサイエンスコーパス: open-angle glaucoma

1 Hg and he was found to have advanced primary open angle glaucoma.

2 linked directly to juvenile- and adult-onset open angle glaucoma.

3 ut there is no clear gender predilection for open angle glaucoma.

4 suggested to be important for progression of open angle glaucoma.

5 Genetics seem to play an important role in open angle glaucoma.

6 heir role in the management of patients with open angle glaucoma.

7 ar diseases, such as keratoconus and primary open angle glaucoma.

8 ent of patients with ocular hypertension and open-angle glaucoma.

9 in lowering intraocular pressure in primary open-angle glaucoma.

10 high prevalence of glaucoma which is largely open-angle glaucoma.

11 in the United States and had newly diagnosed open-angle glaucoma.

12 and 2012 and newly diagnosed and treated for open-angle glaucoma.

13 e the most common genetic factors of primary open-angle glaucoma.

14 set of the prevalent ocular disorder primary open-angle glaucoma.

15 have previously been associated with primary open-angle glaucoma.

16 myocilin, a protein associated with primary open-angle glaucoma.

17 etic retinopathy, uveoretinitis, and primary open-angle glaucoma.

18 (IOP)-lowering medications in patients with open-angle glaucoma.

19 to mutations in the MYOCILIN gene is primary open-angle glaucoma.

20 when selecting treatments for patients with open-angle glaucoma.

21 ay lead to juvenile- and adult-onset primary open-angle glaucoma.

22 a multiple topical drug regimen for primary open-angle glaucoma.

23 procedure for reducing IOP in patients with open-angle glaucoma.

24 at baseline; 68% were diagnosed with primary open-angle glaucoma.

25 rule has limited utility in the diagnosis of open-angle glaucoma.

26 sfunction results in ocular hypertension and open-angle glaucoma.

27 ar disease and 233 eyes of 163 patients with open-angle glaucoma.

28 because TGF-beta2 is associated with primary open-angle glaucoma.

29 ciated with juvenile and adult-onset primary open-angle glaucoma.

30 the most common identifiable risk factor for open-angle glaucoma.

31 d by cataract surgery, increases the risk of open-angle glaucoma.

32 ay lead to juvenile- and adult-onset primary open-angle glaucoma.

33 ix and the most common identifiable cause of open-angle glaucoma.

34 in 58 eyes of 58 patients with suspected or open-angle glaucoma.

35 transgenic mouse model of hereditary primary open-angle glaucoma.

36 6 had ocular hypertension and 14 had primary open-angle glaucoma.

37 be a new risk factor to consider in primary open-angle glaucoma.

38 ce to AH outflow, is a major risk factor for open-angle glaucoma.

39 ular-pressure-lowering drug in patients with open-angle glaucoma.

40 surgery was performed in adults with various open-angle glaucomas.

41 The 5 most prevalent diagnoses were open-angle glaucoma (1.61%), exfoliation glaucoma (0.20%

42 In 30 patients with open-angle glaucoma, 1 eye (median mean deviation [MD],

43 Thirty subjects under treatment for open-angle glaucoma, 23 subjects with ocular hypertensio

44 ed diabetic macular edema and no preexisting open-angle glaucoma, 260 were randomly assigned to recei

45 study included 122 eyes treated for primary open angle glaucoma, 50 eyes (study group) in which, aft

46 ed 354 eyes in 180 subjects (97 with primary open-angle glaucoma, 83 with glaucoma suspicion) who had

47 ecular meshwork is a primary risk factor for open-angle glaucoma, a leading cause of blindness.

48 have identified novel loci for POAG (primary-open-angle glaucoma) (ABCA1, AFAP1, GMDS, PMM2, TGFBR3,

49 ree eyes of 2 patients who developed chronic open-angle glaucoma after Nd:YAG vitreolysis for symptom

50 Inclusion criteria were open-angle glaucoma, age >/= 40 years, and uncontrolled

51 Treatments for open-angle glaucoma aim to prevent vision loss through l

52 2 eyes, comprising 38 glaucomatous eyes with open angle glaucoma and 24 healthy controls, were includ

53 s a highly heritable risk factor for primary open angle glaucoma and currently the only target for gl

54 h Weill-Marchesani-like syndrome and primary open angle glaucoma and ectopia lentis in dogs.

55 e patient with choroidal infarction, primary open angle glaucoma and Flammer syndrome.

56 29 eyes with open angle glaucoma and visual field defects, as well as

57 Sixty-four patients at different stages of open-angle glaucoma and 26 patients with ocular hyperten

58 cts (48 male, 40 female) with a diagnosis of open-angle glaucoma and a median age of 67 years (interq

59 ere collected from 111 patients with primary open-angle glaucoma and an age-matched control group of

60 anuary 2000 and September 2013 on the topics open-angle glaucoma and angle-closure glaucoma.

61 otal of 609 patients (609 eyes) with primary open-angle glaucoma and cataract were included.

62 rther suggest a lack of relationship between open-angle glaucoma and choroidal thickness.

63 urgical algorithms for care in patients with open-angle glaucoma and coexistent cataract remain uncle

64 A 75-year-old pseudophakic man with open-angle glaucoma and diabetic retinopathy developed n

65 GN, SETTING, AND PARTICIPANTS: Patients with open-angle glaucoma and healthy controls were examined b

66 ntia, including Alzheimer's disease, primary open-angle glaucoma and Helicobacter pylori (H.pylori) i

67 h CCT has been implicated, including primary open-angle glaucoma and keratoconus.

68 Consecutive trabeculectomy cases with open-angle glaucoma and no previous incisional glaucoma

69 NTG when compared with patients with primary open-angle glaucoma and nonglaucomatous control subjects

70 trabeculoplasty (SLT) as initial therapy for open-angle glaucoma and ocular hypertension and have dem

71 All patients with newly diagnosed open-angle glaucoma and ocular hypertension were include

72 sc appearance used for the classification of open-angle glaucoma and ocular hypertension, significant

73 rine transporter inhibitor, in patients with open-angle glaucoma and ocular hypertension.

74 he diurnal, but not the nocturnal, period in open-angle glaucoma and ocular hypertension.

75 r lens implantation in patients with primary open-angle glaucoma and ocular hypertension.

76 with an increased prevalence of all forms of open-angle glaucoma and OHTN, whereas hyperopia was asso

77 One eye per patient with primary open-angle glaucoma and stable intraocular pressure (IOP

78 Glaucoma surveillance in eyes with open-angle glaucoma and statistically normal IOP should

79 y 24-2 and 10-2 VFs in patients with primary open-angle glaucoma and to test the hypothesis that pati

80 aucoma Associates of Texas with uncontrolled open-angle glaucoma and underwent gonioscopy-assisted tr

81 Patients with open-angle glaucoma and VFs obtained from reliable exami

82 is and pars plana vitrectomy, and 1 juvenile open-angle glaucoma) and 21 of 70 eyes with nonglaucomat

83 A total of 234 patients (104 with primary open angle glaucoma, and 130 control subjects without an

84 ntil death; 203 patients (65.7%) had primary open-angle glaucoma, and 106 patients (34.2%) had exfoli

85 entosa, while Bardet-Biedl syndrome, primary open-angle glaucoma, and tumor cell invasiveness are lin

86 le have a 2.5-fold increased risk of primary open-angle glaucoma as compared with those in the lowest

87 These mice may be useful as a model for open-angle glaucoma, as well as for assessing the relati

88 To determine the stage of primary open angle glaucoma at presentation at a tertiary eye un

89 l, we enrolled patients with newly diagnosed open-angle glaucoma at ten UK centres (tertiary referral

90 essure in patients with primary or secondary open angle glaucomas, both as an initial therapy or in c

91 e uveitis, diabetic retinopathy, and primary open angle glaucoma, but its role in normal vision is la

92 Genetic studies have linked myocilin to open angle glaucoma, but the functions of the protein in

93 en species play a pathogenic role in primary open angle glaucoma by fostering changes that reduce per

94 However, in eyes with open angle glaucoma, cataract surgery alone may be of li

95 osterior sclera in a canine model of primary open-angle glaucoma caused by the G661R missense mutatio

96 s with a minimum 2-year diagnosis of primary open-angle glaucoma, chronic primary angle-closure glauc

97 in choroidal thickness of eyes with advanced open-angle glaucoma compared to that of fellow eyes with

98 In an institutional setting, a patient with open-angle glaucoma consented to be the recipient of the

99 with many ocular problems, such as secondary open angle glaucoma, corneal dysfunction, cataract, and

100 Myocilin, a causative gene for open angle glaucoma, encodes a secreted glycoprotein wit

101 ERIA: participants with primary or secondary open-angle glaucoma (excluding uveitic) who had undergon

102 hundred seven patients with newly diagnosed open-angle glaucoma from the CIGTS.

103 tively; by 30% and 22%, respectively, in the open-angle glaucoma group compared with the control grou

104 d mean amplitudes in ocular hypertension and open-angle glaucoma groups compared with the control gro

105 eculoplasty (LTP) is routinely used to treat open-angle glaucoma; hence, understanding variations in

106 rs investigated the genetic cause of primary open angle glaucoma in a large four-generation family wi

107 e was being followed and treated for primary open angle glaucoma in our tertiary referral center.

108 benefits and harms of screening for primary open-angle glaucoma in adults.

109 of LOXL1 play a significant role in primary open-angle glaucoma in the Caucasian, African-American,

110 specific risk factors and manifestations of open-angle glaucoma in this rapidly growing population.

111 contribute to the predisposition of primary open-angle glaucoma in various high-risk populations.

112 gle glaucoma (POAG) represent a continuum of open-angle glaucomas, in which a certain level of intrao

113 It may also be important in primary open angle glaucoma influencing aqueous outflow.

114 ed with progression of patients with primary open angle glaucoma is essential to our clinical practic

115 1 single-nucleotide polymorphism and primary open angle glaucoma is found.

116 Primary open angle glaucoma is the most prevalent type of glauco

117 Secondary open-angle glaucoma is a complication of Nd:YAG vitreoly

118 ar meshwork (TM) and the elevation of IOP in open-angle glaucoma is associated with dysfunction and l

119 Primary open-angle glaucoma is characterized by increased resist

120 Primary open-angle glaucoma is the second leading cause of blind

121 14 Normal Tension Glaucoma (NTG), 5 Juvenile Open Angle Glaucoma (JOAG) and 6 (OHT).

122 Juvenile open-angle glaucoma (JOAG) is a severe neurodegenerative

123 llowing cataract surgery (GFCS) and juvenile open-angle glaucoma (JOAG) is variable and with relative

124 ntain any of the previously reported primary open angle glaucoma loci.

125 This study identifies a new primary open angle glaucoma locus, GLC1Q, in a region on chromos

126 Medical and surgical treatments for open-angle glaucoma lower intraocular pressure and reduc

127 ollowing cataract surgery alone in eyes with open angle glaucoma may be limited and transient.

128 es with changing expression in this model of open-angle glaucoma may help elucidate the primary chang

129 < 0.001) in both eyes with incident primary open-angle glaucoma (mean, 10.6%; standard deviation, 22

130 (MetS) with intraocular pressure and primary open angle glaucoma (OAG) have been reported.

131 umor samples were analyzed from 23 eyes with open angle glaucoma (OAG) undergoing glaucoma filtration

132 However, the risk of open-angle glaucoma (OAG) among patients with OSA remain

133 aluate the longitudinal relationship between open-angle glaucoma (OAG) and incident dementia.

134 Coded open-angle glaucoma (OAG) and OAG suspects accounted for

135 beculoplasty (SLT) on medically uncontrolled open-angle glaucoma (OAG) and to evaluate the effects of

136 d (VF) deterioration events in patients with open-angle glaucoma (OAG) by 50% over a 2-year period.

137 personalized forecasts of how patients with open-angle glaucoma (OAG) experience disease progression

138 ection between ocular perfusion pressure and open-angle glaucoma (OAG) has been hypothesized.

139 This study determined the risk factors for open-angle glaucoma (OAG) in adults examined in the Nige

140 Open-angle glaucoma (OAG) is associated with systemic me

141 ice-daily timolol eye drops in patients with open-angle glaucoma (OAG) or ocular hypertension (OHT) t

142 In randomized, controlled trials of open-angle glaucoma (OAG) or ocular hypertension (OHT),

143 oprost ophthalmic solution, in patients with open-angle glaucoma (OAG) or ocular hypertension (OHT).

144 Sequential surgical cases of juvenile open-angle glaucoma (OAG) or primary congenital glaucoma

145 Nine patients had previous diagnoses of open-angle glaucoma (OAG) or suspected glaucoma.

146 lective laser trabeculoplasty (SLT) in early open-angle glaucoma (OAG) patients.

147 iciary person-years and per 10,000 diagnosed open-angle glaucoma (OAG) person-years.

148 iagnosis; 20.0% (n = 330) were identified as open-angle glaucoma (OAG) suspects, 9.2% (n = 151) were

149 or visual field progression in patients with open-angle glaucoma (OAG) were eligible.

150 f Diseases (ICD-9-CM) diagnoses of cataract, open-angle glaucoma (OAG), nonexudative age-related macu

151 ty affect receipt of common tests to monitor open-angle glaucoma (OAG).

152 tect against the development or worsening of open-angle glaucoma (OAG).

153 coherence tomography (OCT) volume scans for open-angle glaucoma (OAG).

154 is an alternative to topical medications for open-angle glaucoma (OAG).

155 a major risk factor for the deterioration of open-angle glaucoma (OAG); medical IOP reduction is the

156 eroid use, myopia, socioeconomic status, and open-angle glaucoma (odds ratio [OR], 0.89; 95% confiden

157 ular hypertension, and patients with primary open angle glaucoma or primary angle closure glaucoma.

158 s who have one of the two forms of glaucoma: open-angle glaucoma or angle-closure glaucoma.

159 were obtained in 57 eyes of 57 subjects with open-angle glaucoma or glaucoma suspicion.

160 ication of Diseases, Ninth Revision, code of open-angle glaucoma or its related entities who underwen

161 Total of 233 patients with open-angle glaucoma or ocular hypertension and with mean

162 tive cohort study of untreated patients with open-angle glaucoma or ocular hypertension at a hospital

163 l of 660 adults with a clinical diagnosis of open-angle glaucoma or ocular hypertension from a referr

164 Forty subjects with open-angle glaucoma or ocular hypertension were admitted

165 A total of 560 patients with primary open-angle glaucoma or ocular hypertension who had insuf

166 eserved with PQ reduced IOP in patients with open-angle glaucoma or ocular hypertension who were into

167 Patients with open-angle glaucoma or ocular hypertension who were into

168 In patients with open-angle glaucoma or ocular hypertension, polyquaterni

169 y and effectively lower IOP in patients with open-angle glaucoma or ocular hypertension, showing sign

170 rz/brim/tim in Mexican patients with primary open-angle glaucoma or ocular hypertension.

171 during late-day time points in patients with open-angle glaucoma or ocular hypertension.

172 ts of glaucoma, but also in individuals with open-angle glaucoma or ocular hypertension.

173 Eligible patients were adults diagnosed with open-angle glaucoma or ocular hypertension.

174 0.005% (50 mug/mL) in patients with primary open-angle glaucoma or ocular hypertension.

175 Adult patients with open-angle glaucoma or ocular hypertension.

176 intraocular pressure (IOP) in patients with open-angle glaucoma or ocular hypertension; however, the

177 I, 1.31-10.13; P = .01), presence of primary open-angle glaucoma (OR, 3.82; 95% CI, 1.60-9.14; P = .0

178 beculectomy surgery in patients with primary open angle glaucoma over a 3-year period of follow-up.

179 ), more glaucoma suspects (p < 0.0001), more open angle glaucoma (p = 0.0006) and fewer other conditi

180 of glaucoma (P < .0001), and a diagnosis of open-angle glaucoma (P = .0003) were associated with inc

181 fold; n=20) in AH derived from human primary open angle glaucoma patients as compared to AH derived f

182 ve, observational cohort study that included open angle glaucoma patients with visually significant c

183 At baseline following washout, open-angle glaucoma patients (n = 75) were administered

184 Open-angle glaucoma patients without prior incisional gl

185 of TGF-beta2 has been found in many primary open-angle glaucoma patients.

186 paracentral visual field defects in primary open-angle glaucoma patients.

187 rgets after a first failed trabeculectomy in open-angle glaucoma patients.

188 The study included patients with primary open angle glaucoma (POAG group, n = 30) and controls (n

189 12 normal controls, 7 patients with primary open angle glaucoma (POAG) and 9 patients with normal te

190 OPTN) are linked to the pathology of primary open angle glaucoma (POAG) and amyotrophic lateral scler

191 les frequencies in primary glaucoma [primary open angle glaucoma (POAG) and primary angle closure gla

192 changes of optic nerve in eyes with primary open angle glaucoma (POAG) by the joint use of optical c

193 tly associated with 6.0% of cases of primary open angle glaucoma (POAG) in patients from Oregon and G

194 oci have been associated with either primary open angle glaucoma (POAG) or heritable ocular quantitat

195 between groups of eyes differing in primary open angle glaucoma (POAG) outcome, how POAG was determi

196 (codon 72 Pro/Arg) and the subset of primary open angle glaucoma (POAG) patients with early loss of c

197 Control TM and most primary open angle glaucoma (POAG) TM were collected from cadave

198 beagle model of autosomal recessive primary open angle glaucoma (POAG) to a 4-Mb interval on chromos

199 ns in the myocilin (MYOC) gene cause primary open angle glaucoma (POAG) with varying age-of-onset and

200 of 30.8 months, including 28 (4.4%) primary open angle glaucoma (POAG), 27 (4.2%) primary angle clos

201 jects were glaucomatous: (67 PXG, 42 Primary Open Angle Glaucoma (POAG), 28 PACG, 14 Normal Tension G

202 Primary open angle glaucoma (POAG), a major cause of blindness w

203 are the most common genetic cause of primary open angle glaucoma (POAG), but the mechanisms underlyin

204 defects also occur in patients with primary open angle glaucoma (POAG), in which there is specific R

205 netically complex disorders, such as primary open angle glaucoma (POAG), may include highly heritable

206 The mean IOP for primary open angle glaucoma (POAG), ocular hypertension (OHT), n

207 ic or glaucoma faculty patients with primary open angle glaucoma (POAG), ocular hypertension (OHTN),

208 n the aqueous humor of patients with primary open angle glaucoma (POAG), pseudoexfoliation syndrome (

209 several characteristics observed in primary open angle glaucoma (POAG).

210 ied 61 patients (80 procedures) with primary open angle glaucoma (POAG).

211 cular meshwork (TM) of patients with primary open-angle glaucoma (POAG) and appears to contribute to

212 ssociation of known loci for IOP and primary open-angle glaucoma (POAG) and identify four new IOP-ass

213 ize the role of osteopontin (OPN) in primary open-angle glaucoma (POAG) and normal eyes.

214 ly examined the global prevalence of primary open-angle glaucoma (POAG) and primary angle-closure gla

215 meshwork (TM) height differs between primary open-angle glaucoma (POAG) and primary angle-closure gla

216 en implicated in the pathogenesis of primary open-angle glaucoma (POAG) based on elevated levels in g

217 age to the optic disc in humans with primary open-angle glaucoma (POAG) can be measured using a novel

218 s) endophthalmitis and risk of a new primary open-angle glaucoma (POAG) diagnosis within 365 days aft

219 ses (TIMPs) in the aqueous humour of primary open-angle glaucoma (POAG) eyes have been described.

220 a genome-wide association study for primary open-angle glaucoma (POAG) in 1,007 cases with high-pres

221 culoplasty (SLT) as sole therapy for primary open-angle glaucoma (POAG) in an Afro-Caribbean populati

222 k, CA) for treating mild-to-moderate primary open-angle glaucoma (POAG) in patients undergoing catara

223 DKN2B genes has been associated with primary open-angle glaucoma (POAG) in several independent studie

224 Primary open-angle glaucoma (POAG) is a blinding disease.

225 Primary open-angle glaucoma (POAG) is a complex disease with a g

226 Primary open-angle glaucoma (POAG) is a genetically complex comm

227 Primary open-angle glaucoma (POAG) is a genetically complex neur

228 Primary open-angle glaucoma (POAG) is a highly prevalent conditi

229 Primary open-angle glaucoma (POAG) is a leading cause of blindne

230 Primary open-angle glaucoma (POAG) is a leading cause of irrever

231 Primary open-angle glaucoma (POAG) is a leading cause of irrever

232 Primary open-angle glaucoma (POAG) is a major cause of blindness

233 Primary open-angle glaucoma (POAG) is a major cause of irreversi

234 r for optic nerve damage in cases of primary open-angle glaucoma (POAG) is an increased intraocular p

235 The primary causative factor of primary open-angle glaucoma (POAG) is elevated intraocular press

236 Primary open-angle glaucoma (POAG) is the second leading cause o

237 venous pressure (RVP) in the eyes of primary open-angle glaucoma (POAG) patients and healthy subjects

238 difference in severity of disease in primary open-angle glaucoma (POAG) patients with a Myocilin (MYO

239 itary determination) was examined in primary open-angle glaucoma (POAG) patients with cataract and no

240 g can cause fibrosis of the TM as in primary open-angle glaucoma (POAG) patients, and is characterize

241 s in ocular hypertensive (OH) and in primary open-angle glaucoma (POAG) patients.

242 American Academy of Ophthalmology's Primary Open-angle Glaucoma (POAG) Preferred Practice Pattern (P

243 erature continues to be that NTG and primary open-angle glaucoma (POAG) represent a continuum of open

244 of aqueous humor from patients with primary open-angle glaucoma (POAG) revealed marked increases in

245 Economic viability of treatments for primary open-angle glaucoma (POAG) should be assessed objectivel

246 The beagle model of primary open-angle glaucoma (POAG) was used to correlate ANGPTL7

247 ce was 1.24% (95% CI, 1.14-1.34) for primary open-angle glaucoma (POAG), 0.39% (95% CI, 0.34-0.45) fo

248 We studied 1 eye of 63 primary open-angle glaucoma (POAG), 30 ocular hypertension (OH),

249 egion previously was associated with primary open-angle glaucoma (POAG), although replication among i

250 the 4 forms of open-angle glaucoma: primary open-angle glaucoma (POAG), normal-tension glaucoma (NTG

251 Primary open-angle glaucoma (POAG), the most common optic neurop

252 In cadaver eyes with primary open-angle glaucoma (POAG), TSP1 is increased in one thi

253 a secondary glaucoma that resembles primary open-angle glaucoma (POAG).

254 at PMH use may decrease the risk for primary open-angle glaucoma (POAG).

255 TM, similar to those associated with primary open-angle glaucoma (POAG).

256 e most common known genetic cause of primary open-angle glaucoma (POAG).

257 orders of blindness, with a focus on primary open-angle glaucoma (POAG).

258 population-specific genetic risk of primary open-angle glaucoma (POAG).

259 nction, which has been implicated in primary open-angle glaucoma (POAG).

260 low autoregulation are implicated in primary open-angle glaucoma (POAG).

261 pressure (IOP) in the evaluation of primary open-angle glaucoma (POAG); and to determine the feasibi

262 ODH) before and after development of primary open-angle glaucoma (POAG); determine the prognostic sig

263 ce interval, 3.3-5.4), consisting of primary open-angle glaucoma (POAG, 3.2%, including high-tension

264 -2013) to estimate effects of T2D on primary open-angle glaucoma (POAG; 3,554 cases).

265 ELINES: Evidence-based update of the Primary Open-Angle Glaucoma Preferred Practice Pattern(R) (PPP)

266 PRIMARY OPEN-ANGLE GLAUCOMA PREFERRED PRACTICE PATTERN(R) GUIDEL

267 closure glaucoma (PACG); 1 of the 4 forms of open-angle glaucoma: primary open-angle glaucoma (POAG),

268 ify possible risk factors leading to primary open angle glaucoma progression and blindness.

269 glandin analog eye drops in treating primary open-angle glaucoma, published between December 2000 and

270 0.77-0.80) and with increases in the odds of open-angle glaucoma ranging from 1.23 (95% CI, 1.20-1.26

271 involved diabetic macular edema and no prior open-angle glaucoma, repeated intravitreous injections o

272 Patients with open-angle glaucoma (Shaffer Grade 3 and 4) and uncontro

273 In open-angle glaucoma, substantial RNFL thinning or struct

274 PRIMARY OPEN-ANGLE GLAUCOMA SUSPECT PREFERRED PRACTICE PATTERN(R

275 osis and management of patients with primary open-angle glaucoma suspect with detailed recommendation

276 In 57 patients with primary open-angle glaucoma, the IOP decreased by 7.7 mmHg (stan

277 In the subjects with open-angle glaucoma, the pattern ERG P50-N95 was found t

278 Risk factors for primary open-angle glaucoma-the most common form of glaucoma-inc

279 or cataract surgery is 4500% and for medical open-angle glaucoma therapy is 4000%.

280 l-tension glaucoma (NTG) is a common form of open-angle glaucoma throughout the world, and yet there

281 etrospective case series of 81 patients with open-angle glaucoma undergoing 110 SLT procedures from N

282 Lucia with medically treated primary open-angle glaucoma underwent a 30-day washout, followed

283 The prevalence of primary open-angle glaucoma was 6.8% overall, increasing from 3.

284 mary glaucoma that had gonioscopy (n = 243), open-angle glaucoma was more common (86 %) than angle-cl

285 Primary open-angle glaucoma was the underlying diagnosis in 342

286 nical questions on the management of primary open-angle glaucoma were derived from practice guideline

287 s) with ocular hypertension or early primary open-angle glaucoma were enrolled.

288 Two hundred and six patients with open-angle glaucoma were examined at 4-month intervals w

289 study, healthy individuals and patients with open-angle glaucoma were prospectively enrolled between

290 itivity sometimes increases in patients with open-angle glaucoma when intraocular pressure (IOP) is d

291 ary 1, 2013, including patients with primary open-angle glaucoma who had a best-corrected visual acui

292 otal of 603 patients (603 eyes) with primary open-angle glaucoma who were using up to 3 glaucoma medi

293 ophotographs from 1 eye of 125 patients with open-angle glaucoma with >/=8 reliable Swedish interacti

294 analysis included 186 patients with primary open-angle glaucoma with a mean age of 59.1 years (range

295 g was performed in 117 patients with primary open-angle glaucoma with a minimum treatment duration of

296 osis and management of patients with primary open-angle glaucoma with an algorithm for patient manage

297 quality of life among patients with primary open-angle glaucoma with structural macular retinal gang

298 FS) is the most common recognizable cause of open-angle glaucoma worldwide.

299 posure is associated with increased risk for open angle glaucoma, yet population-based studies presen

300 as various ocular implications, most notably open angle glaucoma, zonular abnormalities, and cataract