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1 rly-childhood caries, a prevalent and costly oral disease.
2 regime shift"), which promotes dysbiosis and oral disease.
3 f bacteria in the progression of this common oral disease.
4 t likely to develop (any or severe forms of) oral disease.
5 ationship between obesity-associated T2D and oral disease.
6 cious feed-forward loop between systemic and oral disease.
7 potential target for preventing this common oral disease.
8 cer, chronic lung and vascular diseases, and oral disease.
9 -induced effector molecules in resistance to oral disease.
10 identification of meaningful biomarkers for oral disease.
11 a role in the development of tobacco-related oral disease.
12 rvations apply equally to the study of other oral diseases.
13 erns about their role in the pathogenesis of oral diseases.
14 oved intervention strategies for MMP-related oral diseases.
15 ietal relevance of preventing and addressing oral diseases.
16 an oral bacterium implicated in a variety of oral diseases.
17 2) and from a control group (n = 27) without oral diseases.
18 w drugs are developed for pathways common to oral diseases.
19 l associations of DG with various autoimmune oral diseases.
20 eroxidase systems during the pathogenesis of oral diseases.
21 he pathogenesis of different biofilm-related oral diseases.
22 ploit arginine catabolism for the control of oral diseases.
23 their uncontrolled outgrowth can express as oral diseases.
24 ckness or health is a key to combating human oral diseases.
25 ll patients with HIV infection will contract oral diseases.
29 whelming dominance of social determinants on oral disease and the difficulty of translating science i
32 he oral health goal of reducing the level of oral diseases and minimizing their impact is to be achie
34 trates a moderate role of genetic factors in oral diseases, and suggests potential gene-environment i
35 re compatible with the hypotheses that adult oral diseases are associated with the probability of exp
37 and periodontal disease, the most widespread oral diseases, are commonly treated with various oral an
38 To investigate changes in the pattern of oral disease associated with highly active antiretrovira
39 used by Candida albicans, is the most common oral disease associated with human immunodeficiency viru
40 e clinical utility in treating LAP and other oral diseases associated with infection, inflammation, a
41 onal project that identified determinants of oral diseases at the community, family, and individual l
43 nostic that enables rapid quantitation of an oral disease biomarker in human saliva by using a monoli
48 sibility for every aspect of the impact that oral disease could have on the health and welfare of its
50 ntium associated with a higher prevalence of oral diseases (e.g., chronic periodontitis) in aged popu
51 es among older adults have demonstrated that oral disease frequently leads to dysfunction, discomfort
54 e pediatricians on the epidemiology of child oral disease, highlight the importance of good oral heal
56 Periodontal disease is the most widespread oral disease in dogs which if left untreated results in
57 conclude that, in spite of the high rate of oral disease in persons with HIV, many do not use dental
59 baseline, reflecting the natural history of oral disease in these animals, suggests individual varia
61 of intracellular PRRs in the pathogenesis of oral diseases including periodontitis and oral cavity ca
62 (CKD) were investigated to find out whether oral disease inflammatory burden or different etiology (
67 However, recent studies indicate that this oral disease may have profound effects on systemic healt
68 s with current measurement methodologies for oral diseases, measurable specific oral health goals sho
70 in rheumatoid arthritis (RA) patients taking oral disease-modifying antirheumatic drugs (DMARDs).
72 rst study to report benefits of an available oral disease-modifying therapy in patients with early mu
73 rs are at high risk for 2 bacterially driven oral diseases: peri-implant mucositis and peri-implantit
75 search's (NIDR) Division of Epidemiology and Oral Disease Prevention (DEODP) staff and consultants co
77 ve risk of 8 for patients with ocular but no oral disease (pure ocular cicatricial pemphigoid, p < 0.
80 m, an opportunistic bacterial pathogen, from oral disease sites, such as those involved in refractory
83 sition of these biofilms are associated with oral diseases such as dental caries or periodontitis.
84 ved in two well-studied, microbiome-mediated oral diseases, such as butanoate production in periodont
85 vaccine, as well as vaccines to combat other oral diseases, such as dental caries and periodontal dis
89 nce of the DQB1*0301 allele in patients with oral disease was not statistically significant (64%, 7/1
91 AP8, and PLB1 expression was correlated with oral disease, whereas SAP1, SAP3, and SAP6-SAP8 expressi
92 ying mucosal immunology, initially exploring oral diseases, with special emphasis on the immunobiolog
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