ライフサイエンスコーパス: opsoclonus

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1 lonus, tongue and orofacial dyskinesias, and opsoclonus.

2 ebellar syndrome, which in 45% occurred with opsoclonus.

3 The presence of opsoclonus, ataxia, and chorea expands the clinical phen

4 ho presented with a mixed movement disorder (opsoclonus, ataxia, and chorea) as well as seizures refr

5 nt who presented with acute-onset confusion, opsoclonus, chorea, and intractable seizures.

6 ub-acute syndromes (transverse myelitis (1), opsoclonus myoclonus (1)).

7 cute syndromes (transverse myelitis [n = 1], opsoclonus myoclonus [n = 1]).

8 Paraneoplastic opsoclonus myoclonus ataxia (POMA) is a neurologic disor

9 at is an autoimmune target in paraneoplastic opsoclonus myoclonus ataxia (POMA) patients with latent

10 Nova-1, an autoantigen in paraneoplastic opsoclonus myoclonus ataxia (POMA), a disorder associate

11 specific antigens targeted in paraneoplastic opsoclonus myoclonus ataxia (POMA), an autoimmune neurol

12 In patients suffering from paraneoplastic opsoclonus myoclonus ataxia (POMA), Nova-1 and Nova-2 pr

13 nces and its association with paraneoplastic opsoclonus, myoclonus, and ataxia.

14 requency, were brainstem syndrome (including opsoclonus, myoclonus, or both), cerebellar syndrome, my

15 Despite circumstantial evidence that opsoclonus-myoclonus (OM) is often immune mediated, no s

16 A-binding protein Nova-1, the paraneoplastic opsoclonus-myoclonus ataxia (POMA) antigen.

17 with the autoimmune disorder paraneoplastic opsoclonus-myoclonus ataxia (POMA).

18 araneoplastic motor disorder [paraneoplastic opsoclonus-myoclonus ataxia (POMA)].

19 ns in the autoimmune disorder paraneoplastic opsoclonus-myoclonus ataxia and contain K-homology (KH)-

20 nditions, with the possible exception of the opsoclonus-myoclonus ataxia and Lambert-Eaton myasthenic

21 with the autoimmune disorder paraneoplastic opsoclonus-myoclonus ataxia, which is characterized by f

22 Syndromes such as limbic encephalitis or opsoclonus-myoclonus should always raise suspicion of a

23 Most studies on opsoclonus-myoclonus syndrome (OMS) in adults are based

24 araneoplastic cerebellar degeneration, and 1 opsoclonus-myoclonus syndrome).

25 echnique in 2 patients recovering from viral opsoclonus-myoclonus syndrome, comparing saccadic-vergen

26 g on paraneoplastic cerebellar degeneration, opsoclonus-myoclonus, and encephalitides affecting the l

27 icated in the pathogenesis of paraneoplastic opsoclonus-myoclonus-ataxia (POMA).

28 Opsoclonus-myoclonus-ataxia syndrome (OMS) is a severe a

29 eizures and limbic involvement), and two had opsoclonus-myoclonus.

30 ies), neuromyotonia (Caspr2 antibodies), and opsoclonus--myoclonus--ataxia (unknown antigens).

31 uncover a novel phenotype of paraneoplastic opsoclonus that until recently was likely considered idi

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