ライフサイエンスコーパス: oral mucosa

1 sbronchial, 27 lymph node, two skin, and two oral mucosa).

2 but most patients experienced thickening of oral mucosa.

3 the C-terminal domain of DP in the skin and oral mucosa.

4 sible that recolonization may occur from the oral mucosa.

5 e organized similarly to the cells in native oral mucosa.

6 pared with endothelial cells from the normal oral mucosa.

7 chronic, painful, ulcerative lesions of the oral mucosa.

8 mal in the tonsil epithelium, in contrast to oral mucosa.

9 s an important opportunistic pathogen in the oral mucosa.

10 apical region of teeth or perforation of the oral mucosa.

11 anism of invasion of certain bacteria in the oral mucosa.

12 mal papillomaviruses that infect the skin or oral mucosa.

13 hat all of the fragments were present in the oral mucosa.

14 s known of the immune system that serves the oral mucosa.

15 ) and its receptor (uPAR) relative to normal oral mucosa.

16 a basic mechanism of immunoregulation in the oral mucosa.

17 rized by blisters and erosive lesions in the oral mucosa.

18 tobacco smoke on the expression of COX-2 in oral mucosa.

19 nd angiogenesis might be observed in healing oral mucosa.

20 risk factors for SCC of the skin and of the oral mucosa.

21 ence assay directly in KCs comprising murine oral mucosa.

22 unoregulatory cytokines are generated in the oral mucosa.

23 than the amount of compound adsorbed by the oral mucosa.

24 icles, in filiform and fungiform papillae of oral mucosa.

25 invasive squamous carcinomas of the skin and oral mucosa.

26 and histologically dysplastic lesions of the oral mucosa.

27 is a key feature of the privileged repair of oral mucosa.

28 ht into the mechanisms of EBV persistence in oral mucosa.

29 and characterized its expression in skin and oral mucosa.

30 rable early wound closure characteristics of oral mucosa.

31 th pathogenic SIVmac251 administered through oral mucosa.

32 activation in both oral SCC cells and intact oral mucosa.

33 dogs from high-dose viral infection of their oral mucosa.

34 ation, pain, and bleeding of the gingiva and oral mucosa.

35 ns is both a commensal and a pathogen at the oral mucosa.

36 and a different keratinization pattern than oral mucosa.

37 nsformation of epithelial tissues, including oral mucosa.

38 nd to be expressed throughout the airway and oral mucosa.

39 ial for short-term colonization of the mouse oral mucosa.

40 alter the persistence of streptococci on the oral mucosa.

41 factors (i.e., mechanical irritation) in the oral mucosa.

42 specificity tested against bovine and human oral mucosa.

43 esentation of these diseases on the skin and oral mucosa.

44 immunoinflammatory diseases of the skin and oral mucosa.

45 ion against virus infection initiated at the oral mucosa.

46 the periodontal ligament, the cementum, and oral mucosa.

47 ion in full-thickness surgical wounds on rat oral mucosa.

48 antly impaired neutrophil recruitment to the oral mucosa.

49 acent tissues such as the salivary gland and oral mucosa.

50 ected are presumably epithelial cells of the oral mucosa.

51 olves the study of cells exfoliated from the oral mucosa.

52 in gag sequences derived from the blood and oral mucosa.

53 d three-dimensional (3D) models of the human oral mucosa.

54 ve wound-healing phenotypes seen in skin and oral mucosa.

55 lation in tumor samples compared with normal oral mucosa.

56 and applications of tissue-engineered human oral mucosa.

57 >/=1 case of ligneous disease involving the oral mucosa.

58 cosa is more permissive to invasion than the oral mucosa.

59 escence and impaired re-epithelialization of oral mucosa.

60 red oral mucosa closely resembles the normal oral mucosa.

61 and genitourinary tracts in addition to the oral mucosa.

62 ysis done on oral cancers showed that normal oral mucosa (100%, 12 of 12) and 69.1% (47 of 68) of dys

63 forearm skin (64.4%), eyebrow hairs (60.9%), oral mucosa (35.6%), and anal mucosa (33.3%).

64 n) than across the 2 mucosal sites (anal and oral mucosa, 6.9%).

65 The main adverse effects were thickening of oral mucosa (72% in the palifermin group vs. 31% in the

66 ), HPV-12 (beta-1) in forearm skin (23%) and oral mucosa (9.2%), and HPV-76 (beta-3) in anal mucosa (

67 for host defense against C. albicans at the oral mucosa, a recent immunohistochemical evaluation of

68 ed fifty-three biopsies of the supra-implant oral mucosa adjacent to the cover screw of submerged den

69 tudy is to evaluate tissue response of human oral mucosa adjacent to titanium cover screws.

70 or tight adhesion of the whole system to the oral mucosa after application.

71 Most patients with hyperpigmentation of the oral mucosa also had melanonychia.

72 only in superficial epithelial cells of the oral mucosa, although latent proviruses are found in mos

73 We have systematically examined oral mucosa among cGVHD patients and determined that the

74 on abiotic surfaces or on the surface of an oral mucosa analogue.

75 vations were extended to an engineered human oral mucosa and an in vivo rat model of catheter-associa

76 and neck squamous cell carcinoma and normal oral mucosa and annotated gene expression levels to spec

77 Immunofluorescence analysis of oral mucosa and atherosclerotic plaques demonstrate infi

78 based on polymerase chain reaction from his oral mucosa and cutaneous vesicle fluid.

79 y of new alpha-defensins from rhesus macaque oral mucosa and determine the first alpha-defensin struc

80 nd other strategies to treat diseases of the oral mucosa and digestive tract.

81 We apply this technique to the biome of the oral mucosa and find that greater than 25% of recovered

82 ium implant was inserted into the engineered oral mucosa and further cultured to establish epithelial

83 unique population of MSCs derived from human oral mucosa and gingiva, especially their immunomodulato

84 e of different dendritic cell subsets in the oral mucosa and its diseases.

85 regulated in OSCC tissues compared to normal oral mucosa and low levels of SGPL1 mRNA correlated with

86 ophils are essential for host defense at the oral mucosa and neutropenia or functional neutrophil def

87 unique features of cellular structure in the oral mucosa and palatine tonsils, the high rate of oral

88 ulted in elevated levels of cytokines in the oral mucosa and plasma of the SIV-infected macaques.

89 ivo, thereby preserving the integrity of the oral mucosa and protecting from radiation-induced mucosi

90 ing to intercellular adhesion defects in the oral mucosa and skin.

91 e production in wounds of equivalent size in oral mucosa and skin.

92 omedullin may serve a different functions in oral mucosa and skin.

93 several stratified epithelia, including the oral mucosa and skin.

94 tes and may have an immunoregulatory role in oral mucosa and skin.

95 le and in vitro adhesive properties, both to oral mucosa and to teeth surface, were obtained with a b

96 CO2 laser irradiation on biopsies of porcine oral mucosa and underlying bone under conditions that si

97 es from postcapillary venules in the in situ oral mucosa and, if so, whether bradykinin mediated this

98 affect oral hygiene, diagnosis, gingival and oral mucosa, and alveolar bone.

99 ifferentiating epithelial cells of the skin, oral mucosa, and gut, expression is consistently up-regu

100 ntial structural role for K6 isoforms in the oral mucosa, and implicate filiform papillae as being th

101 artments represented by the salivary glands, oral mucosa, and palatine tonsils.

102 dystrophic lesions affecting nails, glands, oral mucosa, and palmar-plantar epidermis.

103 e from the basement membrane of conjunctiva, oral mucosa, and skin.

104 (32% of primary tumors) compared with normal oral mucosa, and that expression correlated significantl

105 al carcinoma-derived cell lines of the skin, oral mucosa, and urogenital tract.

106 of stable stem-cell-like phenotypes, render oral mucosa- and gingiva-derived MSCs a promising altern

107 be used for in vitro reconstruction of human oral mucosa are also discussed.

108 ater adherence of bacterial pathogens to the oral mucosa are associated with the greater frequency of

109 C, suggesting that these immune cells of the oral mucosa are likely to be important for CMV transmiss

110 ular immunity and innate immune functions in oral mucosa are maintained.

111 anti-Candida host defense mechanisms at the oral mucosa are poorly understood.

112 IL-23-dependent manner, and that ILCs in the oral mucosa are the main source for these cytokines.

113 ticles in cells exfoliated from peri-implant oral mucosa around titanium dental implants.

114 ontal parameters and inflammatory lesions of oral mucosa as a characteristic of CD.

115 reporter TOPGAL mice and have identified the oral mucosa as a target tissue for RSpo1.

116 wed that primary epithelial cells from human oral mucosa, as well as an oral epithelial cell line, in

117 previously isolated from the respiratory and oral mucosa, as well as circulating phagocytic cells.

118 tion profiles of bacterial microbiota in the oral mucosa associated with RAS.

119 tential complementary mechanism by which the oral mucosa barrier may be disrupted during HIV-1 infect

120 d three-dimensional (3D) system of the human oral mucosa based on an immortalized human oral keratino

121 ray analysis of primary cultures of 4 normal oral mucosa biopsies, 19 dysplasias, and 16 SCCs.

122 e of CD11b(+)Ly-6G(low)F4/80(-) cells to the oral mucosa but were nonetheless highly susceptible to O

123 ive mRNA expression in histologically normal oral mucosa but with lost or down-regulated expression i

124 s highly expressed in esophagus, tongue, and oral mucosa but, in contrast to cornifin alpha, is not d

125 skin site may be affected (and, rarely, the oral mucosa) but lichen sclerosus is most common in the

126 expressed in OSCC tissues compared to normal oral mucosa by real-time PCR.

127 ive conjunctivitis, erythema of the lips and oral mucosa, changes in the extremities, rash, and cervi

128 sistent, nonproductive, EBV infection of the oral mucosa, characterized by limited expression of repl

129 mistry analyses suggests that the engineered oral mucosa closely resembles the normal oral mucosa.

130 ter-odour, the adsorption of odorants by the oral mucosa could be important but has been little explo

131 To investigate the role of miRNAs in oral mucosa development, we examined mice with mesenchym

132 f the immune response, is upregulated in the oral mucosa during CP compared to its level during gingi

133 phosphorylated PKCzeta expression in normal oral mucosa, dysplasia, and carcinoma as well as SCCHN t

134 dynamic nature of the cell population on the oral mucosa equivalent may be beneficial for intra-oral

135 The oral mucosa equivalent was cultured at an air-liquid int

136 we successfully assembled, ex vivo, a human oral mucosa equivalent, consisting of epidermal and derm

137 wn impaired formation of 3D Ex Vivo Produced Oral Mucosa Equivalents (EVPOME) and closure of an in vi

138 Ex vivo-produced oral mucosa equivalents (EVPOMEs) were fabricated under

139 it skin and to basement membranes in cornea, oral mucosa, esophagus, intestine, kidney collecting duc

140 el system appears appropriate for the use in oral mucosa, especially for sublingual and buccal tissue

141 PP and intestinal mucosa, but not tonsils or oral mucosa, express mucosal addressin cell adhesion mol

142 In the oral mucosa, fibroblasts have a unique phenotype and dem

143 of gross motor patterns, by self-sampling of oral mucosa for assessment of rhythmic expression of the

144 eons face is a lack of sufficient autogenous oral mucosa for reconstruction of the oral cavity.

145 ree-dimensional (3D) reconstruction of human oral mucosa for various in vivo and in vitro application

146 ases of HNSCC but was undetectable in normal oral mucosa from healthy subjects.

147 -fold in HNSCC (n = 24) compared with normal oral mucosa from healthy volunteers (n = 17).

148 acent to HNSCC (n = 10) compared with normal oral mucosa from healthy volunteers.

149 de a high-resolution microendoscopy image of oral mucosa from one volunteer.

150 and pathology in the lymphoid tissues, skin, oral mucosa, gastrointestinal tract, reproductive system

151 mmunoenzyme labeling, we show that the human oral mucosa (gingiva) is infiltrated by large numbers of

152 oral mucosa; only cases metastasizing in the oral mucosa, gingiva, and periodontium were included.

153 tro than fibroblasts isolated from untreated oral mucosa/gingiva.

154 Split-thickness or oral mucosa grafts require more than one surgical proced

155 constitutive expression in the epithelia of oral mucosa, hair follicles, and nail beds.

156 We found that neutrophils from the oral mucosa harvested from morning saliva had released n

157 Tissue-engineered oral mucosa have been further optimized in recent years

158 These results can justify delayed oral mucosa healing in the presence of inflammatory reac

159 Oral mucosa heals faster than does skin, yet few studies

160 s (flaccid blisters and erosions on skin and oral mucosa), histology (epidermal acantholysis), and im

161 GRgpA elicits plasma exudation from in situ oral mucosa in a catalytic site-dependent fashion by ela

162 nsplantation of cultured epithelial cells of oral mucosa in corneal limbal stem cell deficiency was s

163 fection characterized by inflammation of the oral mucosa in direct contact with the denture and affec

164 oH3 produced basement membrane separation of oral mucosa in organ culture.

165 fluence the interaction between odorants and oral mucosa in the oral cavity during a "wine intake-lik

166 cytes maintained their ability to regenerate oral mucosa in vitro after 15 weeks of culture.

167 less tobacco elicits plasma exudation in the oral mucosa in vivo in a specific fashion, and that this

168 EMD improves oral mucosa incisional wound healing by promoting format

169 A levels of six innate/effector genes in the oral mucosa indicated that slower disease progression wa

170 The strength of the aroma-oral mucosa interactions seems to explain these results

171 irst time, the impact of wine composition on oral-mucosa interactions under physiological conditions.

172 However, HIV transmission through the oral mucosa is a rare event.

173 However, little is known as to whether the oral mucosa is able to modulate the local concentration

174 Each type of oral mucosa is believed to develop through discrete mole

175 The oral mucosa is exposed to a high density and diversity o

176 Compared to skin, wound healing in oral mucosa is faster and produces less scarring, but th

177 eviously shown that the chronically infected oral mucosa is in a state of endotoxin tolerance, as evi

178 Radiotherapy-induced damage in the oral mucosa is the result of the deleterious effects of

179 erious effects of radiation, not only on the oral mucosa itself but also on the adjacent salivary gla

180 he lingual mucosa to determine whether minor oral mucosa lesions may enhance susceptibility to CWD in

181 leads to skin blistering (acantholysis) and oral mucosa lesions.

182 icial differentiated epithelial cells of the oral mucosa, many of which appear to be shedding from th

183 he reactions suggest that intake through the oral mucosa might constitute a relevant route of exposur

184 munolocalized to the metanephric mesenchyme, oral mucosa, nasal and cranial cartilage, and brain.

185 that the percentage of residents with normal oral mucosa (odds ratio (OR)=1.81, P=0.027), no visible

186 in amount of COX-2 mRNA was observed in the oral mucosa of active smokers versus never smokers.

187 ion of both ligands was also detected in the oral mucosa of active smokers.

188 test this hypothesis, we applied DHA to the oral mucosa of dogs that had been challenged with the ca

189 Infection of the oral mucosa of human immunodeficiency virus type 1 (HIV-

190 the phosphorylation of EGFR and HER2 in the oral mucosa of mice, and treatment with a dual EGFR and

191 and had a decreased ability to colonize the oral mucosa of mice.

192 ivo situation, we studied gene expression in oral mucosa of neonatal alpha3+/+ and alpha3-/- litterma

193 to the elevated levels of COX-2 found in the oral mucosa of smokers.

194 lin, a ligand of the EGFR, were found in the oral mucosa of smokers.

195 1 microbial species could be identified from oral mucosa of two subjects.

196 (SCC25) were compared with cells from normal oral mucosa (OKF/6) and pre-malignant oral keratinocytes

197 rched for cases of metastatic lesions to the oral mucosa; only cases metastasizing in the oral mucosa

198 No harm to the oral mucosa or systemic health occurred.

199 less of gender, with respect to thickness of oral mucosa overlying the tuberosity.

200 The oral mucosa plays critical roles in protection, sensatio

201 toxin tolerance, as observed in the inflamed oral mucosa, potentiates the innate Ag capture activity

202 Oral mucosa progenitor/stem cells reside as a small-size

203 are detected in primary epithelial cells of oral mucosa, prostate and mammary glands.

204 ed epithelial tissues such as the epidermis, oral mucosa, prostate, and urogenital tract.

205 sclerosis when an onlay procedure utilizing oral mucosa provides the best results using either a one

206 eered devices has demonstrated potential for oral mucosa regeneration.

207 The oral mucosa represents a physical barrier and contains i

208 Wound healing within the oral mucosa results in minimal scar formation compared w

209 developed in squamous epithelia of the skin, oral mucosa, salivary glands, tongue, esophagus, foresto

210 ttle or no methylation was seen in 10 normal oral mucosa samples.

211 Wounds of the oral mucosa show faster closure with less scar formation

212 conjugated with a cell-permeable Tat tag to oral mucosa showed prophylactic and therapeutic effects

213 ces were also detected by PCR in lymph node, oral mucosa, skin, and peripheral blood mononuclear cell

214 The conditional (oral mucosa-specific) beta1 integrin knockout (KO) mice

215 epithelium and salivary glands) and diseased oral mucosa (squamous cell carcinoma and mucoepidermoid

216 diseases from the vaginal (staphylococci) or oral mucosa (streptococci) of the body.

217 Other oral mucosa substitutes, having different cell types and

218 the in vivo effects of EMD on healing of an oral mucosa surgical wound in rats.

219 enal glucocorticoid system is present in the oral mucosa that may play an important role in disease.

220 During inflammation of the oral mucosa, the expression of immunoreactive calprotect

221 the primary carcinogen-activating enzymes in oral mucosa, the use of curcumin as an oral cavity chemo

222 ience mild, transient local reactions in the oral mucosa, these primary reactions rarely necessitate

223 Exposing the oral mucosa to antigen may stimulate immune tolerance.

224 n harboring and disseminating pathogens from oral mucosa to atherosclerosis plaques, which may provid

225 d that this may be one mechanism used in the oral mucosa to attempt to regulate local immune response

226 ranging in severity from mild itching of the oral mucosa to bronchospasm.

227 s cell tumors of the forestomach, esophagus, oral mucosa, tongue, and skin.

228 utively expressed (e.g. hair follicle, nail, oral mucosa, tongue, esophagus, forestomach).

229 cted, all squamous epithelia including skin, oral mucosa, trachea, vaginal epithelium, and the epithe

230 Intraepithelial dysplasia of the oral mucosa typically originates in the proliferative ce

231 Oral mucosa was probed because, like fetal skin, wound r

232 Using a three-dimensional model of the human oral mucosa, we found that E-cadherin was degraded in lo

233 sy of normal-appearing, perilesional skin or oral mucosa when possible.

234 DM) allergens are deposited at the nasal and oral mucosa, where IgA is produced abundantly.

235 replication, is restricted to tissues of the oral mucosa, where it is abundant.

236 n begins in the epidermis of the skin or the oral mucosa, where the virus infects keratinocytes, and

237 different routes of infection, including the oral mucosa which is the most common natural route of in

238 nstitutive expression of pBD-1 in airway and oral mucosa, which is consistent with a lack of consensu

239 ttern of endotoxin tolerance occurs in human oral mucosa with chronic periodontitis (CP).

240 lticellular three-dimensional model of human oral mucosa with induced inflammation promoted MMP12 pro

241 mice developed papillomas exclusively in the oral mucosa within 1 month after tamoxifen treatment.

242 A bilateral oral mucosa wound was created via a crestal incision in

243 ssociated with pathologic alterations of the oral mucosa, yet its direct effects on human keratinocyt