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1  was requested to depict lymphadenopathy and organomegaly.
2 iferative disorder including lymphadenopathy/organomegaly.
3 progressive myeloproliferative disorder with organomegaly.
4 lanine aminotransferase, petechial rash, and organomegaly.
5 ) gene results in widespread hyperplasia and organomegaly.
6 ies, including gender-specific gigantism and organomegaly.
7 compared with those of patients lacking such organomegaly.
8 s with A-MDS were older and had little or no organomegaly.
9 ll of these patients had bulky adenopathy or organomegaly.
10  These mice develop gigantism and widespread organomegaly.
11 linical manifestations including cytopenias, organomegaly and bone disease.
12 tion and maturation block, thrombocytopenia, organomegaly, and extensive fibrosis.
13 tions of ALPS include autoimmune cytopenias, organomegaly, and lymphadenopathy.
14 ohistiocytosis, lymphoproliferative disease, organomegaly, and/or malignancy.
15 anifestations of HLH, including weight loss, organomegaly, anemia, thrombocytopenia, hypercytokinemia
16 g Cux-1 developed multiorgan hyperplasia and organomegaly, but not an overall increase in body size.
17 syndrome (coined to refer to polyneuropathy, organomegaly, endocrinopathy, M protein, and skin change
18 eflects the common features: Polyneuropathy, Organomegaly, Endocrinopathy, Monoclonal protein and Ski
19 ic disorder characterized by polyneuropathy, organomegaly, endocrinopathy, monoclonal protein, and sk
20 y (PN), a clonal plasma cell disorder (PCD), organomegaly, endocrinopathy, skin changes, edema, scler
21 rbid manifestations of the disease including organomegaly, growth and pubertal retardation, and osteo
22 evels and severe mediator symptoms, all with organomegaly had systemic disease, and none without orga
23 egaly had systemic disease, and none without organomegaly had systemic disease.
24 er features, including acanthosis nigricans, organomegaly, hyperandrogenism, and diabetes.
25 (135% of normal birthweight) and generalized organomegaly, kinky tail, postaxial polydactyly, heart a
26           Instead, patients show progressive organomegaly, leukocytosis, anemia, and thrombocytosis.
27  The POEMS syndrome (polyradiculoneuropathy, organomegaly, multiple endocrinopathies, monoclonal prot
28 ndicated a uniform increase in mass, with no organomegaly or associated pathology.
29  included: a bone lesion, Castleman disease, organomegaly (or lymphadenopathy), endocrinopathy, edema
30 did not cause obvious neurological symptoms, organomegaly, or a reduction in lifespan.
31  symptoms including urticaria pigmentosa and organomegaly (P < .02).
32                        We will summarize the organomegaly phenotypes associated with Pten tissue-spec
33 he disease, that is, polyradiculoneuropathy, organomegaly, potentially including coexisting Castleman
34 e reticulo-endothelial systems, with massive organomegaly resulting from the presence of engorged, li
35 p27(Kip1) develop widespread hyperplasia and organomegaly similar to those developed by p18-deficient
36 less than 100 x 10(9)/L, bulky adenopathy or organomegaly, symptomatic hyperviscosity, peripheral neu
37 lets <100 x 10(9)/L, bulky adenopathy and/or organomegaly, symptomatic hyperviscosity, peripheral neu
38 lder age, high white blood cell (WBC) count, organomegaly, T-lineage immunophenotype, ability of leuk
39                                              Organomegaly was a strong indicator of systemic disease.
40                                              Organomegaly was confirmed by means of ultrasound.
41                                              Organomegaly with cardiopulmonary compromise most freque
42         However, these mice developed severe organomegaly with disorganized lymphoid tissues.

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