ライフサイエンスコーパス: optokinetic

1 inetic reflex and evokes the second phase of optokinetic afternystagmus (OKAN-II).

2 Behaviorally, discordant optokinetic and vestibular input induced appropriate hig

3 vement recordings showed decreased gains for optokinetic and vestibulo-ocular reflexes, confirming an

4 ibution suggests that monocular nasotemporal optokinetic asymmetry is partly attributable to subcorti

5 le in maintaining the monocular nasotemporal optokinetic asymmetry seen in patients with infantile es

6 y mutants required nearly an hour to recover optokinetic behavior after return to bright light, where

7 ed calcium imaging in awake zebrafish during optokinetic behavior to record transgenically identified

8 Its applicaton for explaining a variety of optokinetic behaviors in other insects assumes that neur

9 This nonfoveal optokinetic contribution suggests that monocular nasotem

10 ction in visual function on testing with the optokinetic drum and the circadian running wheel.

11 ent of motion responsiveness for pursuit and optokinetic eye movements (optokinetic nystagmus [OKN]).

12 e of virtual reality, vibrotactile feedback, optokinetic flow, YouTube videos, and innovative methods

13 Animals with vision had reduced optokinetic gains by 24 h, while the OKR response for an

14 Moreover, these cells are capable of driving optokinetic head tracking and visually guided behaviour

15 The pigeon flocculus receives visual-optokinetic information and is important for generating

16 Nasally directed optokinetic input to the esodeviated eye can supplement

17 ted rostrally were most persistent following optokinetic input.

18 to generate accurate responses to full-field optokinetic input.

19 were differentially accessed by saccadic and optokinetic inputs.

20 suppresses the perception of this full-field optokinetic motion during active pursuit.

21 s uncovered, exposing it to nasally directed optokinetic motion.

22 ical deviation (DVD), monocular asymmetry of optokinetic nystagmus (MOKN), monocular asymmetry of smo

23 otions; i.e., they give better responses for optokinetic nystagmus (OKN) and visually evoked potentia

24 To evaluate the efficacy of using optokinetic nystagmus (OKN) as an objective measurement

25 Look optokinetic nystagmus (OKN) consists of voluntary tracki

26 e of this study was to characterize vertical optokinetic nystagmus (OKN) in normal human subjects, co

27 Modifying experimental conditions of optokinetic nystagmus (OKN) result in different outcomes

28 designed to elicit smooth pursuit, saccades, optokinetic nystagmus (OKN), vestibulo-ocular reflex (VO

29 The effect of aging on torsional optokinetic nystagmus (tOKN) is unknown.

30 s for pursuit and optokinetic eye movements (optokinetic nystagmus [OKN]).

31 We used the optokinetic nystagmus and pupil size to objectively and

32 The direction of optokinetic nystagmus correlates with visual perception

33 Abnormal vertical optokinetic nystagmus was present in 19 (68%) of 28 subj

34 Asymmetric optokinetic nystagmus, latent nystagmus, and dissociated

35 ion and the role of nasally biased monocular optokinetic nystagmus.

36 during leftward and rightward slow phases of optokinetic nystagmus.

37 es (Macaca mulatta) during the slow phase of optokinetic nystagmus.

38 The optokinetic (OK) stimulus subtended 72 degrees horizonta

39 Visual function was assessed with a rotating optokinetic (OKN) drum at ages 13 and 18 months and neur

40 ent of dye results in significantly improved optokinetic (OKR) ( 43 fold, p < 0.001) and visualmotor

41 Subcortical optokinetic pathways seem to play an important role in m

42 fixation, and a reduction in vestibular and optokinetic quick phases.

43 cued water maze (WM) behavioral test and the optokinetic reflex (OKR) measurement at different times

44 A prime example of such behaviours is the optokinetic reflex (OKR), an innate eye movement mediate

45 haracterized as blind, these mutants lack an optokinetic reflex (OKR), but in another behavioral assa

46 (HOKS) decreases the gain of the horizontal optokinetic reflex and evokes the second phase of optoki

47 ntaneously firing neurons, is engaged during optokinetic reflex compensation for inner ear dysfunctio

48 At 6 d post-fertilization (dpf), no optokinetic reflex could be elicited in no optokinetic r

49 Optokinetic reflex measurements showed that Jimpy mice h

50 e and after disease onset by quantifying the optokinetic reflex responses and to compare them to the

51 The optokinetic reflex resulting in optomotor head tracking

52 Optokinetic reflex was not detectable horizontally.

53 Similarly within the afferent arm of the optokinetic reflex we showed expression in the developin

54 d defective eye movements as measured by the optokinetic reflex.

55 ay of retinal signals into the brainstem for optokinetic reflexes.

56 d those on retinal function were analyzed by optokinetic response (OKR) and electroretinography (ERG)

57 The optokinetic response (OKR) consists of smooth eye moveme

58 The optokinetic response (OKR) to a visual stimulus moving a

59 ly evoked smooth eye movements, known as the optokinetic response (OKR), have been studied in various

60 This behavior, which is termed an optokinetic response (OKR), is a reflex that appears in

61 ations (SOs) and, in several cases, reversed optokinetic response (OKR).

62 The optokinetic response (tracking eye movements) was evoked

63 wo alleles of the recessive lethal mutant no optokinetic response a (noa).

64 Fish deficient in Ribeye a lack an optokinetic response and have shorter synaptic ribbons i

65 was assessed using the electroretinogram and optokinetic response and retinal morphology investigated

66 A red-blind zebrafish mutant, partial optokinetic response b (pob), has been isolated by measu

67 The zebrafish mutant, partial optokinetic response b (pob), was isolated using an N-et

68 physiology of the zebrafish visual mutant no optokinetic response c (nrc) and to identify the genetic

69 o optokinetic reflex could be elicited in no optokinetic response c (nrc) mutant animals under any te

70 Here we describe a zebrafish mutant, no optokinetic response f(w21) (nof), with a nonsense mutat

71 Recovery of Ribeye a levels rescues the optokinetic response, increases the number of PKCalpha-p

72 the whole brains of zebrafish performing the optokinetic response.

73 ysis of optic flow and the generation of the optokinetic response.

74 y optic system (AOS) where they initiate the optokinetic response.

75 d movements in their surroundings, displayed optokinetic responses (OKR) or optomotor responses (OMR)

76 nse to light, of which the optomotor and the optokinetic responses are the most widely studied.

77 Optokinetic responses became unstable but were generally

78 Moreover, bistable optokinetic responses cannot be entirely attributed to s

79 This improvement in optokinetic responses did not necessitate a fixation shi

80 etinal motion input in generating horizontal optokinetic responses in patients with infantile strabis

81 y designed to identify larvae with defective optokinetic responses in red but not white light.

82 viated eye can supplement temporal monocular optokinetic responses in the fixating eye under binocula

83 All patients showed poor temporally directed optokinetic responses that instantaneously improved when

84 Measurement of optokinetic responses to plaid stimuli revealed that mic

85 y was measured behaviorally, using optomotor/optokinetic responses to rotating square-wave stimuli.

86 Subnormal optokinetic responses were found in a subgroup of obliga

87 nced by changes in the direction of elicited optokinetic responses.

88 under certain stimulus conditions to mediate optokinetic signals in the brain.

89 t, gaze holding, convergence, vestibular and optokinetic slow phases, and cancellation of vestibular

90 Long-term horizontal optokinetic stimulation (HOKS) decreases the gain of the

91 Optokinetic stimulation induces nystagmus that can be us

92 These VEPRs are not simple responses to optokinetic stimulation, but are modulated by the config

93 the oculomotor integrator after saccadic or optokinetic stimulation.

94 ike activity (CSA) in response to rotational optokinetic stimuli.

95 = 9) while pairing yaw rotation with a pitch optokinetic stimulus, resulting in cross-axis adaptation

96 ross their retinas, creating a contraversive optokinetic stimulus.

97 nistic motion stimulus from this subcortical optokinetic system facilitates development of the unstab

98 evelop in infancy, this phylogenetically old optokinetic system, which is normally operative in the f

99 OKT thresholds were measured using a virtual optokinetics system.

100 Optokinetic testing was performed in 7 patients with iso

101 65(T/-) mice visual function was measured by optokinetic tracking (OKT) and electroretinography (ERG)

102 ression, selected visual cycle proteins, and optokinetic tracking (OKT) in streptozotocin (STZ)-induc

103 Microbead-injected eyes showed reduced optokinetic tracking as well as cell death.

104 ould improve visual function (evaluated with optokinetic tracking response) of diabetic mice, potenti

105 Visual function was measured using optokinetic tracking to determine spatial frequency and

106 ks of hyperglycemia for visual function with optokinetic tracking weekly visual acuity and monthly co

107 logical (electroretinogram), psychophysical (optokinetic tracking), and pharmacological techniques.

108 opening, in which daily threshold testing of optokinetic tracking, amid otherwise normal visual exper

109 We devised the optokinetic uncover test to examine the role of peripher

110 ysfunction arising due to instability of the optokinetic-vestibular systems.

111 ing nystagmus arises from instability of the optokinetic-vestibular systems.

112 n folium IXcd of the flocculus, such that an optokinetic zone spans a ZII+/- pair: the HA zones span

113 These optokinetic zones relate to the ZII stripes in folium IX