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1 rly-childhood caries, a prevalent and costly oral disease.
2 regime shift"), which promotes dysbiosis and oral disease.
3 f bacteria in the progression of this common oral disease.
4 t likely to develop (any or severe forms of) oral disease.
5 ationship between obesity-associated T2D and oral disease.
6 cious feed-forward loop between systemic and oral disease.
7  potential target for preventing this common oral disease.
8 cer, chronic lung and vascular diseases, and oral disease.
9 -induced effector molecules in resistance to oral disease.
10  identification of meaningful biomarkers for oral disease.
11 a role in the development of tobacco-related oral disease.
12 rvations apply equally to the study of other oral diseases.
13 erns about their role in the pathogenesis of oral diseases.
14 oved intervention strategies for MMP-related oral diseases.
15 ietal relevance of preventing and addressing oral diseases.
16 an oral bacterium implicated in a variety of oral diseases.
17 2) and from a control group (n = 27) without oral diseases.
18 w drugs are developed for pathways common to oral diseases.
19 l associations of DG with various autoimmune oral diseases.
20 eroxidase systems during the pathogenesis of oral diseases.
21 he pathogenesis of different biofilm-related oral diseases.
22 ploit arginine catabolism for the control of oral diseases.
23  their uncontrolled outgrowth can express as oral diseases.
24 ckness or health is a key to combating human oral diseases.
25 ll patients with HIV infection will contract oral diseases.
26                           Chronic, untreated oral disease adversely affects one's systemic health, qu
27                             The link between oral disease and cardiovascular disease was established
28  a scientific model for the understanding of oral disease and its consequences.
29 whelming dominance of social determinants on oral disease and the difficulty of translating science i
30 lyses estimated genetic correlations between oral diseases and cardiovascular disease outcomes.
31 s oral health goals of reducing the level of oral diseases and minimizing their impact by 2020.
32 he oral health goal of reducing the level of oral diseases and minimizing their impact is to be achie
33 he association between different measures of oral diseases and the occurrence of PAD.
34 trates a moderate role of genetic factors in oral diseases, and suggests potential gene-environment i
35 re compatible with the hypotheses that adult oral diseases are associated with the probability of exp
36                                     Clearly, oral diseases are highly prevalent in the globe, posing
37 and periodontal disease, the most widespread oral diseases, are commonly treated with various oral an
38     To investigate changes in the pattern of oral disease associated with highly active antiretrovira
39 used by Candida albicans, is the most common oral disease associated with human immunodeficiency viru
40 e clinical utility in treating LAP and other oral diseases associated with infection, inflammation, a
41 onal project that identified determinants of oral diseases at the community, family, and individual l
42 e is that now antiseptic products will treat oral disease better and oral health will improve.
43 nostic that enables rapid quantitation of an oral disease biomarker in human saliva by using a monoli
44                  Tobacco is a known cause of oral disease but the mechanism remains elusive.
45 disabilities have higher rates of asthma and oral disease, but similar rates of hypertension.
46                     Instead, a wide range of oral diseases can mimic these lesions clinically.
47 surrogate endpoints, and new technologies in oral disease clinical trials.
48 sibility for every aspect of the impact that oral disease could have on the health and welfare of its
49                                   Apparently oral disease could, in fact, contribute to systemic dise
50 ntium associated with a higher prevalence of oral diseases (e.g., chronic periodontitis) in aged popu
51 es among older adults have demonstrated that oral disease frequently leads to dysfunction, discomfort
52                                              Oral disease has implications beyond the mouth and can c
53                                    Childhood oral disease has significant medical and financial conse
54 e pediatricians on the epidemiology of child oral disease, highlight the importance of good oral heal
55                              This pattern of oral disease in a referral clinic suggests that an incre
56   Periodontal disease is the most widespread oral disease in dogs which if left untreated results in
57  conclude that, in spite of the high rate of oral disease in persons with HIV, many do not use dental
58 ate and establish appropriate definitions of oral disease in pregnancy are warranted.
59  baseline, reflecting the natural history of oral disease in these animals, suggests individual varia
60                    Last we underscore select oral diseases in which C. albicans is a contributory mic
61 of intracellular PRRs in the pathogenesis of oral diseases including periodontitis and oral cavity ca
62  (CKD) were investigated to find out whether oral disease inflammatory burden or different etiology (
63         The findings show that experience of oral disease is more deleterious to subjective oral heal
64             Reporting the economic burden of oral diseases is important to evaluate the societal rele
65            Since death as a direct result of oral diseases is rare, DALY estimates were based on year
66 ve disorders, a broad spectrum of collateral oral disease may be encountered.
67   However, recent studies indicate that this oral disease may have profound effects on systemic healt
68 s with current measurement methodologies for oral diseases, measurable specific oral health goals sho
69       Kinase inhibitors have shown promising oral disease-modifying antirheumatic drug potential with
70 in rheumatoid arthritis (RA) patients taking oral disease-modifying antirheumatic drugs (DMARDs).
71                          Teriflunomide is an oral disease-modifying therapy approved for treatment of
72 rst study to report benefits of an available oral disease-modifying therapy in patients with early mu
73 rs are at high risk for 2 bacterially driven oral diseases: peri-implant mucositis and peri-implantit
74                     Vitamin D deficiency and oral diseases (periodontitis, caries, and tooth loss) ar
75 search's (NIDR) Division of Epidemiology and Oral Disease Prevention (DEODP) staff and consultants co
76               While certain craniofacial and oral diseases previously deemed too difficult to tackle
77 ve risk of 8 for patients with ocular but no oral disease (pure ocular cicatricial pemphigoid, p < 0.
78                           Tissue loss due to oral diseases requires the healing and regeneration of t
79 r applications were used for illustration of oral disease risk profiles.
80 m, an opportunistic bacterial pathogen, from oral disease sites, such as those involved in refractory
81 biomarkers/diagnostic tools for systemic and oral disease states.
82                  Novel 3D in vitro models of oral diseases such as cancer, Candida, and bacterial inv
83 sition of these biofilms are associated with oral diseases such as dental caries or periodontitis.
84 ved in two well-studied, microbiome-mediated oral diseases, such as butanoate production in periodont
85 vaccine, as well as vaccines to combat other oral diseases, such as dental caries and periodontal dis
86       Chronic periodontitis (CP) is a common oral disease that confers substantial systemic inflammat
87 ressive periodontitis, a rapidly progressing oral disease that occurs in adolescents.
88                                              Oral disease was associated with excess cardiovascular d
89 nce of the DQB1*0301 allele in patients with oral disease was not statistically significant (64%, 7/1
90                          Moreover, when >/=3 oral diseases were cumulated in the model, the risk incr
91 AP8, and PLB1 expression was correlated with oral disease, whereas SAP1, SAP3, and SAP6-SAP8 expressi
92 ying mucosal immunology, initially exploring oral diseases, with special emphasis on the immunobiolog

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