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1 te the underlying autoimmune pathogenesis of ophthalmopathy.
2 lower lid retraction in patients with Graves ophthalmopathy.
3  in patients with active, moderate-to-severe ophthalmopathy.
4 king increases the risk and relapse rate for ophthalmopathy.
5 a major controllable risk factor for thyroid ophthalmopathy.
6 is for the immunologic attack seen in Graves ophthalmopathy.
7 g to the clinical findings typical of Graves ophthalmopathy.
8 sociated with the appearance or worsening of ophthalmopathy.
9 sic questions about the treatment of thyroid ophthalmopathy.
10 intense inflammation associated with Graves' ophthalmopathy.
11                           Thyroid-associated ophthalmopathy, a condition commonly associated with Gra
12                           Thyroid-associated ophthalmopathy and dermopathy are connective tissue mani
13                  The pathogenesis of thyroid ophthalmopathy and its relation to thyroid gland dysfunc
14  in the etiology and pathobiology of Graves' ophthalmopathy and/or other ocular myopathies.
15 of >/=3 indicating active thyroid-associated ophthalmopathy) and a reduction of 2 mm or more in propt
16                                      Graves' ophthalmopathy (GO) affects 50% to 60% of patients with
17      Appearances of and increases in Graves' ophthalmopathy (GO) have been reported after treatment o
18                                       Graves ophthalmopathy (GO) is an autoimmune disease that typica
19 (GD) is a common thyroid disease, and Graves ophthalmopathy(GO) is the most common extra-thyroidal ma
20            The surgical management of Graves ophthalmopathy includes treatment of globe malposition (
21 se undergoing the orbital changes of Graves' ophthalmopathy is often challenging, even as our underst
22 onnective tissues seen in thyroid-associated ophthalmopathy is unknown.
23         Stickler syndrome (hereditary arthro-ophthalmopathy) is the commonest inherited cause of reti
24 ations, for example, Duane syndrome, Graves' ophthalmopathy, may be particularly problematic.
25                              The restrictive ophthalmopathy of TAO may be associated with more sustai
26                                  The Graves' Ophthalmopathy Quality of Life (GO-QOL) scale was comple
27 l Activity Score, and results on the Graves' ophthalmopathy-specific quality-of-life questionnaire.
28                           Thyroid-associated ophthalmopathy (TAO) is a common and debilitating manife
29                           Thyroid-associated ophthalmopathy (TAO), an autoimmune component of Graves'
30 ed in the pathogenesis of thyroid-associated ophthalmopathy (TAO), the orbital manifestation of GD, r
31 ue remodeling linked with thyroid-associated ophthalmopathy (TAO).
32  This process is known as thyroid-associated ophthalmopathy (TAO).
33 ity of the human orbit to thyroid-associated ophthalmopathy (TAO).
34 c markers for more severe thyroid-associated ophthalmopathy (TAO).
35 le in the pathogenesis of thyroid-associated ophthalmopathy (TAO).
36                      In patients with active ophthalmopathy, teprotumumab was more effective than pla
37 vement of the extraocular muscles in Graves' ophthalmopathy, the absence of N-CAM expression on other
38  fat accumulation, such as occurs in Graves' ophthalmopathy, tissue fibrosis, abnormal wound healing,
39  suggests that it may play a role in thyroid ophthalmopathy treatment and prevention.
40 retrospective review of patients with Graves ophthalmopathy undergoing lower eyelid retraction surger
41 al, specific diagnostic criteria for thyroid ophthalmopathy were established and are included in this
42 Medical records for patients with dysthyroid ophthalmopathy who underwent bilateral lateral rectus re

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