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1 omposite end point of brain damage, nursing home admission, or death (hazard ratio, 0.67; 95% CI, 0.53 to 0.84).
3 ease progression or the development of a new primary cancer or death assessed at 4.5 years after randomization.
9 We determined type, severity, outcome (disability or death), and time course of bleeding requiring medical atte
10 dpoint was time to progression (time of progressive disease or death from any cause), with intention-to-treat analysis.
11 , confirmed by a cardiologist, and a significant LVEF drop, or death of definite or probable cardiac causes.
12 ome; and a composite of doubling of serum creatinine, ESRD, or death between 100 Rtx-treated patients and 103 patients wh
13 The primary outcome was a composite of long-term CV events or death, which was assessed via national health care databas
14 iation with the combined outcome of new neurological events or death (HR, 1.58 [95% CI, 1.11-2.23]).
15 malignant ventricular arrhythmias, end-stage heart failure, or death) compared with carriers of other pathogenic LMNA mut
17 with LBBB randomized to CRT-D, there were differences in HF or death risk and in the degree of reverse remodeling among c
19 sociated with a greater risk of incident HF hospitalization or death at a median follow-up of 608 days.
21 d hazard ratios (HRs) of death and of AIDS-defining illness or death, risk ratios of virological failure, and mean differ
22 isease, defined as the first incident myocardial infarction or death owing to coronary heart disease, and stroke, defined
24 lated from time of exposure to the occurrence of meningioma or death or until December 31, 2010, with logistic regression
27 ignificant change in the incidence of the composite outcome or death-censored cardiovascular events over time (P = 0.41 a
28 ths versus 1.5 months (hazard ratio for disease progression or death, 0.49; 95% CI, 0.39 to 0.61; P<0.001).
30 Secondary end points included time to second progression or death, toxic effects, and quality of life.
34 geneity was associated with an increased risk of recurrence or death (hazard ratio, 4.9; P=4.4x10(-4)), which remained si
35 ability was associated with an increased risk of recurrence or death, a finding that supports the potential value of chro
37 roup and 39% in the placebo group (hazard ratio for relapse or death, 0.47; 95% confidence interval [CI], 0.39 to 0.58; P
38 settings; however, some patients remain at risk of relapse or death for many years after treatment of early-stage diseas
39 cal progression of cancer from low to moderate or high risk or death during 24 months' follow-up) and absence of definite
40 a under the curve, 0.78) and the combined end point of RVAD or death within 14 days (area under the curve, 0.73).
41 rs for RVAD implantation and the combined end point of RVAD or death within 14 days of LVAD were assessed with stepwise l
44 end point (nonfatal myocardial infarction, nonfatal stroke, or death from cardiovascular causes) and death.
47 c renal failure, involvement of the central nervous system, or death), and interventions (ie, renal replacement therapy).
48 sk Triage score were calculated for predicting ICU transfer or death within 48 hours of meeting suspicion criteria.
50 rk of interactions leading to either clearance of the virus or death of the host, depending on the initial dosage of the
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