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1  ciliary band emerge adjacent to the central oral ectoderm.
2 ateral spatial organization of the embryonic oral ectoderm.
3  relations among the regulatory genes of the oral ectoderm.
4 ty effect signaling in the sea urchin embryo oral ectoderm.
5 h, a pocket formed by an invagination of the oral ectoderm.
6  maintenance of nodal gene expression in the oral ectoderm.
7  syndrome I, is the earliest known marker of oral ectoderm.
8  regulating development of the stomodeum, or oral ectoderm.
9 ephalon, and Rathke's pouch, a derivative of oral ectoderm.
10 l in turn depends on Spdri expression in the oral ectoderm.
11 essed specifically in various regions of the oral ectoderm.
12  OER, functions to prevent expression in the oral ectoderm.
13 lops from Rathke's pouch, an invagination of oral ectoderm.
14  induction of multiple pituitary glands from oral ectoderm.
15 of subpopulations of endoderm, mesoderm, and oral ectoderm.
16 that mesomeres are autonomously specified as oral ectoderm.
17 s developmentally derived from a fold in the oral ectoderm and a juxtaposed fold in the neural ectode
18 border of the re-specified posterior-ventral oral ectoderm and by larval stages it is in the same pla
19  also results in additional invaginations of oral ectoderm and can shift the position of Rathke's pou
20  that they are somewhat enriched in cells of oral ectoderm and endoderm.
21 -kb fragment that directs LacZ expression in oral ectoderm and in many of its derivatives.
22 ss expression of the CyIIIa.CAT construct in oral ectoderm and in skeletogenic mesenchyme at differen
23 erepresses the endogenous CyIIIa gene in the oral ectoderm and in the endoderm.
24 rolling region-specific transcription in the oral ectoderm and its derivatives.
25 rives cohorts of regulatory genes within the oral ectoderm and its derived subdomains.
26      During mammalian tooth development, the oral ectoderm and mesenchyme coordinate their growth and
27                    In tooth development, the oral ectoderm and mesenchyme coordinately and reciprocal
28 tions to repress activation of CyIIIa in the oral ectoderm and skeletogenic mesenchyme.
29 ired to prevent ectopic transcription in the oral ectoderm and skeletogenic mesenchyme.
30 pends on reciprocal interactions between the oral ectoderm and the underlying neural-crest-derived me
31 cleavage, but were gradually concentrated in oral ectoderm and vegetal plate territories during gastr
32 rm and then later were largely restricted to oral ectoderm and vegetal plate territories.
33 band of hnf6 expression at the border of the oral ectoderm and where it continues to be expressed thr
34 he stomodeal subdomain emerges inside of the oral ectoderm, and bilateral subdomains defining the lat
35 astomeres and Nodal signaling in presumptive oral ectoderm are necessary and sufficient to initiate p
36 e tip of the archenteron and the presumptive oral ectoderm are not required for the differentiation o
37 anisms regulating Pitx2 transcription in the oral ectoderm are poorly understood.
38 s until their expression is cleared from the oral ectoderm as an indirect consequence of Nodal signal
39 ranscription is activated in the presumptive oral ectoderm at about the 30-cell stage.
40 ntirely zygotic and localized to prospective oral ectoderm at blastula stage.
41 Brac expression first appears broadly in the oral ectoderm at mesenchyme blastula stage and at later
42                         The formation of the oral ectoderm begins with an oral-aboral redox gradient,
43                           Concomitantly, the oral ectoderm beneath where these cartilages develop los
44  throughout the ventral diencephalon and the oral ectoderm, but its expression is subsequently absent
45 utation caused expression of the CAT gene in oral ectoderm cells.
46 ells, cells from the stomodeal region of the oral ectoderm, ciliated band cells and cells from the en
47                                          The oral ectoderm contains at least two types of patterned t
48                     Accumulation of SpGsc in oral ectoderm depends on cell-cell interactions initiate
49 us studies have identified a requirement for oral ectoderm derived Sonic Hedgehog (Shh) in specificat
50 ral ectoderm fate specification and promotes oral ectoderm differentiation.
51 expression of the Nodal ligand in the future oral ectoderm during cleavage, a sequence of regulatory
52 for negative regulation of the LpS1 genes in oral ectoderm during development.
53 rm, but in addition there is a nonautonomous oral ectoderm effect.
54 ency in early head development and pituitary oral ectoderm exhibit craniofacial defects and pituitary
55 pon their clearance explains the dynamics of oral ectoderm gene expression.
56                               The sea urchin oral ectoderm gene regulatory network (GRN) model has in
57  its participation after gastrulation in the oral ectoderm gene regulatory network (GRN), in which it
58 , which selectively prevent transcription of oral ectoderm genes until their expression is cleared fr
59 f 17 different mesodermal genes, 8 different oral ectoderm genes, and 18 other genes expressed specif
60 , the not gene product acts to repress other oral ectoderm genes, contributing crucially to the bilat
61           A clear example is afforded by the oral ectoderm GRN of the sea urchin embryo where cis-reg
62                       Spdri is linked in the oral ectoderm GRN with several other genes encoding tran
63  participates in the central GRN controlling oral ectoderm identity.
64 tely in all cell types including the gut and oral ectoderm in gastrula and larva stage embryos, while
65 hes the transcriptional specification of the oral ectoderm in the sea urchin embryo.
66 er that forms at the boundary of a region of oral ectoderm in which Shh expression is selectively exc
67 the gastrula stage, (2) that the presumptive oral ectoderm is not committed to produce oral structure
68 acent regions indicate that, at tailbud, the oral ectoderm is not specifically required for primary m
69 ansgenic expression of CDKs in the embryonic oral ectoderm is specifically retained in undifferentiat
70 s possibility, we find that specification of oral ectoderm is suppressed when embryos are cultured un
71             Overexpression of Shh in the non-oral ectoderm leads to an expansion of Fgf8, affecting t
72 aling, acting at the Shh boundary within the oral ectoderm, may exert a role in differentiation of ve
73          In addition to disappearance of the oral ectoderm, morphological consequences of alphaSpdri
74                        Ectopic expression in oral ectoderm occurs if P3A2 sites are deleted from CyII
75 pCOUP-TF gene is spatially restricted in the oral ectoderm of the early embryo and, at later stages,
76 regulatory state pattern in the pregastrular oral ectoderm of the embryo.
77 eletogenesis also requires the expression of oral ectoderm patterning information.
78 ent models state that Shh signaling from the oral ectoderm patterns the pituitary after placode induc
79  several further subdivisions into which the oral ectoderm per se is partitioned.
80  lineages including the lens placode and the oral ectoderm (pituitary precursor) cells.
81                                      We used oral ectoderm/Rathke's pouch-specific 5' regulatory sequ
82                                      Neither oral ectoderm regulatory functions nor ciliated band for
83 tate maps, including all spatially expressed oral ectoderm regulatory genes, were established.
84 dal expression is required for expression of oral ectoderm regulatory genes.
85 onal regulatory genes that contribute to the oral ectoderm regulatory state during specification in S
86 ional activator, and SpGoosecoid (SpGsc), an oral ectoderm-restricted transcriptional repressor.
87 ostgastrular sea urchin embryo surrounds the oral ectoderm, separating it from adjacent embryonic ter
88 the main features of nodal expression in the oral ectoderm: since the activity of bZIP factors is red
89 genes LpS1 and LpC2 to be repressed while an oral ectoderm-specific gene, Ecto-V, was expressed in al
90  a negative suppressive signal to inactivate oral ectoderm-specific genes in the prospective aboral e
91 d into polarized embryoids that expressed an oral ectoderm-specific marker uniformly.
92  earlier work to be an essential mediator of oral ectoderm specification in the sea urchin embryo, an
93 ssential for the maintenance of the state of oral ectoderm specification.
94 the repression of spec2a in endomesoderm and oral ectoderm territories.
95 ec2a expression is repressed in endoderm and oral ectoderm territories.
96 odal signaling occurs among all cells of the oral ectoderm territory, and nodal expression is require
97 ry gland are derived from an invagination of oral ectoderm that forms Rathke's pouch.
98 ls trabecular morphogenesis and one from the oral ectoderm that promotes chondrogenesis.
99                                  But, in the oral ectoderm, the same gene participates in the central
100 on of regulatory genes in the central animal oral ectoderm thereby confining their expression to the
101 he same time, accumulates in the presumptive oral ectoderm through pluteus stage.
102 optic chiasm development, causes the rostral oral ectoderm to form an ectopic fold that eventually de
103 raction of cranial neural crest cells to the oral ectoderm, where crest-derived signals were necessar
104  micromeres; second, after about 20 h in the oral ectoderm, where its transcripts remain present at 3

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