1 and cytoplasmic MerA are highly resistant to organic mercury and degrade organic mercury at 10 to 70

2 thylmercury (CH3Hg(+)) is the common form of organic mercury and is more toxic than its inorganic or

3 se results suggest that the toxic effects of organic mercury are focused in microenvironments of the

4 hly resistant to organic mercury and degrade organic mercury at 10 to 70 times higher specific activi

5 ansgenes are required for plants to detoxify organic mercury by converting it to volatile and much le

6 s them to efficiently convert both ionic and organic mercury compounds to the less toxic elemental me

7            To optimize reaction kinetics for organic mercury compounds, the merB gene was engineered

8 er scabbardfish has to be avoided due to the organic mercury concentration.

9                       Bacteria isolated from organic mercury-contaminated sites have developed a syst

10 eagents of different size and charge and the organic mercury derivate, p-(chloromercuri)benzenesulfon

11 e and merB for organomercurial lyase) for an organic mercury detoxification pathway.

12   Although dimercaprol is contraindicated in organic mercury exposures, meso-2,3-dimercaptosuccinic a

13 column and mimic concentrations of dissolved organic mercury in seawater.

14    While present in trace amounts, dissolved organic mercury increases with depth in the water column

15 ent evidence suggests that their efficacy in organic mercury poisoning is uncertain.

16 ve for removal of most tested metal ions and organic mercury species.

17 ts suggest that the diffusion of hydrophobic organic mercury to MerB limits the rate of the coupled r