1 d with a 10-week history of multiple painful oral ulcers.

2                Dermatologic lesions included oral ulcers (100%), genital ulcers (62%), erythema nodos

3 extremity edema (57.1%), dermatitis (25.3%), oral ulcers (24.2%), joint pain (23.0%), pleural effusio

4  muscle and bone aches (20%; grade 3/4, 7%), oral ulcers (4%), diarrhea (6%), and neurologic changes

5 nt (p = 0.012), greater bother from foods or oral ulcers and greater mouth pain, and sensitivity (p <

6             A 25-year-old man presented with oral ulcers and odynophagia.

7 ar rash, discoid rash, photosensitivity, and oral ulcers, and 3 (23%) met the mucocutaneous ACR crite

8      The primary end point was the number of oral ulcers at week 12.

9  aphthous ulcers and Behcet disease prone to oral ulcers failed to induce NETosis, but for different

10                The mean decline in pain from oral ulcers from baseline to week 12 was greater with ap

11  the treatment of erythema nodosum leprosum, oral ulcers, graft versus host disease, and wasting asso

12 ty (P < 0.0001, OR 0.58 [95% CI 0.44-0.76]), oral ulcers (P < 0.0001, OR 0.55 [95% CI 0.42-0.72]), an

13                   The mean (+/-SD) number of oral ulcers per patient at week 12 was significantly low

14  with 2 (3.6%) of 55 controls with recurrent oral ulcers, systemic disease, or no disorders.

15                                              Oral ulcers, the hallmark of Behcet's syndrome, can be r

16 s with Behcet's syndrome who had two or more oral ulcers were randomly assigned to receive 30 mg of a

17         Apremilast was effective in treating oral ulcers, which are the cardinal manifestation of Beh