ライフサイエンスコーパス: oral lesion

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1 area of gingival recession resulting from an oral lesion.

2 sis (PDD) of the malignant transformation of oral lesions.

3 HHV loads were detected for those (n=7) with oral lesions.

4 on reducing the incidence and recurrence of oral lesions.

5 oid gel, which led to the improvement of the oral lesions.

6 entiate this condition from other ulcerative oral lesions.

7 > or = 50 cutaneous lesions and 19 (49%) had oral lesions.

8 served in necrotized regions of samples from oral lesions.

9 sone and other drugs completely resolved the oral lesions after 14 months of therapy.

10 tacrolimus ointment completely resolved the oral lesions after 2 months of therapy.

11 parameters and the presence of opportunistic oral lesions along with records of CD4+ counts and viral

12 oncurrent detection of potentially cancerous oral lesions among HIV-negative patients but not among H

13 direct sequencing, we examined premalignant oral lesion and oral cancer cell lines and found no intr

14 e benefits of tobacco cessation in resolving oral lesions and improving overall periodontal and oral

15 odeficiency subsequent to the development of oral lesions and malnutrition.

16 man herpesvirus 8 [HHV-8]) was determined in oral lesions and oral neoplasms common to persons with h

17 essation revealed complete resolution of the oral lesions and overall improvement of periodontal and

18 orough clinical examination, a biopsy of the oral lesions, and a history of cutaneous psoriasis.

19 at bal mice lack Dsg3 by IF, have typical PV oral lesions, and have a DSG3 gene mutation.

20 tory bowel disease and the importance of the oral lesions as initial presenting signs of systemic dis

21 ivation of Krt16 in mice consistently causes oral lesions as well as PPK-like hyperkeratotic calluses

22 HSV-2 shedding was never associated with an oral lesion, but it was often concurrent with genital HS

23 However, the oral lesions continued to recur over a period of several

24 people with HIV infection fall to experience oral lesions during the course of their disease.

25 Herpes simplex virus type 1 (HSV-1) produces oral lesions, encephalitis, keratitis, and severe infect

26 d 1% pimecrolimus cream twice daily to their oral lesions for 4 weeks, whereas group 2 applied a plac

27 to oral candidiasis (OC), the most prevalent oral lesion in human immunodeficiency virus (HIV).

28 ral human papillomavirus (HPV) infection and oral lesions in 161 human immunodeficiency virus (HIV)-p

29 The oral lesions in DSG3-/- mice reduce their food intake, r

30 tal status and the presence of opportunistic oral lesions in human immunodeficiency virus-positive (H

31 in populations worldwide and cause recurrent oral lesions in up to 40% of infected subjects.

32 Persistent oral lesions, lack of effective oral hygiene, and plaque

33 The presence of these often painful oral lesions may hinder oral hygiene efforts resulting i

34 olvement, the clinician should be aware that oral lesions may occur.

35 of virus within salivary glands adjacent to oral lesions of affected animals.

36 the presence of macrophage subpopulations in oral lesions of VX.

37 with cutaneous psoriasis, that present with oral lesions on the attached gingiva.

38 Oral lesions reflect HIV status and the stage of immunos

39 tion models to identify risk factors for two oral lesions related to human immunodeficiency virus (HI

40 Immunohistochemical analyses of the oral lesions revealed a subepithelial infiltrate that wa

41 HIV-infected individuals frequently have oral lesions that can cause bleeding and release of the

42 ral lichen planus, and all five patients had oral lesions that clinically resembled lichen planus.

43 We speculated that oral lesions (typical in PV patients) might be inhibitin

44 In 27%, the oral lesion was the first sign of a malignant disease.

45 Oral lesions were examined by polymerase chain reaction

46 Oral leukoplakia (OPL) is a heterogeneous oral lesion with an increased oral cancer risk.

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