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1 ion of awake female mice during two types of oromotor activity, licking and bruxing, using specific e
13 pment of this nucleus and the impairments in oromotor control and articulation reported in the KE fam
15 donic taste evaluation, appetitive behavior, oromotor coordination, and inhibitory postingestive feed
16 r Fmr1-KO mice also display appendicular and oromotor deficits comparable to the ataxia and dysarthri
17 , intellectual disability, early feeding and oromotor difficulties, microcephaly and/or craniosynosto
18 al bilateral perisylvian syndrome, featuring oromotor dysfunction, cognitive impairment, and epilepsy
19 long-term fluid-licking assay to investigate oromotor function in Fmr1-KO mice and their wild-type (W
22 inergic and nitrergic neurons overlapped pre-oromotor neurons, but there was sparse double-labeling (
23 roportion of neurons with projections to the oromotor nuclei, these areas of the RF are heterogeneous
24 is characterized by cerebellar dysfunction, oromotor/oculomotor apraxia, emotional lability and muti
25 s that project bilaterally to left and right oromotor pools, which could potentially mediate bilatera
28 a in the brainstem processing that underlies oromotor rejection behaviors and also help substantiate
29 viously demonstrated that quinine-stimulated oromotor rejection reflexes and neural activity (assesse
32 cal assessment of the preterm human neonatal oromotor system has been limited due to their fragile me
33 for the hypothesis that participation of the oromotor system may be essential for laying down the mem
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