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1 te the underlying autoimmune pathogenesis of ophthalmopathy.
2 lower lid retraction in patients with Graves ophthalmopathy.
3 in patients with active, moderate-to-severe ophthalmopathy.
4 king increases the risk and relapse rate for ophthalmopathy.
5 a major controllable risk factor for thyroid ophthalmopathy.
6 is for the immunologic attack seen in Graves ophthalmopathy.
7 g to the clinical findings typical of Graves ophthalmopathy.
8 sociated with the appearance or worsening of ophthalmopathy.
9 sic questions about the treatment of thyroid ophthalmopathy.
10 intense inflammation associated with Graves' ophthalmopathy.
15 of >/=3 indicating active thyroid-associated ophthalmopathy) and a reduction of 2 mm or more in propt
19 (GD) is a common thyroid disease, and Graves ophthalmopathy(GO) is the most common extra-thyroidal ma
21 se undergoing the orbital changes of Graves' ophthalmopathy is often challenging, even as our underst
27 l Activity Score, and results on the Graves' ophthalmopathy-specific quality-of-life questionnaire.
30 ed in the pathogenesis of thyroid-associated ophthalmopathy (TAO), the orbital manifestation of GD, r
37 vement of the extraocular muscles in Graves' ophthalmopathy, the absence of N-CAM expression on other
38 fat accumulation, such as occurs in Graves' ophthalmopathy, tissue fibrosis, abnormal wound healing,
40 retrospective review of patients with Graves ophthalmopathy undergoing lower eyelid retraction surger
41 al, specific diagnostic criteria for thyroid ophthalmopathy were established and are included in this
42 Medical records for patients with dysthyroid ophthalmopathy who underwent bilateral lateral rectus re
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