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1 in patients following radiation therapy for oropharyngeal cancer.
2 tion, are associated with increased risk for oropharyngeal cancer.
3 taining mouthwashes may increase the risk of oropharyngeal cancer.
4 ke and tobacco use are established causes of oropharyngeal cancer.
5 evelop a TNM classification specific to HPV+ oropharyngeal cancer.
6 derive new staging classifications for HPV+ oropharyngeal cancer.
7 ical cancer and a major cause of genital and oropharyngeal cancer.
8 , the presumed precursor of HPV16-associated oropharyngeal cancer.
9 s and pathways significantly associated with oropharyngeal cancer.
10 virus (HPV) develop HPV-related cervical and oropharyngeal cancer.
11 ecognized as important causative factors for oropharyngeal cancer.
12 ded (IGRT) radiotherapy for locally advanced oropharyngeal cancer.
13 finger protein genes on chromosome 19q13 in oropharyngeal cancer.
14 stic factor for survival among patients with oropharyngeal cancer.
15 rade 3, and 5.51 (95% CI, 1.22 to 24.84) for oropharyngeal cancer.
16 revealing the potential infectious nature of oropharyngeal cancer.
17 papillomavirus (HPV) infection is a cause of oropharyngeal cancer.
18 L are important adjuncts to the treatment of oropharyngeal cancer.
19 may have implications in the development of oropharyngeal cancer.
20 luate associations between HPV infection and oropharyngeal cancer.
21 s (HPVs) are a major cause of anogenital and oropharyngeal cancers.
22 worldwide, notably cervical, anogenital, and oropharyngeal cancers.
23 pe associated with cervical, anogenital, and oropharyngeal cancers.
24 ive agents of cervical, anal as well as many oropharyngeal cancers.
25 and reducing the morbidity of treatments for oropharyngeal cancers.
26 ausative agents of anogenital tract and some oropharyngeal cancers.
27 ers and a proportion of other anogenital and oropharyngeal cancers.
28 tained current data for HPV-related oral and oropharyngeal cancers.
29 an papillomavirus (HPV)-associated anal than oropharyngeal cancers.
30 a major cause of noncervical anogenital and oropharyngeal cancers.
31 lomavirus (HPV) infection causes a subset of oropharyngeal cancers.
32 esent more than 10 years before diagnosis of oropharyngeal cancers.
33 are associated with a growing percentage of oropharyngeal cancers.
34 fection, the principal cause of HPV-positive oropharyngeal cancers.
39 L1 seropositivity was highly associated with oropharyngeal cancer among subjects with a history of he
40 al HPV infection is strongly associated with oropharyngeal cancer among subjects with or without the
42 al oncogenic HPV infections and HPV-positive oropharyngeal cancers among men than women arises in par
43 iagnostic samples for 34.8% of patients with oropharyngeal cancer and 0.6% of controls (OR, 274; 95%
44 l study of 100 patients with newly diagnosed oropharyngeal cancer and 200 control patients without ca
45 strongly associated with the development of oropharyngeal cancer and a small minority of oral cavity
46 PK pathway was significantly associated with oropharyngeal cancer and cervical cancer, and TGFbetaR1
48 s been implicated in the rising incidence of oropharyngeal cancer and has led to variety of studies e
49 human papillomavirus 16 (HPV16) and HPV18 in oropharyngeal cancer and hepatitis B and C viruses in li
50 d genes is associated with susceptibility to oropharyngeal cancer and implicates TGFbetaR1/TGFbeta si
52 genes is a determinant of susceptibility to oropharyngeal cancer and other HPV-associated cancers by
53 ohol consumption with respect to the risk of oropharyngeal cancers and cancers of the upper aerodiges
56 can cause cervical and other anogenital and oropharyngeal cancer, and other types of HPV are associa
58 ength and consistency of HPV DNA presence in oropharyngeal cancers bolster the argument that this ass
59 l cancer, and TGFbetaR1 was overexpressed in oropharyngeal cancer, cervical cancer, and HPV(+) head a
60 5% confidence interval (CI), 0.13-0.61] from oropharyngeal cancer, closely followed by high viral loa
61 that mouthwash does not increase the risk of oropharyngeal cancer, confounding due to underascertaine
62 tier integrative computational analysis with oropharyngeal cancer data from a head and neck cancer ge
63 r sexual behaviors that elevated the odds of oropharyngeal cancer developing in a case-control study
64 tivity was present more than 10 years before oropharyngeal cancer diagnosis and was nearly absent in
66 study included patients with non-metastatic oropharyngeal cancer from seven cancer centres located a
67 ence of human papilloma virus (HPV)-positive oropharyngeal cancers has risen rapidly in recent decade
68 tigens in human papillomavirus (HPV)-related oropharyngeal cancer (HPVOPC) are attractive targets for
69 Human papilloma virus-16 (HPV-16) associated oropharyngeal cancer (HPVOPC) is increasing alarmingly i
70 infection is causing an increasing number of oropharyngeal cancers in the United States and Europe.
71 ecent increases in incidence and survival of oropharyngeal cancers in the United States have been att
72 e population-level incidence and survival of oropharyngeal cancers in the United States since 1984 ar
73 papillomavirus (HPV) causes the majority of oropharyngeal cancers in the United States, yet the risk
76 Population-level incidence of HPV-positive oropharyngeal cancers increased by 225% (95% CI, 208% to
80 Our proposed ICON-S staging system for HPV+ oropharyngeal cancer is suitable for the 8th edition of
81 continue, the annual number of HPV-positive oropharyngeal cancers is expected to surpass the annual
82 dence of human papillomavirus (HPV)-positive oropharyngeal cancers is higher and increasing more rapi
83 with a truncated COOH-terminal, and in human oropharyngeal cancer KB cells, which possess wild-type p
86 d system, the International Collaboration on Oropharyngeal Cancer Network for Staging (ICON-S); and a
87 ex partners (26 or more) was associated with oropharyngeal cancer (odds ratio, 3.1; 95% confidence in
88 ents with human papillomavirus (HPV)-related oropharyngeal cancer (OPC) generally present with more a
89 en identified as the cause of the increasing oropharyngeal cancer (OPC) incidence in some countries.
90 iation with or without chemotherapy to treat oropharyngeal cancer (OPC) is supported by evidence from
92 is the principal cause of a distinct form of oropharyngeal cancer (OPCA) that has been rising in inci
95 d alcohol use increased the association with oropharyngeal cancer primarily among subjects without ex
97 PV) integration into the host genome in oral/oropharyngeal cancer, reviewed the literature for HPV-in
100 in a five-generation pedigree and comprises oropharyngeal cancer, skin telangiectases, and mild deve
101 survival rates in p16-positive patients with oropharyngeal cancer support the ongoing efforts to expl
102 ansform cells and contribute to cervical and oropharyngeal cancer, there clearly is much more to lear
103 ampled blood from patients with stage III-IV oropharyngeal cancer undergoing concomitant chemoradioth
104 ll-cause mortality ratio among patients with oropharyngeal cancer was 0.30 (95% CI, 0.13 to 0.67), fo
105 hin 5 months, while the 1-year mortality for oropharyngeal cancer was 57.1% and that for lung cancers
109 an papillomavirus (HPV) cause anogenital and oropharyngeal cancers, whereas cutaneous types (e.g. HPV
110 d by the fact that the patient had developed oropharyngeal cancer, which was treated with chemoradiot
111 V-16) and HPV-18 cause a large proportion of oropharyngeal cancers, which are increasing in incidence
112 bserved HPV prevalence was reweighted to all oropharyngeal cancers within the cancer registries to ac
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