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1 y but not sufficient for the pathogenesis of oral hairy leukoplakia.
2 layer of the tongue epithelium in lesions of oral hairy leukoplakia.
3 atment of EBV-associated diseases other than oral hairy leukoplakia.
4 f oral epithelial EBV in the pathogenesis of oral hairy leukoplakia.
5 (HIV)-seropositive subjects with and without oral hairy leukoplakia, a replicative EBV-associated epi
7 uman immunodeficiency virus (HIV)-associated oral hairy leukoplakia (HLP) and Epstein-Barr virus (EBV
11 logies, such as nasopharyngeal carcinoma and oral hairy leukoplakia, indicating that the virus can in
12 ngue cells lytically infected with EBV (from oral hairy leukoplakia lesions) express much more FAS th
13 such as nasopharyngeal carcinoma (NPC), and oral hairy leukoplakia (OHL) lesions that have lytic inf
14 diseases are due to lytic infection (such as oral hairy leukoplakia) or latent infection (such as nas
15 can produce diverse pathologies ranging from oral hairy leukoplakia to nasopharyngeal carcinoma, from
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