ライフサイエンスコーパス: oral candidiasis

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1 bited potent activity in two mouse models of oral candidiasis.

2 ete deficiency in the neutrophil response to oral candidiasis.

3 abrata are predominant fungi associated with oral candidiasis.

4 ed attenuated virulence in a murine model of oral candidiasis.

5 ather than IL-22 is vital in defense against oral candidiasis.

6 d have promise as therapeutic agents against oral candidiasis.

7 promise as therapeutic agents in humans with oral candidiasis.

8 f human innate immune response in preventing oral candidiasis.

9 roup had opportunistic infections (excluding oral candidiasis and tuberculosis).

10 roviral therapy for the incident outcomes of oral candidiasis and vaginal colonization.

11 Although disseminated, vaginal, and oral candidiasis are all caused by C. albicans species,

12 The most common form of oral candidiasis, denture-associated stomatitis, involve

13 stic fungal pathogen that has been linked to oral candidiasis in AIDS patients, although it has recen

14 lbicans and that is commonly associated with oral candidiasis in human immunodeficiency virus-positiv

15 yte infections (IR, 0.88; 0.67-1.14), and 52 oral candidiasis (IR, 0.93; 95% CI, 0.70-1.22).

16 5-2.1; P jeroveci, IR, 1.3; 95% CI, 1.1-1.6; oral candidiasis, IR, 1.2; 95% CI, 1.0-1.5; cytomegalovi

17 ed findings from this study demonstrate that oral candidiasis may constitute a risk factor for dissem

18 ophil functions and which has been linked to oral candidiasis (OC), the most prevalent oral lesion in

19 next 12-18 months, and patients with either oral candidiasis or hairy leukoplakia and a low CD4:CD8

20 on sequencing to analyze 43 isolates from 11 oral candidiasis patients.

21 except for tuberculosis, and was largest for oral candidiasis, Pneumocystis pneumonia, and toxoplasmo

22 eveloped to investigate whether the onset of oral candidiasis predisposes the host to secondary staph

23 ults suggest that HIV-infected patients with oral candidiasis should be carefully monitored for subse

24 tease Sap6 is important for virulence during oral candidiasis since it degrades host tissues to relea

25 Oral candidiasis specifically, characterized by hyphal i

26 The findings demonstrated that in mice with oral candidiasis, subsequent exposure to S. aureus resul

27 s for HIV-seropositive women was greater for oral candidiasis than for vaginal candidiasis.

28 ough IL-17, confers the dominant response to oral candidiasis through neutrophils and antimicrobial f

29 The markers compared were oral candidiasis (thrush) or fever; serum neopterin leve

30 patients, summary risk was highest (>5%) for oral candidiasis, tuberculosis, herpes zoster, and bacte

31 species of yeast isolated from patients with oral candidiasis, which is frequently a symptom of human

32 f adverse effects (except for an increase in oral candidiasis with FSC and F) was similar among the t

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