ライフサイエンスコーパス: optic neuropathy

1 al vasculitis, 3 papilledema, 2 infiltrative optic neuropathy).

2 wing a variable eye phenotype (i.e. uveitis, optic neuropathy).

3 sive investigation ruled out other causes of optic neuropathy.

4 ogression and the development of compressive optic neuropathy.

5 the most common cause of Leber's hereditary optic neuropathy.

6 e measure was the development of compressive optic neuropathy.

7 gns of chronic angle damage and glaucomatous optic neuropathy.

8 er retina with age and in a model of chronic optic neuropathy.

9 tic role of 5-fluoruracil in 5-FU associated optic neuropathy.

10 tivation and cell death in a rodent model of optic neuropathy.

11 ermitted visualization of the characteristic optic neuropathy.

12 in eye movements, accommodation problems, or optic neuropathy.

13 tic nerve oedema and a subsequent left sided optic neuropathy.

14 e predictive of visual recovery in traumatic optic neuropathy.

15 can lead to the development of glaucomatous optic neuropathy.

16 tion of neurodegeneration that occurs during optic neuropathy.

17 ompressive optic neuropathy in patients with optic neuropathy.

18 e development or progression of glaucomatous optic neuropathy.

19 medial wall contour were not associated with optic neuropathy.

20 tal recti diameter (P = 0.016) predictive of optic neuropathy.

21 development and progression of glaucomatous optic neuropathy.

22 that cause pain, disfigurement, diplopia, or optic neuropathy.

23 to be a significant independent predictor of optic neuropathy.

24 nd in patients with papilledema and ischemic optic neuropathy.

25 from either typical clinical or sub-clinical optic neuropathy.

26 gic manifestations, as in Leber's hereditary optic neuropathy.

27 set retinal dysfunction with similarities to optic neuropathy.

28 he likelihood of neurologic and glaucomatous optic neuropathy.

29 formation of cataracts or radiation-induced optic neuropathy.

30 most common neuro-ophthalmic presentation is optic neuropathy.

31 treatment of patients with Leber hereditary optic neuropathy.

32 d irreversible vision loss from glaucomatous optic neuropathy.

33 omitant epiretinal membrane and glaucomatous optic neuropathy.

34 ive optic neuropathy, and Leber's hereditary optic neuropathy.

35 iations with nonarteritic anterior ischaemic optic neuropathy.

36 eatments for nonarteritic anterior ischaemic optic neuropathy.

37 ctive of bioenergetic loss in cell models of optic neuropathy.

38 ons using standard criteria for glaucomatous optic neuropathy.

39 To produce a mouse model of Leber hereditary optic neuropathy.

40 treatment of patients with Leber hereditary optic neuropathy.

41 ive damage contributes early to glaucomatous optic neuropathy.

42 al ganglion cells in a model of glaucomatous optic neuropathy.

43 diation-induced retinopathy, maculopathy, or optic neuropathy.

44 nal membranes, with and without glaucomatous optic neuropathy.

45 hronic development of bilateral glaucomatous optic neuropathy.

46 , and 3 patients developed an EVD-associated optic neuropathy.

47 agnostic testing in patients with this acute optic neuropathy.

48 ransaminase elevation, and 1 (4.8%) ischemic optic neuropathy.

49 diagnosis and follow-up of patients with an optic neuropathy.

50 perfusion, which suggests early glaucomatous optic neuropathy.

51 ion between amiodarone and the occurrence of optic neuropathy.

52 considered for the novel indication of human optic neuropathy.

53 exposure in the development of glaucomatous optic neuropathy.

54 assess color identification in patients with optic neuropathy.

55 al share of families with unsolved inherited optic neuropathies.

56 tients with glaucomatous and nonglaucomatous optic neuropathies.

57 t than in other quadrants in nonglaucomatous optic neuropathies.

58 skills in the evaluation of nonglaucomatous optic neuropathies.

59 NFL in both glaucomatous and nonglaucomatous optic neuropathies.

60 ns of RNFL loss in different nonglaucomatous optic neuropathies.

61 e a ready source of replacement RGCs in such optic neuropathies.

62 lls (RGCs) die in glaucoma and virtually all optic neuropathies.

63 ically and genetically heterogenous group of optic neuropathies.

64 neurodegeneration in glaucoma and prevalent optic neuropathies.

65 henotyping of the heterogeneous inflammatory optic neuropathies.

66 to acquire retinal images from patients with optic neuropathy: (1) adaptive optics (AO)-flood-illumin

67 ischemic optic neuropathy (25%), compressive optic neuropathy (18.7%) and hereditary optic neuropathy

68 sive optic neuropathy (18.7%) and hereditary optic neuropathy (18.7%).

69 radiation maculopathy (43.1%) and radiation optic neuropathy (20.8%) developing at a mean of 27 mont

70 The most common diagnoses were ischemic optic neuropathy (25%), compressive optic neuropathy (18

71 thy (66% of patients), followed by radiation optic neuropathy (51% of patients).

72 ities (eg, amblyopia, nystagmus, foveopathy, optic neuropathy) accounted for residual postoperative s

73 Acute optic neuritis is the most common optic neuropathy affecting young adults.

74 Optic neuropathy after PPV for macula-sparing primary RR

75 Twenty-two subjects with optic neuropathy (aged 18-65) and 18 control subjects we

76 l changes following murine anterior ischemic optic neuropathy (AION) by using spectral-domain optical

77 report a case of bilateral anterior ischemic optic neuropathy (AION) showing histopathologic evidence

78 pared: 30 normals, 20 with anterior ischemic optic neuropathy (AION), and 25 with papilledema and int

79 Eyes with MME were compared with eyes with optic neuropathy alone and to healthy fellow eyes.

80 eatments for nonarteritic anterior ischaemic optic neuropathy also need further confirmatory studies.

81 lowing future studies on disease modeling of optic neuropathies and development of cell therapies.

82 al purposes to assess disease progression in optic neuropathies and diseases that affect the central

83 nstem features mainly, two had demyelinating optic neuropathies and one had an unclear diagnosis.

84 th that are common in the existing models of optic neuropathies and optic nerve lesions.

85 regarding the use of OCT in nonglaucomatous optic neuropathies and sheds light on common patterns of

86 were misdiagnosed as glaucoma (two ischemic optic neuropathies and two congenital optic disc anomali

87 174 eyes of 110 patients having glaucomatous optic neuropathy and 196 eyes of 101 subjects being norm

88 dred eyes from 74 patients with glaucomatous optic neuropathy and a 24-2 VF with mean deviation bette

89 Glaucoma is a progressive optic neuropathy and a leading cause of blindness.

90 m responsible for RGC early degeneration and optic neuropathy and linking RTN4IP1 functions to mitoch

91 onal deficiency definitively proved to cause optic neuropathy and loss of vision.

92 The study of optic neuropathy and neurologic manifestations of demyel

93 The pattern of RNFL thinning in ischemic optic neuropathy and optic nerve head drusen is more lik

94 trocytes and genes related with glaucomatous optic neuropathy and other neural disorders.

95 tified risk factors associated with ischemic optic neuropathy and prone spinal fusion surgery.

96 o investigate the mechanisms of glaucomatous optic neuropathy and search for IOP-independent neuropro

97 function and death, as in Leber's hereditary Optic Neuropathy and suggests novel therapeutic routes t

98 estions regarding both the mechanisms of the optic neuropathy and the treatment of these patients.

99 gical conditions that may mimic glaucomatous optic neuropathy and to determine which most often lead

100 t additionally lead to ocular motor palsies, optic neuropathies, and orbital pathologies.

101 , death, enucleation, radiation retinopathy, optic neuropathy, and best-corrected visual acuity (BCVA

102 d as POAG (open drainage angle, glaucomatous optic neuropathy, and glaucomatous field defect), glauco

103 s ability to correctly classify glaucomatous optic neuropathy, and HRT's role in monitoring disease p

104 graine, optic nerve head drusen, compressive optic neuropathy, and Leber's hereditary optic neuropath

105 atment of glaucoma, nonglaucomatous forms of optic neuropathy, and perhaps other CNS neurodegeneratio

106 attributed to radiation-induced retinopathy, optic neuropathy, and secondary glaucoma.

107 ermined age, sex, visual acuity, etiology of optic neuropathy, and the temporal and spatial character

108 Optic neuropathies are associated with death of retinal

109 Optic neuropathies are characterised by a loss of retina

110 The mechanisms underlying optic neuropathies are diverse and typically manifest wi

111 Autosomal-recessive optic neuropathies are rare blinding conditions related

112 Disorders of the optic nerves (optic neuropathies) are some of the most common causes o

113 se, including glaucomatous and mitochondrial optic neuropathies, are linked increasingly to dysfuncti

114 l optic neuropathies-especially inflammatory optic neuropathies-are associated with neurological diso

115 scans, was equally predictive of compressive optic neuropathy as the more involved volumetric calcula

116 indicator for nonarteritic anterior ischemic optic neuropathy as well as for retinal vascular events.

117 ntral nervous system (CNS) demyelination and optic neuropathy, as determined by changes in visual evo

118 re, including retinopathy, choroidopathy and optic neuropathy, as well as with an increased risk of o

119 aimed at modifying risk factors for ischemic optic neuropathy associated with prone spinal fusion sur

120 Eyes affected by glaucomatous optic neuropathy at presentation were excluded.

121 Patients with compressive optic neuropathy at the time of presentation were exclud

122 nt differences in prevalence of glaucomatous optic neuropathy between African Americans (AAs) and Cau

123 t the use of idebenone in Leber's hereditary optic neuropathy, but this has not been evaluated in a r

124 nerve head perfusion and cause glaucomatous optic neuropathy by creating transient hypoxemia and inc

125 ewed the charts of consecutive patients with optic neuropathies caused by neurophthalmological condit

126 Glaucomatous optic neuropathy causes blindness through the degenerati

127 oma comprises a distinctive group of chronic optic neuropathies, characterised by the progressive los

128 The glaucomas are a group of progressive optic neuropathies characterized by degeneration of reti

129 Glaucoma is a progressive optic neuropathy characterized by loss of retinal gangli

130 Glaucoma is a multifactorial optic neuropathy characterized by retinal ganglion cell

131 Glaucoma is an optic neuropathy characterized by retinal ganglion cells

132 Glaucoma is an optic neuropathy, commonly associated with elevated intr

133 This raises the possibility that the optic neuropathy could be the result of secondary mtDNA

134 wo novel principles of genetic mechanisms in optic neuropathies: deep intronic OPA1 mutations, which

135 ed on clinical examination with glaucomatous optic neuropathy defined by the presence of neuroretinal

136 atient also developed bilateral glaucomatous optic neuropathy despite a well-controlled intraocular p

137 During the observational period, optic neuropathy developed in 17 amiodarone-treated pati

138 Anterior ischemic optic neuropathy developed in one patient in the group t

139 darone use remained a significant factor for optic neuropathy development among male subjects (HR, 3.

140 iated with a nearly 3-fold increased risk of optic neuropathy development compared with female gender

141 a wide range of other neurodegenerative and optic neuropathy diseases.

142 enlargement in the development of dysthyroid optic neuropathy (DON) in Graves' orbitopathy (GO).

143 Dysthyroid optic neuropathy (DON) is the commonest cause of blindne

144 trial in 85 patients with Leber's hereditary optic neuropathy due to m.3460G>A, m.11778G>A, and m.144

145 Several optic neuropathies-especially inflammatory optic neuropa

146 es such as beriberi, neurological disorders, optic neuropathy, etc.

147 ies who self-registered for the Glaucomatous Optic Neuropathy Evaluation (GONE) Project from December

148 olfactomedin (OLF) domain are linked to the optic neuropathy glaucoma.

149 re, the critical stressor in the most common optic neuropathy, glaucoma.

150 re, the critical stressor in the most common optic neuropathy, glaucoma.

151 on the basis of the presence of glaucomatous optic neuropathy (GON) and 24-2 visual field abnormaliti

152 One eye of 286 patients with glaucomatous optic neuropathy (GON) and 289 age-matched participants

153 ents (57.4 +/- 13.2 years) with glaucomatous optic neuropathy (GON) and 45 eyes of 45 controls (48.0

154 ng healthy eyes from those with glaucomatous optic neuropathy (GON) can be optimized by training with

155 hs of subjects with and without glaucomatous optic neuropathy (GON) followed during the first 13 year

156 ssion in persons with glaucomatous-appearing optic neuropathy (GON) from the Diagnostic Innovations i

157 otographs were graded as either glaucomatous optic neuropathy (GON) or normal by two independent mask

158 dy (ADAGES) with a diagnosis of glaucomatous optic neuropathy (GON) or ocular hypertension (OHT) and

159 the feasibility of identifying glaucomatous optic neuropathy (GON) using IOP corrected and uncorrect

160 One hundred eleven eyes with glaucomatous optic neuropathy (GON), 31 with progressive optic neurop

161 rative diabetic retinopathy and glaucomatous optic neuropathies (GONs).

162 t optic atrophy + group compared to the pure optic neuropathy group, implicating a causal role for th

163 average of 3.2 years follow-up, compressive optic neuropathy had developed in 17% (25/144) of Group

164 At last follow-up, all 7 patients with optic neuropathy had visual acuity less than 20/200, rel

165 gher daily dose did not increase the risk of optic neuropathy (HR, 0.96; 95% CI, 0.91-1.00; P = 0.07)

166 ated patients had a 2-fold increased risk of optic neuropathy (HR, 2.09; 95% CI, 1.13-3.85; P = 0.02)

167 ith prone spinal fusion surgery and ischemic optic neuropathy identified in a large multicenter case-

168 OCT has the potential to monitor progressive optic neuropathies in young children who have difficulty

169 Retinopathy was seen in 31 patients (37.8%), optic neuropathy in 12 (14.6%), and cataracts in 26 (31.

170 taract in 19, glaucoma in 9, nonglaucomatous optic neuropathy in 3, corneal opacities in 3, retinal d

171 We demonstrated an age-related optic neuropathy in AD by OCT, with a significant reduct

172 f ocular complications including uveitis and optic neuropathy in EVD survivors, level of VA impairmen

173 These results demonstrated a higher risk of optic neuropathy in patients treated with amiodarone, es

174 ortant quantifiable predictor of compressive optic neuropathy in patients with optic neuropathy.

175 red as a possible contributing factor in the optic neuropathy in the DBA/2J mouse model of glaucoma.

176 f bilateral optic disc oedema and associated optic neuropathy in the setting of FOLFOX chemotherapy.

177 The treatment of mitochondrial optic neuropathy in the US is only supportive.

178 blindness worldwide, is a neurodegenerative optic neuropathy in which vision loss is caused by loss

179 ic atrophy (ADOA), the most common inherited optic neuropathy, in which retinal ganglion cells (RGCs)

180 umetric univariate predictors of compressive optic neuropathy included medial rectus volume (P = 0.00

181 as a result of axonal injury in a variety of optic neuropathies, including glaucoma.

182 ar geometry are associated with glaucomatous optic neuropathy independently of vascular risk factors

183 vivo neuroprotective effects of NIL against optic neuropathy induced by mitochondrial complex I inhi

184 Expression of human ND4 and rescue of optic neuropathy induced by mutant human ND4.

185 The cause of ischemic optic neuropathy (ION) as the most complex entity of POV

186 It is a sign of optic neuropathy irrespective of its etiology.

187 Radiation-induced optic neuropathy is an expected issue after proton beam

188 Compressive optic neuropathy is associated with significantly thinne

189 Non-arteritic anterior ischemic optic neuropathy is caused by a transient optic nerve is

190 Glaucomatous optic neuropathy is characterized by remodeling of the e

191 Traumatic optic neuropathy is common clinical problem for neuro-op

192 optic disc oedema and associated left sided optic neuropathy is described.

193 complex neurological phenotypes that involve optic neuropathy is puzzling.

194 Leber's hereditary optic neuropathy is the most common mitochondrial DNA di

195 of blindness worldwide and, along with other optic neuropathies, is characterized by loss of retinal

196 open-angle glaucoma (POAG), the most common optic neuropathy, is a heritable disease.

197 gene responsible for 70% of Leber Hereditary Optic Neuropathies (LHON).

198 Leber's hereditary optic neuropathy (LHON) and a multiple sclerosis (MS)-li

199 tic neuropathies, such as Leber's hereditary optic neuropathy (LHON) and Autosomal dominant optic atr

200 l strategy for treatment of Leber hereditary optic neuropathy (LHON) caused by a mutation in the nico

201 ndrial gene responsible for Leber hereditary optic neuropathy (LHON) into the mouse germ line using f

202 Leber hereditary optic neuropathy (LHON) is a disorder characterized by s

203 Leber hereditary optic neuropathy (LHON) is a genetic disorder primarily

204 Leber's hereditary optic neuropathy (LHON) is a maternally inherited blindi

205 Leber's hereditary optic neuropathy (LHON) is a mitochondrial disease affec

206 Leber hereditary optic neuropathy (LHON) is due primarily to one of three

207 natural history of G11778A Leber hereditary optic neuropathy (LHON) is important to determine the op

208 Leber's hereditary optic neuropathy (LHON) is the most common mitochondrial

209 Leber's hereditary optic neuropathy (LHON) is the most common mitochondrial

210 Leber's hereditary optic neuropathy (LHON) is typically characterized by va

211 An animal model of Leber hereditary optic neuropathy (LHON) was produced by introducing the

212 Leber's hereditary optic neuropathy (LHON), a mitochondrial disease, has cl

213 F)-P1ND4v2 in patients with Leber hereditary optic neuropathy (LHON).

214 syndrome (NARP-MILS), and Leber's hereditary optic neuropathy (LHON).

215 n Han Chinese family with Leber's hereditary optic neuropathy (LHON).

216 te the natural history of Leber's hereditary optic neuropathy (LHON).

217 Common mutations for Leber's hereditary optic neuropathy (LHON: G11778A; T14484C; and G3460A) we

218 NA levels responsible for Leber's hereditary optic neuropathy (LHOND), and neurogenic muscle weakness

219 n (2%), partial choroidal ischemia (2%), and optic neuropathy (<1%).

220 Optic neuropathies may range from non-syndromic genetic

221 cell death in rodent glaucoma and traumatic optic neuropathy models.

222 ; n = 3), and nonarteritic anterior ischemic optic neuropathy (n = 1) in the repositioning group, and

223 drome (OIS), non-arteritic anterior ischemic optic neuropathy (NA-AION) and amaurosis fugax (AF).

224 management of nonarteritic anterior ischemic optic neuropathy (NA-AION) and central retinal artery oc

225 euritis (14), nonarteritic anterior ischemic optic neuropathy (NAION) (21), and ONH swelling (average

226 patients with nonarteritic anterior ischemic optic neuropathy (NAION) and its influence on second eye

227 ts with acute nonarteritic anterior ischemic optic neuropathy (NAION) and normal age-related cataract

228 Nonarteritic anterior ischemic optic neuropathy (NAION) is a devastating ocular conditi

229 Nonarteritic anterior ischemic optic neuropathy (NAION) is an optic nerve (ON) stroke a

230 Nonarteritic anterior ischemic optic neuropathy (NAION) is an optic nerve infarct invol

231 To date, non arteritic anterior ischemic optic neuropathy (NAION) is still incurable.

232 Nonarteritic anterior ischemic optic neuropathy (NAION) results from isolated anterior

233 Nonarteritic anterior ischemic optic neuropathy (NAION) results in optic nerve damage w

234 diagnosis of nonarteritic anterior ischemic optic neuropathy (NAION) seen in the Wilmer Eye Network

235 eater risk of nonarteritic anterior ischemic optic neuropathy (NAION) than nondiabetic patients.

236 r unilateral non-arteritic anterior ischemic optic neuropathy (NAION) will eventually develop the sam

237 cerebri), (3) nonarteritic anterior ischemic optic neuropathy (NAION), (4) optic nerve head drusen wi

238 neuritis and nonarteritic anterior ischemic optic neuropathy (NAION), conditions without intracrania

239 ups: normal, glaucoma, nonarteritic ischemic optic neuropathy (NAION), treated proliferative diabetic

240 phy (OCT), in nonarteritic anterior ischemic optic neuropathy (NAION).

241 ge following non-arteritic anterior ischemic optic neuropathy (NAION).

242 ith chronic non-arteritic anterior ischaemic optic neuropathy (NAION).

243 cts, including corneal microdeposits (>90%), optic neuropathy/neuritis (< or =1%-2%), blue-gray skin

244 VEPs can be valuable in diagnosing optic neuropathies, nonorganic visual loss, and assessin

245 1; 95% CI, 1.34-100.56; P = .03) or ischemic optic neuropathy (odds ratio, 5.27; 95% CI, 1.61-17.23;

246 The genetic diagnosis in inherited optic neuropathies often remains challenging, and the em

247 ultiple sclerosis with chronic demyelinating optic neuropathy on stable immunomodulatory therapy.

248 Evidence of glaucomatous optic neuropathy on stereophotographs was used to classi

249 lmic conditions, including anterior ischemic optic neuropathy, optic neuritis/multiple sclerosis, neu

250 tic strabismus, severe gaze dysfunction, and optic neuropathy or cerebral visual impairment (CVI).

251 re was no evidence of radiation retinopathy, optic neuropathy, or cataract.

252 edema (CME), macular scarring, macular hole, optic neuropathy, or macular ischemia.

253 lly other reasons, such as radiation-induced optic neuropathy, or where visual outcome was influenced

254 ion of >/= 10 mmHg and incident glaucomatous optic neuropathy over the first 2 years compared with th

255 n the Ndufs4 KO mouse model of mitochondrial optic neuropathy, papaverine and zolpidem provided signi

256 lence and incidence of post-cataract surgery optic neuropathy (PCSON) in the modern era.

257 optic neuropathy (GON), 31 with progressive optic neuropathy (PGON) 53 with ocular hypertension, and

258 graphic evidence of progressive glaucomatous optic neuropathy (PGON).

259 Docetaxel can cause a toxic optic neuropathy possibly due to an ischemic or neurotox

260 peripheral nervous system, include deafness, optic neuropathy-previously not reported in HSAN IE-larg

261 e mitochondrial disorder, Leber's hereditary optic neuropathy, provides evidence that patients with d

262 ects including postoperative vision changes, optic neuropathy, radiation retinopathy, and cataract.

263 Rodent anterior ischemic optic neuropathy (rAION) is an optic nerve infarct, whic

264 An NAION model, rodent anterior ischemic optic neuropathy (rAION), was used to determine AION-ass

265 -year freedom from radiation retinopathy and optic neuropathy rates were higher in the apex LD than H

266 examined 118 patients with anterior ischemic optic neuropathy referred to a tertiary care center from

267 d be considered in the development of future optic neuropathy-related neuroprotection strategies.

268 The vast majority of optic neuropathies result from retinal ganglion cell (RG

269 hy, and dominant optic atrophy, an inherited optic neuropathy, result from a primary deficiency of mi

270 t common side effects were radiation-induced optic neuropathy, retinopathy, and cataract.

271 of >360 subjects with unexplained inherited optic neuropathy revealed three additional families carr

272 travitreal bevacizumab to suppress radiation optic neuropathy (RON) or maculopathy.

273 To describe the first reported case of toxic optic neuropathy secondary to docetaxel (Taxotere(R)) ch

274 multisystemic, some are tissue specific--eg, optic neuropathy, sensorineural deafness, and type 2 dia

275 OCT-measured RNFL in nonglaucomatous optic neuropathies shows thinning, which may mimic those

276 ement-based therapies for glaucoma and other optic neuropathies.SIGNIFICANCE STATEMENT Glaucoma is th

277 th could lead to novel therapies for chronic optic neuropathies such as glaucoma.

278 des might in susceptible individuals lead to optic neuropathies such as normal tension glaucoma.

279 Inherited mitochondrial optic neuropathies, such as Leber's hereditary optic neu

280 exhibited significantly higher penetrance of optic neuropathy than those carrying only m.11778G > A m

281 Glaucoma is an optic neuropathy that originates with pressure-induced d

282 I have also led to advances in understanding optic neuropathies, the ocular motor system, pseudotumor

283 Given that vitamin B12 deficiency causes an optic neuropathy through unknown mechanisms and that it

284 rsy about the primary treatment of traumatic optic neuropathy (TON) has anchored on final vision foll

285 Traumatic optic neuropathy (TON) is a devastating cause of permane

286 Traumatic optic neuropathy (TON) is an acute injury of the optic n

287 dary to orbital and facial trauma: traumatic optic neuropathy (TON), retrobulbar haemorrhage (RBH) an

288 by several independent methods, glaucomatous optic neuropathy was not associated with a significant c

289 Optic neuropathy was ruled out in all cases.

290 The rate of compressive optic neuropathy was significantly lower and improvement

291 In four families with early-onset recessive optic neuropathy, we identified mutations in RTN4IP1, wh

292 ion, amblyopia, advanced glaucoma, and other optic neuropathies were excluded from the analysis of vi

293 In our study, isquemic and compressive optic neuropathies were the ones that most often did so.

294 Patients with OHT or glaucomatous optic neuropathy were recruited, and photopic full-field

295 with unilateral or asymmetric demyelinating optic neuropathy were retrospectively evaluated.

296 Five types of optic neuropathy were studied: (1) optic neuritis with m

297 O and parkinsonism/dementia with subclinical optic neuropathy widens the phenotypic spectrum of OPA1

298 sociation of nonarteritic anterior ischaemic optic neuropathy with cataract surgery and phosphodieste

299 a trend of increased cumulative incidence of optic neuropathy with longer treatment duration (>41 vs.

300 Early POAG subjects had glaucomatous optic neuropathy with mild, reproducible visual field lo