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1 re followed up with FD-OCT cube scans of the optic disc.
2  on the pathogenesis of rare diseases of the optic disc.
3 eering, Heidelberg, Germany) scanning of the optic disc.
4 led by retrograde biocytin labeling from the optic disc.
5 g the disease based on the appearance of the optic disc.
6 the natural break in Bruch's membrane at the optic disc.
7 ed the entire macula and often encircled the optic disc.
8 ding pituitary hypoplasia and absence of the optic disc.
9 that were near, touching, or surrounding the optic disc.
10 and depends on Shh for its expression at the optic disc.
11 s traction and adhesion above the macula and optic disc.
12  covering a 1.5 x 2-mm2 area centered at the optic disc.
13 ts of age, disease severity, and size of the optic disc.
14  than 4 times as often as progression in the optic disc.
15  1.3, 1.5, and 1.7 mm from the center of the optic disc.
16 l coherence tomography imaging of macula and optic disc.
17 ain axon growth into the fiber layer and the optic disc.
18                   The scans were centered on optic discs.
19  tool for patients with glaucoma with myopic optic discs.
20 6.0, 10.1, 10.2 mm) (P < 0.001), distance to optic disc (3, 5, 5, 5 mm) (P < 0.001), distance to fove
21 nner (2.2 vs. 2.7 mm), more distant from the optic disc (3.2 vs. 2.5 mm) and foveola (4.0 vs. 2.0 mm)
22  87%, P < 0.001), increased mean distance to optic disc (3.3 vs. 5.0 mm, P < 0.001) and foveola (3.1
23 s (N = 80) strabismus (88% versus 64%), pale optic discs (65% versus 27%) and visual field defects (7
24 l glaucoma and optical nerve hypoplasia, and optic disc abnormalities.
25 nd 6.19 +/- 1.80 mm(3), respectively, at the optic disc and 217.4 +/- 43.6 mum and 7.83 +/- 1.55 mm(3
26    The eye underwent 3 scans centered on the optic disc and another 3 scans centered on the macula th
27 red to control eyes was largest close to the optic disc and approximated the level of controls toward
28 rrhages, exudates, neovascularization in the optic disc and elsewhere, drusen, abnormal pigmentation,
29 t basal diameter, and tumor distances to the optic disc and fovea were 7.6, 12.8, 5.2, and 4.6 mm, re
30 etina, depending on location relative to the optic disc and fovea.
31 ach ON at 2 locations (5 mm posterior to the optic disc and just posterior to the optic canal) in pat
32 l coherence tomography (SDOCT) images of the optic disc and macula regions at baseline and at 3 and 6
33       Participants underwent a 3-dimensional optic disc and macula scanning protocol with a prototype
34 dimensional (3D) 6 x 6-mm raster scan of the optic disc and macula, radial, and line scan.
35 e horizontal scans through the center of the optic disc and macula.
36 cle disrupts ERK signaling, which results in optic disc and nerve dysgenesis and, ultimately, ocular
37 xperts on the basis of the appearance of the optic disc and of the corresponding visual field.
38 izontal cross-sectional FD-OCT images of the optic disc and parapapillary retina were obtained in 24
39 focal lesions that were noncontiguous to the optic disc and showed serpiginoid spread.
40                     Normal patterning of the optic disc and stalk and the expression of guidance cues
41 e ocular variables (such as strabismus, pale optic disc and visual field defects) were compared.
42 itis (Uv-N) (n = 143), raised IOP and normal optic disc and/or visual field (Uv-H) (n = 233), and rai
43 ning of the posterior globe, swelling of the optic disc, and bilateral transverse sinus stenosis.
44  by optic nerve degeneration, cupping of the optic disc, and loss of retinal ganglion cells which cou
45 a, coloboma of the iris, retina, choroid and optic disc, and microcornea.
46 xial length, coloboma of the iris and of the optic disc, and severe myopia.
47  the temporal vascular arcades, nasal to the optic disc, and temporal to the macula.
48  algorithm was used to compute 3-dimensional optic disc angiography.
49 nal barrier to fluid migration from cavitary optic disc anomalies can be safely achieved in most pati
50  Fifteen eyes of 15 patients with congenital optic disc anomalies were enrolled in this study.
51                                   Congenital optic disc anomalies were identified in all 15 eyes.
52 chemic optic neuropathies and two congenital optic disc anomalies).
53 structure and the pathogenesis of congenital optic disc anomalies.
54             We describe a distinct excavated optic disc anomaly associated with high myopia and incre
55 ted case of Down syndrome with morning glory optic disc anomaly in literature.
56 ngs compatible with unilateral morning glory optic disc anomaly in the right eye.
57 est naming this entity high myopia-excavated optic disc anomaly.
58 etween CG and the suspects with glaucomatous optic disc appearance (GODA).
59 de in 74.1% of Group A and 75.9% of Group B, optic disc appearance in 85.4% of Group A, and 93% of Gr
60 rol group (CG), patients having glaucomatous optic disc appearance or ocular hypertension, and patien
61 ad intraocular pressure (IOP) >21 mmHg or an optic disc appearance suspicious of glaucoma.
62 nts were suspected to have glaucoma based on optic disc appearance, but the eyes were judged to be no
63 s defined as stable intraocular pressure and optic disc appearance.
64 stalline appearance, and drusen nasal to the optic disc are more likely to have a rare variant in the
65 icant correlation between RNFL thickness and optic disc area (95% confidence interval [CI] = -0.9 to
66 t association between corneal hysteresis and optic disc area (P = .6), cup area (P = .77), vertical c
67 ly been shown to be strongly associated with optic disc area and vertical cup-to-disc ratio, which ar
68                            HRT overestimated optic disc area as compared to SD-OCT.
69                                      Greater optic disc area asymmetry was associated with a larger i
70 s of VCDR variants suggesting that increased optic disc area can significantly contribute to POAG ris
71                               Quality of the optic disc area was better than those of other retinal a
72  pressure (IOP), vertical cup-to-disc ratio, optic disc area, and optic disc cup area.
73 s associated with central corneal thickness, optic disc area, and VCDR were genotyped in 876 OAG case
74                    ONH parameters (including optic disc area, optic cup area, neuroretinal rim area,
75         Five SNPs previously associated with optic disc area, or VCDR, were genotyped in 539 POAG cas
76 lynomial regression model, including age and optic disc area, which accounted for 83.3% of the variat
77 icant association between RNFL thickness and optic disc area.
78 zone areas were expressed as a percentage of optic disc area.
79 tudy chart and a lesion area of less than 12 optic disc areas (DA).
80 sion was detected first as frequently in the optic disc as in the visual field.
81 se had participated in the previous European Optic Disc Assessment Trial (EODAT), a trial on glaucoma
82 testing, autorefraction, A-scan biometry and optic disc assessment.
83 testing, autorefraction, A-scan biometry and optic disc assessment.
84 a (91%), cataract (93%), glaucoma (35%), and optic disc atrophy (25%).
85  high prevalence of cataract, macular edema, optic disc atrophy, and glaucoma.
86 ntral and supero-nasal field remnant and the optic disc became pallor.
87 tal fundus photographs: (1) sharpness of the optic disc border, (2) discontinuity along major vessels
88  microglia persisted and concentrated in the optic disc, but also localized to the retinal periphery.
89 ee 360-degree circular scans centered on the optic disc by the same experienced examiner using the Ci
90 ntraretinal barrier to fluid egress from the optic disc cavitation.
91  degrees -336 degrees ) 2.0 mm away from the optic disc center was the most frequent location where R
92 s were more often gradable in both eyes than optic disc-centered images (P < .001).
93  that reached POAG endpoints for both VF and optic disc change (n = 127) deteriorated more rapidly (-
94 change (n = 74; -0.29 +/- 0.31 dB/y) or only optic disc change (n = 158; -0.12 +/- 0.19 dB/y) had sig
95 s showing only VF change (P = 0.017) or only optic disc change (P < 0.001).
96 es that reached endpoints due to both VF and optic disc change had worse MDRs than eyes displaying ch
97                                     Rates of optic disc change in patients with focal optic disc dama
98 l relationship between them and glaucomatous optic disc changes such as neuroretinal rim thinning/not
99 s repeatable visual field, photography-based optic disc changes, or both.
100 ment of OAG was based on visual field and/or optic disc changes.
101 ble abnormal SAP or glaucomatous progressive optic disc changes.
102 PATIENTS WITHOUT PROGRESSION IN GLAUCOMATOUS OPTIC DISC CHANGES.
103 hat were noted during endoscopy included the optic disc characteristics, anatomic integrity of the re
104 oscopic photographs in the assessment of any optic disc characteristics, for example disc size (mono
105                                              Optic disc colobomas were found in 5 eyes, and the chara
106 (angio-OCT) in morning glory syndrome (MGS), optic disc colobomas, and optic disc pits, and to explor
107  by an automated algorithm from stereoscopic optic disc color photographs of a random sample of 172 s
108                                          The optic disc cube 200 x 200 protocol was used to obtain th
109 erve fiber layer imaging with Cirrus SD-OCT (Optic Disc Cube 200x200) and Heidelberg Retina Tomograph
110  using the fast RNFL program by Stratus, the optic disc cube protocol by Cirrus, and the N-site axona
111 ical cup-to-disc ratio, optic disc area, and optic disc cup area.
112 gnosis, and the comparative diameters of the optic disc cup, experienced observers separately compare
113 tween eyes with presumed large physiological optic disc cupping (pLPC) and eyes with minimal optic di
114  than doubled the known loci associated with optic disc cupping and will allow greater understanding
115 plete success), stable ocular dimensions and optic disc cupping, and no further glaucoma surgery (inc
116 steresis or CCT and quantitative measures of optic disc cupping, suggesting that corneal hysteresis a
117 ssociated with greater visual field loss and optic disc cupping.
118 cause bowing back of the lamina cribrosa and optic disc cupping.
119 f visual field change in patients with focal optic disc damage (mean -0.34, standard deviation [SD] 0
120  of optic disc change in patients with focal optic disc damage (mean -11.70, SD 25.5 x10(-3) mm(2)/ye
121 r) and diffuse (mean +0.01, SD 0.37 dB/year) optic disc damage (P = 0.003, Kruskal-Wallis).
122                          Patients with focal optic disc damage had faster rates of visual field chang
123  on risk factors and presence of progressive optic disc damage on stereophotographs.
124 ure of more than 21 mm Hg and the absence of optic disc damage or abnormal visual field results at th
125 thickness (CCT), and presence of progressive optic disc damage were associated with faster rates of M
126 tic (mean -0.45, SD 20.6 x10(-3) mm(2)/year) optic disc damage, although the differences were not sta
127 red with patients with diffuse and sclerotic optic disc damage, despite similar IOP reductions during
128 ield abnormality or evidence of glaucomatous optic disc damage, or both when not present at baseline.
129 eview determined progression of glaucomatous optic disc damage.
130 change and a tendency toward faster rates of optic disc deterioration when compared with patients wit
131 ickness (P = 0.007) after adjusting for age, optic disc diameter, gender, and refractive error.
132 eral bowing included reduced distance to the optic disc (difference, 1.3 mm; 95% CI, -2.95 mm to 5.51
133 tion) and optic disc (optic nerve head tilt, optic disc dimensions, and peripapillary atrophy) change
134 Eye Study 10-year follow-up who did not have optic disc disease, including glaucoma, were included.
135                             Mean macular and optic disc distance were 4.56 (0-19.9) mm and 4.59 (0-22
136 , pattern dystrophy-like changes (7.5%), and optic disc drusen (2.0%).
137 (19 eyes) with PPE owing to suspected buried optic disc drusen (ODD), and 3 children (6 eyes) with PP
138 tive correlation between the diameter of the optic disc drusen and the global retinal nerve fiber lay
139 demonstrated the internal characteristics of optic disc drusen and their relationship with the lamina
140 streaks, pattern dystrophy-like changes, and optic disc drusen are a consistent finding in seven stud
141 gative correlation between proportion of the optic disc drusen area occupied by optic nerve drusen as
142                     The mean diameter of the optic disc drusen as measured in OCT images was 686.8 (s
143 EDI-OCT and swept source OCT showed multiple optic disc drusen at different levels; most were located
144          Twenty-six eyes of 15 patients with optic disc drusen were evaluated.
145 chemic fundus and retinal lesions and of the optic disc during the acute phase showed no statisticall
146 4%]), (sub)retinal hemorrhage (n = 6 [12%]), optic disc edema (n = 3 [6%]), chorioretinal lesions (n
147 ents with optic nerve head drusen (ONHD) and optic disc edema (ODE) compared with healthy participant
148  and presented with normal visual acuity but optic disc edema and an enlarged blind spot in the right
149                                              Optic disc edema and epiretinal membrane formation was f
150 d the need for remediation for misdiagnosing optic disc edema during end-rotation funduscopic simulat
151 initial evaluation revealed severe bilateral optic disc edema with distal lower-extremity sensory and
152 ncluding uveitis, hemorrhagic complications, optic disc edema, and dry eye syndrome.
153 hthalmic changes, including choroidal folds, optic disc edema, cotton-wool spots, globe flattening, a
154 dings have included unilateral and bilateral optic disc edema, globe flattening, choroidal and retina
155  from 1 degrees to 4 degrees temporal to the optic disc edge using a computer-aided, manual segmentat
156 planation tonometry, gonioscopy, pachymetry, optic disc evaluation, and automated perimetry.
157                                              Optic disc evaluation, which is fundamental to the diagn
158 on, including gonioscopy, dilated fundus and optic disc examination, visual fields, stereo disc photo
159 atures were found, including nonglaucomatous optic disc excavation (20%), relatively low (<10 mmHg) i
160 LPC and individuals with minimal excavation (optic disc excavation within normal limits; control grou
161 ic disc cupping (pLPC) and eyes with minimal optic disc excavation.
162 eripapillary circle (1.7-mm radius) and cube optic disc fdOCT scans were obtained on 208 eyes from 11
163 es), were selected to demonstrate a range of optic disc features from a total of 197 eyes of 197 pati
164 ssess image quality, the ease of visualizing optic disc features important for glaucoma diagnosis, an
165 eles previously associated with POAG or with optic disc features in other cohorts were compared betwe
166  Visual acuity (VA), pupillary reaction, and optic disc findings were assessed at presentation and 3
167 sual field first in 28 eyes (15%) and in the optic disc first in 34 eyes (18%); in 1 eye (1%), it occ
168 ual field first in 163 eyes (52%) and in the optic disc first in 39 eyes (12%); in 1 eye (0%), it was
169 ty, and normal population variability of the optic disc flow index were 1.2%, 4.2%, and 5.0% CV, resp
170 t retinal function in a wide zone around the optic disc, giving rise to massive enlargement of the ph
171  ocular hypertension (OHT) and glaucoma-like optic discs (GLD)- defined as a cup to disc ratio greate
172 ous features, and VF data were combined with optic disc grading to determine "disc plus field defined
173                                              Optic disc grading was available for 25,289 (93 %) eyes
174 sual acuity of worse than 6/12 or suspicious optic discs had detailed examination including Goldmann
175 ucoma progression, eyes with a history of an optic disc hemorrhage (DH) confirmed by stereophotograph
176     To determine the cumulative incidence of optic disc hemorrhage (ODH) before and after development
177 ether phacoemulsification is associated with optic disc hemorrhage in patients with glaucoma.
178 ffected individuals presented with excavated optic discs, high myopia (-1.00 to -16.00 diopters), and
179  (HR, 1.03; 95% CI, 1.01-1.04; P = .001) and optic disc (HR, 1.01; 95% CI, 1.00-1.01; P = .005), and
180                              A subset of 100 optic disc images from both methods were further used to
181                                 Stereo color optic disc images in both digital and 35-mm slide film f
182 its role in anterior segment vasculature and optic disc imaging has been limited thus far.
183 l in 133 eyes (94.3%), were noncontiguous to optic disc in 122 eyes (86.52%), and involved the macula
184 er was prominent on the temporal side of the optic disc in ADOA (P <0.0001), but there was considerab
185 luorescence that extended nasally beyond the optic disc in both eyes.
186 wed no filling of the entire choroid and the optic disc in both groups of animals.
187 l ganglion cell (RGC) axons grow towards the optic disc in the central retina, where they turn to exi
188         Congenital cavitary anomalies of the optic disc, including typical coloboma, optic pit (and o
189  45 degrees areas centered on the macula and optic disc is 4.7%.
190 mation with indistinct borders, vascular and optic disc leakage, vessel wall staining, or capillary n
191                                          The optic disc locations of the RNFL defects were identified
192 ns were independently evaluated for quality: Optic disc, macula, and superior and inferior vascular a
193            The peculiar features of cavitary optic disc maculopathy can be explained only by consider
194                                     Cavitary optic disc maculopathy develops when fluctuating pressur
195 retinal rim assessment based on the clinical optic disc margin (DM) lacks a sound anatomic basis for
196 rane opening (BMO), rather than conventional optic disc margin (DM)-based assessment or retinal nerve
197                  On fundus examination, left optic disc margin was blurred.
198                                 Conventional optic disc margin-based neuroretinal rim measurements la
199 ripapillary retinal nerve fiber layer (RNFL)/optic disc measurements and a reliable 24-2 SITA-Standar
200 re than 3 mm (P = 0.002), mean distance from optic disc more than 4 mm (P<0.001), and mean maximal ba
201 s a diagnostic challenge because of atypical optic disc morphology and visual field defects that can
202 whereas glaucoma eyes without progression in optic disc morphology lost 1.18 mum per year in RNFL thi
203                                              Optic disc morphology might not be a potential anatomica
204 D) (spherical equivalent) and typical myopic optic disc morphology, with and without glaucoma, were r
205 s was compared with morphological changes of optic disc morphology.
206  visual field mean deviation (MD) and global optic disc neuroretinal rim area with follow-up time.
207       A cross-sectional study comprising 189 Optic Disc (OD) centred retinal images of healthy and di
208 B colorectal cancer, who developed bilateral optic disc oedema and associated left sided optic neurop
209                To report a case of bilateral optic disc oedema and associated optic neuropathy in the
210 tal fundus images centered on the macula and optic disc of 213 patients were graded.
211 re was impaired perfusion and leakage at the optic disc on fluorescein angiography immediately after
212 ial on glaucoma diagnostic accuracy based on optic discs only.
213 and myopic choroidal neovascularization) and optic disc (optic nerve head tilt, optic disc dimensions
214 rative evidence for vitreous traction on the optic disc or macula was seen in any eye.
215 , shorter distance between the tumor and the optic disc (P=0.026), subretinal fluid (P=0.035), thickn
216 e posterior globe [p=0.491], swelling of the optic disc [p=0.881], and bilateral dural sinus stenosis
217                                              Optic disc pallor in 9 eyes (12%) and peripapillary nodu
218 me-wide association meta-analysis of IOP and optic disc parameters and validated our findings in mult
219 erated by the new SSADA, repeatably measures optic disc perfusion and may be useful in the evaluation
220 rence tomography (FDOCT) was used to measure optic disc, peripapillary retinal nerve fiber layer (NFL
221                                          The optic disc, peripapillary retinal nerve fiber layer (NFL
222 ane opening (BMO) algorithm and stereoscopic optic disc photograph readings by glaucoma specialists.
223 tographs for glaucoma likelihood, monoscopic optic disc photographs did not appear to represent a sig
224    For expert observers in the evaluation of optic disc photographs for glaucoma likelihood, monoscop
225              Each participant matched stereo optic disc photographs of 40 healthy and 48 glaucomatous
226                    A series of 42 monoscopic optic disc photographs of healthy and glaucomatous eyes
227 thalmologists correctly matched stereoscopic optic disc photographs to their corresponding visual fie
228 ologists for correctly matching stereoscopic optic disc photographs to their visual fields was 58.7%.
229 ntral corneal thickness (CCT) were measured; optic disc photographs were analyzed; and multivariable
230                 Localized LC lesions seen on optic disc photographs were identified as focal LC defec
231                                              Optic disc photographs were read by 2 masked glaucoma sp
232                                 Stereoscopic optic disc photographs, acquired for each individual, we
233 tructed 3-dimensionally, and superimposed on optic disc photographs.
234 TS) participants annually using stereoscopic optic disc photographs.
235 pendently assessed optic disc progression in optic disc photographs.
236 essment by 3 clinicians of visual fields and optic disc photographs.
237  by glaucoma experts on masked evaluation of optic disc photographs.
238                    All eyes had stereoscopic optic disc photography and in vivo LC imaging using enha
239 metry (SAP), Cirrus SD-OCT, and stereoscopic optic disc photography within 6 months.
240 nitored by VF testing, quantitative imaging, optic disc photography, and tonometry at 11 visits.
241  testing included bilateral color fundus and optic disc photography, fundus autofluorescence, automat
242 andard automated perimetry, and stereoscopic optic disc photography.
243                  Initial and last acceptable optic disc photos were analyzed.
244  analyze the morphologic changes seen in the optic disc pit and evaluate the source of subretinal flu
245 was performed on a 15-year-old boy with deep optic disc pit and foveal detachment, before and for 10
246 partial thickness fenestration radial to the optic disc pit was associated with retinal anatomic and
247                                              Optic disc pit with associated maculopathy is a known en
248 he gap in the lamina cribrosa present in the optic disc pit, supporting the hypothesis of cerebrospin
249 ckness inner retinotomy just temporal to the optic disc pit.
250 ectomy for foveal detachment associated with optic disc pit.
251  of serous macular detachment resulting from optic disc pits as well as combined surgery.
252 or serous macular detachment associated with optic disc pits were evaluated in this study.
253 ry syndrome (MGS), optic disc colobomas, and optic disc pits, and to explore possible correlations be
254    The rate of rim area loss in eyes with an optic disc POAG endpoint was significantly faster than i
255 litative analysis of the optic nerve sheath, optic disc, posterior globe, and pituitary gland morphol
256   Evaluation of visual field progression and optic disc progression during an 8-year follow-up period
257         Three graders independently assessed optic disc progression in optic disc photographs.
258 illedema, 37 eyes with congenitally elevated optic disc (pseudopapilledema), and 34 normal eyes met t
259 0.95, 0.99, 0.87, and 0.93 for 3D macula, 3D optic disc, radial, and line scans, respectively.
260 0, and 0.91, respectively, for 3D macula, 3D optic disc, radial, and line scans.
261 e Ocular Hypertension Treatment Study (OHTS) Optic Disc Reading Group and the OHTS Endpoint Committee
262 nts (CGE) was delivered to the tumor and the optic disc received a minimum of 50 CGE.
263 s, greater cup-to-disc area ratio, and lower optic disc rim area (P < 0.001 for all).
264 tinal thickness, cup-to-disc area ratio, and optic disc rim area.
265 ous visual field loss combined with matching optic disc rim thinning and an enlarged cup-to-disc (C:D
266 ON was determined based on morphology of the optic disc, rim and retinal nerve fiber layer at the tim
267                                              Optic disc, RNFL, and VF showed no statistically signifi
268 ng was the most common artifact on 3D scans (optic disc scan, 7%; macula scan, 9%), whereas segmentat
269 ty-two normal subjects underwent macular and optic disc scanning in both eyes with Cirrus high-defini
270 inating the closure of the optic fissure and optic disc specification, which is necessary for the out
271 uded change in corneal diameter and clarity, optic disc status, refraction, need for anti-glaucoma th
272 try [SAP] and by masked assessment of serial optic disc stereophotographs by expert graders.
273 igher proportion of eyes with progression by optic disc stereophotographs compared with the OLS metho
274                                              Optic disc stereophotographs were co-localized to SD-OCT
275  annually with standard automated perimetry, optic disc stereophotographs, and scanning laser polarim
276 ed with standard achromatic perimetry (SAP), optic disc stereophotographs, confocal scanning laser op
277 a was diagnosed based on a masked grading of optic disc stereophotographs.
278 nic uveitis (9 patients), isolated bilateral optic disc swelling (1 patient), and Parinaud syndrome (
279             Posterior segment signs included optic disc swelling (95.0%) and retinal striae (85.0%).
280 tracranial pressure to findings in eyes with optic disc swelling caused by optic neuritis and nonarte
281 tive afferent pupillary defect, (4) observed optic disc swelling, and (5) no other etiology being fou
282  included anterior and intermediate uveitis, optic disc swelling, and white-yellowish choroidal lesio
283 tereoscopic photographs and OCT scans of the optic discs taken during the same visit were compared.
284 im health according to the appearance of the optic disc, the clinically visible surface of the ONH.
285 ctly by regulating axon guidance cues at the optic disc through patterning of the optic stalk.
286 uxtapapillary choroidal melanoma (</=1 mm to optic disc) treated with plaque radiotherapy from Octobe
287 er and thickness, distance to the foveola or optic disc, tumor calcification, central macular thickne
288  underwent circular OCT scans centred at the optic disc using a Spectralis OCT (Heidelberg Engineerin
289                   We induced ischemia at the optic disc via laser-activated photochemical thrombosis,
290 d for three definitions of glaucoma based on optic disc, visual field, and a combination of both.
291 ty to the foveola was 3.0 mm and that to the optic disc was 1.8 mm.
292                                          The optic disc was imaged with SD OCT at approximately 4-mon
293    Severe PCO was defined if the view of the optic disc was obscured, or neodymium-yttrium-aluminum-g
294    A curved demarcation line inferior to the optic disc was observed on SW-AF images in 31/32 subject
295 papillary RNFL circular scan centered on the optic disc was obtained from right eyes of 25 preterm ch
296 optical coherence tomography scanning of the optic disc, was carried out between January 2013 and Nov
297                           The fundus and the optic disc were evaluated by repeated ophthalmoscopy, co
298                     Stereophotographs of the optic disc were obtained at baseline and compared with t
299 l coherence tomography scans surrounding the optic disc were performed in each eye of patients and su
300 ith 2 healthy subjects with normal-appearing optic disc within 5 years of age.

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