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1 aplastic haemangiopericytoma compressing the optic chiasm.
2 mbryonic brain during formation of the mouse optic chiasm.
3 of cell groups that lie within the nerve and optic chiasm.
4 rve outgrowth, including path-finding at the optic chiasm.
5 commissure, which is located adjacent to the optic chiasm.
6 olfactory tract, mammillothalamic tract, or optic chiasm.
7 halmia and hypoplasia of the optic nerve and optic chiasm.
8 l axon divergence associated with the albino optic chiasm.
9 days after birth (P24) at the centre of the optic chiasm.
10 equired for efficient RGC decussation at the optic chiasm.
11 athfinding of the ganglion cell axons at the optic chiasm.
12 ultimately affects axonal growth through the optic chiasm.
13 s a missorting of RGC axons as they exit the optic chiasm.
14 ome biogenesis and axonal growth through the optic chiasm.
15 lmic disorders affecting the optic nerve and optic chiasm.
16 tes, and anomalous axonal pathfinding at the optic chiasm.
17 axial planes 1-mm thick and parallel to the optic chiasm.
18 with the RGCs themselves, most likely at the optic chiasm.
19 nas still project axons to the brain via the optic chiasm.
20 ltered in the developing Foxg1-/- retina and optic chiasm.
21 Foxg1 is also expressed at the optic chiasm.
22 r contralateral targets, thereby forming the optic chiasm.
23 ral diencephalon during the formation of the optic chiasm.
24 ws from the ventrotemporal retina toward the optic chiasm.
25 ons either cross or avoid the midline at the optic chiasm.
26 ial manifestation of disorders involving the optic chiasm.
27 ons either cross or avoid the midline at the optic chiasm.
28 A/OPCs isolated from cortex, optic nerve and optic chiasm.
29 on for retinal axon growth in the developing optic chiasm.
30 pond by making axonal guidance errors at the optic chiasm.
31 lion cells with uncrossed projections at the optic chiasm.
32 knockout mice and analyzed their retinas and optic chiasms.
33 ecision to cross or avoid the midline at the optic chiasm, a critical guidance maneuver that establis
35 e retina and in the region of the developing optic chiasm, a ventral midline structure in which retin
36 erns of heparan sulfation on RGCs and at the optic chiasm and (2) this differential sulfation directs
37 estigated with reference to disorders of the optic chiasm and anophthalmia (absence of the eyes).
41 rocesses, such as pathfinding of RGCs at the optic chiasm and hippocampal long-term potentiation and
42 (RGC) fibers affects the organization of the optic chiasm and lateral geniculate nuclei (LGN) in huma
43 of the optic nerves, chiasm and tracts, and optic chiasm and LGN volume compared with controls (P <
44 directly control RGC axon divergence at the optic chiasm and may additionally function as a general
51 lateral and contralateral projections at the optic chiasm and the subsequent segregation of retinal i
53 , the body of the optic stalk and nerve, the optic chiasm and ventral diencephalon, and the anterior
54 sal and temporal retinal fibers cross at the optic chiasm, and (2) ocular dominance columns normally
55 ovided identical tracing of the optic nerve, optic chiasm, and optic tracts to the level of the later
56 ptic stalk, cross the ventral midline at the optic chiasm, and terminate in the optic tectum of the z
57 spinal cord, the hindbrain and midbrain, the optic chiasm, and the median eminence in the forebrain.
58 n in significant numbers and fail to form an optic chiasm; and (4) axons in multiple commissural trac
59 n, the mechanisms for axon divergence in the optic chiasm are discussed in the context of other popul
60 tricular and subventricular zones and in the optic chiasm, areas that are rich in oligodendrocyte (OL
61 in determining the relative position of the optic chiasm at the ventral midline of the developing hy
63 e, loss of axonal staining progressed to the optic chiasm by 7 days and remained undetectable at 2 we
65 e and in the pattern of decussation at their optic chiasm, demonstrating that a melanin-related agent
66 nal ganglion cell (RGC) axons at the midline optic chiasm determines whether RGCs project to ipsilate
67 ription factor known for its role in eye and optic chiasm development, causes the rostral oral ectode
78 al axons and the cellular composition of the optic chiasm in albino mice are similar to those of norm
79 Vema is localized to the floor plate and the optic chiasm, intermediate targets located at the ventra
87 axons away from its ligand, ephrinB2, at the optic chiasm midline, and a transcription factor Zic2, t
88 cell (RGC) axons from nasal retina cross the optic chiasm midline, whereas temporal retina axons do n
90 at directs the ipsilateral projection at the optic chiasm, misrouted RGCs target the appropriate reti
91 ation associated with axonal behavior at the optic chiasm must affect ganglion cells in a cell-extrin
96 Brn3b(-/-) mice but missing were entirely in optic chiasms of Brn3b/Brn3c double knockout mice, sugge
97 Retinal axons cross the neuraxis to form the optic chiasm on the hypothalamus in a position defined b
98 lved both in determining the position of the optic chiasm on the ventral diencephalon (presumptive hy
99 ) project axons along the optic nerve to the optic chiasm on the ventral surface of the hypothalamus.
101 on of Vema in the developing spinal cord and optic chiasm resembles the expression patterns of a vari
106 ents this increase, abolishes glutamate- and optic chiasm stimulation-induced phase delays of the SCN
107 al hypothalamus, and in a site dorsal to the optic chiasm that included the suprachiasmatic nucleus.
108 nd therapeutic interventions that damage the optic chiasm, the pituitary stalk and the hypothalamic a
110 abnormal retinal decussation patterns at the optic chiasm: their uncrossed projections are smaller an
111 ficient mice initially fail to grow from the optic chiasm to form optic tracts and are delayed tempor
112 ganglion cell (RGC) axons diverge within the optic chiasm to project to opposite sides of the brain.
113 whether to cross or avoid the midline at the optic chiasm to project to targets on both sides of the
114 s with ephrin-B2 on radial glia cells at the optic chiasm to repulse VT axons away from the midline a
115 agonists, and electrical stimulation of the optic chiasm to SCN brain slices to determine the effect
116 of retinal ganglion cell (RGC) axons at the optic chiasm to the appropriate hemisphere, a pattern cr
117 ding from the area immediately caudal to the optic chiasm to the level of the posterior hypothalamus.
118 sion that retinal ganglion cells make at the optic chiasm, to either cross or avoid the midline.
120 ct retinal axon growth and divergence at the optic chiasm, we cocultured mouse retinal and chiasm exp
121 Ipsilateral and misrouted projections at the optic chiasm were overproduced in Brn3b(-/-) mice but mi
123 eview, we compare guidance mechanisms at the optic chiasm with those in other midline models and high
124 rgence of retinal ganglion cell axons at the optic chiasm, with strictly controlled numbers projectin
126 embryos deficient in GAP-43 have an enlarged optic chiasm within which RGC axons were reportedly stal
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